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1 llitus, Hashimoto's thyroiditis, and primary ovarian failure).
2 elopment of specialized therapies to prevent ovarian failure.
3 ) but could also without (type II) premature ovarian failure.
4 racterized by sensorineural hearing loss and ovarian failure.
5 fects, including kyphoscoliosis, and primary ovarian failure.
6 bnormal follicular development and premature ovarian failure.
7 to recover and are sterile, due to premature ovarian failure.
8 ion of its function in ovaries specially for ovarian failure.
9 subfertile and exhibited signs of premature ovarian failure.
10 ized with craniofacial defects and premature ovarian failure.
11 ivors of childhood cancer develop persistent ovarian failure.
12 an further develop parkinsonism or premature ovarian failure.
13 AS in older males and females, and premature ovarian failure.
14 eterioration often associated with premature ovarian failure.
15 and more common conditions such as premature ovarian failure.
16 ncentrations had returned to those seen with ovarian failure.
17 f chemotherapy and radiotherapy is premature ovarian failure.
18 sed significantly in the women who developed ovarian failure.
19 sponsible for environmental toxicant-induced ovarian failure.
20 other metabolic diseases, and for causes of ovarian failure.
21 vous system white matter disease and primary ovarian failure.
22 oss, 31.9% (95% CI, 28.0%-35.8%) for primary ovarian failure, 31.1% (95% CI, 27.3%-34.9%) for Leydig
23 licular initiation plays a role in premature ovarian failure, a common cause of infertility and prema
24 ight into the causes of idiopathic premature ovarian failure, a disease that burdens many infertile c
25 s well as long-term complications of primary ovarian failure and cognitive dysfunction, which are die
26 in young women facing the risk of premature ovarian failure and infertility as a sequel to the treat
27 female mice, the event that drives premature ovarian failure and infertility in female cancer patient
30 exposure of females to BaP causes premature ovarian failure and ovarian tumorigenesis and that embry
33 (AOD) is a probable cause of human premature ovarian failure, and a potential complication of contrac
34 es of Turner syndrome include short stature, ovarian failure, and a variety of other anatomic and phy
35 be at risk of uterine artery embolization or ovarian failure, and in those in whom the ovarian artery
36 increased destruction of oogonia, premature ovarian failure, and ovarian tumorigenesis after transpl
38 sm spectrum disorder in childhood, premature ovarian failure, and the neurodegenerative disorder, fra
39 eficits on the fragile-X spectrum; premature ovarian failure; and a newly described, neurodegenerativ
40 asing hormone agonists, chemotherapy-induced ovarian failure, androgen deprivation therapy, and aroma
41 ng the "postmenopausal" stage of accelerated ovarian failure (AOF) in young female mice treated with
45 The primary study end point was the rate of ovarian failure at 2 years, with ovarian failure defined
47 survivors included older age at assessment, ovarian failure at a younger age, treatment with cranial
48 s, the development of methods for postponing ovarian failure at menopause may represent an attractive
52 l ovotoxicants plays a role in the premature ovarian failure commonly associated with infertility and
54 the rate of ovarian failure at 2 years, with ovarian failure defined as the absence of menses in the
56 ozygous mutant females displayed a premature ovarian failure due to a depletion of the germ cell pool
57 linical end points: development of premature ovarian failure, end-stage renal disease (ESRD), doublin
58 ulation is depleted through natural cycling, ovarian failure follows increasing periods of acyclity.
60 iable spectrum of somatic features including ovarian failure, heart and renal abnormalities, microgna
62 mong the 35 women who were defined as having ovarian failure, highly significant bone loss was observ
63 ally occurring BMP15 mutation leads to early ovarian failure in humans, and BMP15 has been shown to a
65 aluable for predicting the risk of premature ovarian failure in lupus nephritis patients treated with
72 ndergo a true menopause, female mice exhibit ovarian failure long before death because of chronologic
75 evaluate the effects of chemotherapy-induced ovarian failure on bone loss and markers of skeletal tur
76 alkylators can place women at risk for acute ovarian failure or premature menopause and men at risk f
78 ung women who are facing a risk of premature ovarian failure (POF) caused by radiation or chemotherap
79 or an SNP on MCM8 associated with premature ovarian failure (POF) diminishes the functional activity
80 nsanguineous family with inherited premature ovarian failure (POF) identified a homozygous frameshift
81 d not prevent chemotherapy-induced premature ovarian failure (POF) in patients with lymphoma, but may
82 ations cause gonadal dysgenesis or premature ovarian failure (POF) in women, as well as eyelid/forehe
88 he impact of treatment regimens on premature ovarian failure (POF) occurrence and motherhood, includi
91 lkylating agent doses were at risk for acute ovarian failure, premature menopause, and small-for-gest
93 35 with complete primary end-point data, the ovarian failure rate was 8% in the goserelin group and 2
94 ith chemotherapy appeared to protect against ovarian failure, reducing the risk of early menopause an
95 ificantly lower risk of developing premature ovarian failure (relative risk 0.10; 95% confidence inte
96 yopathy; demyelinating neuropathy; premature ovarian failure; short stature; hearing loss; pigmentary
97 east cancer who develop chemotherapy-induced ovarian failure should have their bone density monitored
99 in relation to models that ascribe premature ovarian failure to interruption of ovarian genes or to a
100 oid loss, ovariectomy and chemically-induced ovarian failure, to evaluate kainate-induced seizure act
102 nsanguineous family with inherited premature ovarian failure, we identified a homozygous 1-bp deletio
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