ライフサイエンスコーパス: overfeeding

1 ctor (TNF), was changed in adipose tissue by overfeeding.

2 may contribute to the hypophagic response to overfeeding.

3 sue from humans gaining weight (4-7 kg) with overfeeding.

4 y underfeeding and to avoid complications of overfeeding.

5 4; 2243 +/- 253 mg.kg-1.min-1) carbohydrate overfeeding.

6 dictions for overweight subjects may lead to overfeeding.

7 t oxidation were reciprocally related during overfeeding.

8 pEE did not increase during the first day of overfeeding.

9 he metabolic chamber during the last week of overfeeding.

10 izer under conditions of acute refeeding and overfeeding.

11 to estimate EI during weight gain induced by overfeeding.

12 ts receiving nutrition on CVVH may result in overfeeding.

13 ot the increase of IHCLs induced by fructose overfeeding.

14 reased by caloric restriction and reduced by overfeeding.

15 imize nutrition and protect individuals from overfeeding.

16 port in obese, diabetic patients rather than overfeeding.

17 hosphatase in the liver following short term overfeeding.

18 insulin and leptin resistance by short-term overfeeding.

19 ain containing (ADIPOQ)] were changed by SFA overfeeding.

20 balance of approximately 275 g after 96 h of overfeeding.

21 ptin and insulin ensued after just 3 days of overfeeding.

22 c rate or in thermogenesis from meals during overfeeding.

23 2 g/d (control) to a maximum of 10 g/d after overfeeding.

24 In 2 studies of controlled overfeeding (1 inpatient study and 1 outpatient study),

25 gy imbalance in six lean men on mixed diets (overfeeding: 16.5 MJ/d, +33%, n = 3; underfeeding: 3.5 M

26 Changes in body weight were +1.8 kg (overfeeding), -6.0 kg (CR), and +3.5 kg (refeeding).

27 , and a larger response to high-carbohydrate overfeeding all correlated with weight gain.

28 Neonatal overfeeding also perturbs the development of neural proj

29 Overfeeding also resulted in reduced hunger ratings and

30 ic exposure during pregnancy, and adolescent overfeeding, also during pregnancy.

31 Before and after overfeeding and after hypocaloric diet, metabolic variab

32 ion of PN exceeding energy needs may lead to overfeeding and associated complications.

33 d 3.1 +/- 2.1 (mean +/- SD) kg body fat with overfeeding and lost 2.4 +/- 1.7 kg body fat with underf

34 ise, FGF21 is produced from the liver during overfeeding and mitigates peripheral insulin resistance.

35 epEE) were measured on days 1, 14, and 56 of overfeeding and on day 57 while consuming the baseline d

36 aintaining normal blood glucose, by avoiding overfeeding and providing insulin therapy when necessary

37 rease in response to extra energy intake.SFA overfeeding and PUFA overfeeding induce distinct epigene

38 r avidly accumulates fat during carbohydrate overfeeding and support a role for DNL in the pathogenes

39 ophagic signaling in response to involuntary overfeeding and support the hypothesis that MCs are impo

40 unteracted most of the effects of short-term overfeeding and under-activity at the whole-body level a

41 Carbohydrate intake (540 and 83 g/d for overfeeding and underfeeding, respectively) exerted dire

42 Fat oxidation (59 and 177 g/d for overfeeding and underfeeding, respectively) was not sens

43 e to dietary fat intake (150 and 20 g/d, for overfeeding and underfeeding, respectively), rather, its

44 ate oxidation (551 and 106 g/d at day 12 for overfeeding and underfeeding, respectively).

45 .1% and 84.0% of the energy imbalance during overfeeding and underfeeding, respectively.

46 seems to normalize the anabolic response to overfeeding and, if these findings are confirmed, may be

47 omponents were modest; TEE changed by +6.2% (overfeeding) and -10.5% (underfeeding).

48 asting, a smaller EE response to low-protein overfeeding, and a larger response to high-carbohydrate

49 istinct responses of different fat depots to overfeeding, and fat-cell number increases in certain de

50 termine whether such responses to fasting or overfeeding are associated with future weight change.

51 xacerbates obesity by provoking compensatory overfeeding as one way to restore reward sensitivity.

52 Subsequent overfeeding, as a preventative measure against hypoglyce

53 Short-term overfeeding blunts the central effects of fatty acids on

54 Energy expenditure (EE) increases with overfeeding, but it is unclear how rapidly this is relat

55 n random order: energy balance (control) and overfeeding by 50% of energy requirements with fat (O(fa

56 vo lipogenesis was 2- to 3-fold higher after overfeeding by 50% than after the control treatment in a

57 novo lipogenesis was measured during 96 h of overfeeding by 50% with either sucrose or glucose and du

58 However, lower-body fat responded to overfeeding by fat-cell hyperplasia, with adipocyte numb

59 Overfeeding can lead to multiple metabolic and clinical

60 t is poorly recognized and may contribute to overfeeding complications.We aimed to quantify the syste

61 rectly predicted resistance to fat gain with overfeeding (correlation coefficient = 0.77, probability

62 inpatient individuals, calculated EI during overfeeding determined from DeltaES in FM and FFM was (m

63 free-living individuals, estimated EI during overfeeding determined from DeltaES in FM and FFM was 41

64 Overfeeding did not impair insulin signaling in WAT, but

65 ttenuates the effects of short-term fructose overfeeding, dietary conditions known to increase intrah

66 energy balance, fasting, and four different overfeeding diets with 200% energy requirements was meas

67 Increased EE in response to overfeeding dissipated 7.9% of the energy excess with a

68 Ten volunteers underwent 8 weeks of overfeeding, during which they gained an average weight

69 Overfeeding EI was estimated as the sum of baseline ener

70 The estimated overfeeding EI was then compared with the actual EI cons

71 Carbohydrate overfeeding for 3 wk induced a >10-fold greater relative

72 ses in total energy expenditure (TEE) during overfeeding have been small (0.9 +/- 0.8 MJ/d) and accou

73 In summary, overfeeding impairs hypothalamic insulin action, which m

74 aloric energy intake and after 2 days of 30% overfeeding in a counterbalanced design.

75 ently by dietary fat composition and general overfeeding in a randomized trial.We studied the effects

76 is, and regional fat responses to short-term overfeeding in BMI-matched overweight/obese individuals

77 n can predict weight increase in response to overfeeding in humans.

78 g a period of weight gain as a result of 40% overfeeding in individuals who are inpatients or free-li

79 A high degree of overfeeding in lean individuals was accompanied by a gre

80 steatohepatitis (SH) caused by intragastric overfeeding in mice.

81 REE increases significantly after short-term overfeeding in patients with HIV lipodystrophy.

82 NEAT) mediate resistance to weight gain with overfeeding in sedentary adults.

83 SleepEE was unchanged by overfeeding in the low-protein diet group, and baseline

84 (photomicrographs) and number in response to overfeeding in upper- and lower-body s.c. fat depots of

85 Overfeeding increased POMC mRNA in the ARC by 180% relat

86 Carbohydrate overfeeding increased weight (+/-SEM) by 2% (1.8 +/- 0.3

87 extra energy intake.SFA overfeeding and PUFA overfeeding induce distinct epigenetic changes in human

88 Conversely, in a model of overfeeding-induced insulin resistance, CB(1) antagonism

89 of food intake that accompanies involuntary overfeeding is an effective regulatory response to posit

90 responses to oleic acid following short term overfeeding is likely to contribute to the rapid onset o

91 ts for the variability in weight gain during overfeeding is the energy expended in physical activity.

92 Two days of overfeeding led to significant attenuation of these resp

93 re during fasting and smaller increases with overfeeding, lose less weight during caloric restriction

94 Controlling hyperlipidemia and preventing overfeeding may improve outcomes more than tight control

95 The decrease in walking that accompanied overfeeding occurred to a similar degree in the lean (-1

96 also affected by many other factors, such as overfeeding of bees with sucrose, harvesting prior to ma

97 he 2 groups to examine the overall effect of overfeeding on the DNA methylation in adipose tissue.The

98 We examined the effects of short-term overfeeding on the neuronal responses to food-related vi

99 We examined whether overfeeding overweight subjects with simple sugars incre

100 The importance of not overfeeding patients has been highlighted.

101 ture during fasting and a larger response to overfeeding predicted more weight loss over 6 weeks, eve

102 Furthermore, after 3 d of overfeeding, REE increased significantly in patients wit

103 Short-term overfeeding replicated this transcriptional pattern, sug

104 fat accumulation and body composition during overfeeding saturated fatty acids (SFAs) or polyunsatura

105 ALERIE) weight-loss study, the Bouchard Twin overfeeding study, and the Minnesota Starvation Experime

106 The type of carbohydrate overfeeding (sucrose or glucose) had no significant effe

107 n subjects enrolled in a trial of 8 weeks of overfeeding to promote fattening, adipocyte expansion re

108 xcess with a variation in EE of <1.7% across overfeeding treatments (NS).

109 there was no significant difference between overfeeding treatments.

110 xposed during our early evolution, including overfeeding, underactivity, aging, artificial lighting a

111 Three days of overfeeding virtually obliterated the metabolic and anor

112 With overfeeding, walking distance decreased by 1.5 miles/day

113 Overfeeding was prescribed as 140% of baseline energy re

114 For rats in the second group, the overfeeding was stopped, and they were infused intracere

115 24-h EE during fasting and the increase with overfeeding were correlated.

116 gy expenditure responses to fasting and 200% overfeeding were measured in a whole-room indirect calor

117 In conclusion, forced overfeeding with a high-fat diet in mice induces obesity

118 prevent the formation of new fat cells upon overfeeding with dietary cholesterol.

119 ) and macronutrient oxidation in response to overfeeding with different types of dietary carbohydrate

120 difference in fat balance during controlled overfeeding with fat, fructose, glucose, or sucrose.

121 De novo lipogenesis increases after overfeeding with glucose and sucrose to the same extent

122 re, and body protein did not increase during overfeeding with the low protein diet.

123 at de novo lipogenesis would increase during overfeeding, would vary depending on the type of carbohy