ライフサイエンスコーパス: osteoprogenitor cell

1 lation (EFS)-enhanced osteogenic response in osteoprogenitor cells.

2 tion and differentiation of chondrocytes and osteoprogenitor cells.

3 een the proliferation and differentiation of osteoprogenitor cells.

4 nd pericytes were postulated to be potential osteoprogenitor cells.

5 in regulating osteogenic differentiation of osteoprogenitor cells.

6 d molecular mechanisms, we studied C3H10T1/2 osteoprogenitor cells.

7 us mRNA in mouse pluripotent mesenchymal and osteoprogenitor cells.

8 lial cells can induce the differentiation of osteoprogenitor cells.

9 may directly regulate the differentiation of osteoprogenitor cells.

10 new blood vessels can serve as a conduit for osteoprogenitor cells.

11 ion of the nearby periosteum and a source of osteoprogenitor cells.

12 PYK2 regulates the differentiation of early osteoprogenitor cells across species and that inhibitors

13 accounted for by increased proliferation of osteoprogenitor cells and bone formation resulting from

14 ession is suppressed in nonosseous cells and osteoprogenitor cells and during the early proliferative

15 Osteoprogenitor cells and endosteal-lining osteopontin(+

16 ication is a regulated process that involves osteoprogenitor cells and frequently complicates common

17 ne marrow (BMT) leads to engraftment of both osteoprogenitor cells and hematopoietic cells; however,

18 is highly expressed in chondroprogenitor and osteoprogenitor cells and in vitro experiments suggest t

19 e marrow derived MSC differentiation towards osteoprogenitor cells and inhibited Notch signaling in a

20 d demonstrate that its functions map to both osteoprogenitor cells and mature osteoblasts.

21 chitosan potentiates the differentiation of osteoprogenitor cells and may facilitate the formation o

22 ein fingerprints from single native MC3T3-E1 osteoprogenitor cells and MC3T3-E1 cells transfected wit

23 ) transcription and delay differentiation in osteoprogenitor cells and patient-derived bone.

24 rix mineralization and calcium deposition by osteoprogenitor cells and primary mesenchymal stem cells

25 o apoptosis, leading to decreased numbers of osteoprogenitor cells and subsequently reduced bone form

26 tase subunits was confirmed in human primary osteoprogenitors cells, and a significant increase in en

27 idics enables efficient sorting of committed osteoprogenitor cells, as distinct from these mesenchyma

28 ncy enhances differentiation and activity of osteoprogenitor cells, as does expressing a PYK2-specifi

29 d halt the recruitment or the advancement of osteoprogenitor cells at the sites where sutures should

30 cytes with the growth and differentiation of osteoprogenitor cells by simultaneously modulating Bmp4

31 for their effects on the differentiation of osteoprogenitor cells (C2C12) and the proliferation and

32 In line with this, osteoprogenitor cell cultures from the Sirt1(DeltaOsx1)

33 ed the role of p27 during differentiation of osteoprogenitor cells derived from the bone marrow (BM)

34 me of our data differ from current models of osteoprogenitor cell differentiation and emphasize compo

35 nd molecular evidence for Dlx3 in regulating osteoprogenitor cell differentiation and for both positi

36 hosphatase activity suggests that effects on osteoprogenitor cell differentiation are the result of a

37 of the biology of bone graft remodeling and osteoprogenitor cell differentiation.

38 feration and differentiation in CNCC-derived osteoprogenitor cells during intramembranous bone format

39 e also active in the programming of arterial osteoprogenitor cells during vascular and valve calcific

40 n periosteal (HPO) cells were chosen because osteoprogenitor cells found in bone repair typically com

41 iferation versus osteoblast differentiation, osteoprogenitor cells from the skulls of Tgfbr2(f/f) emb

42 In contrast, p53-null osteoprogenitor cells have increased proliferation, incr

43 egulate the proliferation and recruitment of osteoprogenitor cells; however, CTGF is down-regulated a

44 as the use of concentrated blood products or osteoprogenitor cells in conjunction with grafts, have b

45 o affected the proliferation and survival of osteoprogenitor cells in osteogenic condensations, leadi

46 TGFbeta IIR is required for proliferation of osteoprogenitor cells in the CNC-derived frontal bone an

47 suggest that the endothelium is a source of osteoprogenitor cells in vascular calcification that occ

48 but also to modulate the differentiation of osteoprogenitor cells in vitro and in vivo.

49 naling affected the functional activities of osteoprogenitor cells, including the RUNX2-mediated tran

50 The differentiation of osteoprogenitor cells, indicated by alkaline phosphatase

51 and blocks BMP2-mediated differentiation of osteoprogenitor cells into osteoblasts.

52 /progenitor cells (MSCs)--rather than mature osteoprogenitor cells--into osteoblasts, resulting in ne

53 In addition, the differentiation of marrow osteoprogenitor cells is regulated by leptin.

54 Deletion of Pten in osteoprogenitor cells led to increased numbers of osteob

55 rized the response of 2T9 cells, an immature osteoprogenitor cell line derived from the calvariae of

56 In this study we examined an immature osteoprogenitor cell line for its potential utility in m

57 Microarray analysis of MC3T3 cells, an osteoprogenitor cell line, revealed that EGFR signaling

58 were initiated using MC3T3-E1 cells, a mouse osteoprogenitor cell line.

59 These osteoprogenitor cells may be derived from the circulatio

60 This increased pool of osteoprogenitor cells may be susceptible to additional t

61 ted from combinations of such materials with osteoprogenitor cells or osteoinductive factors such as

62 There is evidence that the size of the osteoprogenitor cell population determines the rate of c

63 an active process that can originate from an osteoprogenitor cell population in the adventitia.

64 the developing bone, consequently promoting osteoprogenitor cell proliferation and decreasing differ

65 ng that allows for independent regulation of osteoprogenitor cell proliferation and differentiation.

66 Moreover, Panx3 also inhibits osteoprogenitor cell proliferation and promotes cell cyc

67 In calvarial cultures we reduced osteoprogenitor cell proliferation; however, we did not

68 n osteoblasts, and overexpression of Dlx3 in osteoprogenitor cells promotes, while specific knock-dow

69 , the presence of Runx2 in actively dividing osteoprogenitor cells suggests that the protein may also

70 row cells can serve as an abundant source of osteoprogenitor cells that are capable of repairing cran

71 lial cells but required for specification of osteoprogenitor cells that differentiate into preosteobl

72 Significantly, PTEN is found in osteoprogenitor cells that give rise to bone-forming ost

73 Fgfr2 is expressed only in proliferating osteoprogenitor cells; the onset of differentiation is p

74 ically caused by impaired differentiation of osteoprogenitor cells, they also suggest that increased

75 te rate of osteoblastic differentiation from osteoprogenitor cell to terminally differentiated osteoc

76 cular endothelium has a role in contributing osteoprogenitor cells to the calcific lesions.

77 lates the vascular endothelium to contribute osteoprogenitor cells to the vascular calcification.

78 The outcome of various osteoprogenitor-cell transplantation protocols was asses