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1 tly discovered a novel function for MDSCs as osteoclast progenitors.
2 identify the quiescent PPARgamma(+) cells as osteoclast progenitors.
3 F-kappaB and JNK activation in RANKL-treated osteoclast progenitors.
4 essed by transfected cell lines and purified osteoclast progenitors.
5 vating TRAF6-dependent signaling pathways in osteoclast progenitors.
6 sarcoma oncogene family, protein B (MAFB) in osteoclast progenitors.
8 ly expressed by isolated bone marrow-derived osteoclast progenitors and by mature osteoclasts in vivo
9 steoclastogenesis both directly by acting on osteoclast progenitors and indirectly by increasing expr
10 ed bone formation, but increased bone marrow osteoclast progenitors and osteoclast numbers on bone su
11 lity that estrogen exerts a direct effect on osteoclast progenitors, and does so through the estrogen
12 calcium concentrations, increased committed osteoclast progenitors, and mature osteoclasts as well a
14 ings not only identify the long-sought-after osteoclast progenitors but also establish unprecedented
15 owever, the role of B cells is not to act as osteoclast progenitors but may be to act as osteoclast s
17 flammatory signals led to the recruitment of osteoclast progenitor cell potential from ex vivo-isolat
18 mediated by enhancing expression of RANK in osteoclast progenitor cells and by upregulating secretio
19 f Rac1 restored the pathological increase in osteoclast progenitor cells in Nf1+/- mice and was suffi
21 examine the effects of estrogen loss at the osteoclast progenitor colony forming unit-granulocyte ma
22 unique cell surface phenotype of clonogenic osteoclast progenitors (colony-forming unit-osteoclast [
25 dies demonstrated that B cells do not act as osteoclast progenitors in estrogen-replete or estrogen-d
26 r primary M-MuLV infection of osteoclasts or osteoclast progenitors in the bone marrow, and they sugg
28 The signaling pathway activated by WNT16 in osteoclast progenitors is noncanonical, whereas the path
32 rescent protein) reporter mice, we show that osteoclast progenitors reside specifically in the PPARga
35 y support the contention that in addition to osteoclast progenitors such as CFU-GM, earlier hematopoi
36 mulating factor 1 (CSF1) is known to promote osteoclast progenitor survival, but its roles in osteocl
37 aired osteoblast mineralization and enhanced osteoclast-progenitor survival, leading to increased ost
38 g tumor progression, in this case as a novel osteoclast progenitor that specifically drives bone meta
39 hat PKD activity promotes differentiation of osteoclast progenitors through increased expression of D
40 eased osteoclast formation was observed when osteoclast progenitors were cocultured with oim/oim-deri
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