ライフサイエンスコーパス: oryzae

1 rculans) to 34.9% (K. lactis), and 19.5% (A. oryzae).

2 and bacterial blight (Xanthomonas oryzae pv. oryzae).

3 nfection of bacterium Xanthomonas oryzae pv. oryzae.

4 t the virulence of compatible isolates of M. oryzae.

5 athogens: Magnaporthe oryzae and Xanthomonas oryzae.

6 dent disease susceptibility to X. oryzae pv. oryzae.

7 13 induction by 42 isolates of X. oryzae pv. oryzae.

8 C is essential for viability of Magnaporthe oryzae.

9 a model plant pathogenic fungus, Magnaporthe oryzae.

10 plain enhanced susceptibility to Magnoporthe oryzae.

11 pment of infection structures in Magnaporthe oryzae.

12 thogens Pseudomonas syringae and Xanthomonas oryzae.

13 f laccase (LAC) originating from Aspergillus oryzae.

14 culans, Kluyveromyces lactis and Aspergillus oryzae.

15 enhanced susceptibility of rice plants to M. oryzae.

16 on etiologic agent of mucormycosis, Rhizopus oryzae.

17 known to inactivate plant MAP kinases in M. oryzae.

18 infection by the fungal pathogen Magnaporthe oryzae.

19 y step in the infection cycle of Magnaporthe oryzae.

20 growth, development and pathogenicity in M. oryzae.

21 ra protected DKA mice from infection with R. oryzae.

22 uencing of the rice blast fungus Magnaporthe oryzae.

23 re core components of the Tig1 complex in M. oryzae.

24 fection by the rice blast fungus Magnaporthe oryzae.

25 lungs infected with voriconazole-exposed R. oryzae.

26 apid evolution within Xanthomonas oryzae pv. oryzae.

27 f MoGSK1 produced deformed appressoria in M. oryzae.

28 thods for Aspergillus fumigatus and Rhizopus oryzae.

29 pdate our understanding of the biology of M. oryzae.

30 ytoplasmic localization as MoGsk1 does in M. oryzae.

31 rp1 as the yeast Vrp1 homolog in Magnaporthe oryzae.

32 rmation plays a critical role in Magnaporthe oryzae.

33 as validated in the hemibiotroph Magnaporthe oryzae.

34 ll-to-cell movement of invasive hyphae in M. oryzae.

35 involving the rice blast fungus Magnaporthe oryzae.

36 llular ROS signalling and pathogenesis in M. oryzae.

37 cally to infection by pathogenic Pyricularia oryzae.

38 two thioredoxin genes, TRX1 and TRX2, in M. oryzae.

39 without subsequent elicitation with Rhizopus oryzae.

40 formation and plant infection in Magnaporthe oryzae.

41 growth, conidiation and pathogenicity of M. oryzae.

42 mechanism of the blight disease caused by M. oryzae.

43 ented with Monascus purpureus or Aspergillus oryzae.

44 (ETFDH) in the rice blast fungus Magnaporthe oryzae.

45 bation with caspofungin (32 microg/mL for R. oryzae; 0.0625 microg/mL for other isolates) increased e

46 The main fungal species were Rhizopus oryzae (32%) and Lichtheimia species (29%).

47 erboard assays for 4 clinical isolates of R. oryzae (48-hour fractional inhibitory concentration inde

48 o examine the effects of statins on Rhizopus oryzae, a common cause of mucormycosis.

49 ar signature of domestication on Aspergillus oryzae, a fungus used to ferment several Japanese food p

50 factor of RNAP of host bacterium Xanthomonas oryzae, a major rice pathogen.

51 for use in the rice blast fungus Magnaporthe oryzae, allowing rapid generation of transformants in wh

52 is involved in all three MAPK pathways in M. oryzae although its functions differ in each pathway.

53 annotation improvements for A. nidulans, A. oryzae and A. fumigatus genomes based on recently availa

54 ase of rice caused by the fungus Magnaporthe oryzae and can result in loss of a third of the annual g

55 irox effectively chelated iron from Rhizopus oryzae and demonstrated cidal activity in vitro against

56 r infectious growth and conidiogenesis in M. oryzae and highlighted that chromatin modification is an

57 Development of M. oryzae and initiation of infection are critically depend

58 riculture, but also because both Magnaporthe oryzae and its host are amenable to advanced experimenta

59 including Zymoseptoria tritici, Magnaporthe oryzae and Neurospora crassa, exhibited PAMP activity, i

60 We review recent studies of M. oryzae and other relevant appressorium-forming fungi whi

61 pathogens Xanthomonas oryzae pathovar (pv.) oryzae and pv. oryzicola produce numerous transcription

62 Gram-negative E. coli and plant pathogens X. oryzae and X. campestris, as well as against human funga

63 nced susceptibility to the rice pathogens M. oryzae and Xanthomonas oryzae pv oryzae (Xoo).

64 gainst two major rice pathogens: Magnaporthe oryzae and Xanthomonas oryzae.

65 t, as well as enhanced resistance to both M. oryzae and Xoo.

66 hanced resistance to rice blast (Magnaporthe oryzae) and bacterial blight (Xanthomonas oryzae pv. ory

67 Thus, FTR1 is a virulence factor for R. oryzae, and anti-Ftr1p passive immunotherapy deserves fu

68 he normal physiology and pathogenicity of M. oryzae, and it sheds new light on the mechanism of the b

69 s to MAP kinase kinase kinase in Magnaporthe oryzae, and urate oxidase (designated ClUrase) were func

70 acearum, Xanthomonas campestris, Xanthomonas oryzae, and Xylella fastidiosa T2S also occurs in nonpat

71 s species: Aspergillus nidulans, Aspergillus oryzae, Aspergillus fumigatus and Aspergillus niger.

72 The reductive aminase from Aspergillus oryzae (AspRedAm) was combined with a single alcohol deh

73 dependent reductive aminase from Aspergillus oryzae (AspRedAm, Uniprot code Q2TW47) that can catalyse

74 ne susceptible and the other resistant to M. oryzae, at multiple time points during the fungal infect

75 pproximately 40-50% with B. circulans and A. oryzae beta-galactosidases, and at 95% lactose depletion

76 inhardtii, Mucor circinelloides and Rhizopus oryzae but was dissimilar to the non-oleaginous Aspergil

77 fungal development and plant infection in M. oryzae by regulation of fatty acid metabolism, turgor es

78 Extraction of R. oryzae by the manual method resulted in a limit of detec

79 gens Xanthomonas vesicatoria and Xanthomonas oryzae, capable of inhibiting disease symptoms in detach

80 the first case of a successfully treated A. oryzae catheter-associated bloodstream infection in an i

81 Xanthomonas oryzae pv. oryzae causes bacterial blight of rice (Oryza sativa L.)

82 Here, we report that Magnaporthe oryzae CKS1 encodes a cyclin-dependent kinase subunit, w

83 Additionally, expression of R. oryzae CotH was increased within hours of incubation wit

84 hreshold of posaconazole activity against R. oryzae could be achieved with 2-fold lower drug concentr

85 report that a purine-requiring mutant of M. oryzae could develop functional appressoria, penetrate h

86 ologous recombination, but multinucleated R. oryzae could not be forced to segregate to a homokaryoti

87 ional heterologous expression in Aspergillus oryzae coupled with isolation and detailed structural el

88 ity, stability, and structure of Aspergillus oryzae cutinase and compare it to the well-studied enzym

89 These structural features of A. oryzae cutinase are proposed to result in an improved hy

90 lity specifically to strains of X. oryzae pv oryzae dependent on AvrXa7 or PthXo3 for virulence.

91 e rice blast disease, the fungus Magnaporthe oryzae develops a pressurized dome-shaped cell called an

92 ast disease, the fungal pathogen Magnaporthe oryzae develops a specialized infection structure called

93 resistance to PthXo2-dependent X. oryzae pv. oryzae due to promoter variations of OsSWEET13 in japoni

94 sativa) NLR RGA5 recognizes the Magnaporthe oryzae effector AVR-Pia through direct interaction.

95 also mediate recognition of the unrelated M. oryzae effector AVR-Pia, indicating that the correspondi

96 t factors that interact with the Magnaporthe oryzae effector AVR-Pii.

97 uired for the recognition of the Magnaporthe oryzae effector AVR1-CO39.

98 Here, we show that the M. oryzae effector AvrPiz-t interacts with the bZIP-type tr

99 Development of the M. oryzae effector-secreting biotrophic interfacial complex

100 The rice blast fungus Magnaporthe oryzae elaborates a specialized cell called an appressor

101 ts corroborate a model whereby X. oryzae pv. oryzae enhances the release of sucrose from host cells i

102 Additionally, R. oryzae exposed to lovastatin showed macroscopic loss of

103 statins had MICs of >64 microg/mL against R. oryzae Exposure of R. oryzae to statins decreased germli

104 Slp1 is required by M. oryzae for full virulence and exerts a significant effec

105 icant reductions in cutaneous lesions and R. oryzae fungal burden, compared with animals that receive

106 is work, the effect of SSF with the Rhizopus oryzae fungus on the phenolic acid content of rice bran

107 lus fumigatus, Aspergillus terreus, Rhizopus oryzae, Fusarium solani, Fusarium oxysporum, Scedosporiu

108 also showed, for the first time, that the X. oryzae gene indeed encoded an enzyme with alpha-L: -arab

109 We demonstrate that Magnaporthe oryzae generates NO during germination and in early deve

110 ordinary diversity of Xanthomonas oryzae pv. oryzae genotypes and races that have been isolated from

111 R. oryzae germination, DNA fragmentation, susceptibility to

112 he limit of detection of A. fumigatus and R. oryzae GHE in bronchoalveolar lavage (BAL) fluid with ei

113 cluster was reconstructed within Aspergillus oryzae giving production of pleuromutilin in an ascomyce

114 oid triggering plant defences that impact M. oryzae growth and BIC development.

115 Consequently, R. oryzae has a 2- to 10-fold enrichment in gene families a

116 We conclude that M. oryzae has a robust anti-oxidant defence system and main

117 receptors for Candida albicans and Rhizopus oryzae has been demonstrated in experimental animal mode

118 Fermentation by using Aspergillus oryzae has been reported to increase antioxidant activit

119 The genome sequence of M. oryzae has provided insight into how genome structure an

120 of the devastating rice pathogen Magnaporthe oryzae impaired for de novo methionine biosynthesis.

121 t of the important rice pathogen Magnaporthe oryzae in leaf cells.

122 at FTR1 is required for full virulence of R. oryzae in mice.

123 olimus, alone and in combination, against R. oryzae in vitro, using multiple methods (ie, hyphal meta

124 cies (ROS) and enhances susceptibility to M. oryzae, indicating that AvrPiz-t functions to suppress p

125 The rice blast fungus Magnaporthe oryzae infects plants with a specialized cell called an

126 hanced GRP78 expression and the resulting R. oryzae invasion and damage of endothelial cells in a rec

127 l resistant and susceptible plants during M. oryzae invasion discovered distinct pathways triggered i

128 A. oryzae IOC 3999/1998 expressed beta-glucosidase activity

129 efatted soybean flour (DSF) with Aspergillus oryzae IOC 3999/1998 or Monascus purpureus NRRL 1992 was

130 iron permease gene (FTR1) is required for R. oryzae iron transport in iron-depleted environments.

131 Acidovorax oryzae is a bacterium that has never before been reporte

132 The rice blast fungus Magnaporthe oryzae is a model for studying fungal-plant interactions

133 The fungus Magnaporthe oryzae is a serious pathogen of rice and other grasses.

134 disease by strains of Xanthomonas oryzae pv. oryzae is dependent on major transcription activation-li

135 ted and the distribution of the operon in X. oryzae is investigated in over 100 isolates.

136 ycogen synthase kinase 3 (GSK3) MoGSK1 in M. oryzae is regulated by Mps1 MAP kinase, particularly und

137 Magnaporthe oryzae is the causal agent of rice blast disease, the mo

138 Rhizopus oryzae is the most common cause of mucormycosis, an angi

139 Magnaporthe oryzae is the most damaging fungal pathogen of rice (Ory

140 The blast fungus Magnaporthe oryzae is the most devastating pathogen of cultivated ri

141 m-negative bacterium, Xanthomonas oryzae pv. oryzae, is a tyrosine sulfotransferase.

142 epeat protein, confers resistance against X. oryzae isolates by recognizing multiple TALEs.

143 -galactosidase preparations from Aspergillus oryzae, Kluyveromyces lactis and Bacillus circulans.

144 f sialyl Lewis(a), Lewis(x), and Aspergillus oryzae lectin [AOL] and binding of wheat germ agglutinin

145 splasia, abnormal DNA ploidy and Aspergillus oryzae lectin) can identify patients at high risk for de

146 ion of the hybrid synthetases in Aspergillus oryzae led to the production of reprogrammed compounds i

147 mated deleterious substitutions along the A. oryzae lineage were lower than those in the wild sister

148 r terephthalate-to-adipate ratio by Rhizopus oryzae lipase and Fusarium solani cutinase.

149 rs by two fungal esterases (FsC and Rhizopus oryzae lipase) at different temperatures.

150 -1 (50.5%); Ncdcl-2 (38.0%)] and Magnaporthe oryzae [MDL-1 (45.6%); MDL-2 (38.0%)], respectively.

151 t pathogen, the rice blast fungusMagnaporthe oryzae(Mo), was expressed inPichia pastoris.Mo-MnLOX was

152 so increased fungal growth and attenuated R. oryzae neutrophil-mediated damage.

153 m-negative bacterium, Xanthomonas oryzae pv. oryzae; NH1, the rice ortholog of NPR1, a key regulator

154 We show that the M. oryzae NMO2 gene is required for mitigating damaging lip

155 s relative to control mycelial RNAs using M. oryzae oligoarrays.

156 tivation (SSC) time of rice bran by Rhizopus oryzae on gamma-oryzanol recovery and its antioxidant pr

157 tructure of the DBD of PCG2, the Magnaporthe oryzae orthologue of MBP1, bound to MCB-DNA.

158 show that the rice blast fungus Magnaporthe oryzae overcomes this first line of plant defense by sec

159 The rice pathogens Xanthomonas oryzae pathovar (pv.) oryzae and pv. oryzicola produce n

160 oc (> 90%), but absent in the other major X. oryzae pathovar.

161 sponse to diverse TAL effectors from both X. oryzae pathovars.

162 reviously, we showed that during Xanthomonas oryzae phage Xp10 infection, the phage protein P7 inhibi

163 cription regulator P7 encoded by Xanthomonas oryzae phage Xp10.

164 These results suggest that the X. oryzae pv. oryzae PhoPQ TCS functions in virulence and in the produ

165 eport that the rice blast fungus Magnaporthe oryzae possesses two distinct secretion systems to targe

166 Piz-t from the rice blast fungus Magnaporthe oryzae preferentially accumulates in the specialized str

167 The enzyme from A. oryzae produced the highest yield and specific productiv

168 es and soluble protein fractions of Azospira oryzae PS, as well as soluble protein fractions encapsul

169 ase, acetylxylan esterase, and a Xanthomonas oryzae putative a-L: -arabinofuranosidase.

170 the rice pathogens M. oryzae and Xanthomonas oryzae pv oryzae (Xoo).

171 susceptibility specifically to strains of X. oryzae pv oryzae dependent on AvrXa7 or PthXo3 for virul

172 is a susceptibility (S) gene for Xanthomonas oryzae pv oryzae, the causal agent of bacterial blight,

173 a) xylem and mesophyll pathogens Xanthomonas oryzae pv. oryzae (Xoo) and pv. oryzicola (Xoc) encode n

174 ion pathway of the rice pathogen Xanthomonas oryzae pv. oryzae (Xoo) in a semi-isolated environment r

175 The biotrophic pathogen Xanthomonas oryzae pv. oryzae (Xoo) produces a sulfated peptide name

176 esistance to a broad spectrum of Xanthomonas oryzae pv. oryzae (Xoo) races that cause bacterial bligh

177 of the bacterial blight pathogen Xanthomonas oryzae pv. oryzae (Xoo) that deliver the TAL effector Av

178 and delivered by the pathogenic Xanthomonas oryzae pv. oryzae (Xoo), in revealing the new function o

179 XA3 receptor confer immunity to Xanthomonas oryzae pv. oryzae (Xoo), the causal agent of bacterial l

180 nfection by pathogenic bacterium Xanthomonas oryzae pv. oryzae (Xoo), which causes a vascular disease

181 ce to bacterial blight caused by Xanthomonas oryzae pv. oryzae (Xoo).

182 ce to bacterial blight caused by Xanthomonas oryzae pv. oryzae (Xoo).

183 of rice (Oryza sativa) caused by Xanthomonas oryzae pv. oryzae (Xoo).

184 ssociated with pathogens such as Xanthomonas oryzae pv. oryzae (Xoo).

185 to most strains of the bacterium Xanthomonas oryzae pv. oryzae (Xoo).

186 terial blight diseases caused by Xanthomonas oryzae pv. oryzae (Xoo).

187 Xanthomonas oryzae pv. oryzae causes bacterial blight of rice (Oryza

188 recessive resistance to PthXo2-dependent X. oryzae pv. oryzae due to promoter variations of OsSWEET1

189 The results corroborate a model whereby X. oryzae pv. oryzae enhances the release of sucrose from h

190 n the extraordinary diversity of Xanthomonas oryzae pv. oryzae genotypes and races that have been iso

191 preventing disease by strains of Xanthomonas oryzae pv. oryzae is dependent on major transcription ac

192 These results suggest that the X. oryzae pv. oryzae PhoPQ TCS functions in virulence and i

193 X. oryzae pv. oryzae requires a regulatory two-component sy

194 tor for the sulfated form of the Xanthomonas oryzae pv. oryzae secreted protein Ax21.

195 Xanthomonas oryzae pv. oryzae strain PXO99(A) induces the expression

196 tor, XA21, confers resistance to Xanthomonas oryzae pv. oryzae strains producing the type one system-

197 X. oryzae pv. oryzae therefore modulates the expression of

198 ram-negative bacterial pathogen, Xanthomonas oryzae pv. oryzae upon recognition of a small protein, A

199 out strain and found it to be impaired in X. oryzae pv. oryzae virulence and no longer able to activa

200 cating that PhoP controls a key aspect of X. oryzae pv. oryzae virulence through regulation of hrpG.

201 he oryzae) and bacterial blight (Xanthomonas oryzae pv. oryzae).

202 rom the Gram-negative bacterium, Xanthomonas oryzae pv. oryzae, is a tyrosine sulfotransferase.

203 sed by the gamma-proteobacterium Xanthomonas oryzae pv. oryzae, which utilizes a group of type III TA

204 ays after infection of bacterium Xanthomonas oryzae pv. oryzae.

205 thXo2-dependent disease susceptibility to X. oryzae pv. oryzae.

206 led OsSWEET13 induction by 42 isolates of X. oryzae pv. oryzae.

207 icity and rapid evolution within Xanthomonas oryzae pv. oryzae.

208 to the Gram-negative bacterium, Xanthomonas oryzae pv. oryzae; NH1, the rice ortholog of NPR1, a key

209 e bacterial leaf streak pathogen Xanthomonas oryzae pv. oryzicola (Xoc) contains a homologous operon.

210 The rice bacterial pathogen Xanthomonas oryzae pv. oryzicola (Xoc) has been demonstrated to cont

211 e bacterial leaf streak pathogen Xanthomonas oryzae pv. oryzicola (Xoc).

212 Xanthomonas oryzae pv. oryzicola causes bacterial leaf streak of ric

213 that a native TAL effector from Xanthomonas oryzae pv. oryzicola drives expression of a target with

214 as campestris pv. raphani strain 756C and X. oryzae pv. oryzicola strain BLS256, pathogens that infec

215 ce to the strains in the African clade of X. oryzae pv. oryzicola, representing the first dominant re

216 derivative flg22(Xo) peptides of Xanthomonas oryzae pvs. oryzae (Xoo) and oryzicola (Xoc).

217 wo fungal CFI protein classes in Magnaporthe oryzae: Rbp35/CfI25 complex and Hrp1.

218 Lyophilized protease from A. oryzae reached 1251.60 U/g and yield of 155010.66 U/kg o

219 i such as the rice blast fungus (Magnaporthe oryzae) remains unclear.

220 X. oryzae pv. oryzae requires a regulatory two-component system (TCS)

221 the hemibiotrophic rice pathogen Magnaporthe oryzae requires plant defence suppression to facilitate

222 Here, we show that plant infection by M. oryzae requires two independent S-phase cell-cycle check

223 soy flour fermented with M. purpureus or A. oryzae, respectively.

224 xposure to lovastatin on the virulence of R. oryzae RESULTS: All statins had MICs of >64 microg/mL ag

225 Recent genome sequencing of Rhizopus oryzae revealed evidence of a whole-genome duplication e

226 Comparative evaluation of CpaDs from A. oryzae RIB40 and A. flavus NRRL3357 indicated the import

227 on factor p7 interacts with host Xanthomonas oryzae RNA polymerase beta' subunit and prevents both pr

228 amino acid residues at the N terminus of X. oryzae RNAP beta'.

229 succinate) (PBS) and the lipase of Rhizopus oryzae (RoL), we detected complete hydrolysis of PBS thi

230 te fermentation (Bacillus subtilis, Rhizopus oryzae, Saccharomyces cerevisiae, Lactobacillus helvetic

231 haliana (Arabidopsis), and the monocotyledon Oryzae sativa (rice).

232 hyphae (IH) of the blast fungus Magnaporthe oryzae secrete effectors to alter host defenses and cell

233 sulfated form of the Xanthomonas oryzae pv. oryzae secreted protein Ax21.

234 Aspergillus oryzae showed stability at all pH values studied.

235 Here, we report that a M. oryzae sirtuin, MoSir2, plays an essential role in rice

236 We review the M. oryzae species-specific and cultivar-specific avirulence

237 The rice blast fungus Magnaporthe oryzae spreads in rice biotrophically early during infec

238 In the murine model, infection with a R. oryzae strain preexposed to voriconazole was associated

239 Xanthomonas oryzae pv. oryzae strain PXO99(A) induces the expression of the hos

240 were expressed in TAL effector-deficient X. oryzae strain X11-5A, and assessed in 21 rice varieties.

241 infection with a voriconazole-nonexposed R. oryzae strain.

242 confers resistance to Xanthomonas oryzae pv. oryzae strains producing the type one system-secreted mo

243 isolates of Zygomycetes, including Rhizopus oryzae strains, we found no evidence that bacterial endo

244 d in the crystallographic analysis of the A. oryzae structure: (i) an additional disulfide bond and (

245 es, we enriched and cultivated anti-Rhizopus oryzae T cells from healthy individuals.

246 The roles of X. oryzae TAL effectors in diverse rice backgrounds, howeve

247 led two related non-LTR retrotransposons (M. oryzae Telomeric Retrotransposons or MoTeRs) inserted in

248 inoculated with the race IB49 of Magnaporthe oryzae that recognizes Pi-ta.

249 For R. oryzae, the automated method was more sensitive for DNA

250 ptibility (S) gene for Xanthomonas oryzae pv oryzae, the causal agent of bacterial blight, and the re

251 Magnaporthe oryzae, the causal agent of blast disease, is one of the

252 A mutant of Rhizopus oryzae, the most common cause of mucormycosis, with redu

253 amage of human endothelial cells by Rhizopus oryzae, the most common etiologic species of Mucorales,

254 In Magnaporthe oryzae, the Mst11-Mst7-Pmk1 MAP kinase pathway is essent

255 vo activity of posaconazole against Rhizopus oryzae, the Mucorales species most commonly associated w

256 X. oryzae pv. oryzae therefore modulates the expression of multiple ho

257 ession by RNAi compromised the ability of R. oryzae to acquire iron in vitro and reduced its virulenc

258 urface signals are recognized by Magnaporthe oryzae to activate the Pmk1 MAP kinase that is essential

259 t, gene silencing of CEBiP in rice allows M. oryzae to cause rice blast disease in the absence of Slp

260 cose, and iron, augmenting the ability of R. oryzae to invade and subsequently damage endothelial cel

261 th anti-CotH Abs abolished the ability of R. oryzae to invade host cells and protected DKA mice from

262 In contrast, exposure of Rhizopus oryzae to itraconazole, amphotericin B, or caspofungin a

263 Exposure of R. oryzae to statins at concentrations below their MICs dec

264 4 microg/mL against R. oryzae Exposure of R. oryzae to statins decreased germling formation, induced

265 ong telomeric repeat sequence of Aspergillus oryzae together with reverse-transcription-PCR and ident

266 bacterial pathogen Pseudomonas syringae pv. oryzae, transgenic EFR wheat lines had reduced lesion si

267 e bacterial pathogen, Xanthomonas oryzae pv. oryzae upon recognition of a small protein, Ax21, that i

268 me isolates of the rice pathogen Xanthomonas oryzae use truncated versions of TALEs (which we term in

269 and found it to be impaired in X. oryzae pv. oryzae virulence and no longer able to activate the resp

270 vivo infections of rice demonstrate that M. oryzae virulence is enhanced, quite paradoxically, when

271 PhoP controls a key aspect of X. oryzae pv. oryzae virulence through regulation of hrpG.

272 ion antitermination mechanism of Xanthomonas oryzae virus Xp10 protein p7, which binds host RNA polym

273 A. oryzae was further established as a platform for bio-con

274 ere the causative species was identified, R. oryzae was present in 85% of rhinocerebral forms compare

275 culans, Kluyveromyces lactis and Aspergillus oryzae) was analysed in detail, at 4 and 40 degrees C.

276 activity of beta-galactosidase (Aspergillus oryzae) was evaluated.

277 genesis of the rice blast fungus Magnaporthe oryzae, we identified MoGlo3 as an ArfGAP protein that i

278 tter understanding of redox regulation in M. oryzae, we measured the amount and redox potential of gl

279 Clinical strains of R. oryzae were exposed to lovastatin, atorvastatin, and sim

280 ed by heterologous expression in Aspergillus oryzae, whereas tropB and tropC were successfully expres

281 ment of pathogenic fungi such as Magnaporthe oryzae which causes rice blast.

282 gamma-proteobacterium Xanthomonas oryzae pv. oryzae, which utilizes a group of type III TAL (transcri

283 between the domesticated fungus Aspergillus oryzae, whose saccharification abilities humans have har

284 s essential for growth and development of M. oryzae with extensive downstream targets in addition to

285 lg22(Xo) peptides of Xanthomonas oryzae pvs. oryzae (Xoo) and oryzicola (Xoc).

286 d mesophyll pathogens Xanthomonas oryzae pv. oryzae (Xoo) and pv. oryzicola (Xoc) encode numerous sec

287 of the rice pathogen Xanthomonas oryzae pv. oryzae (Xoo) in a semi-isolated environment represented

288 e biotrophic pathogen Xanthomonas oryzae pv. oryzae (Xoo) produces a sulfated peptide named RaxX, whi

289 o a broad spectrum of Xanthomonas oryzae pv. oryzae (Xoo) races that cause bacterial blight disease.

290 erial blight pathogen Xanthomonas oryzae pv. oryzae (Xoo) that deliver the TAL effector AvrXa27.

291 red by the pathogenic Xanthomonas oryzae pv. oryzae (Xoo), in revealing the new function of two previ

292 or confer immunity to Xanthomonas oryzae pv. oryzae (Xoo), the causal agent of bacterial leaf blight.

293 pathogenic bacterium Xanthomonas oryzae pv. oryzae (Xoo), which causes a vascular disease in rice, o

294 rial blight caused by Xanthomonas oryzae pv. oryzae (Xoo).

295 rial blight caused by Xanthomonas oryzae pv. oryzae (Xoo).

296 yza sativa) caused by Xanthomonas oryzae pv. oryzae (Xoo).

297 athogens M. oryzae and Xanthomonas oryzae pv oryzae (Xoo).

298 ith pathogens such as Xanthomonas oryzae pv. oryzae (Xoo).

299 ains of the bacterium Xanthomonas oryzae pv. oryzae (Xoo).

300 ht diseases caused by Xanthomonas oryzae pv. oryzae (Xoo).