ライフサイエンスコーパス: osteoid

1 y of the bone in which spindle cells produce osteoid.

2 lasts display polarised behaviour to deposit osteoid.

3 thin dentin, lack of acellular cementum, and osteoid accumulation in alveolar bone.

4 uding radiolucencies and resorptive lesions, osteoid accumulation on the alveolar bone crest, and sig

5 non-mineralized polymer scaffold (19 +/- 8% osteoid and 10 +/- 2% mineralized tissue).

6 biopsy specimens revealed numerous areas of osteoid and bone formation around FDBA particles, with n

7 Although D2J mice showed decreased osteoid and mineralization surfaces, their osteoblasts w

8 ed with decreased trabecular bone volume and osteoid and osteoblast surfaces in postmenopausal osteop

9 nd length of trabecular surface covered with osteoid and up-regulates bone marker gene (OPN, Cbfa1, C

10 there was no significant difference in total osteoid between the two samples, suggesting that increas

11 lastic bone resorption with simultaneous new osteoid/bone formation in the absence of ascorbate (vita

12 collagen synthesis and the formation of new osteoid/bone.

13 sed as the osteoblast differentiates into an osteoid cell or osteocyte, first appearing on the formin

14 In contrast, new bone and osteoid formation and osteoblast numbers were increased

15 Osteoid formation and osteocalcin expression were descri

16 y, and more distorted growth plate with more osteoid formation in the trabecular region.

17 lized tissue generation, but not necessarily osteoid formation.

18 oci of endosteally localized cells and human osteoid generation.

19 rocomputer tomography), and the formation of osteoids (histologically).

20 al heads, supporting a greater proportion of osteoid in the diseased tissue.

21 tly, osteoblast development and synthesis of osteoid in the nascent bone collar was uncoupled from th

22 SC) clusters, leading to formation of marrow osteoid islets accompanied by high levels of angiogenesi

23 Cancellous bone volume and osteoid markers correlated with bone mineral density mea

24 ates, adjusted apposition rates) and static (osteoid markers, osteoblast number) parameters of bone f

25 the splenic nodules revealed the presence of osteoid matrices and osteocytes trapped within mineraliz

26 rate significantly increased regeneration of osteoid matrix (32 +/- 7% of total tissue area; mean +/-

27 tion, shown by accumulation of unmineralized osteoid matrix and interglobular patterns of protein dep

28 The majority (55 cases) demonstrated osteoid matrix mineralization; 17 showed marked minerali

29 trengthened by observations showing that the osteoid matrix that is responsible for implant osseointe

30 the presence of transformed cells producing osteoid matrix, even if these cells comprise a minority

31 the primary spongiosa with reduced immature osteoid (new bone formation) and overall length, which l

32 the block specimens exhibited no evidence of osteoid or active bone formation, but large marrow space

33 tions revealed no differences in osteoblast, osteoid, or osteoclast surface areas.

34 MR imaging features of osteoid osteoma (edema, hyperemia, and nidus vasculariza

35 ded radiofrequency ablation [RA] therapy for osteoid osteoma (OO).

36 ndings that were diagnostic for nonvertebral osteoid osteoma and no contraindications to MR imaging-g

37 se, lower extremity injuries in toddlers and osteoid osteoma are emphasized.

38 ale; mean age, 21 years) with a diagnosis of osteoid osteoma based on clinical and imaging findings.

39 at MR-guided focused ultrasound treatment of osteoid osteoma can be performed safely with a high rate

40 olinium-enhanced MR imaging demonstrated the osteoid osteoma equally well in eight of 11 patients and

41 ing, the edema and hyperemia associated with osteoid osteoma gradually disappeared in all lesions.

42 One osteoid osteoma had peak enhancement in the venous phase

43 Biopsy results revealed osteoid osteoma in 10 patients, chondroblastoma in one,

44 rable treatment option for the management of osteoid osteoma in children and young adults.

45 bone location, bone segment, location of the osteoid osteoma in relation to the native cortex, nidus

46 ) of 11 patients had peak enhancement of the osteoid osteoma in the arterial phase with early partial

47 CT-guided percutaneous RF ablation of osteoid osteoma is a safe and effective technique.

48 echniques were scored for conspicuity of the osteoid osteoma relative to the surrounding bone.

49 d, 263 patients who were suspected of having osteoid osteoma underwent 271 ablation procedures.

50 y ablation is now the standard treatment for osteoid osteoma, as the procedure can be performed with

51 wever, in the 10 patients with biopsy-proved osteoid osteoma, puncture of the tumor caused the mean c

52 t of a routine strategy for the treatment of osteoid osteoma.

53 increase significantly at needle puncture of osteoid osteoma.

54 Osteoid osteomas can be imaged with greater conspicuity

55 Diaphyseal osteoid osteomas demonstrate a lower ratio of nidus mine

56 The nidus mineralization ratio of osteoid osteomas increases significantly with pain durat

57 dolinium-enhanced MR images demonstrated the osteoid osteomas significantly better than the nonenhanc

58 In long bones, diaphyseal osteoid osteomas were significantly less mineralized tha

59 es of 11 patients with pathologically proven osteoid osteomas who underwent nonenhanced MR imaging, d

60 six patients with histopathologically proven osteoid osteomas, complete clinical files, and CT data w

61 compass enchondromas, aneurysmal bone cysts, osteoid osteomas, giant-cell lesions of bone, bone sarco

62 Except for percentage osteoid perimeter, there was no difference in bone histo

63 lines, and that binding results in decreased osteoid production in vitro.

64 lytic lesion formation by causing decreased osteoid production in vivo.

65 radiographic mineralization and histological osteoid production, the differentiation state of tumors

66 terized by severe osteoporosis and decreased osteoid production.

67 lume, trabecular number, osteoblast surface, osteoid surface, and bone-formation rate.

68 d mineralization lag time, as well as higher osteoid surface, osteoblastic surface, resorption surfac

69 It also increased osteoid surfaces and, importantly, bone formation rates.

70 ng decrease in the rate of mineralization of osteoid that occurred despite an unexpected osteoblast a

71 hat Dkk2-null mice have increased numbers of osteoids, these data indicate that Dkk2 has a role in la

72 e investigate osteoblast 'burial' within the osteoid they deposit.

73 one at 6 wk revealed significant increase in osteoid thickness, osteoblast number, erosion surface wi

74 stologic components (fibrous, chondroid, and osteoid), tumor grade, and marrow involvement.

75 Risedronate did associate with increased osteoid volume and trabecular thickness in male particip

76 The transport of osteoid water across the mineralized matrix of bone was

77 collagen type I is the main component of the osteoid, we hypothesized that the bone vasculature guide

78 atrix markers and existence of unmineralized osteoid were seen at M3 and M14.