ライフサイエンスコーパス: ova

1 trospective analysis of the results of 2,704 ova and parasite (O & P) examinations performed on stool

2 hat by females), sperm become less abundant, ova become relatively less rare, and competition between

3 using intranasal house dust mite or aerosol ova-albumin challenge, and chloroquine or quinine were t

4 e of inactivation increased until nearly all ova were nonviable by day 24.

5 stages until day 16, after which nearly all ova were observed to be nonviable.

6 ated changes in CRF1 expression in brain and ova of stressed female rats and in the brain of their ne

7 re physically destroyed during digestion and ova can be inactivated faster if their development cycle

8 oung adult mice and absence of follicles and ova in older mice, along with interstitial stromal cell

9 showed moderate cellular immune reaction and ova (viable and/or calcified).

10 re unchanged in gynogenetic and androgenetic ova; the latter contain two maternal and two paternal ge

11 s: in individual blastomeres of androgenetic ova, both paternal Snrpn alleles were active (Snrpn was

12 cell-mediated immunity to provide tumor (B16-ova-Melanoma) protection in around 40% of vaccinated mic

13 ected into the perivitelline space of bovine ova.

14 Ovalbumin-specific Treg cells (ova-Tregs) were isolated from patients' peripheral blood

15 ransduced mice maintained stable circulating ova levels without evidence of an immune response.

16 In contrast, ova exposed to aerobic conditions did not become dormant

17 rmation of liver granulomas around deposited ova.

18 In addition, only 35% of fully developed ova exposed to the anaerobic environment retained their

19 ression of Snrpn and Igf2r in normal diploid ova was unchanged in individual blastomeres of triploid

20 bility by day 16 compared to 65% for dormant ova.

21 imens were positive for G. lamblia by ELISA, ova and parasite test, and/or direct fluorescent-antibod

22 cquired resistance impairs tick engorgement, ova production, and viability.

23 Ep2(-/-) ova could be fertilized in vitro, suggesting that in add

24 erm cells that can navigate to and fertilize ova.

25 The ability to successfully fertilize ova relies upon the swimming ability of spermatozoa.

26 ales will vary in their ability to fertilize ova on the basis of sperm viability alone.

27 esulting from culture of in vitro fertilized ova were transduced with a self-inactivating lentiviral

28 oratories (40% for stool culture and 45% for ova and parasite [O&P] examinations) had restrictions fo

29 s (determined by traditional examination for ova and parasites) preserved in ECOFIX compared to their

30 ultiple stool examinations were negative for ova and parasites.

31 ction of tumors expressing either a foreign (ova) antigen or fully syngeneic tumor antigens (on Panc0

32 tive (Snrpn was not expressed in gynogenetic ova), and in individual gynogenetic and androgenetic bla

33 eing present in the sperm that fertilize her ova.

34 affects CRF1 expression in brain but also in ova, pointing to a possible mechanism of transgeneration

35 creted transgene product (ovalbumin, ova) in ova-specific T-cell receptor (TCR) transgenic mice by he

36 normally present at high levels primarily in ova cytoplasm of developing ovarian follicles, and in th

37 As early as 90min post injection, ova-micelle conjugates were associated with 28% and 55%

38 to form granulomas in response to S. mansoni ova even after T cell activation, suggesting a requireme

39 ve studied the immune response to S. mansoni ova in Stat4- and Stat6-deficient mice, which lack Th1 a

40 IL-10 in animals inoculated with S. mansoni ova, larvae from the filarial helminth Brugia malayi, or

41 iated with the immune response to S. mansoni ova.

42 es of larval miracidia within the S. mansoni ova.

43 The development of Schistosoma mansoni ova-induced granulomas is regulated by cytokines secrete

44 -term selection ovulates on average 6.7 more ova than its randomly selected control line.

45 mice have impaired Th2 responses in a murine ova-induced asthma model, while Th1 responses are normal

46 MI (97/103) and MII (6/103) nondisjunctional ova were strikingly different.

47 ific expression of Snrpn and Igf2r in normal ova were unchanged in gynogenetic and androgenetic ova;

48 Approximately 65% of ova were able to retain their viability for up to 16 day

49 In particular, administration of ova from the pig whipworm Trichuris suis (T. suis; TSO)

50 e results of serologic tests or detection of ova) and 17 healthy controls were included in the study.

51 assessment methods supports the efficacy of ova-Tregs; this immune therapy approach warrants further

52 ingle male will compete for fertilization of ova with spermatozoa from another male when present in t

53 ered the "gold standard" for the fixation of ova and parasites in the preparation of permanently stai

54 Injections of ova-Tregs were well tolerated, with 54 adverse events (2

55 supporting immune-suppressive mechanisms of ova-Tregs.

56 tinal inflammation and reduces the number of ova within the gut.

57 eated stool examinations for the presence of ova and parasites, in practice this test is not sensitiv

58 ic B cells (P = 0.003) and the production of ova-specific antibodies (P = 0.03) after immunization.

59 les must compete to fertilize a given set of ova.

60 were randomly assigned to receive placebo or ova treatment.

61 ization with the soluble antigen, ovalbumin (ova), administered i.p. or orally without adjuvant, acti

62 to a secreted transgene product (ovalbumin, ova) in ova-specific T-cell receptor (TCR) transgenic mi

63 hine evolution resulted in multiple ovulated ova with each cycle, and that these changes triggered ad

64 evere inflammation directed against parasite ova, whereas IL-4Ralpha expression by non-BM-derived cel

65 extent of hepatic damage caused by parasite ova during natural infection with Schistosoma mansoni, b

66 GF-beta mAbs had little effect upon parasite ova-induced intestinal pathology or development of alter

67 istosoma mansoni infection in which parasite ova induce granulomas whose cellular content is 50% eosi

68 osomiasis mansoni, schistosome worms produce ova that incite focal Th2-type granulomatous inflammatio

69 Following adoptive transfer into RIP-ova(high) recipients, PD-1(-/-) OT-I T cells expanded in

70 the control of the rat insulin promoter (RIP-ova(high)).

71 lls adoptively transferred into the same RIP-ova(high) recipient mouse.

72 , a recombinant Listeria expressing OVA (rLM-ova) was generated.

73 resolved discrepant results by using routine ova and parasite examination (O&P) and on immunofluoresc

74 ferent males compete to fertilize a female's ova, is a widespread and fundamental force in the evolut

75 r more males compete to fertilize a female's ova.

76 of being transmitted to each of the mother's ova and, therefore, would appear to have equal interests

77 Schistosoma ova were identified exclusively among African refugees a

78 normal granulomatous response to schistosome ova during the course of natural infection.

79 host cellular immune response to schistosome ova is a risk factor for urinary tract morbidity in area

80 local granulomatous response to schistosome ova nor the systemic response to soluble egg Ag switches

81 Results suggest that some ova are physically destroyed during digestion and ova ca

82 RL were proportional to the numbers of sperm/ova and are indicative of the stage of the reproductive

83 specimens) that were tested by the standard ova and parasite (O&P) examination as the "gold standard

84 umber of unnecessary stool cultures (STCUL), ova/parasite (O&P) examinations, and Giardia/Cryptospori

85 overed STHs; and viability assessment of STH ova.

86 ion supports further investigation of T suis ova in patients with immune-mediated diseases, particula

87 okine responses were not affected by T. suis ova treatment.

88 ion of T. suis-specific cytokines in T. suis ova-treated participants, allergen-specific cytokine res

89 b-cohort of randomized participants (T. suis ova-treated, n = 12, Placebo-treated, n = 10).

90 ed three weekly doses of 2500 Trichuris suis ova (n = 45) or placebo (n = 44) over 6 months.

91 Trichuris suis ova (TSO) have been tested for therapeutic application i

92 ess the safety or efficacy of Trichuris suis ova in allergies, inflammatory bowel diseases, multiple

93 Patients received 2500 Trichuris suis ova or placebo orally at 2-week intervals for 12 weeks.

94 sts that cytokines induced by Trichuris suis ova treatment do not alter allergic reactivity to pollen

95 Trichuris suis ova were obtained from the US Department of Agriculture.

96 Eggs of the pig whipworm (Trichuris suis ova) have been shown to be safe in multiple studies.

97 t difference in inactivation of Ascaris suum ova in digesters operated at different solids residence

98 f embryonated and unembryonated Ascaris suum ova in six laboratory-scale mesophilic (35 degrees C) an

99 idual blastomeres of triploid and tetraploid ova.

100 subsequent mating events from accessing the ova.

101 es at the second meiotic division, after the ova has been fertilized by a C57BL/6 sperm bearing a Y c

102 ype hypersensitivity reaction induced by the ova.

103 erminus of MBP with six amino acids from the ova peptide, and replacement of the lysine side chain in

104 nd organic contaminants were measured in the ova and embryonic tissues and compared to concentrations

105 ernal DDK Om allele in only a portion of the ova of F1 females.

106 ain symptomatic after a negative result, the ova and parasite examination and special stains for othe

107 ntly cycled and ovulated normally, and their ova could be fertilized; however, the embryos did not pr

108 ning TCR+CD4+ cell population was anergic to ova antigen in vitro and enriched for CD25+ cells.

109 lop an exaggerated granulomatous response to ova trapped in the bladder wall, with associated urinary

110 ells and splenocytes were hypo-responsive to ova as early as day 10 after gene transfer.

111 racytoplasmic sperm injection into wild-type ova and implantation of the fertilized eggs into foster

112 able to complete fertilization of uninfected ova, but a rescue function allows infected eggs to devel

113 with age, even after adjustment for urinary ova excretion.

114 eosinophils, epithelial erosion, and viable ova.

115 In contrast, concomitant VSV-ova infection induced OT-I-mediated epithelial cell dest

116 dogenous CD8 cells were nonresponsive to VSV-ova infection.

117 Morphologically, T. vulpis ova resemble those of the human whipworm (T. trichiura)

118 ther vaccinia virus expressing ovalbumin (VV-ova) or peptide-pulsed dendritic cells.

119 we used an adoptive transfer system wherein ova-specific T cells were infused into a syngeneic host,

120 e occurred in 13 of 30 patients (43.3%) with ova treatment compared with 4 of 24 patients (16.7%) giv

121 inhibited IFN-gamma in mice inoculated with ova.

122 , neither E- nor P-selectin was found within ova.

123 Worm ova accumulation in the intestines of Arg I-deficient bo

124 t lethal intestinal pathology caused by worm ova in mice infected with the human parasite Schistosoma

125 imeras develop a marked accumulation of worm ova within the ileum but impaired fecal egg excretion co