1 Our analyses identify recurrent mutations in 4 genes not prev
2 In addition,
our calculations identify the exciton absorption bands in tra
3 Thus,
our data identify a key cellular trafficking factor important
4 Thus,
our data identify a previously unsuspected mechanism by which
5 hem with two existing models that conditionally delete Tcf4
Our data identify a set of overlapping phenotypes that may se
6 Taken together,
our data identify cerebellar astrocytes as key responders to
7 Our data identify different receptor regions that are importa
8 Thus,
our data identify Flower as a key protein mediating granule e
9 Our data identify Fyn and Lyn as important factors that promo
10 Our data identify GA as an attractive drug repurposing candid
11 Our data identify Gag-protease as a major determinant of subt
12 Our data identify GFRAL as a new regulator of body weight and
13 Moreover,
our data identify mIndy as a target gene of IL-6 and determin
14 Our data identify Sema7A as a key regulator of adult hippocam
15 Collectively,
our data identify the canonical autophagy pathway in DCs as a
16 Our findings identify a core learning process disrupted by br
17 Collectively,
our findings identify a molecular decoy naturally generated d
18 Taken together,
our findings identify a new chromatin-based epigenetic mechan
19 Our findings identify a novel role for MC1-R in macrophage ch
20 Our findings identify a potential pathogenic mechanism attrib
21 In conclusion,
our findings identify a previously unidentified role for pero
22 mice infected with wild-type P. aeruginosa Taken together,
our findings identify a previously unknown mechanism by which
23 Our findings identify a previously unrecognized molecular mec
24 Our findings identify a unique function for Themis2 in differ
25 Our findings identify an architecture and fate of stomata in
26 Together,
our findings identify context-specific miRNA-regulated checkp
27 Our findings identify elevation of A3A expression in AML cell
28 Our findings identify kindlin-2 as a key protein that couples
29 Our findings identify NDPK-C as an essential requirement for
30 Our findings identify PABPC1 as a direct regulator of cardiac
31 Therefore
our findings identify PKC-theta as a critical factor for ILC2
32 Our findings identify RCL1 as a novel candidate gene for depr
33 essential for many metabolic pathways in eukaryotic cells,
our findings identify the phosphorylation of sPPases as a pot
34 Taken together,
our results identify a neuronal mechanism for controlling int
35 Our results identify a novel intracellular signaling module t
36 Our results identify a repertoire of toxins with different mo
37 Our results identify cell-adhesion molecule-mediated inhibiti
38 Our results identify DsbA-L as a critical regulator of mitoch
39 Our results identify MCL-1 as a critical prosurvival protein
40 Together,
our results identify Nur77 as a critical intracellular target
41 Our results identify promoter shape as a molecular trait that
42 Overall,
our results identify RXR as a regulator in the myeloid cell-a
43 Our results identify SAP as an inhibitor of PD-1 function and
44 Our results identify that MCAK promotes fast MT growth speeds
45 Overall,
our results identify the CXCL2/MIF-CXCR2 axis as an important
46 Our results identify the ERK1/2 pathway as a direct regulator
47 Overall,
our results identify the impact of different risk factors, wh
48 Our results identify viral transformation-driven synthetic le
49 Our studies identify a complex regulation of 5-HT homeostasis
50 In summary,
our studies identify distinct expression patterns and roles i