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   1 (human transferrin, human serum albumin, and ovalbumin).                                             
     2 d desensitization to the model food allergen ovalbumin.                                              
     3 rcutaneous challenge by the protein allergen ovalbumin.                                              
     4  airway epithelium were tolerized to inhaled ovalbumin.                                              
     5 deled in mice sensitized and challenged with ovalbumin.                                              
     6 ngth) produced a Th2-biased response against ovalbumin.                                              
     7 H2 responses after allergic sensitization to ovalbumin.                                              
     8 nternalized by dendritic cells and delivered ovalbumin.                                              
     9 ntraperitoneally toward the primary antigen, ovalbumin.                                              
    10 ild-type (WT) mice sensitized and exposed to ovalbumin.                                              
    11 ration of antibodies after immunization with ovalbumin.                                              
    12 roteases which were not accessible in native ovalbumin.                                              
    13 n was prepared by conjugating [Asp3]MC-RY to ovalbumin.                                              
    14 e of Galbeta(1-->4)GlcNAc ligands on chicken ovalbumin.                                              
    15 pidly after sensitization and challenge with ovalbumin.                                              
    16 ollowed by injection of 2,4,6-trinitrophenyl-ovalbumin.                                              
    17 from mice sensitized to native or aggregated ovalbumin.                                              
  
  
  
  
  
    23 3beta) sequences following immunization with ovalbumin administered with complete Freund's adjuvant (
    24 eally injected into ovalbumin-sensitized and ovalbumin aerosol-challenged leukotriene C4 synthase-def
    25 accurately identify a CD4 T cell response to ovalbumin against a background response to the complex m
    26 entification using LC-MS/MS highlighted that ovalbumin aggregation rendered a number of peptide bonds
    27 als were exposed repeatedly to aspiration of ovalbumin alone or together with environmental adjuvants
  
    29  model of allergic asthma was developed with ovalbumin-alum in female Cd39 wild type (Cd39(+/+) ) and
    30    Mice were sensitized with intraperitoneal ovalbumin-alum, followed by intranasal challenge with ov
  
    32 cross-presentation in vitro relative to free ovalbumin, an unconjugated physical mixture of ovalbumin
  
    34 erformed intranasally or epicutaneously with ovalbumin and a secondary antigen, keyhole limpet hemocy
  
    36 nd exposed to two separate allergic stimuli (ovalbumin and Aspergillus extract), genetic removal of M
  
  
    39 of glycopeptides from fetuin, glycophorin A, ovalbumin and gp120 tryptic digests were used to build a
  
    41 ferent murine models of experimental asthma (ovalbumin and house dust mite); miRNAs deregulated in bo
  
  
    44 LDI-MS identified proteolytic fragments from ovalbumin and lysozyme, exhibiting varied resistance to 
  
  
  
    48 tion against the anaphylaxis associated with ovalbumin and peanut allergy and affects the epigenome o
  
    50 mpared to immunization with soluble protein, ovalbumin and polymer mixture, and the control micelle w
    51 albumin, an unconjugated physical mixture of ovalbumin and polymer, and a non-pH-responsive micelle-o
  
  
    54 is, estimated by the disappearance of intact ovalbumin and the appearance of soluble peptides, was gr
    55 oups for reversible conjugation of thiolated ovalbumin, and a tercopolymer ampholytic core-forming bl
    56 f polymer type (proteins, i.e. caseinate and ovalbumin, and polysaccharides, i.e. alginate and methyl
    57 ngly, serpins are homologs of the well-known ovalbumin antigen and are associated with autoimmunity. 
    58 d standards from an Abl kinase sensor and an ovalbumin antigenic peptide were then employed to identi
  
    60 s shown by both the astringency method (with ovalbumin as a precipitant) and the tannins determinatio
    61 f these mice with house dust mite extract or ovalbumin as allergens led to exacerbated inflammation, 
  
  
    64 yptophan depletion in myofibrillar proteins, ovalbumin, beta-lactoglobulin, soy protein and human ser
    65 revealed that calcium inhibited glycation of ovalbumin by a mixed non-competitive mechanism in both d
    66 A knockout mice (P < .005) and in vivo after ovalbumin challenge (P < .05), higher levels of apoptosi
  
    68    Sensitised mice were exposed to prolonged ovalbumin challenge or continuous house-dust mite exposu
  
    70 ypertrophy in the lungs of a murine model of ovalbumin-challenged allergic asthma, leading to improve
    71 mulin plasmids were detected in the lungs of ovalbumin-challenged asthmatic mice up to 27days after a
  
  
    74  Approximately 90% of initially internalized ovalbumin-conjugated micelles were retained in cells aft
  
  
    77 umin ratio, yolk index, air cell size, and S-ovalbumin content of UC were significantly the most supe
  
  
    80  systemically applied (64)Cu-labeled chicken ovalbumin (cOVA)-TCR transgenic T cells into the pulmona
    81 s on proteins (as caseins, whey proteins and ovalbumin) declared in the label of several sport nutrit
  
  
    84 of heat-induced aggregation on the extent of ovalbumin digestion and the nature of peptides released 
  
    86 otein effects with bovine serum albumin, (S-)ovalbumin, egg white, whole egg, defatted egg yolk, whea
    87 sters, which were mainly composed of chicken ovalbumin-encapsulated AuNCs, can recognize ricin B beca
  
  
  
    91 an in vivo mouse model of nasal tolerance to ovalbumin, F. prausnitzii A2-165 enhanced ovalbumin-spec
    92 ansgenic CD4(+) T-cell receptor specific for ovalbumin failed to develop HF and adverse remodeling.  
  
  
  
    96  suggest that the final coating of dissolved ovalbumin from egg white after long exposure to air, whi
  
    98 n of non-entrapped drug present) or protein (ovalbumin >90% reduction of OVA present) and organic sol
    99 tactin, Cytochrome C, holo hemoglobin-alpha, ovalbumin, human transthyretin, avidin, bovine serum alb
   100 sing the HOD antigen (hen egg lysozyme [HEL]-ovalbumin-human transmembrane Duffy(b)) were transfused 
   101 d that the AuNCs@ew were mainly derived from ovalbumin, i.e., the major protein in egg white, encapsu
   102 Cs purified from the draining lymph nodes of ovalbumin-immunized mice favored the generation of antig
   103 the TH2-inducing antigens house dust mite or ovalbumin in a model of allergic airway inflammation or 
  
   105 bumin-dependent colitis in mice that express ovalbumin in biliary epithelia (ASBT-OVA mice) and cross
  
  
  
   109 valuating model antigens such as chicken egg ovalbumin in mouse models but their relevance to humans 
   110 ompared to the native secondary structure of ovalbumin in solution of circa 33% alpha-helix and beta-
  
   112 bumin followed by intranasal challenges with ovalbumin induced airway hyper-reactivity, which was red
  
  
  
   116 n-1) PSCs was studied in an in vivo model of ovalbumin-induced allergic inflammation and an in vitro 
  
  
  
   120  isothiocyanate contact hypersensitivity and ovalbumin-induced dermatitis as models for TH2-mediated 
  
   122 pleted of Ly49A/D/G(+) NK cell subsets in an ovalbumin-induced model of allergic airway disease (OVA-
  
   124  Haemophilus respiratory infection-mediated, ovalbumin-induced severe, steroid-resistant allergic air
   125 ytial virus respiratory tract infections and ovalbumin-induced, severe, steroid-insensitive allergic 
  
  
   128  knockout mice with EpCAM-deficient LCs with ovalbumin led to increased induction of type 2 Ova-speci
   129  an aqueous two-phase system (ATPS) using an ovalbumin-methylcellulose system (OMCS) in comparison to
   130 of activation of CD8+ T cells in an in vitro ovalbumin model and illustrated that acid-labile polymer
  
   132 orted that K14-mOVA mice expressing membrane ovalbumin (mOVA), driven by the keratin 14 (K14) promote
   133 immune-modulating properties of MVA-encoding ovalbumin (MVA-OVA) on the allergen-specific immune resp
  
  
   136 control vs sensitized and/or challenged with ovalbumin, n=3-5/group) 6 hour, 24 hour, and 48 hour aft
   137 etween heat-induced aggregates of ovalbumin (ovalbumin nanoparticles, OVAn) and linoleic acid (LA) we
  
   139 wed by five oral dosing with purified AHC or ovalbumin on alternate days and continued on HFD for ano
   140 pendence of the apparent molecular weight of ovalbumin on buffer concentration are extrapolated to ze
   141 o dog-associated house dust protects against ovalbumin or cockroach allergen-mediated airway patholog
   142  this murine model was exposed to allergens (ovalbumin or house dust mite extract) to decipher in viv
   143 RAs) and assessed their abilities to reverse ovalbumin or peanut allergies in mouse models, as well a
   144 C-RAs to half-maximally sensitized mice with ovalbumin or peanut allergy reduced anaphylactic respons
   145 ols for food allergy induction, using either ovalbumin or peanut extract as allergens for sensitizati
   146 fluenza and, once recovered, subjected to an ovalbumin- or house dust mite-induced experimental asthm
   147 lly block nociceptors, substantially reduced ovalbumin- or house-dust-mite-induced airway inflammatio
   148 I cells and cognate peptide stimulation with ovalbumin (OVA) 323-339 plus the inflammatory adjuvant, 
  
  
  
  
   153  SIGIRR(-/-) C57BL/6 mice were sensitized to ovalbumin (OVA) and challenged with OVA aerosol to induc
   154 -deficient (miR-155(-/-) ) mice were used in ovalbumin (OVA) and house dust mite (HDM) models of AAI.
   155 xpression and activity were evaluated in the ovalbumin (OVA) and house dust mite (HDM) murine models.
  
   157 dine or activated using dimaprit in both the ovalbumin (OVA) and house dust mite extract (HDM) murine
  
   159 sh) mice were epicutaneously sensitized with ovalbumin (OVA) and then challenged orally with OVA.    
   160 tudy of labeling of thiolic groups of native ovalbumin (OVA) as a function of protein concentration. 
  
  
   163  evidence in a transgenic mouse system using ovalbumin (OVA) as a model surrogate TRA that the de nov
  
   165 ose on anaphylactic symptoms induced by oral ovalbumin (OVA) challenge in sensitized mice were invest
  
   167 rghei parasites expressing the model antigen ovalbumin (OVA) either in the parasite cytoplasm or on t
   168 e epicutaneously sensitized female mice with ovalbumin (OVA) followed by epicutaneous sensitization a
  
  
   171 6 mice by intraperitoneal sensitization with ovalbumin (OVA) in combination with intranasal (i.n) exp
   172 tes uptake and processing of soluble antigen ovalbumin (Ova) in primary human macrophages and dendrit
  
  
   175 model in which instillation of ClO(-) before ovalbumin (OVA) induces AHR without bronchial inflammato
   176 80% sulforhodamine b (SRB) and model antigen ovalbumin (OVA) into murine, swine, and human skin withi
  
  
   179 fractionally coated with a powder mixture of ovalbumin (OVA) model allergen, CpG, and 1,25-dihydroxyv
   180 c T cell receptor (TCR) with specificity for ovalbumin (OVA) on CD4(+)-T cells and cMy-mOVA mice expr
   181   While allergenic challenge with a low-dose ovalbumin (OVA) only produced a small bronchospasm (~2-f
  
   183 ary during the sensitization phase of either ovalbumin (OVA) or house dust mite (HDM) acute asthma mo
   184 to wild-type recipient mice, challenged with ovalbumin (OVA) or house dust mite (HDM), and accessed f
   185 ay inflammation in mice administering either ovalbumin (OVA) or house dust mite extract (HDM) for sen
  
  
  
   189 e that received an irrelevant immunodominant ovalbumin (OVA) peptide, OVA323-339, mice that received 
   190 rformed by applying a skin patch soaked with ovalbumin (OVA) plus CpG, and its suppressor activity wa
  
  
  
   194 e and IP knockout mice were aerosolized with ovalbumin (OVA) to induce immune tolerance prior to immu
  
  
   197 y, amyloids assembled from the model protein ovalbumin (OVA) were found to release the precursor prot
  
  
  
   201 N microparticles co-loaded with the antigen, ovalbumin (OVA), and adjuvant, CpG 1826, (PNSN(OVA + CpG
   202 /-) mice were sensitized and challenged with ovalbumin (OVA), and the development of AAD was ascertai
  
   204 s an IgG1 B cell receptor (BCR) specific for ovalbumin (OVA), the epitope of which can be mimicked us
   205 ) was used to produce exosomes together with ovalbumin (OVA), the resulting Dexo vaccine strongly sti
   206 the dose and penetration of a model vaccine, Ovalbumin (OVA), to depths of 500mum into porcine skin. 
  
  
   209 XCL1 (KC) expression were also determined in ovalbumin (OVA)-challenged wild-type female recipient mi
  
   211 to T and B cell responses of unimmunized and ovalbumin (OVA)-immunized BALB/c mice, and furthermore, 
  
   213 of TH subsets in vitro and on development of ovalbumin (OVA)-induced airway hyperresponsiveness (AHR)
   214 s was assessed in whole lungs from mice with ovalbumin (OVA)-induced allergic airway inflammation (AA
   215 his study, we used a neonatal mouse model of ovalbumin (OVA)-induced allergic airway inflammation to 
  
   217  to determine the effect of Ptx3 deletion on ovalbumin (OVA)-induced allergic inflammation in a murin
   218 nt susceptibility to pneumococcal infection, ovalbumin (OVA)-induced allergic lung inflammation (ALI)
  
  
  
   222    Basophils were purified from the lungs of ovalbumin (OVA)-sensitized and OVA-challenged (OVA-immun
  
  
  
  
  
  
  
  
  
  
  
  
  
   236 o-mid mass proteins (Trypsin Inhibitor (TI); Ovalbumin (OVA); Bovine Serum Albumin (BSA)), we observe
   237  CAF09 and a surface-adsorbed model antigen [ovalbumin (OVA)] showed that a significantly larger frac
   238 ntial allergenicity of food allergens, using ovalbumin (OVA, an egg white allergen) as a model allerg
   239 ntracellular uptake of model cancer antigen (ovalbumin, OVA) by THP-1-differentiated macrophage-like 
   240 formation between heat-induced aggregates of ovalbumin (ovalbumin nanoparticles, OVAn) and linoleic a
   241 hree proteins with high sequence similarity: ovalbumin, ovalbumin-related protein Y (OVAY), and ovalb
   242 ntioxidant peptides, which were derived from ovalbumin, ovotransferrin and cystatin were isolated fro
  
   244 O4@Al2O3 MNPs are functionalized with pigeon ovalbumin (POA), which contains Gal-alpha(1-->4)-Gal-bet
  
   246 ctron tomography to measure the structure of ovalbumin precipitate particles salted out with ammonium
   247 fic antibodies and enhanced proliferation of ovalbumin-reactive T cells associated with increased acc
  
   249 ns with high sequence similarity: ovalbumin, ovalbumin-related protein Y (OVAY), and ovalbumin-relate
  
  
  
   253  nanostructures comprised of crystallites of ovalbumin self-assemble into a well-defined bicontinuous
   254 ype (WT) and EP2(-/-) mice were subjected to ovalbumin sensitization and acute airway challenge.     
   255 ng IL-4 and IL-13 expression were induced by ovalbumin sensitization and challenge prior to acute lun
   256 n response to Aspergillus fumigatus, but not ovalbumin sensitization and challenge, are activated by 
  
  
  
  
   261  ex vivo, were intratracheally injected into ovalbumin-sensitized and ovalbumin aerosol-challenged le
  
  
  
  
  
   267 involved in asthmatic-like attacks using the ovalbumin-sensitized murine model of the disease, and id
  
   269 se, cholangitis developed in mice expressing ovalbumin simultaneously in biliary epithelia and entero
  
   271 2 but significantly less IL-10 production by ovalbumin-specific CD4(+) T cells, resulting in prolifer
  
  
   274 rausnitzii A2-165 stimulated BMDCs increased ovalbumin-specific T cell proliferation and reduced the 
   275 to ovalbumin, F. prausnitzii A2-165 enhanced ovalbumin-specific T cell proliferation and reduced the 
   276 s examined by assessment of proliferation of ovalbumin-specific T cells in rat insulin promoter-membr
  
  
  
  
   281 by co-cultures of DCs from infected mice and ovalbumin-specific, MHC class II-restricted alpha/beta (
  
   283      This work illustrates the links between ovalbumin structure after heating and allergenicity pote
  
   285 d tolerance induced by inhalation of soluble ovalbumin, suppressing the normal generation of forkhead
  
  
   288 cells in rat insulin promoter-membrane-bound ovalbumin transgenic mice after sham or IRI procedures. 
   289  the present work, we determined that murine ovalbumin-transgenic (OT-1) CD8(+) T cells recognize the
   290 as explored by stimulating OTI-Tc cells with ovalbumin-transgenic antigen-presenting cells in the pre
   291 ia hepatic nuclear factor 4alpha and chicken ovalbumin upstream promoter transcription factor I/II.  
   292 n partially through up-regulation of chicken ovalbumin upstream promoter transcription factor II (COU
   293 ale mouse embryos lacking Coup-tfII (chicken ovalbumin upstream promoter transcription factor II) in 
   294 m stem/progenitor cells that express chicken ovalbumin upstream promoter transcription factor II.    
  
  
  
  
  
   300 gens HDM and papain and a classical allergen ovalbumin was evaluated in vivo in mice deficient for TS
  
   302 nulation capacities of native and aggregated ovalbumin were measured with sera from egg-allergic chil
  
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