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1 (human transferrin, human serum albumin, and ovalbumin).
2 d desensitization to the model food allergen ovalbumin.
3 rcutaneous challenge by the protein allergen ovalbumin.
4 airway epithelium were tolerized to inhaled ovalbumin.
5 deled in mice sensitized and challenged with ovalbumin.
6 ngth) produced a Th2-biased response against ovalbumin.
7 H2 responses after allergic sensitization to ovalbumin.
8 nternalized by dendritic cells and delivered ovalbumin.
9 ntraperitoneally toward the primary antigen, ovalbumin.
10 ild-type (WT) mice sensitized and exposed to ovalbumin.
11 ration of antibodies after immunization with ovalbumin.
12 roteases which were not accessible in native ovalbumin.
13 n was prepared by conjugating [Asp3]MC-RY to ovalbumin.
14 e of Galbeta(1-->4)GlcNAc ligands on chicken ovalbumin.
15 pidly after sensitization and challenge with ovalbumin.
16 ollowed by injection of 2,4,6-trinitrophenyl-ovalbumin.
17 from mice sensitized to native or aggregated ovalbumin.
23 3beta) sequences following immunization with ovalbumin administered with complete Freund's adjuvant (
24 eally injected into ovalbumin-sensitized and ovalbumin aerosol-challenged leukotriene C4 synthase-def
25 accurately identify a CD4 T cell response to ovalbumin against a background response to the complex m
26 entification using LC-MS/MS highlighted that ovalbumin aggregation rendered a number of peptide bonds
27 als were exposed repeatedly to aspiration of ovalbumin alone or together with environmental adjuvants
29 model of allergic asthma was developed with ovalbumin-alum in female Cd39 wild type (Cd39(+/+) ) and
30 Mice were sensitized with intraperitoneal ovalbumin-alum, followed by intranasal challenge with ov
32 cross-presentation in vitro relative to free ovalbumin, an unconjugated physical mixture of ovalbumin
34 erformed intranasally or epicutaneously with ovalbumin and a secondary antigen, keyhole limpet hemocy
36 nd exposed to two separate allergic stimuli (ovalbumin and Aspergillus extract), genetic removal of M
39 of glycopeptides from fetuin, glycophorin A, ovalbumin and gp120 tryptic digests were used to build a
41 ferent murine models of experimental asthma (ovalbumin and house dust mite); miRNAs deregulated in bo
44 LDI-MS identified proteolytic fragments from ovalbumin and lysozyme, exhibiting varied resistance to
48 tion against the anaphylaxis associated with ovalbumin and peanut allergy and affects the epigenome o
50 mpared to immunization with soluble protein, ovalbumin and polymer mixture, and the control micelle w
51 albumin, an unconjugated physical mixture of ovalbumin and polymer, and a non-pH-responsive micelle-o
54 is, estimated by the disappearance of intact ovalbumin and the appearance of soluble peptides, was gr
55 oups for reversible conjugation of thiolated ovalbumin, and a tercopolymer ampholytic core-forming bl
56 f polymer type (proteins, i.e. caseinate and ovalbumin, and polysaccharides, i.e. alginate and methyl
57 ngly, serpins are homologs of the well-known ovalbumin antigen and are associated with autoimmunity.
58 d standards from an Abl kinase sensor and an ovalbumin antigenic peptide were then employed to identi
60 s shown by both the astringency method (with ovalbumin as a precipitant) and the tannins determinatio
61 f these mice with house dust mite extract or ovalbumin as allergens led to exacerbated inflammation,
64 yptophan depletion in myofibrillar proteins, ovalbumin, beta-lactoglobulin, soy protein and human ser
65 revealed that calcium inhibited glycation of ovalbumin by a mixed non-competitive mechanism in both d
66 A knockout mice (P < .005) and in vivo after ovalbumin challenge (P < .05), higher levels of apoptosi
68 Sensitised mice were exposed to prolonged ovalbumin challenge or continuous house-dust mite exposu
70 ypertrophy in the lungs of a murine model of ovalbumin-challenged allergic asthma, leading to improve
71 mulin plasmids were detected in the lungs of ovalbumin-challenged asthmatic mice up to 27days after a
74 Approximately 90% of initially internalized ovalbumin-conjugated micelles were retained in cells aft
77 umin ratio, yolk index, air cell size, and S-ovalbumin content of UC were significantly the most supe
80 systemically applied (64)Cu-labeled chicken ovalbumin (cOVA)-TCR transgenic T cells into the pulmona
81 s on proteins (as caseins, whey proteins and ovalbumin) declared in the label of several sport nutrit
84 of heat-induced aggregation on the extent of ovalbumin digestion and the nature of peptides released
86 otein effects with bovine serum albumin, (S-)ovalbumin, egg white, whole egg, defatted egg yolk, whea
87 sters, which were mainly composed of chicken ovalbumin-encapsulated AuNCs, can recognize ricin B beca
91 an in vivo mouse model of nasal tolerance to ovalbumin, F. prausnitzii A2-165 enhanced ovalbumin-spec
92 ansgenic CD4(+) T-cell receptor specific for ovalbumin failed to develop HF and adverse remodeling.
96 suggest that the final coating of dissolved ovalbumin from egg white after long exposure to air, whi
98 n of non-entrapped drug present) or protein (ovalbumin >90% reduction of OVA present) and organic sol
99 tactin, Cytochrome C, holo hemoglobin-alpha, ovalbumin, human transthyretin, avidin, bovine serum alb
100 sing the HOD antigen (hen egg lysozyme [HEL]-ovalbumin-human transmembrane Duffy(b)) were transfused
101 d that the AuNCs@ew were mainly derived from ovalbumin, i.e., the major protein in egg white, encapsu
102 Cs purified from the draining lymph nodes of ovalbumin-immunized mice favored the generation of antig
103 the TH2-inducing antigens house dust mite or ovalbumin in a model of allergic airway inflammation or
105 bumin-dependent colitis in mice that express ovalbumin in biliary epithelia (ASBT-OVA mice) and cross
109 valuating model antigens such as chicken egg ovalbumin in mouse models but their relevance to humans
110 ompared to the native secondary structure of ovalbumin in solution of circa 33% alpha-helix and beta-
112 bumin followed by intranasal challenges with ovalbumin induced airway hyper-reactivity, which was red
116 n-1) PSCs was studied in an in vivo model of ovalbumin-induced allergic inflammation and an in vitro
120 isothiocyanate contact hypersensitivity and ovalbumin-induced dermatitis as models for TH2-mediated
122 pleted of Ly49A/D/G(+) NK cell subsets in an ovalbumin-induced model of allergic airway disease (OVA-
124 Haemophilus respiratory infection-mediated, ovalbumin-induced severe, steroid-resistant allergic air
125 ytial virus respiratory tract infections and ovalbumin-induced, severe, steroid-insensitive allergic
128 knockout mice with EpCAM-deficient LCs with ovalbumin led to increased induction of type 2 Ova-speci
129 an aqueous two-phase system (ATPS) using an ovalbumin-methylcellulose system (OMCS) in comparison to
130 of activation of CD8+ T cells in an in vitro ovalbumin model and illustrated that acid-labile polymer
132 orted that K14-mOVA mice expressing membrane ovalbumin (mOVA), driven by the keratin 14 (K14) promote
133 immune-modulating properties of MVA-encoding ovalbumin (MVA-OVA) on the allergen-specific immune resp
136 control vs sensitized and/or challenged with ovalbumin, n=3-5/group) 6 hour, 24 hour, and 48 hour aft
137 etween heat-induced aggregates of ovalbumin (ovalbumin nanoparticles, OVAn) and linoleic acid (LA) we
139 wed by five oral dosing with purified AHC or ovalbumin on alternate days and continued on HFD for ano
140 pendence of the apparent molecular weight of ovalbumin on buffer concentration are extrapolated to ze
141 o dog-associated house dust protects against ovalbumin or cockroach allergen-mediated airway patholog
142 this murine model was exposed to allergens (ovalbumin or house dust mite extract) to decipher in viv
143 RAs) and assessed their abilities to reverse ovalbumin or peanut allergies in mouse models, as well a
144 C-RAs to half-maximally sensitized mice with ovalbumin or peanut allergy reduced anaphylactic respons
145 ols for food allergy induction, using either ovalbumin or peanut extract as allergens for sensitizati
146 fluenza and, once recovered, subjected to an ovalbumin- or house dust mite-induced experimental asthm
147 lly block nociceptors, substantially reduced ovalbumin- or house-dust-mite-induced airway inflammatio
148 I cells and cognate peptide stimulation with ovalbumin (OVA) 323-339 plus the inflammatory adjuvant,
153 SIGIRR(-/-) C57BL/6 mice were sensitized to ovalbumin (OVA) and challenged with OVA aerosol to induc
154 -deficient (miR-155(-/-) ) mice were used in ovalbumin (OVA) and house dust mite (HDM) models of AAI.
155 xpression and activity were evaluated in the ovalbumin (OVA) and house dust mite (HDM) murine models.
157 dine or activated using dimaprit in both the ovalbumin (OVA) and house dust mite extract (HDM) murine
159 sh) mice were epicutaneously sensitized with ovalbumin (OVA) and then challenged orally with OVA.
160 tudy of labeling of thiolic groups of native ovalbumin (OVA) as a function of protein concentration.
163 evidence in a transgenic mouse system using ovalbumin (OVA) as a model surrogate TRA that the de nov
165 ose on anaphylactic symptoms induced by oral ovalbumin (OVA) challenge in sensitized mice were invest
167 rghei parasites expressing the model antigen ovalbumin (OVA) either in the parasite cytoplasm or on t
168 e epicutaneously sensitized female mice with ovalbumin (OVA) followed by epicutaneous sensitization a
171 6 mice by intraperitoneal sensitization with ovalbumin (OVA) in combination with intranasal (i.n) exp
172 tes uptake and processing of soluble antigen ovalbumin (Ova) in primary human macrophages and dendrit
175 model in which instillation of ClO(-) before ovalbumin (OVA) induces AHR without bronchial inflammato
176 80% sulforhodamine b (SRB) and model antigen ovalbumin (OVA) into murine, swine, and human skin withi
179 fractionally coated with a powder mixture of ovalbumin (OVA) model allergen, CpG, and 1,25-dihydroxyv
180 c T cell receptor (TCR) with specificity for ovalbumin (OVA) on CD4(+)-T cells and cMy-mOVA mice expr
181 While allergenic challenge with a low-dose ovalbumin (OVA) only produced a small bronchospasm (~2-f
183 ary during the sensitization phase of either ovalbumin (OVA) or house dust mite (HDM) acute asthma mo
184 to wild-type recipient mice, challenged with ovalbumin (OVA) or house dust mite (HDM), and accessed f
185 ay inflammation in mice administering either ovalbumin (OVA) or house dust mite extract (HDM) for sen
189 e that received an irrelevant immunodominant ovalbumin (OVA) peptide, OVA323-339, mice that received
190 rformed by applying a skin patch soaked with ovalbumin (OVA) plus CpG, and its suppressor activity wa
194 e and IP knockout mice were aerosolized with ovalbumin (OVA) to induce immune tolerance prior to immu
197 y, amyloids assembled from the model protein ovalbumin (OVA) were found to release the precursor prot
201 N microparticles co-loaded with the antigen, ovalbumin (OVA), and adjuvant, CpG 1826, (PNSN(OVA + CpG
202 /-) mice were sensitized and challenged with ovalbumin (OVA), and the development of AAD was ascertai
204 s an IgG1 B cell receptor (BCR) specific for ovalbumin (OVA), the epitope of which can be mimicked us
205 ) was used to produce exosomes together with ovalbumin (OVA), the resulting Dexo vaccine strongly sti
206 the dose and penetration of a model vaccine, Ovalbumin (OVA), to depths of 500mum into porcine skin.
209 XCL1 (KC) expression were also determined in ovalbumin (OVA)-challenged wild-type female recipient mi
211 to T and B cell responses of unimmunized and ovalbumin (OVA)-immunized BALB/c mice, and furthermore,
213 of TH subsets in vitro and on development of ovalbumin (OVA)-induced airway hyperresponsiveness (AHR)
214 s was assessed in whole lungs from mice with ovalbumin (OVA)-induced allergic airway inflammation (AA
215 his study, we used a neonatal mouse model of ovalbumin (OVA)-induced allergic airway inflammation to
217 to determine the effect of Ptx3 deletion on ovalbumin (OVA)-induced allergic inflammation in a murin
218 nt susceptibility to pneumococcal infection, ovalbumin (OVA)-induced allergic lung inflammation (ALI)
222 Basophils were purified from the lungs of ovalbumin (OVA)-sensitized and OVA-challenged (OVA-immun
236 o-mid mass proteins (Trypsin Inhibitor (TI); Ovalbumin (OVA); Bovine Serum Albumin (BSA)), we observe
237 CAF09 and a surface-adsorbed model antigen [ovalbumin (OVA)] showed that a significantly larger frac
238 ntial allergenicity of food allergens, using ovalbumin (OVA, an egg white allergen) as a model allerg
239 ntracellular uptake of model cancer antigen (ovalbumin, OVA) by THP-1-differentiated macrophage-like
240 formation between heat-induced aggregates of ovalbumin (ovalbumin nanoparticles, OVAn) and linoleic a
241 hree proteins with high sequence similarity: ovalbumin, ovalbumin-related protein Y (OVAY), and ovalb
242 ntioxidant peptides, which were derived from ovalbumin, ovotransferrin and cystatin were isolated fro
244 O4@Al2O3 MNPs are functionalized with pigeon ovalbumin (POA), which contains Gal-alpha(1-->4)-Gal-bet
246 ctron tomography to measure the structure of ovalbumin precipitate particles salted out with ammonium
247 fic antibodies and enhanced proliferation of ovalbumin-reactive T cells associated with increased acc
249 ns with high sequence similarity: ovalbumin, ovalbumin-related protein Y (OVAY), and ovalbumin-relate
253 nanostructures comprised of crystallites of ovalbumin self-assemble into a well-defined bicontinuous
254 ype (WT) and EP2(-/-) mice were subjected to ovalbumin sensitization and acute airway challenge.
255 ng IL-4 and IL-13 expression were induced by ovalbumin sensitization and challenge prior to acute lun
256 n response to Aspergillus fumigatus, but not ovalbumin sensitization and challenge, are activated by
261 ex vivo, were intratracheally injected into ovalbumin-sensitized and ovalbumin aerosol-challenged le
267 involved in asthmatic-like attacks using the ovalbumin-sensitized murine model of the disease, and id
269 se, cholangitis developed in mice expressing ovalbumin simultaneously in biliary epithelia and entero
271 2 but significantly less IL-10 production by ovalbumin-specific CD4(+) T cells, resulting in prolifer
274 rausnitzii A2-165 stimulated BMDCs increased ovalbumin-specific T cell proliferation and reduced the
275 to ovalbumin, F. prausnitzii A2-165 enhanced ovalbumin-specific T cell proliferation and reduced the
276 s examined by assessment of proliferation of ovalbumin-specific T cells in rat insulin promoter-membr
281 by co-cultures of DCs from infected mice and ovalbumin-specific, MHC class II-restricted alpha/beta (
283 This work illustrates the links between ovalbumin structure after heating and allergenicity pote
285 d tolerance induced by inhalation of soluble ovalbumin, suppressing the normal generation of forkhead
288 cells in rat insulin promoter-membrane-bound ovalbumin transgenic mice after sham or IRI procedures.
289 the present work, we determined that murine ovalbumin-transgenic (OT-1) CD8(+) T cells recognize the
290 as explored by stimulating OTI-Tc cells with ovalbumin-transgenic antigen-presenting cells in the pre
291 ia hepatic nuclear factor 4alpha and chicken ovalbumin upstream promoter transcription factor I/II.
292 n partially through up-regulation of chicken ovalbumin upstream promoter transcription factor II (COU
293 ale mouse embryos lacking Coup-tfII (chicken ovalbumin upstream promoter transcription factor II) in
294 m stem/progenitor cells that express chicken ovalbumin upstream promoter transcription factor II.
300 gens HDM and papain and a classical allergen ovalbumin was evaluated in vivo in mice deficient for TS
302 nulation capacities of native and aggregated ovalbumin were measured with sera from egg-allergic chil
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