1 igh-dose mannitol in an attempt to induce an osmotic diuresis.

2  significantly, resulting in natriuresis and osmotic diuresis.

3 the proximal tubule, leading to polyuria and osmotic diuresis.

4 e inner medullary interstitium, resulting in osmotic diuresis.

5  mechanism is the prevention of urea-induced osmotic diuresis.

6    However, concerns that SGLT2i may provoke osmotic diuresis and activate vasopressin led to systema

7 indicate that in isolation, glucose-elicited osmotic diuresis and glucose transporter upregulation ra

8 renal water reabsorption, limits natriuretic osmotic diuresis, and results in concurrent extracellula

9                                   To prevent osmotic diuresis by the large quantity of urea generated

10 nuously elevated solute excretion, long-term osmotic diuresis does not occur in humans with SGLT2 inh

11  increased glomerular filtration rate (GFR), osmotic diuresis, hypertrophy, and megalin and cubilin d

12 es selective renal vasodilation and a marked osmotic diuresis in the innervated kidney.

13    SGLT2 inhibition promotes natriuresis and osmotic diuresis, leading to plasma volume contraction a

14     Our model predicts that glucose-elicited osmotic diuresis per se raises albumin excretion only sl

15 or both canagliflozin doses vs 22 [5%]), and osmotic diuresis-related events (pollakiuria: 12 [3%] fo

16 r 2 (SGLT2) inhibitor empagliflozin promotes osmotic diuresis via urinary glucose excretion and there

17 r 2 (SGLT2) inhibitor empagliflozin promotes osmotic diuresis via urinary glucose excretion, likely l