1 Our data are consistent with a causal relationship between BM
2 Our data are consistent with a genetic architecture for PTSD
3 In both [CO2]-mediated responses,
our data are consistent with a mechanism in which ABA increas
4 Instead,
our data are consistent with a model in which a DNA-bound for
5 Our data are consistent with a model in which breathing stimu
6 Our data are consistent with a model in which CCL27 drives ba
7 Our data are consistent with a model in which H3K27Me2 is a r
8 Our data are consistent with a model in which inhibition of s
9 Our data are consistent with a model in which PHLPP modifies
10 Our data are consistent with a model in which subsets of RNPs
11 Collectively,
our data are consistent with a model in which the beta subuni
12 Our data are consistent with a model in which the highly redu
13 knockout shifts splicing toward the fully spliced isoform,
our data are consistent with a model in which unproductive sp
14 Our data are consistent with a model of systemic and transgen
15 Together,
our data are consistent with a model where MICOS, mitochondri
16 Therefore,
our data are consistent with a model whereby augmented and or
17 Overall,
our data are consistent with a model wherein a single molecul
18 Our data are consistent with a phase transition model of carc
19 est that BNF in tropical forest has been overestimated, and
our data are consistent with a recent top-down estimate of gl
20 Our data are consistent with a simultaneous and synergistic t
21 Our data are consistent with a unique mode of MT interaction
22 Our data are consistent with a view that DNA bound proteins u
23 Our data are consistent with an age-related decline in autoph
24 Furthermore,
our data are consistent with an association between transient
25 Our data are consistent with an inverse association between n
26 We conclude that
our data are consistent with contrast-invariant orientation t
27 Our data are consistent with current models of affine strain
28 Our data are consistent with electroneutral entry of Na(+) oc
29 Our data are consistent with GAS6 being protective during EAE
30 Our data are consistent with neuropathological studies and fu
31 Our data are consistent with Plasmodium VITs playing a major
32 Our data are consistent with RocA interfacing with the CovR/S
33 Our data are consistent with RSV nonstructural proteins 1 and
34 ed sex ratios under different conditions, we also show that
our data are consistent with sl-CSD.
35 Finally,
our data are consistent with the brain forming a sanctuary si
36 Our data are consistent with the central medial thalamus acti
37 Our data are consistent with the domain rotation model but in
38 Furthermore,
our data are consistent with the FRUITFULL (FUL) MADS-domain
39 Our data are consistent with the hypothesis that RNA viruses
40 Our data are consistent with the hypothesis that the circannu
41 Our data are consistent with the idea that binding of Ena/VAS
42 Our data are consistent with the idea that FKBP8 and Hsp90bet
43 Furthermore,
our data are consistent with the idea that HSV counters STING
44 regulation of the inflammatory response in the cornea, and
our data are consistent with the idea that HSV modulates STIN
45 Our data are consistent with the idea that the CUL4A/B-DDB1-C
46 Our data are consistent with the model that REF/Aly is involv
47 tin expression and crown-like structures in adipose tissue,
our data are consistent with the notion that adipose tissue i
48 Our data are consistent with the putative th locus increasing
49 Our data are consistent with the scenario where cytochrome c
50 We show that
our data are consistent with V2/V3 input conferring structure