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1 ment of refractory detrusor overactivity and overactive bladder.
2 involved in the management of patients with overactive bladder.
3 ecent developments in pharmacotherapy of the overactive bladder.
4 ted pig bladder strips, an in vitro model of overactive bladder.
5 evere lower urinary tract symptoms including overactive bladder.
6 and are obvious targets for treatment of the overactive bladder.
7 openers may have utility in the treatment of overactive bladder.
8 sorders of urine storage and voiding such as overactive bladder.
11 on is required to identify the true cause of overactive bladder, allowing new targeted treatments to
12 -five urine specimens (from 41 patients with overactive bladder and 24 controls) were examined using
13 drugs remain the first-line treatment of the overactive bladder and a favorable efficacy/tolerability
14 dysfunctional voiding, Botox injections for overactive bladder and an adult anticholinergic for over
17 cantly less likely to receive a diagnosis of overactive bladder and more likely to receive a diagnosi
19 nation with an alpha-blocker, in men with an overactive bladder and summarize the efficacy and safety
20 d clinical trials, to establish the cause of overactive bladder and to determine the best method of m
24 from urothelium results in incontinence and overactive bladder due to abnormal mechanotransduction;
25 ere are persistent urinary storage symptoms (overactive bladder) following therapy with an alpha-bloc
26 New theories and modified definitions of overactive bladder have been proposed, structured eviden
28 r existing anticholinergics, in treating the overactive bladder in children need closer scrutiny.
29 l nerve neuromodulation for the treatment of overactive bladder in patients who do not respond to opt
33 ur understanding of the basic science of the overactive bladder it is becoming clear that the control
38 al treatment of men with incontinence due to overactive bladder or to stress urinary incontinence pub
39 veral potential targets for treatment of the overactive bladder, particularly within the mechanosenso
40 on the Urogenital Distress Inventory and the Overactive Bladder Questionnaire (both P <0.001) at both
41 both before and after RTx as measured by the Overactive Bladder Questionnaire and International Prost
42 nd 2010 were asked to complete the validated Overactive Bladder Questionnaire based on patient sympto
43 inumtoxinA showed greater improvement in the Overactive Bladder Questionnaire SF for symptom bother (
44 om baseline in urinary symptom scores in the Overactive Bladder Questionnaire Short Form (SF); range,
45 00, higher scores indicating worse symptoms; Overactive Bladder Satisfaction questionnaire; range, 0-
47 rusor instability with subsequent obstructed/overactive bladder symptom complexes not dissimilar to t
50 hat in men with persistent storage symptoms (overactive bladder symptoms), clinically meaningful impr
52 ection and a control group the prevalence of overactive bladder syndrome (OAB), and how it was associ
55 gastroesophageal reflux disease (GERD), and overactive bladder syndrome (OBS), as well as other gast
57 ower urinary tract storage disorders such as overactive bladder syndrome and urinary incontinence sig
58 lower urinary tract symptoms, including the overactive bladder syndrome, and that combination of the
59 tment of nonobstructed urinary retention and overactive bladder syndrome, especially when accompanied
62 he treatment of lower urinary tract symptoms/overactive bladder syndrome/and detrusor overactivity.
63 ive bladder and an adult anticholinergic for overactive bladder that underwent testing in children; e
65 oms and nonobstructive pattern recognized as overactive bladder type has also been successfully evalu
66 actions is pivotal to the disease process in overactive bladder, urge incontinence, and spinal cord i
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