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1 ens preparation differed from the pattern of overflow from chromaffin cells and there was also some t
2 nt sensitization by regulating dopamine (DA) overflow from DA neuron terminals in the nucleus accumbe
3 lation of H(3)R or A(1)R reduced ischemic NE overflow from H(3)R(+/+) hearts by 50%, only A(1)R, but
7 ogenase, which in turn is driven by fumarate overflow from purine nucleotide breakdown and partial re
9 l in the superfusate, representing adenosine overflow from the retinas, and the adenosine level in re
10 (DeltaAd) adipocytes and the resultant lipid overflow from WAT led to marked hepatosteatosis, dyslipi
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