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1 ust be instituted to avoid renal toxicity or osteonecrosis of the jaw.
2 e pathogenesis of the bisphosphonate-induced osteonecrosis of the jaw.
3 ic sites, such as the bisphosphonate-related osteonecrosis of the jaw.
4 zoledronic acid, who unexpectedly developed osteonecrosis of the jaw.
5 ons, tumor metastasis and infections such as osteonecrosis of the jaw.
6 el alendronate period, adjudicated events of osteonecrosis of the jaw (1 event each in the romosozuma
7 as associated with higher rates of confirmed osteonecrosis of the jaw (35 [4%]) than was clodronic ac
8 sion under Adalimumab therapy presented with osteonecrosis of the jaw after suspended oral and intrav
12 or hypocalcemia, and there were no cases of osteonecrosis of the jaw and no adverse reactions to the
20 te proactive adjudication of every potential osteonecrosis of the jaw by an international expert pane
21 acid group, there were 17 confirmed cases of osteonecrosis of the jaw (cumulative incidence, 1.1%; 95
24 d case series described clinical features of osteonecrosis of the jaw in patients with cancer who wer
26 eons described 104 patients with cancer with osteonecrosis of the jaw in the medical literature and i
28 res indicate worse disability), incidence of osteonecrosis of the jaw, kidney dysfunction, skeletal m
34 ta regarding atrial fibrillation, bone pain, osteonecrosis of the jaw (ONJ), atypical fractures, and
35 issues, inappropriate femoral fractures, and osteonecrosis of the jaw (ONJ), the pathophysiological m
41 udies have linked bisphosphonate therapy and osteonecrosis of the jaws (ONJ), but neither causality n
42 ing bisphosphonates are at greatest risk for osteonecrosis of the jaws; these patients represent 94%
45 2%] of 697 vs 172 [24%] of 704) but rates of osteonecrosis of the jaw were low in both groups (nine [
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