ライフサイエンスコーパス: osteoblast number

1 This suggests that FSH negatively regulates osteoblast number.

2 ing to increased mineral apposition rate and osteoblast number.

3 ar bone with thinner cortices, and decreased osteoblast number.

4 one volume and BFR as well as osteoclast and osteoblast numbers.

5 rsus non-Tg siblings, arising from increased osteoblast numbers.

6 tis, clopidogrel had little effect on MSC or osteoblasts numbers.

7 olume (32%), trabecular thickness (29%), and osteoblast numbers (68%) as well as a 40% decrease in os

8 ytes stimulated bone formation by increasing osteoblast number and activity, which was due in part to

9 formation because no significant changes in osteoblast number and bone formation rate were observed.

10 in bone mass was due to an early increase in osteoblast number and bone formation rate, mediated by a

11 ading to, among other changes, a decrease in osteoblast number and bone formation.

12 Osteoblast number and bone resorption were not altered.

13 Effects of mobilization on osteoblast number and function depend on the function of

14 evere osteopenia due to a marked decrease in osteoblast number and function, although bone resorption

15 Hey2 transgenic females exhibited reduced osteoblast number and function, but no changes in bone r

16 last numbers, with no significant effects on osteoblast number and function.

17 es (expressing Osterix1) exhibited increased osteoblast number and high bone mass that was maintained

18 dvancing age in mice is because of decreased osteoblast number and is associated with increased oxida

19 -R(-/-) bones exhibit a striking increase in osteoblast number and matrix accumulation.

20 Osteoblast number and mineral apposition rate were signi

21 reased bone mass was the result of increased osteoblast number and osteoblast activity with unaltered

22 s from a combination of Wnt-driven increased osteoblast number and resorption-dependent osteoblast ac

23 In contrast, osteoblast number and surface and bone formation rate in

24 OG2-DeltaFosB mice demonstrated increased osteoblast numbers and an osteosclerotic phenotype but n

25 ment with anti-sclerostin antibody increased osteoblast numbers and bone formation rate but did not i

26 , PTH administration significantly increased osteoblast numbers and bone formation rate in both contr

27 showed significantly reduced osteoclast and osteoblast numbers and exhibited an osteopenic bone phen

28 deficient in PP5 phosphatase have increased osteoblast numbers and high bone formation, which result

29 This was associated with increased osteoblast numbers and osteoblast activity based on bone

30 rden, mouse IL-6, and osteoclasts, increased osteoblast number, and inhibited bone destruction as mea

31 ed osteoblast function without a decrease in osteoblast numbers, and by increased bone resorption tha

32 emonstrate low bone mineral density, reduced osteoblast numbers, and increased osteoclast numbers.

33 , increased osteoclastogenesis and decreased osteoblast number, as quantified by histomorphometry and

34 revention was attributable to maintenance of osteoblast numbers because inhibition of serotonin recep

35 ate osteoblast apoptosis, thereby increasing osteoblast number, bone formation rate, and bone mass, b

36 were associated with low osteoclast number, osteoblast number, bone formation rate, and wall width i

37 Osteoblast number, bone volume, and trabecular thickness

38 icantly reduced these changes; SCI decreased osteoblast numbers, but increased expression in these ce

39 ed greater bone volume by twofold and higher osteoblast number by threefold, compared with the contro

40 Teriparatide increases osteoblast numbers by suppressing osteoblast apoptosis a

41 We show that osteoblast numbers decrease by 55% in myelodysplasia and

42 d significant increase in osteoid thickness, osteoblast number, erosion surface with osteoclasts, and

43 Besides increasing osteoblast number in the trabecular bone, deletion of Bm

44 reduced bone formation because of decreased osteoblast numbers in response to ibandronate.

45 osteoblasts lining trabecular bone, whereas osteoblast numbers in similarly treated mice lacking the

46 Osteoblast numbers in SR625 group were significantly hig

47 of duodenal serotonin, a hormone suppressing osteoblast numbers, inhibited loss of osteoblasts.

48 ce have low bone mass accompanied by reduced osteoblast numbers, low bone formation rates, and increa

49 In vivo, MAGP1Delta mice exhibit normal osteoblast number, mineralized bone surface, and bone fo

50 varia in vivo model, cause a 50% decrease in osteoblast number (N.Ob-BS) and a 32% decrease in minera

51 were no differences in adipocyte number and osteoblast number or activity.

52 s and bone resorption, without a decrease in osteoblast number or bone formation.

53 steoclast (OC) numbers, but had no change in osteoblast numbers or bone formation rate.

54 position rates) and static (osteoid markers, osteoblast number) parameters of bone formation were dec

55 characterized by significant depressions in osteoblast number, perimeters, bone formation rates, and

56 o reduced osteoclast formation and increased osteoblast numbers, respectively.

57 of HBD peptide to Igfbp2(-/-) mice increased osteoblast number, suppressed marrow adipogenesis, resto

58 mice, which was due to a marked increase in osteoblast number that was likely to be driven by hyperp

59 Osteoblast number was markedly decreased and osteogenic

60 Although CFU-fibroblast (CFU-F) and total osteoblast numbers were decreased, the bone formation ra

61 R625, and SR900 groups, significantly higher osteoblast numbers were detected than in control group (

62 contrast, new bone and osteoid formation and osteoblast numbers were increased significantly vs. untr

63 Interestingly, osteoblast numbers were maintained despite the anti-reso

64 clast numbers and progressive bone loss, but osteoblast numbers were not coordinately increased, as i

65 Consistently, significantly elevated osteoblast numbers were noted.

66 found that the TIEG(-/-) mice had increased osteoblast numbers with no increased bone formation para

67 mr(-/-)) also had reduced bone formation and osteoblast number within the injury site.