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1 stic bone formation and indirectly regulates osteoclastic bone resorption.
2 together have established that TSH inhibits osteoclastic bone resorption.
3 ovariectomy-induced bone loss by inhibiting osteoclastic bone resorption.
4 r of NF-kappaB ligand (RANKL), which induces osteoclastic bone resorption.
5 FK) is, in contrast, a negative regulator of osteoclastic bone resorption.
6 ctor kappaB ligand, an essential mediator of osteoclastic bone resorption.
7 ance between osteoblastic bone formation and osteoclastic bone resorption.
8 and metastasize into bone tissue by inducing osteoclastic bone resorption.
9 s from inflammatory reactions that stimulate osteoclastic bone resorption.
10 xpression of lysosomal enzymes essential for osteoclastic bone resorption.
11 d by osteoblasts and seems to be involved in osteoclastic bone resorption.
12 ance between osteoblastic bone formation and osteoclastic bone resorption.
13 ys its so far uncharacterized action against osteoclastic bone resorption.
14 nhanced expression of cytokines that promote osteoclastic bone resorption.
15 hosphonates and their activity in inhibiting osteoclastic bone resorption.
16 L-6), an inflammatory cytokine implicated in osteoclastic bone resorption.
17 which mediates the subsequent stimulation of osteoclastic bone resorption.
18 creases bone mass as the result of defective osteoclastic bone resorption.
19 nt bone loss because of a marked increase in osteoclastic bone resorption.
20 tastases that are characterized by excessive osteoclastic bone resorption.
21 is the major protease responsible for human osteoclastic bone resorption.
22 Superoxide production contributes to osteoclastic bone resorption.
23 tion and sealing zone formation required for osteoclastic bone resorption.
24 I collagen is necessary for PTH induction of osteoclastic bone resorption.
25 formation of the sealing zone, required for osteoclastic bone resorption.
26 family of diseases characterized by impaired osteoclastic bone resorption.
27 d a rising ambient Ca2+ interact to regulate osteoclastic bone resorption.
28 ast precursors with a subsequent increase in osteoclastic bone resorption.
29 reases in osteoblastic matrix deposition and osteoclastic bone resorption.
30 cathepsin O2 may play a major role in human osteoclastic bone resorption.
31 rparts, HIV-1 transgenic rats undergo severe osteoclastic bone resorption, a consequence of an imbala
34 and atorvastatin (ATV) are known to inhibit osteoclastic bone resorption and have been proposed to h
35 nflammatory bone loss through stimulation of osteoclastic bone resorption and inhibition of osteoblas
36 thermore, PTHrP caused a coupled increase in osteoclastic bone resorption and new bone formation that
37 loma bone disease is caused by uncoupling of osteoclastic bone resorption and osteoblastic bone forma
38 e of tumor implantation profoundly inhibited osteoclastic bone resorption and prevented hypercalcemia
39 -9 signaling, resulting in the inhibition of osteoclastic bone resorption and prostate cancer bone me
40 The bone disease is mediated by increased osteoclastic bone resorption and suppressed bone formati
41 an aminobisphosphonate, is known to inhibit osteoclastic bone resorption and was proposed to have os
43 ase of bone is based on giving inhibitors of osteoclastic bone resorption, and bisphosphonates are th
44 nes during osteoclastogenesis, and prevented osteoclastic bone resorption but did not impair osteobla
45 264.7 cells suggested that the inhibition of osteoclastic bone resorption by these compounds did not
47 oblasts takes advantage of the regulation of osteoclastic bone resorption exerted by osteoblasts.
49 ncreased recognition of factors that promote osteoclastic bone resorption in cancer patients led us t
51 tives that have potent inhibitory effects on osteoclastic bone resorption in vitro and that prevent o
52 teoblastic activity to match the increase in osteoclastic bone resorption induced by estrogen deficie
53 nd 100 microg/l), reversed the inhibition of osteoclastic bone resorption induced by high extracellul
56 position, whereas in modern humans extensive osteoclastic bone resorption is found in the same region
58 ata suggest that the actions of IFN-gamma on osteoclastic bone resorption may be mediated by its effe
59 remodeling, osteoblastic bone formation, and osteoclastic bone resorption, mediated via the TSH recep
60 nment of the tooth because of the failure of osteoclastic bone resorption on the coronal tooth surfac
63 sive loss of bone mass resulting from excess osteoclastic bone resorption relative to osteoblastic bo
64 itory effect of Pyk2(Y402F), suggesting that osteoclastic bone resorption requires both c-Src kinase
67 ortisol inhibits acid-induced, cell-mediated osteoclastic bone resorption through a decrease in osteo
68 he aryl hydrocarbon receptor (Ahr) to induce osteoclastic bone resorption through the activation of c
69 wn to enhance osteoblastogenesis and inhibit osteoclastic bone resorption, thus promoting tissue rege
70 s process of osteoblastic bone formation and osteoclastic bone resorption to maintain normal bone mas
72 osteoblastic bone formation and by increased osteoclastic bone resorption, we assessed whether oxidiz
73 ovariectomy-induced bone loss by inhibiting osteoclastic bone resorption, whereas flurbiprofen at si
74 ive phosvitin potently inhibited PTH-induced osteoclastic bone resorption with simultaneous new osteo
75 xamination revealed that ibandronate reduced osteoclastic bone resorption, with increased apoptosis i
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