ライフサイエンスコーパス: overeating

1 ouths' social network is likely to reinforce overeating.

2 e, physiological satiety signals and promote overeating.

3 eating disorders and more moderate forms of overeating.

4 d alpha-MSH secretion in an attempt to limit overeating.

5 susceptibility to weight gain in response to overeating.

6 d cues, which might decrease risk for future overeating.

7 in responsivity may result from a period of overeating.

8 ture rather than the induction of compulsive overeating.

9 ve neurons in adult mice caused obesity from overeating.

10 eward region responsivity increases risk for overeating.

11 dency toward certain foods may contribute to overeating.

12 g and reward systems differently to moderate overeating.

13 bitory control signalling, which facilitates overeating.

14 vulnerability factor or a result of previous overeating.

15 tiety cues may prevent at-risk children from overeating.

16 t overall energy intake at meals and promote overeating.

17 hysiology of food that may predispose toward overeating.

18 lsive behavior as it applies to pathological overeating: (1) habitual overeating; (2) overeating to r

19 ies to pathological overeating: (1) habitual overeating; (2) overeating to relieve a negative emotion

20 metabolic environment in vivo, resulting in overeating after food deprivation in mice.

21 Overeating and binge eating assessed via questionnaire e

22 arting to binge drink frequently, while both overeating and binge eating predicted starting to use ma

23 for a common mechanism underlying compulsive overeating and compulsive drug taking.

24 It has been proposed that overeating and concomitant opioid release could downregu

25 fying subpopulations with increased risk for overeating and dietary obesity and detecting early signs

26 nt neurological underpinnings for compulsive overeating and drug addiction.

27 The role of diet composition in response to overeating and energy dissipation in humans is unclear.

28 ric intake and energy expenditure, fostering overeating and further weight gain.

29 n of CNTF treatment does not result in binge overeating and immediate rebound weight gain.

30 We all know that overeating and lack of exercise are risk factors for man

31 ing the behaviours that lead to pathological overeating and obesity are poorly understood.

32 rochemical pathways involved in pathological overeating and obesity are poorly understood.

33 g problems facing contemporary society, from overeating and obesity to impulsive sexual behavior and

34 (HF) food over time, which may contribute to overeating and obesity, has not been determined.

35 on in reward circuits may be associated with overeating and obesity.

36 on in this pathway could potentially lead to overeating and obesity.

37 nally representative US sample suggests that overeating and oversleeping can be used to identify an a

38 urrent theories consider obesity a result of overeating and sedentary life style and most efforts to

39 s that obesity does not have to be caused by overeating and sedentary life-style but may be the resul

40 sexual cues is associated with indulgence in overeating and sexual activity, respectively, and provid

41 ivity of reward circuitry increases risk for overeating and that this overeating may further attenuat

42 This may contribute to overeating and the development of obesity.

43 er satiating power and may result in passive overeating and therefore weight gain.

44 gested that insufficient sleep may stimulate overeating and thus play a role in the current epidemic

45 ptin levels, a consequence which may lead to overeating and weight gain in individuals or populations

46 nditure by doubly labeled water prior to the overeating and weight stabilization periods and at weeks

47 se studies can increase our understanding of overeating and, perhaps in some cases, the development o

48 nt of acute energy needs, thereby promoting "overeating" and the consequent development of an energy

49 atment for chronic food cravings, compulsive overeating, and binge eating that may represent a phenot

50 es that are linked to a sedentary lifestyle, overeating, and genetic factors.

51 es override signals from the body to promote overeating, and indicate OX1R antagonism as a potential

52 oses animals to systemic insulin resistance, overeating, and obesity.

53 e and treatments for phobias, addiction, and overeating are discussed.

54 These data do not support overeating as a cause of weight gain among breast cancer

55 r psychological factors, suggesting that the overeating associated with weight gain might be physiolo

56 fancy, evidence of learning disabilities and overeating behavior due to an abnormal satiety response

57 Emotional overeating behavior may occur in the context of negative

58 he absence of hunger and lead to maladaptive overeating behavior.

59 cated as a putative causal factor in chronic overeating, binge eating, and obesity.

60 lamus are well known for producing long-term overeating, but past research has excluded this site as

61 w the evidence that some forms of obesity or overeating could be considered a food addiction and argu

62 eating, recurrent and persistent episodes of overeating coupled with a lack of control over eating, a

63 relieve a negative emotional state; and (3) overeating despite aversive consequences.

64 dipose tissue occurs in response to cold and overeating (diet induced), and there is an inverse relat

65 for therapeutic interventions for compulsive-overeating disorder.

66 The growing prevalence of overeating disorders is a key contributor to the worldwi

67 es hunger state and pathways associated with overeating disorders.

68 used to study maladaptive behaviors, such as overeating, drug taking, or relapse.

69 Emotional overeating (EOE) has been associated with increased obes

70 rol is an important indicator of severity of overeating episodes.

71 somatosensory brain regions, contributing to overeating, few studies have examined the relative role

72 Hyperphagia continued in maturity, with overeating greatest during the mid- to late-dark cycle.

73 The food addiction model of overeating has been proposed to help explain the widespr

74 whether there is a genetic susceptibility to overeating high-fat diets has not been resolved.

75 Is starvation in anorexia nervosa (AN) or overeating in bulimia nervosa (BN) a form of addiction?

76 accurately recognize hunger signals, whereas overeating in bulimia nervosa could represent an exagger

77 to restricted eating in anorexia nervosa and overeating in bulimia nervosa.

78 Alcohol intake associates with overeating in humans.

79 ested that higher levels of binge eating and overeating in males at age 13 y likely cause higher BMI

80 t and thrombospondin receptor, in triggering overeating in mice with central BDNF depletion.

81 The neurobiological mechanisms underlying overeating in obesity are not understood.

82 mals affording a potential target to control overeating in people.

83 eural network that mediates this cue-induced overeating in sated rats include lateral hypothalamus, a

84 ll activity is essential for ethanol-induced overeating in the absence of societal factors, in single

85 s of the posterodorsal amygdala (PDA) elicit overeating in their own right.

86 This overeating is a clinical concern, but its causes are puz

87 seful for short periods when difficulty with overeating is anticipated, such as during vacations.

88 sivity of these regions may increase risk of overeating, it is unclear whether this is an initial vul

89 ct appetite in children, which could lead to overeating later in the day.

90 Thus, we tested whether overeating leads to reduced striatal responsivity to pal

91 Mounting evidence suggests that overeating may be conceptualized within the same behavio

92 ing worldwide trend in obesity, suggest that overeating may be the largest avoidable cause of cancer

93 rthermore, higher levels of binge eating and overeating may cause higher BMI in later life.

94 decreased reward sensitivity, implying that overeating may contribute to reduced striatal responsivi

95 increases risk for overeating and that this overeating may further attenuate responsivity of reward

96 uggest that initiating a prolonged period of overeating may increase striatal responsivity to food cu

97 Neither overeating nor binge eating was associated with starting

98 tor subtypes in obesity induced by voluntary overeating of a palatable diet.

99 d cause overeating or would be the effect of overeating of carbohydrate and fat.

100 aberrant eating, which posits that recurrent overeating of palatable food is similar to addictive beh

101 ntermittent access to palatable food exhibit overeating of palatable food upon renewed access and an

102 creased propensity to develop uncontrollable overeating of palatable food.

103 induced HP food motivation and engagement in overeating of such foods to enhance risk of weight gain

104 baseline intake or food restriction-induced overeating of the standard chow diet.

105 often narrows to the question of whether the overeating of these foods causes the same long-term neur

106 This suggests that behaviors, such as overeating or drug taking, might be especially prone to

107 Discontinuing unhealthy behaviors, such as overeating or drug use, depends upon an individual's abi

108 l variant frontotemporal dementia identified overeating or increased sweet food preference in 80 (78%

109 a relatively low protein intake would cause overeating or would be the effect of overeating of carbo

110 erventions for disorders such as drug abuse, overeating, or pathological gambling.

111 s in glucose levels might reduce the risk of overeating, particularly in environments inundated with

112 adult brain induced a hitherto unrecognized overeating phenotype and caused overweight that was prev

113 to be fixed in cave populations in which the overeating phenotype is present.

114 Overeating produced significantly less weight gain in th

115 may not be initial vulnerability factors, as overeating reduces D2 receptor density, D2 sensitivity,

116 ogical factors necessary for ethanol-induced overeating remain unclear, and societal causes have been

117 zards associated with calorie restriction or overeating, respectively.

118 ognized that some lifestyle patterns such as overeating result in metabolic syndrome, which may play

119 In conclusion, overeating SFAs promotes hepatic and visceral fat storag

120 den of disease is associated with behaviors--overeating, smoking, excessive alcohol consumption, and

121 eward region responsivity increases risk for overeating, suggesting qualitatively distinct reward sur

122 ble foods may explain, at least in part, why overeating tasty foods is so irresistible even in the fa

123 gion responsivity to food, which may lead to overeating that produces blunted dopamine signaling and

124 Despite overeating, they weighed less and had significantly redu

125 , and low mu-opioid availability may promote overeating to compensate decreased hedonic responses in

126 cal overeating: (1) habitual overeating; (2) overeating to relieve a negative emotional state; and (3

127 is increasing evidence that the pathological overeating underlying some forms of obesity is compulsiv

128 One potentially important difference between overeating versus drug abuse refers to the sensory stimu

129 fully adjusted models, binge eating, but not overeating, was associated with incident overweight/obes

130 ely causes higher levels of binge eating and overeating, weight and shape concerns, and weight-contro

131 d to a chronic food restriction challenge by overeating, which resulted in prerestriction, rather tha

132 crease their susceptibility to opportunistic overeating while at the same time making food intake les

133 Although any overeating, with or without loss of control, predicted t

134 Infrequent binge eating or purging or overeating without a loss of control were reported by 31