ライフサイエンスコーパス: otoacoustic emission

1 r functions (auditory brainstem response and otoacoustic emissions).

2 y, compressive nonlinearity, and spontaneous otoacoustic emission.

3 conduction as well as numerous properties of otoacoustic emissions.

4 earing, sounds that are known as spontaneous otoacoustic emissions.

5 of the sensory input as well as spontaneous otoacoustic emissions.

6 stimulus, the inner ear emits sounds called otoacoustic emissions.

7 y brainstem responses and distortion product otoacoustic emissions.

8 oscillations that might underlie spontaneous otoacoustic emissions.

9 itive, sharply tuned hearing and spontaneous otoacoustic emissions.

10 t of middle-ear immittance, and recording of otoacoustic emissions.

11 Distortion product otoacoustic emission amplitudes of Coch(G88E/G88E) mice

12 Tone-evoked distortion product otoacoustic emission and auditory brainstem response mea

13 itory brainstem response, distortion product otoacoustic emission and cochlear microphonics tests, an

14 t with large reduction in distortion product otoacoustic emission and severe hearing loss at high fre

15 mutants show only minimal distortion product otoacoustic emissions and 70-80 dB threshold shifts in a

16 function was assessed via distortion product otoacoustic emissions and auditory brainstem responses (

17 function was assessed via distortion product otoacoustic emissions and auditory brainstem responses,

18 However, previous recordings of otoacoustic emissions and cochlear microphonic potential

19 They lack distortion product otoacoustic emissions and cochlear microphonic responses

20 ddle ear muscle reflexes, distortion product otoacoustic emissions and cochlear microphonics, as well

21 ferent function measures (distortion product otoacoustic emissions and contralateral suppression) wer

22 ms, to evaluate the feasibility of including otoacoustic emissions and extended high frequency audiom

23 in the presence of normal distortion product otoacoustic emissions and normal audiometric thresholds.

24 Since the discovery of otoacoustic emissions and outer hair cell (OHC) motility

25 not involved in the backward propagation of otoacoustic emissions and that sounds exit the cochlea p

26 sed on the measurement of stimulus-frequency otoacoustic emissions and, unlike previous noninvasive p

27 ochlear receptor outer hair cell activities (otoacoustic emissions) and absent or abnormally delayed

28 r microphonic potentials, distortion product otoacoustic emissions, and basilar membrane motion indic

29 ct otoacoustic emissions (DPOAEs), transient otoacoustic emissions, and the hearing-in-noise test (HI

30 stem evoked responses and distortion product otoacoustic emissions are, for most frequencies, normal

31 Such sounds, otoacoustic emissions, are widely used for diagnosis of

32 oscopy, tympanometry, and distortion product otoacoustic emissions as near the time of admission as w

33 e amount of change in the distortion product otoacoustic emission (at 2f(1)-f(2)) just after onset of

34 iological tests including distortion product otoacoustic emissions, auditory brainstem responses, env

35 Two possible sources for otoacoustic emissions, both localized within individual

36 Click-evoked otoacoustic emissions (CEOAEs) are echo-like waveforms e

37 es were unrelated to the modest variation in otoacoustic emissions, cochlear tuning, or the residual

38 r, the olivocochlear efferents, by examining otoacoustic emissions created by the normal ear, which c

39 se-induced suppression of distortion product otoacoustic emissions derived from outer hair cell trans

40 functionally assessed by distortion product otoacoustic emission (DPOAE) analysis.

41 function was evaluated by distortion product otoacoustic emission (DPOAE) and auditory brainstem resp

42 cally tuned effect on the distortion product otoacoustic emission (DPOAE) and the cochlear whole-nerv

43 Distortion product otoacoustic emission (DPOAE) assay and acoustic brainste

44 We used distortion product otoacoustic emission (DPOAE) measurements to monitor coc

45 Distortion product otoacoustic emission (DPOAE) testing was performed on 97

46 with the cubic 2f(1)-f(2) distortion product otoacoustic emissions (DPOAE) at the start of the study

47 Here we used 2f1-f2 distortion product otoacoustic emissions (DPOAE) measurements to refine the

48 ainstem response (ABR) or distortion product otoacoustic emissions (DPOAE) or is being challenged by

49 instem response (ABR) and distortion product otoacoustic emissions (DPOAE) to assess hearing recovery

50 se -- ABR thresholds, and distortion-product otoacoustic emission -- DPOAE magnitudes), and were clus

51 teral suppression (CS) of distortion product otoacoustic emissions (DPOAEs) in humans and CBA mice.

52 (0.5 to 8 kHz) and evoked distortion product otoacoustic emissions (DPOAEs) were conducted for 32 pat

53 stem responses (ABRs) and distortion product otoacoustic emissions (DPOAEs) were unaffected by loss o

54 hlear status, assessed by distortion product otoacoustic emissions (DPOAEs), and to further clarify t

55 audiometry, tympanometry, distortion-product otoacoustic emissions (DPOAEs), transient otoacoustic em

56 instem response (ABR) and distortion product otoacoustic emissions (DPOAEs).

57 outer hair cell function [distortion product otoacoustic emissions (DPOAEs)].

58 oltage, low-mid-frequency distortion-product-otoacoustic-emissions (DPOAEs), and passive basilar memb

59 mpound action potentials, distortion product otoacoustic emissions) during efferent fiber activation,

60 as assessed through the contralateral evoked otoacoustic emission (EOAE) amplitude attenuation effect

61 However, the fundamental question of how the otoacoustic emission exits the cochlea remains unanswere

62 and how the inner ear-generated sound, i.e., otoacoustic emission, exits the cochlea, we created a so

63 -frequency audiometry and distortion product otoacoustic emissions for ototoxicity monitoring in chil

64 aneous (SOAE) and stimulus-frequency (SFOAE) otoacoustic emissions from a bird (barn owl, Tyto alba)

65 afness by P25 and reduced distortion product otoacoustic emissions from P15 onward.

66 y brainstem responses and distortion product otoacoustic emissions from these mice displayed wild-typ

67 se thresholds and reduced distortion-product otoacoustic emissions, in the presence of normal endococ

68 Electrically evoked otoacoustic emission is a manifestation of reverse trans

69 le the exact mechanism for the production of otoacoustic emissions is not known, active motion of ind

70 n auditory threshold, and distortion product otoacoustic emission measurements indicate that this mil

71 roperties, as measured by distortion product otoacoustic emissions, neither before nor after noise ex

72 Neonatal hearing test results, including otoacoustic emission (OAE) data, were sought for all neo

73 Otoacoustic emissions (OAEs) are faint sounds generated

74 We used otoacoustic emissions (OAEs) as a method to quantify the

75 Otoacoustic emissions or OAEs (reflections of cochlear e

76 We suggest that otoacoustic emissions originate from phase coherence in

77 Furthermore, stimulus-frequency otoacoustic emissions (SFOAEs), sounds emitted by the ea

78 We also observed spontaneous otoacoustic emissions (SOAEs) in 70% of the homozygous m

79 e pharmacological sensitivity of spontaneous otoacoustic emissions (SOAEs) in a lizard, the Tokay gec

80 Otoacoustic emissions, sounds generated by the inner ear

81 Furthermore, otoacoustic emissions, sounds generated inside the cochl

82 e mice, mutant mice showed reduced or absent otoacoustic emissions, suggesting cochlear outer hair ce

83 e mice progressively lost distortion product otoacoustic emissions, suggesting defects in outer hair

84 hearing revealed subtle differences in their otoacoustic emissions, suggesting that the expression of

85 r hair cell function (cochlear microphonics, otoacoustic emissions, summating potentials) and auditor

86 hearing screening was performed by means of otoacoustic emission testing and auditory brain stem res

87 and the relatively robust distortion product otoacoustic emissions that are found in elderly subjects

88 The morphology of sensory hair cells and otoacoustic emissions that depend on the integrity of ha

89 anical activity in hair cells is spontaneous otoacoustic emission, the unprovoked emanation of sound

90 These spontaneous otoacoustic emissions, the most striking manifestation o

91 bly modulated by drugs that affect mammalian otoacoustic emissions, the salicylates and the aminoglyc

92 surement of auditory brainstem responses and otoacoustic emissions to assess cochlear presynaptic and

93 Otoacoustic emissions were absent in patients with a mod

94 Otoacoustic emissions were not impaired pointing to norm

95 Distortion product otoacoustic emissions were not recordable from Clrn1(-/-

96 rve action potential, and stimulus frequency otoacoustic emissions were recorded from 12 days after b