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1 ) versus long bone ossification formation (2 ossification centers).
2 stribution that decreased toward the primary ossification center.
3  promote synchondrosis closure and fusion of ossification centers.
4 ay in the formation of primary and secondary ossification centers.
5 and formation and vascularization of primary ossification centers.
6 normally occurs in growth plates and primary ossification centers.
7 nchronous ossification of one of the sternal ossification centers 1-4 (P >.003) and of occurrence of
8  thus impairing the formation of the primary ossification center and causing severe limb shortening.
9 eal demarcation, visibility of the secondary ossification center and its physis, and metaphyseal undu
10 eal demarcation, visibility of the secondary ossification center and its physis, prominence of the pe
11 physis and physis, and bone of the secondary ossification center and juxtaphyseal metaphysis), signal
12 c mice showed delayed formation of secondary ossification center and localized increase of bone mass
13 clast numbers were reduced in both secondary ossification center and proximal metaphysis.
14  depicted five regions between the secondary ossification center and the metaphysis corresponding his
15 d wider with delayed and malformed secondary ossification centers and an irregular and highly expande
16 layed invasion of blood vessels into primary ossification centers and delayed removal of terminal hyp
17 ociated with poor vascularization of primary ossification centers and disrupted endochondral ossifica
18 one formation would accelerate the fusion of ossification centers and limit the endochondral bone gro
19  the physis, epiphyseal cartilage, secondary ossification center, and metaphysis was qualitatively as
20 ed protein accumulation in marrow, secondary ossification centers, and periosteum.
21 ic zone, the delay in formation of secondary ossification centers, and the achondroplasia-like phenot
22 of another novel imprinted transcript, Ocat (ossification center-associated transcript).
23 vascular invasion and formation of the early ossification center at least in part by interfering with
24 lowed by the formation of a new endochondral ossification center at the distal end of the bone stump.
25 f provisional calcification of the secondary ossification center, (b) physis of the secondary ossific
26 fication center, (b) physis of the secondary ossification center, (c) epiphyseal cartilage, (d) physi
27                             The endochondral ossification center contains proliferating chondrocytes
28 delayed invasion of vessels into the primary ossification center, demonstrating a significant role of
29                       In addition, secondary ossification centers do not form in the central regions
30 have neither craniosynostosis nor additional ossification centers in interfrontal suture and displaye
31       Using the polarity of the endochondral ossification centers induced by BMP2 at two different am
32 zone comes to be subdivided by the secondary ossification center into distinct articular and growth c
33                              Missing sternal ossification centers occur most commonly at segments 2 a
34 le due to repetitive strain on the secondary ossification center of the tibial tuberosity.
35 asts migrate from perichondrium into primary ossification centers of cartilage templates of future bo
36  embryos from days 11 to 17, particularly in ossification centers of the embryonic skeleton at day 15
37  physis, epiphyseal cartilage, and secondary ossification center (P < .05).
38 portant role in the formation of the primary ossification centers (POCs) and secondary ossification c
39 he chondrocytes of the prospective secondary ossification center precludes its development.
40  hypertrophic cartilage in growth plates and ossification centers; proliferative chondrocytes also st
41 in a delay in the formation of the secondary ossification center (SOC).
42 ry ossification centers (POCs) and secondary ossification centers (SOCs) of mammalian long bones.
43 structures by stimulating a new endochondral ossification center that utilizes an existing network of
44 adiograph: the appearance, size and shape of ossification centers, the width and the shape of growth
45    During vascular invasion and formation of ossification centers, these Nes(+) cells were closely as
46 ication orientation in the condylar ramus (1 ossification center) versus long bone ossification forma

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