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1 ) versus long bone ossification formation (2 ossification centers).
2 stribution that decreased toward the primary ossification center.
3 promote synchondrosis closure and fusion of ossification centers.
4 ay in the formation of primary and secondary ossification centers.
5 and formation and vascularization of primary ossification centers.
6 normally occurs in growth plates and primary ossification centers.
7 nchronous ossification of one of the sternal ossification centers 1-4 (P >.003) and of occurrence of
8 thus impairing the formation of the primary ossification center and causing severe limb shortening.
9 eal demarcation, visibility of the secondary ossification center and its physis, and metaphyseal undu
10 eal demarcation, visibility of the secondary ossification center and its physis, prominence of the pe
11 physis and physis, and bone of the secondary ossification center and juxtaphyseal metaphysis), signal
12 c mice showed delayed formation of secondary ossification center and localized increase of bone mass
14 depicted five regions between the secondary ossification center and the metaphysis corresponding his
15 d wider with delayed and malformed secondary ossification centers and an irregular and highly expande
16 layed invasion of blood vessels into primary ossification centers and delayed removal of terminal hyp
17 ociated with poor vascularization of primary ossification centers and disrupted endochondral ossifica
18 one formation would accelerate the fusion of ossification centers and limit the endochondral bone gro
19 the physis, epiphyseal cartilage, secondary ossification center, and metaphysis was qualitatively as
21 ic zone, the delay in formation of secondary ossification centers, and the achondroplasia-like phenot
23 vascular invasion and formation of the early ossification center at least in part by interfering with
24 lowed by the formation of a new endochondral ossification center at the distal end of the bone stump.
25 f provisional calcification of the secondary ossification center, (b) physis of the secondary ossific
26 fication center, (b) physis of the secondary ossification center, (c) epiphyseal cartilage, (d) physi
28 delayed invasion of vessels into the primary ossification center, demonstrating a significant role of
30 have neither craniosynostosis nor additional ossification centers in interfrontal suture and displaye
32 zone comes to be subdivided by the secondary ossification center into distinct articular and growth c
35 asts migrate from perichondrium into primary ossification centers of cartilage templates of future bo
36 embryos from days 11 to 17, particularly in ossification centers of the embryonic skeleton at day 15
38 portant role in the formation of the primary ossification centers (POCs) and secondary ossification c
40 hypertrophic cartilage in growth plates and ossification centers; proliferative chondrocytes also st
42 ry ossification centers (POCs) and secondary ossification centers (SOCs) of mammalian long bones.
43 structures by stimulating a new endochondral ossification center that utilizes an existing network of
44 adiograph: the appearance, size and shape of ossification centers, the width and the shape of growth
45 During vascular invasion and formation of ossification centers, these Nes(+) cells were closely as
46 ication orientation in the condylar ramus (1 ossification center) versus long bone ossification forma
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