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1 One subject had evidence of orthostatic hypotension.
2 eral vascular resistance, often resulting in orthostatic hypotension.
3 ents were peripheral oedema, hypotension, or orthostatic hypotension.
4 oreceptor-cardiac reflex function and causes orthostatic hypotension.
5 m serve to protect against the occurrence of orthostatic hypotension.
6 Five of the LBD cases and all MSA cases had orthostatic hypotension.
7 action can be beneficial in the treatment of orthostatic hypotension.
8 ls, anemia, fatigue, nausea or vomiting, and orthostatic hypotension.
9 tive heart failure, cardiac arrhythmias, and orthostatic hypotension.
10 with standing, in the absence of significant orthostatic hypotension.
11 hort over 4 years; of those who died, 52 had orthostatic hypotension.
12 ious and safe in the treatment of neurogenic orthostatic hypotension.
14 It might be possible to improve treatment of orthostatic hypotension acutely with water imbibation an
15 gns of sympathetic neurocirculatory failure (orthostatic hypotension and abnormal blood-pressure resp
16 Screening autonomic function tests indicated orthostatic hypotension and confirmed chronic autonomic
17 ide an explanation for the predisposition to orthostatic hypotension and intolerance in well-trained
21 betes increases the risk of hypertension and orthostatic hypotension and raises the risk of cardiovas
23 comes in patients with coexistent neurogenic orthostatic hypotension and supine hypertension, clinici
25 erapy in patients with coexistent neurogenic orthostatic hypotension and supine hypertension; and the
27 mediate benefits of treatment for neurogenic orthostatic hypotension and the long-term risks of supin
28 sed a significant increase in heart rate and orthostatic hypotension, and 20% of the nortriptyline-tr
29 imates of impaired BP stabilization, initial orthostatic hypotension, and orthostatic hypotension are
30 c blood pressure (BP) stabilization, initial orthostatic hypotension, and orthostatic hypotension bas
31 re likely to have mild cognitive impairment, orthostatic hypotension, and RBD at baseline, and at pro
32 ients with PD for mild cognitive impairment, orthostatic hypotension, and RBD even at baseline visits
35 zation, initial orthostatic hypotension, and orthostatic hypotension based on beat-to-beat blood pres
36 thesis that short-term alcohol intake causes orthostatic hypotension because of an impairment in the
43 and all with Parkinson's disease-associated orthostatic hypotension, have a loss of cardiac sympathe
49 The best cluster solution found was based on orthostatic hypotension, mild cognitive impairment, rapi
54 Patients with Parkinson's disease (PD) and orthostatic hypotension (OH) (PD+OH) or with pure autono
55 arkinsonism and non-motor features including orthostatic hypotension (OH) and cognitive impairment.
62 sed on HUT results, we divided patients into orthostatic hypotension (OH), postural tachycardia syndr
63 n the heart and other organs, manifesting as orthostatic hypotension (OH; also known as postural hypo
64 available concerning the predictive value of orthostatic hypotension on mortality in ambulatory elder
67 cterised by autonomic failure, manifested as orthostatic hypotension or urogenital dysfunction, with
69 measured in patients with PD with or without orthostatic hypotension (PD+OH, PD-No-OH); in patients w
71 or autonomic failure in Parkinson disease), orthostatic hypotension reflects sympathetic neurocircul
72 uncomplicated faint, situational syncope, or orthostatic hypotension should receive electrocardiograp
73 n, upper gastrointestinal tract dysfunction, orthostatic hypotension, sweating abnormalities, or erec
74 gene encoding alpha-synuclein, also features orthostatic hypotension, sympathetic neurocirculatory fa
75 apeutic requirements for managing neurogenic orthostatic hypotension that manifests with falls or cog
76 supine hypertension when treating neurogenic orthostatic hypotension; the effectiveness of nocturnal
77 e symptomatic autonomic failure (symptomatic orthostatic hypotension, urinary incontinence, or both)
78 There was no effect by center, severity of orthostatic hypotension, use of fludrocortisone or compr
79 sk factors were identified across 6 domains: orthostatic hypotension, visual impairment, impairment o
82 t stroke, coronary heart disease and cancer, orthostatic hypotension was a significant independent pr
83 Program's fourth examination (1991 to 1993), orthostatic hypotension was assessed in relation to subs
86 ed mortality rates in those with and without orthostatic hypotension were 56.6 and 38.6 per 1000 pers
87 ous events, two of which (hallucinations and orthostatic hypotension) were deemed related to study dr
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