ライフサイエンスコーパス: ovariectomize

1 OMSC, orofacial mesenchymal stem cell; OVX, ovariectomized.

2 e (XE991) (40 mum), in both intact males and ovariectomized, 17beta-estradiol (E2)-treated females.

3 12-week-old Fisher 344 rats were ovariectomized 2 weeks before flight and randomized into

4 Treatment of ovariectomized 3xTg-AD mice with estrogen, but not proge

5 hs) and aged (22-23 months) female rats were ovariectomized 7 days prior to a 48-h administration of

6 hs) and aged (22-23 months) female rats were ovariectomized 7 days prior to implantation of silastic

7 Mammary carcinogenesis in ovariectomized ACI rats requires continuous exposure to

8 mary carcinogenesis, six-week-old intact and ovariectomized ACI rats were continuously exposed to low

9 Also, ovariectomized ACI rats were treated with high-dose estr

10 ne, low-dose estrogen plus progesterone, and ovariectomized ACI rats with high-dose estrogen plus pro

11 carcinogenesis induced solely by hormones in ovariectomized ACI rats.

12 male mice or testosterone administration to ovariectomized adult female mice.

13 Ovariectomized adult female rats were treated with a sin

14 the right lateral dorsal hippocampus area of ovariectomized adult rat.

15 Ovariectomized, adult Sprague-Dawley rats were implanted

16 Ovariectomized adults were subjected to a hormone replac

17 terotropic and ER-mediated gene responses in ovariectomized AF2ERKI female mice in the same manner as

18 Young adult female Sprague-Dawley rats were ovariectomized and bilaterally implanted via stereotaxic

19 edly protected against bone mass loss in the ovariectomized and dexamethasone treated rat osteoporosi

20 Female C57Bl6/J mice were ovariectomized and implanted with estradiol- or oil-secr

21 Lister Hooded rats were ovariectomized and implanted with mini-pumps containing

22 IL-6 levels in splenocyte supernatants from ovariectomized and male mice; elevated splenic IL-6 and

23 Ovariectomized and ovariectomized-osteoporosis rats were

24 BM progenitors in castrated male rats and in ovariectomized and proestrus female rats.

25 ntreated (control), laparatomized (sham), or ovariectomized and received a deficient diet (OVX-Diet).

26 Sprague-Dawley adult female rats were ovariectomized and then injected over 2 days with 17beta

27 d aged (about 22-month-old) female rats were ovariectomized and then, 4 weeks later, subcutaneously i

28 Young and middle-aged female monkeys were ovariectomized and treated with conjugated equine estrog

29 ue when subjects were either ovary-intact or ovariectomized and treated with estradiol, estradiol plu

30 e experiment, rats were left ovary-intact or ovariectomized and were then irradiated with 2.5, 5, 10,

31 These female rats were ovariectomized, and one subgroup was administered estrad

32 Cohorts of XX and XO gonadally intact, ovariectomized, and ovariectomized females supplemented

33 Female rats (15 months) were ovariectomized, and, 14 weeks later, adeno-associated vi

34 Cyclic estradiol treatment in aged ovariectomized animals restored MSB frequencies to level

35 polyphenols has translated the findings from ovariectomized animals to postmenopausal osteopenic wome

36 ral stroke and can be modelled using aged or ovariectomized animals.

37 n the DSA task compared to vehicle-implanted ovariectomized animals.

38 as this was reversed in the estrogen-treated ovariectomized animals.

39 Ovariectomized ApoE(-/-):Ins2(+/Akita) mice presented ch

40 In addition, ovariectomized ARKO hosts with wild-type ovary transplan

41 fferences, subsets of mice were castrated or ovariectomized at 5 weeks of age.

42 Female hamsters were ovariectomized at adulthood and housed in either a stand

43 Female Long Evans hooded rats were ovariectomized at middle age (11-12 months) and placed i

44 In vivo biodistribution studies in female ovariectomized athymic (NCr) nu/nu mice bearing GPR30-ex

45 to stimulate the growth of mammary tumors in ovariectomized athymic nude mice implanted with estrogen

46 evel analyses were investigated in immature, ovariectomized C57BL/6 mice treated with 300 mg/kg o, p'

47 METHODS AND We treated ovariectomized C57BL/6J mice with the ER-beta selective

48 d by 28% (P<0.01 compared to vehicle-treated ovariectomized controls), and all cells expressing detec

49 onsiveness ex vivo and in vivo compared with ovariectomized controls.

50 cant reduction in trabecular bone volume but ovariectomized Dmp1-ERalpha(-/-) female mice displayed a

51 (muscimol; 100ng/side) receptor agonists in ovariectomized, estradiol-primed rats.

52 This occurs on a daily basis in ovariectomized, estradiol-treated (OVX+E) mice; GnRH neu

53 Here, we used an established ovariectomized, estradiol-treated (OVX+E) mouse model ex

54 study the effect of IL-27 supplementation on ovariectomized estrogen-deficient mice on various immune

55 ment of endometriosis-like lesions in 63% of ovariectomized estrogen-supplemented Tgfb1-null mutant m

56 , hepatosteatosis developed only in male and ovariectomized female aLivGHRkd mice.

57 cts in response to letrozole were similar in ovariectomized female and male mice, with, however, diff

58 (housed individually) or pair housed with an ovariectomized female before induction of stroke, via tr

59 ed from the lungs of E2- and placebo-treated ovariectomized female C57BL/6 mice following infection.

60 Therefore, we used an ovariectomized female guinea pig model to measure the di

61 ffects whether it is administered to aged or ovariectomized female mice and emphasizes the need to co

62 17beta-Estradiol-treated ovariectomized female mice demonstrated increased lung l

63 found that supplementation of estrogen into ovariectomized female mice enhanced M2 polarization in v

64 te for the first time that E2 replacement in ovariectomized female mice improves stroke-induced perip

65 t recognition and object placement memory in ovariectomized female mice in an ERK-dependent manner, s

66 Immediately after training, ovariectomized female mice received bilateral DH infusio

67 ERalpha-deficient, or ERalpha-AF1-deficient ovariectomized female mice were fed a high-fat diet and

68 ential for vitamin D(3)-mediated protection, ovariectomized female mice were given E(2) or placebo an

69 Wild-type, ovariectomized female mice were treated with placebo or

70 In sham-operated or ovariectomized female mice, 17beta-E2, P4, 17beta-E2+P4,

71 Ovariectomized female mice, implanted with 17beta-E2 or

72 ced object recognition and spatial memory in ovariectomized female mice, whereas the GPER antagonist

73 EPO treatment also reduced weight gain in ovariectomized female mice, while the effect was abrogat

74 d object recognition memory consolidation in ovariectomized female mice.

75 at 1, 6 and 24 h post-ischaemia to aged and ovariectomized female mice.

76 ntracerebroventricular) inhibited feeding in ovariectomized female mice.

77 lly suppresses binge-like eating behavior in ovariectomized female mice.

78 ubstantially suppressed binge-like eating in ovariectomized female mice.

79 periment 1, E2 benzoate-induced LH surges in ovariectomized female monkeys were severely attenuated b

80 ME, also directly influences GnRH release in ovariectomized female monkeys, in which the ovarian sour

81 tal mice were either housed in pairs with an ovariectomized female or socially isolated for the durat

82 nitiated 14 days prior to global ischemia in ovariectomized female rats acts via the IGF-1 receptor t

83 d in bone from 5-wk-old intact and 10-wk-old ovariectomized female rats fed SPI diets.

84 tracerebral injection of 17beta-estradiol to ovariectomized female rats immediately after ischemia re

85 To control estradiol cyclical variability, ovariectomized female rats received empty or estradiol f

86 Ovariectomized female rats were tested for rotational be

87 Adult ovariectomized female rats were treated with 3 days of h

88 Ovariectomized female rats were used to test the possibi

89 In ovariectomized female rats, E2 rapidly (within 10 minute

90 signaling cascade to protect CA1 neurons in ovariectomized female rats.

91 prefrontal cortex (dlPFC) of young and aged ovariectomized female rhesus monkeys with and without E

92 ochemical distribution of aromatase in young ovariectomized female rhesus monkeys with or without lon

93 on enhances voluntary alcohol consumption in ovariectomized female rodents and that increased alcohol

94 ochemical effects of chronic E2 treatment of ovariectomized female wild-type and Kal7(KO) mice.

95 Indeed, in ovariectomized females and in male mice, the dominance o

96 itis (EAE) in intact female mice, but not in ovariectomized females or males.

97 Ovariectomized females supplemented with 17beta-estradio

98 and XO gonadally intact, ovariectomized, and ovariectomized females supplemented with estrogen were e

99 In contrast, ovariectomized females treated with estradiol and proges

100 Treatment of adult ovariectomized females with an aromatized metabolite of

101 In ovariectomized females, Kiss1 neurons were scattered thr

102 ellular activity similar to that observed in ovariectomized females, suggesting that estradiol-induce

103 vitamin D(3)-mediated EAE resistance in the ovariectomized females.

104 ater in E2-treated compared with oil-treated ovariectomized females.

105 genous progesterone treatment in males or in ovariectomized females.

106 Two-month-old female Sprague-Dawley rats, ovariectomized for 1 week, were given subcutaneous injec

107 amined in macaques that were ovary-intact or ovariectomized for 3 years living in a relatively natura

108 All mice were ovariectomized; half were estradiol replaced.

109 nterestingly, this effect is not observed in ovariectomized heterozygous BDNF Val66Met females, sugge

110 ter transplantation under kidney capsules of ovariectomized hosts, treated follicles developed to the

111 n a postmenopausal breast cancer model using ovariectomized, immune-competent female mice orthotopica

112 cells grafted subcutaneously into syngeneic ovariectomized immunocompetent mice, we found that E2 po

113 In ovariectomized-induced bone loss mouse model and RANKL-i

114 demonstrated oral efficacy in rat models of ovariectomized-induced thermoregulatory dysfunction and

115 22, were tested in a telemetric rat model of ovariectomized-induced thermoregulatory dysfunction and

116 d oral efficacy in a telemetric rat model of ovariectomized-induced thermoregulatory dysfunction, a m

117 ting (physiological bone loss condition) and ovariectomized (induction of surgical menopause) animals

118 Both intact and ovariectomized Kal7(KO) female mice exhibited decreased

119 e of absent gonadal estrogen, as chronically ovariectomized Kiss1r KO females developed obesity, hype

120 rata oriens, lucidum, and radiatum of CA3 in ovariectomized mice 6 h after administration.

121 n of Phd2 and Phd3 was sufficient to protect ovariectomized mice against bone loss without disrupting

122 ling was increased in female but not male or ovariectomized mice and was associated with increased di

123 rtantly, uterine ILC2s were nearly absent in ovariectomized mice and were increased in wild-type mice

124 Together, our findings establish ovariectomized mice as a model for UTIs in menopausal wo

125 Although ovariectomized mice continue to develop high-grade serou

126 Estrogen treatment of ovariectomized mice decreased I(K,total) (46.4+/-3.0 to

127 e surge of estrogen as estrogen treatment of ovariectomized mice did not alter the 26 S proteasome ac

128 7 days post-ischaemia, progesterone-treated ovariectomized mice did not differ significantly in perf

129 compared with shams, whereas vehicle-treated ovariectomized mice displayed a significant functional i

130 As expected, estradiol-implanted ovariectomized mice exhibited increased plasma estradiol

131 rmined morphometrically in male, female, and ovariectomized mice exposed to 6 months of cigarette smo

132 Anesthetized, ovariectomized mice had optical fibers implanted in the

133 We found that ovariectomized mice had significantly higher bacteriuria

134 In ovariectomized mice IL-17A and IL-23R expression was inc

135 asting; bone marrow-derived macrophages from ovariectomized mice implanted with estrogen exhibited en

136 ol (17-betaE) treatment of latently infected ovariectomized mice induces viral reactivation, as demon

137 In vivo, MPA treatment of latently infected ovariectomized mice inhibited IFN-gamma production and l

138 In uterine tissues of ovariectomized mice injected with P(4), miR-200 expressi

139 vestrant, decreased MCF7 xenograft growth in ovariectomized mice more potently than each drug alone.

140 ort-term effects of estradiol replacement in ovariectomized mice on apoE expression and markers for c

141 ordings of GnRH neurons in brain slices from ovariectomized mice revealed a slow ADP (sADP) after act

142 Finally, UPEC-infected ovariectomized mice showed a significant increase in qui

143 ed that 17-beta estradiol supplementation of ovariectomized mice significantly enhanced BM-EPC-induce

144 IL-27 treatment in ovariectomized mice suppressed Th17 cell differentiation

145 omian glands were obtained from young adult, ovariectomized mice that were administered 17beta-estrad

146 lume ( approximately 20%) in both normal and ovariectomized mice treated with SFN for 5 weeks compare

147 homeostasis after infection, was aberrant in ovariectomized mice with defective superficial urothelia

148 ngly, bone loss is prevented by treatment of ovariectomized mice with either antioxidants or CTLA4-Ig

149 ass, and bone strength in normal mice and in ovariectomized mice with established bone loss.

150 re made of GnRH neurons in brain slices from ovariectomized mice with ionotropic GABA and glutamate r

151 e and peripheral and CNS immune responses in ovariectomized mice with or without sustained, controlle

152 In ovariectomized mice, 5-, 10-, and 20-Hz activation of AR

153 In a further study of ovariectomized mice, allowed to survive for 7 days post-

154 onger excited preoptic kisspeptin neurons in ovariectomized mice, an effect that was fully restored b

155 nd functional impacts of E4 on the vagina of ovariectomized mice, and we determined the molecular mec

156 ry-enhancing effects in middle-aged and aged ovariectomized mice, and whether these effects depend on

157 Whereas in ovariectomized mice, at 48 h post-ischaemia, progesteron

158 estored after administration of estradiol in ovariectomized mice, demonstrating that the sex-specific

159 In ovariectomized mice, estrogen treatment resulted in down

160 When injected into ovariectomized mice, the FSH antibody attenuates bone lo

161 Using ovariectomized mice, we first demonstrate that intraperi

162 Using adoptive-transfer experiments and ovariectomized mice, we highlighted the role of female s

163 c disturbances in wt and ERalphaAF-1 degrees ovariectomized mice, whereas these actions were totally

164 ed growth of established MCF-7 xenografts in ovariectomized mice, whereas treatment with the neutrali

165 expression markedly increased in the bone of ovariectomized mice, which was blocked by bisphosphonate

166 liver, whereas such benefit was abolished in ovariectomized mice.

167 othalamic cultures from young or aged female ovariectomized mice.

168 ion following cerebral ischaemia in aged and ovariectomized mice.

169 comparing cortical bone in sham-treated and ovariectomized mice.

170 uced cell proliferation is partly reduced in ovariectomized mice.

171 to enhance novel object recognition in young ovariectomized mice.

172 ene expression in the hypothalamus in female ovariectomized mice.

173 ocytes from male, non-pregnant, pregnant and ovariectomized mice.

174 d a dose-dependent decrease in FSH levels in ovariectomized mice.

175 endogenous LH pulse parameters measured from ovariectomized mice.

176 covery of Opa(+) gonococci does not occur in ovariectomized mice; therefore, the reproductive cycle p

177 Ovariectomized Mig-6(d/d) mice exhibit this hyperplastic

178 Ovariectomized Mig-6(d/d) mice treated with E2 developed

179 ficient Min/+ colons, as well as colons from ovariectomized Min/+ mice (OvxMin/+) and E(2)-treated Ov

180 Aged ovariectomized monkeys treated with vehicle displayed si

181 In young ovariectomized monkeys without E treatment, presynaptic

182 ulated release of GnRH and E2 in the S-ME of ovariectomized monkeys, (2) electrical stimulation of th

183 utive and attention processes in middle-aged ovariectomized monkeys.

184 Previously, we reported cyclic ET in aged, ovariectomized NHPs increased spine density on dlPFC neu

185 to address this hypothesis using a long-term ovariectomized non-human primate (NHP) model, the cynomo

186 Fluorescent ER(+) MCF-7 tumors were grown in ovariectomized nude mice supplemented with estradiol.

187 Therefore, we used ovariectomized oil- or E2-treated EGFP (enhanced green f

188 as few PR-labeled profiles were detected in ovariectomized, oil-replaced females.

189 Food-restricted ovariectomized or sham-operated c-fos-GFP rats were trai

190 The mice were ovariectomized or sham-operated, and 5 weeks after surge

191 male Japanese macaques (Macaca fuscata) were ovariectomized or tubal-ligated (n=5/group) and returned

192 deficient, anti-IL-6 receptor alpha-treated, ovariectomized, or male mice; undetectable IL-6 levels i

193 ens of female C57BL/6 mice that were intact, ovariectomized, or ovariectomized with E2 replacement ex

194 ould be an effective enriched milk powder in ovariectomized-osteoporosis and ovariectomized rats as a

195 Ovariectomized and ovariectomized-osteoporosis rats were used as a menopaus

196 ne = ingestion of caffeine/sham surgery); 3) ovariectomized (OVX) = non-ingestion of caffeine/ovariec

197 le rats, the effects of exogenous ghrelin in ovariectomized (OVX) and estradiol (E2)-treated female r

198 C, the average core temperature (T(CORE)) of ovariectomized (OVX) control rats was significantly elev

199 PKG activity was not enhanced in ovariectomized (OVX) female mice as a result of cardiac

200 1-/-) mice, control Spp1+/+ (C57BL/6J) mice, ovariectomized (OVX) female mice, and estrogen-treated m

201 d the regulation of Hdac4 in the amygdala of ovariectomized (OVX) female mice.

202 onal antibody (Scl-Ab) in aged male rats and ovariectomized (OVX) female rats were used to study the

203 wing acquisition, intact male and intact and ovariectomized (OVX) female rats with and without estrad

204 Ovariectomized (OVX) female rats with and without estrad

205 he dorsolateral prefrontal cortex (dlPFC) in ovariectomized (OVX) female rhesus monkeys, and that E i

206 saline was administered to intact male rats, ovariectomized (OVX) females and OVX females treated wit

207 cement in male, female (freely cycling), and ovariectomized (OVX) females treated with either estroge

208 Intact males, females, and ovariectomized (OVX) females with and without estradiol

209 tion after vascular injury is exaggerated in ovariectomized (OVX) human C-reactive protein transgenic

210 Ovariectomized (OVX) mice treated or not with estradiol

211 molecular mechanisms mediating bone loss in ovariectomized (OVX) mice, a model of human menopause, u

212 Ovariectomized (ovx) mice, but not AIB1 x ERalpha-/- mic

213 We have shown that ovariectomized (Ovx) monkeys treated with estradiol (E)

214 ir in an estrogen-deprived animal model, the ovariectomized (Ovx) mouse, principally by dampening the

215 Two-month-old rats were ovariectomized (OVX) or had maxillary molars removed fro

216 Ovariectomized (OVX) rats (n=5-6/group) were treated wit

217 Ovariectomized (OVX) rats 24 h (but not 6 or 72 h) after

218 trus vs estrus), (2) pLTF would be absent in ovariectomized (OVX) rats and in physiological condition

219 ue inflammation, and the cecal microbiota in ovariectomized (OVX) rats bred for low-running capacity

220 es aldosterone binding, in male rats, and in ovariectomized (OVX) rats given estradiol benzoate (EB)

221 at increases circulating levels of AngII, in ovariectomized (OVX) rats treated with oestradiol benzoa

222 Treatment of ovariectomized (OVX) rats with estradiol benzoate (EB) c

223 Following implantation in ovariectomized (ovx) rats, the Sr(++)-cross-linked const

224 nucleus of the solitary tract (NTS) of lean ovariectomized (OVX) rodents.

225 However, in ovariectomized (OVX) WT and in ERbeta(-/-) mice, there w

226 ps: 1) young intact, 2) old intact, 3) young ovariectomized (OVX), 4) old OVX, 5) young OVX plus estr

227 Mice were ovariectomized (OVX), and a subset was treated with estr

228 studies were undertaken in brain slices from ovariectomized (OVX), diestrous, and proestrous kisspept

229 After task acquisition, animals were ovariectomized (OVX).

230 owth also increased mammary tumorigenesis in ovariectomized polyomavirus middle T-antigen mice.

231 program even in the absence ovarian E in an ovariectomized, progesterone-supplemented pregnant mouse

232 enting hot flushes in the morphine-dependent ovariectomized rat model and results in the restoration

233 Long-term ovariectomized rats also displayed a robust hyperinducti

234 14 T magnetic field exposure was compared in ovariectomized rats and ovariectomized rats with estradi

235 lk powder in ovariectomized-osteoporosis and ovariectomized rats as a model of menopause-osteoporosis

236 chronic 17beta-estradiol (E2) replacement of ovariectomized rats enhanced Kal7 expression in the hipp

237 ase in the trabecular bone formation rate in ovariectomized rats following oral administration.

238 In ovariectomized rats irradiated with 15 Gy, estradiol inc

239 diol or progesterone into the hippocampus of ovariectomized rats on serotonin (5-HT) clearance, as we

240 Pretreatment with beta-estradiol in ovariectomized rats protects hilar interneurons against

241 E(2)-treated ovariectomized rats received an injection of antisense o

242 We show in ovariectomized rats receiving estradiol or control treat

243 If so, exogenous hormonal priming to ovariectomized rats should eliminate fluoxetine's effect

244 nt of low rates of responding (DRL) tasks in ovariectomized rats that were given chronic estradiol vi

245 e was no difference between ovary-intact and ovariectomized rats that were irradiated at lower doses.

246 RANK was significantly expressed in ovariectomized rats treated with etidronate.

247 mia protected the hippocampus from damage in ovariectomized rats via phosphorylation of cyclic-AMP re

248 of spaceflight on bone microarchitecture in ovariectomized rats was bone-and bone compartment-specif

249 on lowering NET2C concentrations in urine in ovariectomized rats was demonstrated.

250 evant insult, as the CA3 region of long-term ovariectomized rats was profoundly hypersensitive to the

251 ity in hippocampal neurons of young and aged ovariectomized rats were determined.

252 Ovariectomized rats were given ten injections of E2 at 4

253 In Experiment 2, ovariectomized rats were SC administered propylene glyco

254 Ovariectomized rats were subchronically treated with 10

255 In another experiment, ovariectomized rats were treated continuously with three

256 Adult (6 mo) and aged (22 mo) ovariectomized rats with (OP) and without (Ovx) 17- exis

257 sure was compared in ovariectomized rats and ovariectomized rats with estradiol replacement.

258 formation and protects against bone loss in ovariectomized rats, an established preclinical model of

259 Leptin did not alter LSNA or HR in ovariectomized rats, but its effects were normalized wit

260 agonism manifests as tissue-selectivity: in ovariectomized rats, Cl-4AS-1 mimics DHT while TFM-4AS-1

261 In ovariectomized rats, osteoclasts were noticed in the roo

262 In ovariectomized rats, the compound lowers cholesterol, ma

263 In ovariectomized rats, there was more c-Fos expressed in t

264 By testing different classes of compounds in ovariectomized rats, we established that ligands that tr

265 ng, memory and ischemic neuronal survival in ovariectomized rats.

266 loped a standardized oral osteotomy model in ovariectomized rats.

267 and apposition sides of the periodontium in ovariectomized rats.

268 to inhibit serotonin transporter function in ovariectomized rats.

269 normal cycling rats and in estrogen-treated ovariectomized rats.

270 responses in a dose-dependent manner in the ovariectomized rats.

271 mammary carcinogenesis in intact but not in ovariectomized rats.

272 in a 3 mm femur osteoporotic defect model in ovariectomized rats.

273 cently stained femur samples from normal and ovariectomized rats.

274 st cancer cells and in the mammary glands of ovariectomized rats.

275 oreover, endocrine function was recovered in ovariectomized recipients, including elevated levels of

276 , and decreased bone turn over markers in an ovariectomized rodent model for postmenopausal osteoporo

277 e-dependent manner, to treat osteoporosis in ovariectomized rodents because of an isolated increase i

278 Estradiol treatment of ovariectomized rodents is known to affect the morphology

279 of young (4-5 months) and old (22-24 months) ovariectomized Sprague-Dawley female rats.

280 To test this, ovariectomized, steroid-primed rats were injected (i.p.)

281 inistration and extinction, female rats were ovariectomized to isolate estrogen effects on reinstatem

282 bone loss phenotype was seen in young adult ovariectomized transgenic mice by microcomputed tomograp

283 m unmoved objects after a 60-min delay while ovariectomized vehicle-treated females and gonadally int

284 However, adult ovariectomized Vgat-Cre;Lepr(lox/lox) mice displayed sig

285 he XE991-sensitive M-current by threefold in ovariectomized (vs oil-treated) female mice.

286 ta in regulating transcription and learning, ovariectomized wild-type (WT) and ERalpha and ERbeta kno

287 the development of preneoplastic lesions in ovariectomized wild-type (WT) and ERbeta knockout (ERbet

288 gene responses were increased in D3-treated ovariectomized wild-type animals, in a manner similar to

289 en-like effects of ICI in different tissues, ovariectomized wild-type mice and mice with mutations in

290 pithelial and stromal cells of the uterus in ovariectomized wild-type mice, but not in PRKO mice.

291 In ovariectomized wild-type mice, tamoxifen significantly r

292 anxiety, motor, and nociceptive behavior of ovariectomized, wildtype (WT), and ERss knockout (ssERKO

293 To achieve this goal, 2-month-old ovariectomized Wistar rats were injected bilaterally, in

294 Young adult female FVB/NJ mice were ovariectomized with (OVX+E, n=6) or without (OVX, n=8) e

295 /6 mice that were intact, ovariectomized, or ovariectomized with E2 replacement exhibited no differen

296 olism, and glucose intolerance compared with ovariectomized WT females.

297 Infarct sizes were equivalent between ovariectomized XX and XO mice, between intact XX and XO

298 d XO mice, and between estrogen-supplemented ovariectomized XX and XO mice.

299 Ovariectomized young and aged female mice were maintaine

300 bserved in the cholinergic neurons in either ovariectomized young or middle-aged monkeys.