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1                        The meiotic status of oviductal and remaining follicular oocytes was evaluated
2 wever, selective ablation of CTNNB1 from the oviductal ciliated cells did not affect embryo transport
3                                    Defective oviductal embryo transport arising from aberrant endocan
4                                     Impaired oviductal embryo transport is also observed in wild-type
5 contraction and relaxation crucial to normal oviductal embryo transport.
6 es of the female reproductive tract, such as oviductal, endometrial and cervical epithelia, and show
7 ture anaphase II onset upon removal from the oviductal environment.
8 on of the R273H p53 mutant protein in murine oviductal epithelial (MOE) cells enhanced proliferation
9 and was also increased in FT explants and in oviductal epithelial cell line OE-E6/E7 infected with C.
10                        FT explants (n=4) and oviductal epithelial cells (cell line OE-E6/E7) were tre
11 ted that disruption of estrogen signaling in oviductal epithelial cells alters ciliary function and i
12 hat ciliary length and beat frequency of the oviductal epithelial cells are regulated through estroge
13 ype showed significantly enhanced binding to oviductal epithelium.
14 -head agglutination and lack affinity to the oviductal epithelium.
15 e the promotion of flagellar detachment from oviductal epithelium.
16 area between the OSE, mesothelium and tubal (oviductal) epithelium, as a previously unrecognized stem
17 port, the other relatively simple (synthetic oviductal fluid plus albumin).
18 d coitus in female mice; prolactin-triggered oviductal fluid secretion clears the oviduct of debris,
19 h contains 5.5 mM d-glucose, or in synthetic oviductal fluid, in the presence but not in the absence
20 ased on the electrolyte concentration of the oviductal fluid.
21 -40, followed by macrophage chitotriosidase, oviductal glycoprotein, and macrophage YM-1.
22 e and humans produces uterine, cervical, and oviductal malformations.
23 ration with adrenergic receptors coordinates oviductal motility for normal journey of embryos into th
24 pendent technique, called Genome-editing via Oviductal Nucleic Acids Delivery (GONAD).
25                          Ovulation sites and oviductal oocytes were routinely observed in controls.
26 d by counting ovulation sites and collecting oviductal oocytes.
27  antimicrobial and antiinflammatory drugs on oviductal pathology in chronic chlamydial upper genital
28                         Thus, suppression of oviductal protease activity mediated by estrogen-epithel
29 nt cannabinoid signaling impedes coordinated oviductal smooth muscle contraction and relaxation cruci
30 ation, adenomyosis, and fibrosis, as well as oviductal smooth muscle hypertrophy.
31 e infertility, in part because of defects in oviductal transport and reduced numbers of follicles.
32      Estrogen receptor alpha is required for oviductal transport of embryos.
33 one" required for normal embryo development, oviductal transport, implantation, and pregnancy (see th
34 h-/- mice rescued developmental defects, not oviductal transport, implying that embryonic and materna
35  for normal development of embryos and their oviductal transport.
36 -expression in HGSC cells and PTEN-deficient oviductal tumors may have the potential to induce apopto

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