ライフサイエンスコーパス: our data suggest that

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1 Our data suggest that ApoA1 combined with 9alpha,11ss-PGF2 re

2 Our data suggest that CTCF and BORIS cooperate with additiona

3 Our data suggest that defective self-renewal ability and leuk

4 Our data suggest that eradication of HCV in coinfected patien

5 Our data suggest that interorgan signaling plays an important

6 Our data suggest that miR-323-3p acts in a negative feedback

7 Our data suggest that NCR peptides are host determinants of s

8 Our data suggest that nivolumab is active in relapsed/refract

9 Our data suggest that patients who have an efficiently-wired

10 Our data suggest that polymorphisms in the CYP2R1 and GC gene

11 Our data suggest that recessive mutations in FOXI1 can explai

12 Our data suggest that SERPINA3 may be involved in the pathoge

13 Our data suggest that SHP2 is an important regulator of fibro

14 Our data suggest that Src kinase may represent a potential ta

15 Our data suggest that the decay of platelet mRNAs is slowed b

16 Our data suggest that the implantation reaction that occurs i

17 Our data suggest that the LPAI H7N2 virus requires further ad

18 Our data suggest that the subiculum-containing detour loop is

19 Our data suggest that the wide spectrum of clinical outcomes,

20 Our data suggest that therapeutic anticoagulation is not nece

21 Our data suggest that Top3beta is the major topoisomerase for

22 Our data suggest that UCSs do not develop from EACs via EMT.

23 Our data suggest that VISTA represents another compensatory i

24 Our data suggest that Wnt-induced dissociation of the Axin/GS

25 Our data suggest that ZIKV, when produced from mammalian cell

26 Additionally, our data suggest that many heritable L1 insertions originate

27 Altogether, our data suggest that the altered response of B cells in tole

28 does not linearly equate with transcriptional kinetics, and our data suggest that SMAD2 recruits multiple co-factors duri

29 thalamic input to L4 in M1 has largely been neglected, but our data suggest that, as in S1, the thalamic input is amplif

30 Collectively, our data suggest that JNK activity promotes positive energy b

31 Collectively, our data suggest that NGD and subsequent quality control are

32 Collectively, our data suggest that patients should be stratified according

33 Collectively, our data suggest that reduced Cx43 HC function could promote

34 In conclusion, our data suggest that persistent EBOV infection in rhesus mon

35 s, e.g., uniquely patterned array and short turnover cycle, our data suggest that chicken gustatory tissue provides an id

36 Interestingly, our data suggest that the following base on the template stra

37 Moreover, our data suggest that adaptive NK cells are more long-lived t

38 Moreover, our data suggest that internalization of Notch via endocytosi

39 Overall, our data suggest that CT-optimal, affective touch enhances su

40 Overall, our data suggest that hepsin, like matriptase, induces potent

41 increasing cAMP and enhancing cholangiocyte proliferation; our data suggest that a TGR5 antagonist alone or concurrently

42 Therefore, our data suggest that Nox4 mediates LPS-TLR4 signaling in hum

43 Together our data suggest that pTRS1 inhibits PKR by binding to conser

44 Together, our data suggest that pharmacological inhibition of necroptos

45 Together, our data suggest that PON-2 regulates ENaC activity by modula

46 Together, our data suggest that SK1 is important for islet revasculariz

47 Together, our data suggest that the heptad repeat LIZ contributed to TM

48 Taken together, our data suggest that instability in the genetic architecture

49 Taken together, our data suggest that p300-mediated acetylation of HIPK2 incr

50 Contrary to the current view, our data suggest that reciprocal repression of GATA6 and NANO

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