ライフサイエンスコーパス: oviduct

1 tected within the egg/cumulus complex in the oviduct.

2 ital tract, but fail to cause disease in the oviduct.

3 nd applied to eggs as they progress down the oviduct.

4 nce of allurin in the serosal capsule of the oviduct.

5 t contact with eggs as they move through the oviduct.

6 and the passage of early embryos through the oviduct.

7 concurrently with the apoptotic index of the oviduct.

8 al activity and restrict expression to avian oviduct.

9 e oviduct, and then persists apically in the oviduct.

10 two key reproductive tissues, the uterus and oviduct.

11 e ovary as well as on eggs isolated from the oviduct.

12 s induced eightfold by estrogen in the chick oviduct.

13 l aspect of the epithelium in the uterus and oviduct.

14 and adult lung, choroid plexus, testis, and oviduct.

15 passage of pre-implantation embryos down the oviduct.

16 on, and passage of the early embryo down the oviduct.

17 otic transformation of the proximal 25% into oviduct.

18 geneity from vesiculosomes shed from chicken oviduct.

19 and physical cues provided by the egg or the oviduct.

20 and suggesting it is expressed in the adult oviduct.

21 f the apoptosis-inducing factor Trail in the oviduct.

22 he stream that guides sperm migration in the oviduct.

23 ease from the ovary and its passage into the oviduct.

24 s such as airways, brain ventricles, and the oviduct.

25 tus induces fluid flow to guide sperm in the oviduct.

26 onse that did not prevent reinfection of the oviduct.

27 cy of late apoptotic/dead neutrophils in the oviduct.

28 CR-positive samples were from the uterus and oviducts.

29 on-induced swelling of the oocyte within the oviducts.

30 eonates had ovaries they lacked a uterus and oviducts.

31 cilitates the spread of both variants to the oviducts.

32 which regulates muscle contraction in locust oviducts [21]; and the FMRF amide dromyosuppressin, whic

33 ale reproductive tract and also a uterus and oviducts, a phenocopy of MIS ligand-deficient male mice.

34 Few eggs are detected in the oviduct after stimulation with gonadotropins, and no two

35 h following estrogen treatment in the chick oviduct, an extremely estrogen-responsive reproductive t

36 embryos spend the first few days within the oviduct and are transported to the uterus, where they im

37 aled constitutive and inducible NOS in human oviduct and cumulus (the cellular layer investing the oo

38 oth muscle cells of reproductive tracts, the oviduct and ductus epididymis.

39 ating gonadal steroid control, typical of an oviduct and egg specific gene.

40 icit significant chronic inflammation of the oviduct and fails to induce hydrosalpinx.

41 implantation embryo floats freely within the oviduct and is capable of developing into a blastocyst i

42 ique pattern of expression in the uterus and oviduct and its regulation by estrogen, a principal repr

43 on, and there was a significant reduction in oviduct and mesosalpinx pathology at late time points.

44 behavior required for progression along the oviduct and penetration of the zona pellucida.

45 of Wnt7a that mediates the patterning of the oviduct and proper differentiation of the uterus.

46 Oviduct and somatic ovarian telomere length declined mor

47 al processes such as oocyte transport in the oviduct and sperm penetration.

48 mRNA is expressed almost exclusively in the oviduct and that its expression is increased 2.5-fold by

49 tracts, with the uterus anteriorized towards oviduct and the vas deferens anteriorized towards epidid

50 fection, thereby preventing infection of the oviduct and thus the major disease sequelae associated w

51 lacking estrogen receptor alpha (ERalpha) in oviduct and uterine epithelial cells have impaired ferti

52 as preferentially recruited to the upper GT (oviduct and uterine horn) over the lower GT (cervical-va

53 The changes observed in the oviduct and uterus are accompanied by a postnatal loss o

54 rtile because of abnormal development of the oviduct and uterus, both of which are Mullerian duct der

55 lower levels of neutrophil chemokines in the oviducts and decreased production of neutrophil chemokin

56 sozymes show defects in transport within the oviducts and in binding to zonae pellucidae.

57 e ovaries, followed by egg movement down the oviducts and the deposition of eggs onto the substratum.

58 s progressively as the egg moves through the oviducts and the uterus.

59 of the Mullerian ducts leads to retention of oviducts and uteri in males.

60 ucing male pseudohermaphrodites that possess oviducts and uteri.

61 strogen-treated and estrogen-withdrawn chick oviducts and was subjected to differential display analy

62 roductive tissues (male hemipenes and female oviduct) and brain.

63 g fusion, migration from the uterus into the oviduct, and binding to the egg zona pellucida.

64 tor binding in the cockroach hindgut, locust oviduct, and fruit fly crop are similar.

65 ntaneous muscle contractions of the hindgut, oviduct, and heart.

66 pathway were regulated by estrogen in mouse oviduct, and inhibition of this pathway in a whole ovidu

67 ghly stage-specific manner in the uterus and oviduct, and its expression is restricted to the surface

68 ssful ovulation, transport of the COC to the oviduct, and its fertilization, depend on the interactio

69 o expressed in lung, eye, intestine, uterus, oviduct, and male reproductive tract.

70 vel of acute inflammation in the endocervix, oviduct, and mesosalpingeal tissues than in infected wil

71 ium in the region between the uterus and the oviduct, and then persists apically in the oviduct.

72 lting two-cell embryos were transferred into oviducts, and live mice were born.

73 ge of oocytes from the ovary into the narrow oviducts, and osmotic pressure caused by hydration-induc

74 ed from the endocervix to the uterine horns, oviducts, and ovaries in a temporal fashion before the o

75 in the uterus; proliferative lesions of the oviduct; and persistent vaginal cornification.

76 development and differentiation of the chick oviduct are exquisitely dependent upon estrogen, this se

77 The muscle cells surrounding the oviducts are multinuclear with highly organized sarcomer

78 pairs, the male ejaculatory duct and female oviduct, are known to be developmentally homologous.

79 alian preimplantation embryos develop in the oviduct as individual entities, and can develop and surv

80 ooperation may assist sperm passage into the oviduct as well as sperm-egg interactions.

81 ated epithelial cells lining the bronchi and oviduct, as well as in the developing spermatocytes in t

82 alpinx even when directly delivered into the oviduct at a high dose.

83 ferentiate CBF in different locations of the oviduct at different post-conception stages.

84 s compared with controls in both ovaries and oviducts at 6 mo.

85 greater numbers of viable chlamydiae in the oviducts at lower inoculating doses, and the number of o

86 njunctivitis in the cervix, endometrium, and oviducts at various times following a primary intravagin

87 m not only resulted in more infection in the oviduct but also stimulated more inflammatory infiltrati

88 sion of ER mRNA was prominent in most of the oviduct but not in the luminal epithelium.

89 ovaries, dTdc2 mutants release eggs into the oviducts but are unable to deposit them.

90 patches in the region between the uterus and oviduct, but is confined to the basal aspect of the epit

91 implantation embryos within the intact mouse oviduct by a simple electroporation method, and result i

92 non-oviduct tissue and in estrogen-deprived oviduct by a strong repressor site located from -130 to

93 sting that a rapid but transient invasion of oviduct by chlamydial organisms can prevent the developm

94 d to the GI tract even when delivered to the oviduct by intrabursal inoculation.

95 g oogenesis and delivering the oocyte to the oviduct by ovulation.

96 Elevated protease activity in cKO oviducts causes premature degradation of the zona pelluc

97 ds, present the first in vivo mapping of the oviduct CBF in its native context, and demonstrate the a

98 of the Ov gene was done by treating primary oviduct cell cultures with dimethyl sulfate.

99 gions were made and transfected into primary oviduct cell cultures.

100 beta production-capable TLR expressed by the oviduct cell lines, we were not able to determine whethe

101 purified BMP-7 induces apoptosis in primary oviduct cells.

102 ine endometrium, as well as epithelia of the oviduct, cervix, and vagina.

103 n the number of CD4, CD8, and B cells in the oviduct compared to the number of these cells at the sam

104 ious studies suggested that a barrier in the oviduct confines sperm and Acp36DE to a limited area nea

105 erse the uterus but do not progress into the oviduct, contributing to the infertility of fertilin bet

106 amide hydrolase (FAAH) in mouse embryos and oviducts creates locally an appropriate "anandamide tone

107 amide hydrolase (FAAH) in mouse embryos and oviducts creates locally an appropriate "anandamide tone

108 t, and inhibition of this pathway in a whole oviduct culture system resulted in a decreased embryo tr

109 ubsequent growth of OA synaptic sites at the oviduct, demonstrating that seminal proteins can contrib

110 y enhanced the incidence of hydrosalpinx and oviduct dilatation compared to those of TNF-alpha(-/-) m

111 ence of oviduct dilatation; however, reduced oviduct dilatation was observed for "controllers," i.e.,

112 ncluding the development of hydrosalpinx and oviduct dilatation.

113 Animals in both groups developed evidence of oviduct dilatation; however, reduced oviduct dilatation

114 smid-deficient strains are protected against oviduct disease upon challenge with virulent C. muridaru

115 g assays demonstrated that regression of the oviduct during estrogen withdrawal involves apoptosis, w

116 cy and absolute number of neutrophils in the oviducts during acute infection.

117 The chicken oviduct ecto-ATP diphosphohydrolase (ATPDase), a member

118 and is related to both the chicken liver and oviduct ecto-ATPDase enzymes characterized earlier, as w

119 The deduced primary sequence of the chicken oviduct ecto-ATPDase indicates a protein of 493 amino ac

120 equence comparison revealed that the chicken oviduct ecto-ATPDase is equally distant from the two ect

121 -ATPDase are similar to those of the chicken oviduct ecto-ATPDase that we have previously purified an

122 properties, the relationship of the chicken oviduct ecto-ATPDase with other reported ATPDases and ec

123 tudy of a purified ecto-ATPDase, the chicken oviduct ecto-ATPDase, with respect to ATP and ADP, and a

124 hows only minor differences from that of the oviduct ecto-ATPDase.

125 The ejaculatory duct/oviduct enhancer retains the ability to activate express

126 Although slightly larger than the 80-kDa oviduct enzyme, the two ecto-ATPDases are nearly identic

127 rface of ciliated epithelial cells of: lung, oviduct, epididymis, ductus deferens, and seminiferous t

128 the critical pattern recognition receptor in oviduct epithelial (OE) cells that is stimulated during

129 For this report a murine oviduct epithelial cell line was derived in order to det

130 The infected oviduct epithelial cell lines also secreted the immunomo

131 Oviduct epithelial cell lines infected with C. muridarum

132 We generated cloned murine oviduct epithelial cell lines without viral or chemical

133 Utilizing cloned murine oviduct epithelial cell lines, we previously identified

134 ute phase cytokines by C. muridarum-infected oviduct epithelial cell lines.

135 Because murine oviduct epithelial cells express TLR3 but not TLRs 4, 7,

136 mydia-induced IFN-beta synthesis in infected oviduct epithelial cells implicates a novel ligand that

137 infection induces IFN-beta synthesis in the oviduct epithelial cells in a TRIF-dependent manner.

138 arked only a fraction of ovarian surface and oviduct epithelial cells in wild-type tissues.

139 As expected, oviduct epithelial cells infected by Chlamydia muridarum

140 eased production of neutrophil chemokines by oviduct epithelial cells infected with CM3.1 in vitro.

141 for infection-induced IFN-beta secretion by oviduct epithelial cells remains to be determined.

142 C. muridarum-infected murine oviduct epithelial cells secrete the inflammatory cytoki

143 gene (STING) protein in HeLa cells and mouse oviduct epithelial cells significantly decreased IFN-bet

144 nt-negative TLR3 mutants, and TLR3-deficient oviduct epithelial cells to show that the IFN-beta secre

145 n the Chlamydia-induced IFN-beta response in oviduct epithelial cells, we used small interfering RNA,

146 of IFN-beta production by Chlamydia-infected oviduct epithelial cells.

147 macrophage cell line and in primary chicken oviduct epithelial cells.

148 The oviduct epithelial lines did not secrete IFN-beta in res

149 these in vitro studies predict that infected oviduct epithelium contributes significantly to host inn

150 on of a large number of embryos in the mouse oviduct, eventually leading to pregnancy failure.

151 Incubation of human sperm with oviduct explants induced sperm protein S-nitrosylation r

152 irect effects on preimplantation embryos via oviduct expression of embryotrophic cytokines.

153 , and wild-type embryos transferred into cKO oviducts fail to develop normally unless rescued by conc

154 ulated oocytes that can be fertilized in the oviduct (Figure 1).

155 ted with increased organism ascension to the oviduct following the intrauterine inoculation.

156 tibody and T cell responses that protect the oviduct from pathology despite a lack of sterilizing imm

157 elicited recall responses that protected the oviduct from pathology despite low-level reinfection of

158 s unknown if these signals are necessary for oviduct function in supporting fertilization and preimpl

159 senchyme disrupted the normal coiling of the oviduct in the knockout embryo, resembling the phenotype

160 f CD4 cells to the upper genital tract (GT) (oviducts) in comparison to the lower GT (cervix) during

161 led an important role of the interactions of oviduct infection with inflammatory responses in chlamyd

162 CBA/J mice developed a delayed and extensive oviduct infection.

163 The severity of oviduct inflammation and dilatation resulting from these

164 ntravaginal inoculation, suggesting that the oviduct inflammation can be induced by plasmid-independe

165 nts, despite their ability to activate acute oviduct inflammation, are attenuated in inducing tubal f

166 with the fact that eggs progressing down the oviduct initially show evidence of allurin being incorpo

167 n embryo development and passage through the oviduct into the uterus are prerequisites for implantati

168 which correlated with a rapid but transient oviduct invasion by C. muridarum with a peak infection o

169 monstrates that the function of BMP-7 in the oviduct involves the induction of apoptosis and that est

170 The tubular gland of the chicken oviduct is an attractive system for protein expression a

171 her sterilizing immunity at the level of the oviduct is essential for protection because of the possi

172 f Gld expression in the ejaculatory duct and oviduct is presented.

173 etory cells in the luminal epithelium of the oviduct, is displayed on the ciliary layer and then mech

174 a swimming pattern of mammalian sperm in the oviduct, is essential for fertilization in vivo.

175 the apical epithelial surface at the uterine-oviduct junction.

176 We observe that the oviduct lacks a clear demarcation from the anterior uter

177 med within the isthmic regions of the female oviducts, leading to a conjecture in the literature that

178 a preadaptation to the fetal feeding on the oviduct lining of viviparous caecilians.

179 , we found significantly more neutrophils in oviduct lumen of A/J mice on days 7 and 10, which correl

180 at adequate live chlamydial infection in the oviduct may be necessary to induce hydrosalpinx.

181 have a direct role in sperm-zona binding or oviduct migration; alternatively, the effects on these f

182 agonist on the cockroach hindgut and locust oviduct, mimicked the effect of dromyosuppressin on the

183 virulent variant, could be isolated from the oviducts more often and in greater numbers than the atte

184 The posterior VNC contains Ilp7+ oviduct motoneurons, whose innervation and morphology ar

185 We report further that oviduct muscle relaxation can be induced by activating O

186 of CB1 and beta2-adrenergic receptors in the oviduct muscularis implies that a basal endocannabinoid

187 identified a mating-dependent relaxation of oviduct musculature, for which ovulin is a necessary and

188 e, defective for function of the nonstriated oviduct myoepithelial sheath, and defective for epiderma

189 tor (RBF) was originally isolated from avian oviduct nuclear matrix.

190 mation of hydrosalpinx-a surrogate marker of oviduct occlusion and infertility.

191 iggered oviductal fluid secretion clears the oviduct of debris, lowers viscosity, and generates the s

192 ar Pullorum colonized both the ovary and the oviduct of hens and led to 6% of laid eggs being infecte

193 Infusion of hCG into the oviduct of steroid-hormone-treated ovariectomized baboon

194 ation was induced by chlamydial infection in oviducts of C3(-/-) mice, explaining why the C3(-/-) mic

195 vulation, cumulus masses were present in the oviducts of homozygous mutant mice, but zona-free eggs w

196 Finally, the oviducts of infected infertile mice showed evidence of c

197 At 3 and 6 mo, ovaries and oviducts of recuperated offspring had increased mitochon

198 segmented genomes that replicate only in the oviducts of some species of parasitic wasps and are requ

199 t with this observation, eggs recovered from oviducts of superovulated, adult mZP3-/- females also la

200 less invasive when delivered directly to the oviduct on day 7 after inoculation.

201 did intercaruncular endometrium, myometrium, oviduct, ovary, fetal bladder, or fetal kidney.

202 terone (P<0.001) and is reduced in regressed oviducts (P<0.001) demonstrating gonadal steroid control

203 athogenesis of Chlamydia trachomatis-induced oviduct pathological sequelae is not well understood.

204 cing Chlamydia-specific CD8(+) T cells cause oviduct pathological sequelae.

205 al clearance rates but significantly reduced oviduct pathology (hydrosalpinx) compared to that of wil

206 nd pathology in the uterine horns but normal oviduct pathology after infection.

207 the murine genital tract but does not elicit oviduct pathology because it fails to activate Toll-like

208 ronic infection in MyD88 KO mice resulted in oviduct pathology comparable to that of WT mice, increas

209 tested and found to display no reduction in oviduct pathology compared with control mice.

210 ta has been implicated in the development of oviduct pathology during Chlamydia muridarum genital inf

211 ay, in IL-1beta secretion and development of oviduct pathology during genital chlamydial infection.

212 n-CD8(+) T cells contribute significantly to oviduct pathology following genital chlamydial infection

213 on-CD8(+) cells cooperates to induce optimal oviduct pathology following genital chlamydial infection

214 more, IFNAR(-/-) mice developed less chronic oviduct pathology in comparison to that in WT mice.

215 N-beta has been implicated as an effector of oviduct pathology resulting from genital chlamydial infe

216 ither WT or TNFR1 KO CD8(+) T cells restored oviduct pathology to WT levels in both KO groups.

217 rophil infiltration, and reduced severity of oviduct pathology upon C. muridarum genital infection.

218 ns of Chlamydia muridarum are protected from oviduct pathology upon challenge with wild-type C. murid

219 uridarum infection but significantly reduced oviduct pathology, compared with WT animals.

220 -1R-deficient mice had significantly reduced oviduct pathology, which was associated with decreased n

221 earance of infection and in the mediation of oviduct pathology.

222 idarum clearance, greater dissemination, and oviduct pathology.

223 mmatory mediators and development of chronic oviduct pathology.

224 ce, while C57BL/6 mice are more resistant to oviduct pathology.

225 ect to both chlamydial clearance and reduced oviduct pathology.

226 n earlier than control mice and develop less oviduct pathology.

227 m, CM3.1, does not induce the development of oviduct pathology.

228 Motile cilia in the mammalian oviduct play a key role in reproduction, such as transpo

229 receptor-binding factor (RBF) for the avian oviduct progesterone (Pg) receptor (PR) has previously b

230 tial sequences of RF-hsp 70 and p60, a chick oviduct protein that shows 70% identity to the human pro

231 mechanism of restricted sperm entry into the oviduct rather than in sperm-egg interaction.

232 deleterious anamnestic T cell response upon oviduct reinfection.

233 , the mechanisms mediating regression of the oviduct remain unknown.

234 trogen-mediated cellular function within the oviduct remains unclear.

235 No bursa membrane is formed, and the oviduct remains uncoiled.

236 ng genital Chlamydia muridarum infection and oviduct sequelae.

237 he SREC that is involved in ejaculatory duct/oviduct-specific expression.

238 cal granules, nor is it the mouse homolog of oviduct-specific glycoprotein.

239 ed in various tissues including lung, brain, oviduct, testis, and embryonic kidney.

240 infiltration and cytokine production in the oviduct than the intravaginal inoculation, suggesting th

241 is secreted from the upper two thirds of the oviduct that includes the pars recta and the proximal pa

242 loops in the uterus and in the upper common oviduct that relax and constrict throughout sperm storag

243 dence exists regarding effects of BPA on the oviduct, the placenta, and pubertal development.

244 in estrogen-responsive tissues such as chick oviduct, the regulation of chMRP1 gene expression is con

245 becomes impermeable as it proceeds down the oviducts; the process is complete by the time the egg is

246 alter the lipid microenvironment within the oviduct, thereby affecting sperm motility.

247 cilia and their activity in the lumen of the oviduct through tissue layers represents a major challen

248 Ov is repressed in non-oviduct tissue and in estrogen-deprived oviduct by a str

249 sites in chicken progesterone receptor using oviduct tissue minces labeled with [32P]PO4 under nonequ

250 tory infiltration and cytokine production in oviduct tissue, suggesting that C5 may contribute to chl

251 ive chlamydial organisms were recovered from oviduct tissues of both C5(-/-) and C5(+/+) mice, sugges

252 Uterine and oviduct tissues were assessed for transcription of MMP g

253 al-vaginal tissues) and upper genital tract (oviduct tissues) to increasing inoculating doses.

254 gest that IRF members also repress Ov in non-oviduct tissues.

255 nation of Crisp protein expression in the Xt oviduct using RT-PCR showed that of five documented Xt C

256 loping Mullerian duct that gives rise to the oviduct, uterus and upper region of the vagina of the fe

257 In females, Mullerian ducts develop into the oviduct, uterus, cervix and upper vagina, whereas Wolffi

258 the emergence of distinct cell types in the oviduct, uterus, cervix and vagina and is dependent upon

259 of the Mullerian ducts, the primordia of the oviducts, uterus and upper vagina.

260 ctive tract organs of mammals, including the oviducts, uterus, cervix and upper vagina, are derived f

261 organisms were directly inoculated into the oviduct via an intrabursal injection, which was accompan

262 ducts that constitute a major portion of the oviduct wall.

263 , and expression levels in the magnum of the oviduct were constant over at least 16 months in transge

264 n of the bacteria from the endocervix to the oviduct, where an overly aggressive inflammatory respons

265 d to the tubular glands of the magnum of the oviduct, where egg white synthesis occurs, with around 1

266 sion of jeltraxin mRNA was restricted to the oviduct, which distinguishes it as the first serum-relat

267 arum developed visible hydrosalpinges in the oviduct while the remaining 13 did not, although all inf

268 How oocytes are transferred into an oviduct with a receptive environment remains poorly know

269 lia beat frequency (CBF) in the intact mouse oviduct with micro-scale spatial resolution.

270 utrophils were eliminated from the blood and oviducts with this treatment, immature neutrophils and h