コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 ling can substitute for the influence of the outflow tract.
2 locity-time integral of the left ventricular outflow tract.
3 with a striking increase in the size of the outflow tract.
4 VEGF-C to stimulate vessel growth around the outflow tract.
5 V internal dimension were measured in the RV outflow tract.
6 ied the focal origin in the left ventricular outflow tract.
7 evelops from the slowly conducting embryonic outflow tract.
8 the AV endocardial cushions and the cardiac outflow tract.
9 ions in the pharyngeal apparatus and cardiac outflow tract.
10 particular, for the formation of the cardiac outflow tract.
11 ond heart field cells that contribute to the outflow tract.
12 r migration to the heart or septation of the outflow tract.
13 nd in the correct positioning of the cardiac outflow tract.
14 delay in the anterolateral right ventricular outflow tract.
15 displacement into the left ventricular (LV) outflow tract.
16 al arches and the septation of the heart and outflow tract.
17 ginally smaller in BAs than in WAs (proximal outflow tract, 30.9+/-5.5 versus 32.8+/-5.3 mm, P<0.001;
18 , we found aberrant myocardialization of the outflow tract, a process also known to be EMT dependent.
19 iagnosis for syndromic and nonsyndromic left outflow tract abnormalities and implications for at-risk
21 versus 0 mV); (2) delayed right ventricular outflow tract activation (82+/-18 versus 37+/-11 ms); (3
22 ryological context for understanding cardiac outflow tract alignment and membranous ventricular septa
23 lesion, supporting the idea that AHF-derived outflow tract alignment defects may constitute an embryo
24 function in the AHF results in a spectrum of outflow tract alignment defects ranging from overriding
26 ng premature ventricular contractions of the outflow tract alternating with papillary muscle or fasci
27 entricular contractions originating from the outflow tract alternating with the papillary muscle or f
28 -CMR data analysis included left ventricular outflow tract and aortic valve segmentation, and extract
30 the second heart field, pharyngeal endoderm, outflow tract and atrioventricular endocardial cushions
31 ct was the most common CHD observed, whereas outflow tract and atrioventricular septal defects were t
33 sect Hand2-dependent defects specifically in outflow tract and cardiac cushion independent of Hand2 f
34 uber ciliopathy syndromes, including cardiac outflow tract and cochlea defects associated with PCP pe
38 xplants with isolated pharyngeal endoderm or outflow tract and found that outflow tract co-cultures p
40 ritruncal blood vessels encircle the cardiac outflow tract and invade the aorta, but the underlying p
42 rise to three cardiovascular lineages in the outflow tract and myocardium in the distal ventricle.
44 participate in the remodeling of the cardiac outflow tract and pharyngeal arch arteries during cardio
46 age-sensitive role in the differentiation of outflow tract and right ventricle from progenitors of th
49 second heart field (AHF), gives rise to the outflow tract and the majority of the right ventricle an
50 regulates the signaling processes leading to outflow tract and valve morphogenesis and ventricular tr
53 tracellular matrix homeostasis in HDAC3-null outflow tracts and semilunar valves, and pharmacological
55 hose that affect the proper alignment of the outflow tracts and septation of the ventricles are a hig
58 om the epicardial right ventricular apex, RV outflow tract, and LV free wall, as well as premature at
59 tis, in situ stents in the right ventricular outflow tract, and presence of outflow tract irregularit
63 l was used for soft tissue structures of the outflow tract, aortic root, and noncalcified valve cusps
64 tive primary repair; their right ventricular outflow tracts are characterized by mild residual obstru
69 teria can help distinguish right ventricular outflow tract arrhythmias originating from ARVD/C compar
71 s in lower vertebrates contain a rudimentary outflow tract but not a right ventricle, the existence a
72 y run in parallel along the left ventricular outflow tract, but in the Nkx2-5(+/-)/Sspn(KO) mutant th
74 eal endoderm or outflow tract and found that outflow tract co-cultures prevented SAG-induced prolifer
75 arteriosus in basal actinopterygians, to an outflow tract commanded by the non-valved, elastic, bulb
76 o difference in E12 in the right ventricular outflow tract compared with the right-left ventricular o
79 function in patients with right ventricular outflow tract conduit dysfunction; the impact of this te
81 implantation in obstructed right ventricular outflow tract conduits in 2010 after a multicenter trial
86 misalignment of the aortic arch arteries and outflow tract, contributing to development of double out
87 ects including pharyngeal arch artery (PAA), outflow tract, craniofacial and thymic abnormalities.
89 oppler gradient across the right ventricular outflow tract decreased from 41.9 +/- 27.9 mm Hg to 19.1
90 sistent truncus arteriosus, a severe cardiac outflow tract defect also seen in human congenital heart
92 ls that some Isl1 derivatives in the cardiac outflow tract derive from Wnt1-expressing neural crest p
94 es of cardiac progenitor differentiation and outflow tract development and has implications for under
95 e myocyte cell-cell adhesions during cardiac outflow tract development contributes to impaired outflo
96 hich is required for aortic arch and cardiac outflow tract development, and is a known genetic intera
98 (HR, 0.94 per 1%; P=0.02), right ventricular outflow tract diameter (HR, 1.08 per 1 mm; P=0.01), mitr
100 a mean (SD) follow-up of 6.4 (2.5) years, RV outflow tract dimension increased from 35 mm (interquart
103 myofibres and collagen fibres to the apex-to-outflow-tract direction was consistent with this also be
104 tics of both syndromic and nonsyndromic left outflow tract disorders is hoped to lead to improved ide
105 ution involves the transition from a cardiac outflow tract dominated by a multi-valved conus arterios
106 nsformative technology for right ventricular outflow tract dysfunction with the potential to expand t
109 lation without wall motion abnormalities; RV outflow tract ectopy; and exercise-induced T-wave pseudo
110 eir migration to the proximal aspects of the outflow tract endocardial cushions, resulting in the fai
111 inding EGF-like growth factor to Jag1-mutant outflow tract explant cultures rescued the hyperprolifer
114 in gestation and display defects in cardiac outflow tract formation, atrial and ventricular septatio
116 cells required for proper patterning of the outflow tract, generation of the appropriate number of n
119 ficacy of both SA and SM in left ventricular outflow tract gradient (LVOTG) reduction seems comparabl
120 essure ratio (P<0.001) and right ventricular outflow tract gradient (P=0.004) than those with no tear
123 h significant reduction in right ventricular outflow tract gradient and the RV:Ao ratio when compared
124 g patients with significant (>/=30 mm Hg) RV outflow tract gradient and/or other residual hemodynamic
127 ce interval, 1.02-2.30) and left ventricular outflow tract gradient progression (hazard ratio, 1.45;
128 IVSd was not related to left ventricular outflow tract gradient reduction at rest (P=0.883) or du
131 estent and lower discharge right ventricular outflow tract gradient were associated with longer freed
132 athy (HCM) exhibit elevated left ventricular outflow tract gradients (LVOTGs) and appear to have a wo
133 zation data in 162 consecutive patients with outflow tract gradients (median [interquartile range], 9
135 ASA had equal effects on left ventricular outflow tract gradients and symptoms throughout the spec
137 iomyopathy was frequently observed (proximal outflow tract >/=32 mm; 45.0% of BAs, 58.5% of WAs).
139 446), and latest endocardial site was in the outflow tract in 13 of 18 ARVD patients versus 4 of 6 co
143 t ventricular outflow tract, and presence of outflow tract irregularities at the implant site were as
144 enting of the ventricular septum or systemic outflow tract is feasible and effective in the short ter
146 ant mice, this latter process fails, and the outflow tract is shortened and misaligned as a result.
148 initial SHF population incorporates into the outflow tract, it is replenished from the surrounding pr
150 lmonary left ventricle, and left ventricular outflow tract (LVOT) conduit dysfunction has not been st
152 criteria for distinguishing left ventricular outflow tract (LVOT) from right ventricular outflow trac
153 e mitral valve could reduce left ventricular outflow tract (LVOT) obstruction and associated mitral r
154 rtrophic cardiomyopathy and left ventricular outflow tract (LVOT) obstruction, but without basal sept
155 (VAs) originating from the left ventricular outflow tract (LVOT) sometimes require catheter ablation
156 (VAs) originating from the left ventricular outflow tract (LVOT) sometimes require catheter ablation
157 (VAs) originating from the left ventricular outflow tract (LVOT), an alternative approach from the a
158 ve patients who experienced left ventricular outflow tract (LVOT)/annular/aortic contained/noncontain
159 associated with ascending aortic dilatation, outflow tract malrotation, overriding aorta, double outl
160 activity in the epicardial right ventricular outflow tract may be beneficial in patients with Brugada
161 results indicate that BMP signaling from the outflow tract modulates hedgehog-induced proliferation i
162 ar genetics of neural crest contributions to outflow tract morphogenesis and cell differentiation.
163 r the control of Bmp signaling that promotes outflow tract myocardial differentiation from cardiac pr
164 ow tract development contributes to impaired outflow tract myocardialization and displacement of the
165 ore, impaired NMII-B motor activity inhibits outflow tract myocardialization, leading to mislocalizat
167 the free wall of the right ventricular (RV) outflow tract (n=8), lateral RV (n=44), RV apex (n=61),
168 . 24 +/- 6 mm; p < 0.001) and less prevalent outflow tract obstruction (19% vs. 34%; p = 0.015); 2) h
169 thy with severe symptomatic left ventricular outflow tract obstruction (47+/-11 years, 63% male) intr
171 y, there was mild residual right ventricular outflow tract obstruction (mean gradient, 24+/-13 mm Hg)
172 tions, including relief of right ventricular outflow tract obstruction (n=5), pulmonary arterioplasty
173 the use of paroxetine and right ventricular outflow tract obstruction (relative risk, 1.07; 95% CI,
174 between paroxetine use and right ventricular outflow tract obstruction and between sertraline use and
175 ntly accompanied by dynamic left ventricular outflow tract obstruction and symptoms of dyspnea, angin
176 sk of Ebstein's anomaly (a right ventricular outflow tract obstruction defect) in infants and overall
179 g surgery for the relief of left ventricular outflow tract obstruction have low event rates during lo
180 ssociation of symptoms with left ventricular outflow tract obstruction in HCM, there exist paradoxica
182 tral valve was discovered as the cause of LV outflow tract obstruction in the M-mode echocardiography
183 whereas proximally, severe left ventricular outflow tract obstruction is associated with an addition
185 ern was evident, which is associated with LV outflow tract obstruction loss and right ventricle systo
186 mic pulmonary hypertension, left ventricular outflow tract obstruction or dilated cardiomyopathy.
187 ands were younger with less left ventricular outflow tract obstruction than G- probands, however, had
188 .9 years) with significant right ventricular outflow tract obstruction underwent BMS followed by PPVI
189 ntry, including 249 in whom left ventricular outflow tract obstruction was absent both at rest and fo
190 remia and flow reserve in our study, whereas outflow tract obstruction was not an independent determi
191 rated vigorous left ventricular function, no outflow tract obstruction, and no aortic valve insuffici
192 ns in this series were for right ventricular outflow tract obstruction, highlighting the importance o
193 ly seen in association with left ventricular outflow tract obstruction, itself part of a spectrum of
201 95% CI 3.60-25.91%), while left ventricular outflow tract obstruction/mid-ventricular obstruction (L
203 strategy consisting of relief of inflow and outflow tract obstructions, resection of endocardial fib
207 he current knowledge of the genetics of left outflow tract of the heart, including the aortic stenosi
214 ifferentiation, cardiomyocyte proliferation, outflow tract (OFT) and atrioventricular septation, and
215 dial cells to mesenchymal cells (EMT) at the outflow tract (OFT) but not atrioventricular canal (AVC)
217 play key roles in development of the cardiac outflow tract (OFT) for establishment of completely sepa
218 vious genetic studies in mice indicated that outflow tract (OFT) formation requires Dvl1 and 2, but i
221 ecades, the mechanisms underlying RA-induced outflow tract (OFT) malformations are not understood.
223 pharyngeal arch arteries (PAAs). and cardiac outflow tract (OFT) requires multipotent neural crest ce
224 mouse identifies common progenitors for the outflow tract (OFT), LV, atrium and SV but not the right
231 ght ventricle (P=0.037) and left ventricular outflow tract (P<0.001) and higher in left ventricle-rig
234 00 ventricular extrasystoles (or >500 non-RV outflow tract) per 24 h; and symptoms, ventricular tachy
235 lve replacement in dilated right ventricular outflow tracts, permitting lower risk, nonsurgical pulmo
236 ignaling is required for EMT in the proximal outflow tract (pOFT) but not atrioventricular canal (AVC
237 In patients referred for left ventricle outflow tract premature ventricular contraction ablation
238 ght consecutive patients with left ventricle outflow tract premature ventricular contraction were inc
239 s (mean age 44 +/- 14 years, 21 female) with outflow tract premature ventricular contractions (PVCs)/
240 d October 2008, 64 patients who underwent RV outflow tract procedures in early childhood had more tha
241 Cranial pSHF cells also contribute to the outflow tract: proximal and distal at 4 somites, and dis
242 perior imaging of the right ventricular (RV) outflow tract, pulmonary arteries, aorta, and aortopulmo
243 n evaluated in response to right ventricular outflow tract PVCs with fixed short, fixed long, and var
244 ects in children requiring right ventricular outflow tract reconstruction typically involves multiple
247 rs), 32 patients underwent right ventricular outflow tract reintervention for obstruction (n=27, with
248 ntractions originating in the left ventricle outflow tract represent a significant subgroup of patien
249 f the Melody TPV to patients with nonconduit outflow tracts (right ventricular outflow tract [RVOT])
252 placed epicardially on the right ventricular outflow tract (RVOT) before video-assisted thoracoscopic
255 es have indicated that the right ventricular outflow tract (RVOT) is likely to be the site of electro
256 atients with postoperative right ventricular outflow tract (RVOT) obstruction or pulmonary regurgitat
257 outflow tract (LVOT) from right ventricular outflow tract (RVOT) origin in patients with idiopathic
259 and cardiomyopathy-related right ventricular outflow tract (RVOT) ventricular arrhythmias (VAs) is cr
261 imings across the right ventricle (RV) body, outflow tract (RVOT), and left ventricle were calculated
262 e usually localized to the right ventricular outflow tract (RVOT), presumably below the pulmonic valv
263 nonconduit outflow tracts (right ventricular outflow tract [RVOT]) has the potential to vastly expand
264 an isolated subepicardial right ventricular outflow tract scar serving as a substrate for fast VT in
266 aniofacial cartilaginous structures, cardiac outflow tract septation and thymic and dorsal root gangl
267 on of the second heart field (SHF) and heart outflow tract septation defects are combined, although t
268 modeling of the pharyngeal arch arteries and outflow tract septation during heart development, but th
269 haryngeal arch artery remodeling and cardiac outflow tract septation during vertebrate development.
270 ng of pharyngeal arch arteries and defective outflow tract septation resulting in the formation of a
271 c neural crest-derived cells, which form the outflow tract septum, migrated into the outflow tract an
273 diac phenotype (119-113 Ma) and suggest that outflow tract simplification in actinopterygians is comp
274 defects that are preceded by a reduction in outflow tract size and loss of caudal pharyngeal arch ar
277 ) (r = 0.880; p < 0.0001), right ventricular outflow tract stroke volume (r = 0.660; p < 0.0001), and
279 were placed into the right ventricular apex/outflow tract through a subclavian vein puncture with a
280 ng the left ventricle is reversed toward the outflow tract through rotating reversal flow around the
282 the ventricular septum or subvalvar systemic outflow tract, using 1 of the following 3 delivery appro
283 as ([LA emptying fraction x left ventricular outflow tract-velocity time integral] / [indexed LA end-
284 ct (RVOT) origin in patients with idiopathic outflow tract ventricular tachycardia (OTVT) and lead V(
286 uctural heart disease, most left ventricular outflow tract ventricular tachycardias (VTs) have a foca
287 ent of specific anatomical structures (e.g., outflow tract, ventricular septum, and atrial septum) th
288 ultiple cardiovascular defects affecting the outflow tract, ventricular septum, atrioventricular cush
289 irect transcriptional target of MEF2C in the outflow tract via an AHF-restricted Tdgf1 enhancer.
290 maximal Doppler velocity in left ventricular outflow tract (VmaxAo) measured using either approach, a
291 hologies (MMs) of inducible left ventricular outflow tract VT may indicate a scar-related VT that can
292 patients referred for ablation of sustained outflow tract VT without overt structural heart disease,
293 tural heart disease, 24 had left ventricular outflow tract VT, 10 had MM VT, and 14 had a single VT (
294 l pole morphogenesis, identifying defects in outflow tract wall and cushion morphology that preceded
295 terior right free wall and right ventricular outflow tract, which increased after flecainide from 17.
296 erved that VEGF-C is widely expressed in the outflow tract, while cardiomyocytes develop specifically
297 that CXCL12 is present at high levels in the outflow tract, while peritruncal endothelial cells (ECs)
298 bserved exclusively in the right ventricular outflow tract with the following properties (in comparis
299 ection flow velocity in the left ventricular outflow tract, with consequent loss of flow momentum.
300 (SHF) gives rise to the right ventricle and outflow tract, yet its evolutionary origins are unclear.
WebLSDに未収録の専門用語(用法)は "新規対訳" から投稿できます。