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1 who underwent above-knee amputation to treat osteogenic sarcoma.
2 at may play a role in metastatic activity in osteogenic sarcomas.
3 origins, breast and prostatic carcinomas and osteogenic sarcoma, also demonstrated very similar cytot
6 oid leukemia, 36 for colon carcinoma, 33 for osteogenic sarcoma, and 12 for female genital cancers.
7 l analysis of the arrest, we generated U2-OS osteogenic sarcoma cell clones in which p16 transcriptio
9 abundantly expressed on primary osteoblasts, osteogenic sarcoma cell lines, and primary fibroblasts.
10 edly reduced in primary fibroblasts and U2OS osteogenic sarcoma cells by treatment with small molecul
12 of multiple tumor cell lines including U2OS osteogenic sarcoma cells, SY5Y neuroblastoma cells, and
13 ock arsenite-induced transformation of human osteogenic sarcoma (HOS) cells to anchorage-independence
14 have been performed on rhabdomyosarcomas and osteogenic sarcomas, including cell lines and animal mod
15 s, cholangiocarcinomas, hepatoblastomas, and osteogenic sarcomas), individual lineages yielded tumors
16 reatment of U2-OS cells, a wt-p53-containing osteogenic sarcoma line, and Saos-2 cells, a p53-negativ
17 t of U2-OS cells, a wild-type p53-containing osteogenic sarcoma line, with a common p53 inducer, etop
18 rcoma line, and Saos-2 cells, a p53-negative osteogenic sarcoma line, with etoposide, a potent induce
19 ed magnetic resonance (MR) images of primary osteogenic sarcoma (n = 19) and Ewing sarcoma (n = 10) w
21 erstand the early molecular events involving osteogenic sarcoma (OGS), we have initiated a program to
22 essenger RNA (mRNA) in three of four primary osteogenic sarcoma (OGS)-derived cell lines and in eight
26 y alteration in gene expression pertinent to osteogenic sarcoma which was achieved by employing the m
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