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1 -tetrahydronaphth-1-ylamino]-(2S)-2-propanol oxalate).
2 ilizing vanadinite and precipitating lead as oxalate.
3 on and renal excretion of both phosphate and oxalate.
4 um speciation, and the significant effect of oxalate.
5  least in part via the formation of ammonium oxalate.
6 by making cations unavailable to precipitate oxalate.
7 solution with the concomitant degradation of oxalate.
8 emperature and ambient pressure catalyzed by oxalate.
9  increasing amounts of Mn(III), carbadox and oxalate.
10 d with a solution of 0.05 mol L(-1) ammonium oxalate.
11 d measured transcellular secretion of [(14)C]oxalate.
12 tite and goethite at pH 7 in the presence of oxalate.
13 lating absorption or endogenous synthesis of oxalate.
14 mations of the corresponding N-phthalimidoyl oxalates.
15  weeks of treatment with either escitalopram oxalate (10-20 mg/d) or 16 sessions of manual-based cogn
16                                              Oxalate (11.09 mg/100 g) content was the major anti-nutr
17 (6.2-69.7% by BSW and 10.6-57.3% by BNW) and oxalate (14.7-88.9% by BSW and 14.5-87.3% by BNW) but sa
18              Phenolic ( approximately 0.2%), oxalate (2.2-3.4%) and saponin (2.6-3.0%) contents were
19                          The fraction of [Fe(oxalate)2](-) and [Fe(pyruvate)](2+) is significantly hi
20 is simple but elegant mechanism explains how oxalate, a molecule that humans and most animals cannot
21 ry oxalate results from passive paracellular oxalate absorption as modified by oxalate back secretion
22 , urine and biopsy studies is complicated by oxalate accumulation in chronic renal failure, and heigh
23 autosomal recessive disease characterized by oxalate accumulation in the kidneys and other organs.
24 n a cellular model system that recapitulated oxalate accumulation, exposure to DECA reduced oxalate a
25 alate accumulation, exposure to DECA reduced oxalate accumulation, similar to pyridoxine treatment th
26    In the presence of both DFOB and oxalate, oxalate acted synergistically with DFOB to increase the
27                           The combination of oxalate adaptation and oxalate supplementation in the ch
28                   The formation of insoluble oxalate after incubation at 25 degrees C for 30min is a
29                              The presence of oxalate alone caused the release of Cr, but not of Fe, f
30 plexed) Ni(2+) in solution suggests that the oxalate also alters Ni adsorption affinity.
31 ility field, with cationic lead(II) and lead oxalate also occurring.
32 pletely suppressed in the presence of 0.01 M oxalate, an organic ligand that can exist in GCS sites.
33 oxalate, and (4) a mixture of 0.2 M ammonium oxalate and 0.1 M ascorbic acid at 96 degrees C) are app
34 ure and enthalpy of vaporization of ammonium oxalate and adipate.
35 rapped: the covalent adducts between TPP and oxalate and between TPP and CO2.
36 effect of potassium citrate on urine calcium oxalate and calcium phosphate supersaturation and stone
37                         Furthermore, calcium oxalate and calcium phosphate supersaturation were highe
38 d phosphate levels lead to increased calcium oxalate and calcium phosphate supersaturation.
39 trations to wild-type (WT) plants, but lower oxalate and CaOx(c) concentrations.
40 ar pathway that senses and tightly regulates oxalate and citrate levels and may control Ca(2+)-oxalat
41 he molecular mechanisms that control urinary oxalate and citrate levels are not understood completely
42 adiolabeling was performed by pumping (89)Zr-oxalate and DFO-Bz-trastuzumab into the microfluidic rea
43 ptation of E. coli K-12 at pH 5.5 with 50 mM oxalate and inclusion of 25 mM oxalate in pH 3.0 minimal
44 speed suggesting that an important route for oxalate and malonate entry in cloudwater is via some com
45 ium(IV) complex (NBu4)2[ReBr4(ox)] (1) (ox = oxalate and NBu4(+) = tetra-n-butylammonium cation) has
46 .24mgkg(-1)FW, respectively), in addition to oxalate and phytate (14+/-9and0.17+/-0.02mg/100gFW, resp
47 evaluated and the values of phenols, tannin, oxalate and phytate contents were 0.02-0.32, 0.04-0.53,
48 ess copper, excreted approximately 1.9x more oxalate and produced approximately 1.75x less water-solu
49 o negligible values, also to reduce phytate, oxalate and saponin contents, simultaneously enhanced th
50 se cases was an inverse relationship between oxalate and several metals (Fe, Mn, K, Na, Mg, Ca), espe
51  calcium, and potassium and with lower urine oxalate and supersaturation for calcium oxalate and uric
52 monstrate that competition between dissolved oxalate and the mineral surface for Ni overwhelms the en
53 ion causes the accumulation of intracellular oxalate and the primary hyperoxaluria type 1 (PH1).
54  in tomato, a model plant for fleshy fruits; oxalate and threonate are accumulated in leaves, as is o
55 n 50% of the total Fe(III) is coordinated by oxalate and up to 20% of total Fe(III) is bound in the n
56 rine oxalate and supersaturation for calcium oxalate and uric acid.
57                 Three case flights show that oxalate (and no other organic acid) concentrations drop
58 ium dihydrogen phosphate, (3) 0.2 M ammonium oxalate, and (4) a mixture of 0.2 M ammonium oxalate and
59 trations of sea salt crustal tracer species, oxalate, and malonate were positively correlated with lo
60                    Urine citrate, phosphate, oxalate, and pH levels were higher and urine calcium lev
61  levels; however, the increases in urine pH, oxalate, and phosphate levels lead to increased calcium
62 g of lithogenic substrates, such as calcium, oxalate, and phosphate, and of inhibitors of crystalliza
63 tion did not increase in proportion to total oxalate, and the phytate concentration in all foods was
64 enol, salicylic acid, catechol, maleic acid, oxalate, and urea), the DeltaEE obtained during As(III)
65 rphous calcium carbonate cystoliths, calcium oxalates, and silica phytoliths.
66 ructed from Jahn-Teller distorted Cu(2+) and oxalate anions, showing a strong antiferromagnetic inter
67                                        Alkyl oxalates are new bench-stable alcohol-activating groups
68 nt ECL enhancement in the presence of sodium oxalate as co-reactant in PBS at pH 7.2).
69                  ECL was also observed using oxalate as the co-reactant, which was dissolved in the a
70                        We theorize that this oxalate ATR could enhance the pathogenesis of virulent E
71                     Unlike other SCOAs, this oxalate ATR is not a part of the RpoS regulon but appear
72 racellular oxalate absorption as modified by oxalate back secretion mediated by the SLC26A6 oxalate t
73      Because aluminum citrate blocks calcium oxalate binding and toxicity in human kidney cells, it m
74             Thus, in order to reduce soluble oxalate, bran samples (wheat, oat and barley) and bean s
75 lysis of the Cu(2+) interactions through the oxalate-bridges suggests a stripe mode pattern of coupli
76 t strategies target the formation of calcium oxalate but not its interaction with kidney tissue.
77  transported to the cytosol and converted to oxalate by lactate dehydrogenase, leading to kidney fail
78 plexes with cations, which increases soluble oxalate by making cations unavailable to precipitate oxa
79 etermined by atomic absorption spectroscopy, oxalate by titrimetry, phytate and tannin by colorimetri
80 uction of carbon dioxide to products such as oxalate (C2O4(2-)) is an active area of research, as the
81  suggest that photodissociation of iron(III) oxalate can lead to the formation of volatile oxidation
82            The dimerized species of CO2(*-), oxalate, can also be determined quantitatively.
83 FR2 in human and murine kidneys with calcium oxalate (CaOx) nephrocalcinosis-related CKD compared wit
84              Nephrocalcinosis, acute calcium oxalate (CaOx) nephropathy, and renal stone disease can
85  be stabilized within the conjugated uranium oxalate-carboxylate sheet.
86      However, the presence of ligands (i.e., oxalate, citrate, pyrophosphate) greatly retards the oxi
87 d mineral speciation as a function of pH and oxalate closely correlated with experimental conditions
88 ed significant reduction (P<0.05) in soluble oxalate compared to bean samples.
89 s smoothly with CO2 to generate the bridging oxalate complex [(TiX3 )2 (mu2 -C2 O4 -kappaO:kappaO'')]
90 s leads to the stabilization of a bismuth(I) oxalate complex and results in a core ion switch.
91                       Importantly, iron(III) oxalate complexes absorb near-UV radiation (lambda > 350
92                             Photolysis of Fe-oxalate complexes further enhances Fe dissolution with t
93 on photoreactivity in terms of iron-aqua and oxalate complexes, are not in accordance with our result
94 y the photodissociation of aqueous iron(III) oxalate complexes.
95 ely to involve -OH(-) exchange from zirconyl oxalate complexes.
96 ggests that this is dominantly the result of oxalate complexing and solubilizing Ni.
97  The oat bran sample had the highest soluble oxalate concentration at 79+/-1.3 mg/100 g, while total
98 ient to contribute to an increase in soluble oxalate concentration by binding calcium.
99                                      Soluble oxalate concentration did not increase in proportion to
100             Notably, ALN-GO1 reduced urinary oxalate concentration up to 50% after a single dose in t
101 hytase in all samples, but effect on soluble oxalate concentration varied.
102 mers due to its tendency to increase urinary oxalate concentration.
103  of calcium ions and this influences soluble oxalate concentration.
104 ed together with their soluble and insoluble oxalate concentrations in order to investigate the effec
105 t 79+/-1.3 mg/100 g, while total and soluble oxalate concentrations in the food samples studied range
106 thogenesis of virulent E. coli consumed with oxalate-containing foods like spinach.
107 modeling was associated with the decrease in oxalate content in transgenic fruit.
108 2-OXDC fruit showed up to a 90% reduction in oxalate content, which correlated with concomitant incre
109 verage) without affecting vegetables growth, oxalate contents and marketable quality.
110 ntion of starch, PEF treatment reduced tuber oxalate contents by almost 50% in some tissues and could
111                        With time, a shift to oxalate coordination of bioaccumulated copper occurred i
112 high oxalate production resulting in calcium oxalate crystal formation and deposition in the kidney a
113 h this, deficiency of RIPK3 or MLKL prevents oxalate crystal-induced acute kidney injury.
114 y intravital microscopy in models of IRI and oxalate crystal-induced acute kidney injury.
115                                Also in human oxalate crystal-related acute kidney injury, dying tubul
116  Oxalate-producing plants accumulate calcium oxalate crystals (CaOx(c)) in the range of 3-80% w/w of
117                                      Calcium oxalate crystals are widespread among animals and plants
118                      In combination, calcium oxalate crystals in leaves can act as a biochemical rese
119 ellular matrix (ECM) from the fungus, and Ca oxalate crystals.
120 the wild type, resulting in the formation of oxalate crystals.
121                                              Oxalate decarboxylase (OxDC) catalyzes the Mn-dependent
122                                              Oxalate decarboxylase (OxDC) catalyzes the Mn-dependent
123 alyzed, in a substrate-specific reaction, by oxalate decarboxylase (OXDC), forming formic acid and ca
124 anced expression level of Cmoxdc1 coding for oxalate decarboxylase.
125                                  The calcium oxalate deficient 5 (cod5) mutant of Medicago truncatula
126 cidity-mimicking phenotypes such as improved oxalate degradation and increased antifungal activity at
127 reas plays a repressor role in regulation of oxalate degradation and possibly antifungal activity of
128                         Improved efficacy in oxalate degradation by DeltaCmpacC-29 was consistent wit
129         On the basis of the stoichiometry of oxalate degradation in the PMS/CuFe(2)O(4), the radical
130   All species examined have abundant calcium oxalate deposits around the veins.
131 te-minus mutants, whereas supplementing with oxalate did not.
132                                         Zinc oxalate dihydrate was formed through oxalate excretion b
133 ch subsequently was transformed to manganese oxalate dihydrate.
134   As a result, the hydrogenation of dimethyl oxalate (DMO) to ethylene glycol, an important reaction
135 le of OXDC and the metabolic consequences of oxalate down-regulation in a heterotrophic, oxalic acid-
136 f box model studies we show that the loss of oxalate due to the photolysis of iron oxalato complexes
137     Aspergillus niger also precipitated lead oxalate during growth in the presence of lead carbonate,
138 on of CFTR stimulated SLC26A6-mediated Cl(-)-oxalate exchange in Xenopus oocytes.
139 es CM significantly reduced (>32.5%) urinary oxalate excretion and stimulated (>42%) distal colonic o
140  24 of 26 patients had steatorrhea and urine oxalate excretion averaged 69 mg/day, with a positive co
141    Zinc oxalate dihydrate was formed through oxalate excretion by the test fungi and the mineral surf
142  of the study was to determine whether urine oxalate excretion correlates with elements of fat balanc
143                     The reduction in urinary oxalate excretion in hyperoxaluric mice treated with O.
144                                Average urine oxalate excretion was 61 mg/day; there was no correlatio
145                        Fat balance and urine oxalate excretion were measured simultaneously in 26 sev
146  oxalate secretion, thereby reducing urinary oxalate excretion, via an unknown secretagogue.
147 ysis and lead oxalate formation depending on oxalate excretion.
148 s no correlation between fecal fat and urine oxalate excretion.
149 tive correlation between fecal fat and urine oxalate excretions (r = 0.71, P < .001).
150 dings confirmed the role of mineral-specific oxalate exudation in ectomycorrhizal weathering to disso
151 mide, duloxetine hydrochloride, escitalopram oxalate, fluoxetine hydrochloride, mirtazapine, nortript
152 ting the electrogenic exchange of sulfate or oxalate for chloride or bicarbonate and electroneutral c
153 te acetylhydrolase gene in H915-1 eliminated oxalate formation but neither influence on pH decrease n
154  formation reaction commonly anticipated for oxalate formation by reductive coupling of CO2 on low-va
155 ing on organic phosphate hydrolysis and lead oxalate formation depending on oxalate excretion.
156                                              Oxalate forms mononuclear bidentate ligand with surface
157 ent oxalate oxidoreductase (OOR) metabolizes oxalate, generating two molecules of CO2 and two low-pot
158 were defined as fecal fat >7 g/day and urine oxalate &gt;40 mg/day.
159 e coupled in a similar fashion through their oxalate half esters.
160 he citrate transporter NaDC-1 in citrate and oxalate homeostasis.
161 ophiles such as ethyl glyoxalate and diethyl oxalate in aqueous medium leads to the formation of benz
162  These results suggest that the dominance of oxalate in diacid concentrations measured in ambient aer
163                                      Soluble oxalate in foods is major concern for kidney stone forme
164  addition to the well-characterized C2O4(2-) oxalate in Li2C2O4 viable covalent CO2-based nets emerge
165 .5 with 50 mM oxalate and inclusion of 25 mM oxalate in pH 3.0 minimal challenge medium separately co
166                         The presence of iron oxalate in seed aerosol is found to inhibit aerosol grow
167 ted in increased survival over adaptation or oxalate in the challenge medium alone.
168 ading to the deposition of insoluble calcium oxalate in the kidney.
169   This research highlights the importance of oxalate in vanadinite bioweathering and suggests a gener
170                                        Thus, oxalate increases the relative reactivity ratio of bioti
171 umulation of weathered calcium with secreted oxalate, increasing significantly in sequence: quartz, g
172  a different therapeutic approach to calcium oxalate-induced injury.
173 te defects in growth, stress resistance, and oxalate/insecticidal compound production, only a small d
174                  Before and after RYGB, high oxalate intake contributed to the severity of hyperoxalu
175 redox coupling of tert-alkyl N-phthalimidoyl oxalate intermediates with electron-deficient alkenes is
176 ed activity of oxalate oxidase that converts oxalate into CO2 Similar results were also observed unde
177 from air and fixes it into oxalate, with the oxalate ion bridging between two copper atoms.
178                                    The bound oxalate ion is released as oxalic acid on treatment with
179 ysis demonstrated that Zr(IV) interacts with oxalate ions, and the fluoride adsorption mechanism is l
180 e of the (13)C(2) pair in [1-(18)O,(13)C(2)]-oxalate is 2-3 times longer than the spin-lattice relaxa
181 bon radicals from tert-alkyl N-phthalimidoyl oxalates is proposed that is based on earlier pioneering
182 ng of which occurs in the hereditary calcium oxalate kidney stone disease primary hyperoxaluria type
183 or inhibiting the crystallisation of calcium oxalate kidney stones in susceptible individuals.
184 1) in two unrelated individuals with calcium oxalate kidney stones.
185  (AGT), which allows glyoxylate oxidation to oxalate leading to the deposition of insoluble calcium o
186 ecretion, Cftr(-/-) mice had serum and urine oxalate levels 2.5-fold greater than those of wild-type
187 DECA and pyridoxine was additive in reducing oxalate levels.
188 bers and if PEF treatment could reduce tuber oxalate levels.
189 ed by Mn(II) and Cr(III) ions linked through oxalate ligands and a layer of [Fe(sal2-trien)](+) compl
190 ilibrated with aqueous solutions of acetate, oxalate, malonate, or citrate at 50 degrees C and 90 bar
191                                          The oxalate-minus mutants accumulated fumaric acid, produced
192                           Here, we generated oxalate-minus mutants of S. sclerotiorum using two indep
193                                          The oxalate-minus mutants retained pathogenicity on plants,
194 ng the host tissue enhanced virulence of the oxalate-minus mutants, whereas supplementing with oxalat
195  to (13)C(2) singlet states in a symmetrical oxalate molecule by substituting one or more (16)O nucle
196 catalyzes the Mn-dependent conversion of the oxalate monoanion into CO2 and formate.
197 catalyzes the Mn-dependent conversion of the oxalate monoanion into CO2 and formate.
198   Here we examine this phenomenon in calcium oxalate monohydrate (COM) crystallization, a model syste
199 e proteins in the crystallization of calcium oxalate monohydrate (COM), the most prominent constituen
200                                 Some calcium oxalate monohydrate (whewellite) was formed with natural
201      Here we report that crystals of calcium oxalate, monosodium urate, calcium pyrophosphate dihydra
202                 This investigation shows the oxalate motif is maintained when the concentration of li
203 eased incidence of hyperoxaluria and calcium oxalate nephrolithiasis.
204       We report two cases of renal allograft oxalate nephropathy in patients with remote histories of
205 ases in the transplant population, allograft oxalate nephropathy is likely to be an increasing cause
206 cation of bariatric surgery, with consequent oxalate nephropathy leading to chronic kidney disease an
207 lay progressive forms of nephrocalcinosis in oxalate nephropathy, such as primary hyperoxaluria.
208  in renal pathology are nephrocalcinosis and oxalate nephropathy.
209 re required to recognize and treat allograft oxalate nephropathy.
210 urgery, renal allografts are also at risk of oxalate nephropathy.
211 les as a fundamental initiating mechanism of oxalate nephropathy.
212 [Fe(sal2-trien)](+) complex cation into a 2D oxalate network in the presence of different solvents re
213 HBr3, or CH2Br2) intercalated between the 2D oxalate network.
214 could potentially aid the development of low oxalate oca-based foods.
215   Here we investigate effects of acetate and oxalate on alkali feldspar-brine interactions in a simul
216 gree of Al-Si order may affect the effect of oxalate on feldspar dissolution: a promotion of ~500% in
217  the effects of organic ligands (acetate and oxalate) on biotite dissolution and surface morphologica
218 eatment randomization to either escitalopram oxalate or cognitive behavior therapy for 12 weeks.
219             In the presence of both DFOB and oxalate, oxalate acted synergistically with DFOB to incr
220              Several forms of InsP(n) in the oxalate-oxalic acid extracted sediment were identified.
221 )) in sediment samples was tested, utilizing oxalate-oxalic acid extraction followed by determination
222       Tomato waste was treated with ammonium oxalate/oxalic acid by conventional extraction (CE), und
223 l decomposition and by increased activity of oxalate oxidase that converts oxalate into CO2 Similar r
224  the acidic conditions that are required for oxalate oxidase to function.
225                         Combining an enzyme, oxalate oxidase, and an organic oxidation catalyst, 4-am
226       Thiamine pyrophosphate (TPP)-dependent oxalate oxidoreductase (OOR) metabolizes oxalate, genera
227 Cl), only produced by P. javanicus, and lead oxalate (PbC2O4), produced by A. niger and P. javanicus.
228 cium than other GLVs, because of low fibres, oxalate, phytate and tannin content.
229 toxic substances (nitrate, nitrite, cyanide, oxalate, phytate, and trypsin inhibitor) in tubers of Je
230 ntent of calcium absorption inhibitors, like oxalate, phytate, tannin and dietary fibres, and evaluat
231 s using co-precipitation of metal salts into oxalate precursors and subsequent thermal decomposition.
232 k in the study region due to the ubiquity of oxalate precursors, clouds, and metal emissions from shi
233                                        Alkyl oxalates, prepared from their corresponding alcohols, ar
234 ether calcium would combine with the soluble oxalate present in the spinach.
235                                              Oxalate-producing plants accumulate calcium oxalate crys
236 n UV-induced mutants that concomitantly lost oxalate production and pathogenicity.
237 demonstrate the ability of ALN-GO1 to reduce oxalate production in preclinical models of PH1 across m
238 ency in this enzyme leads to abnormally high oxalate production resulting in calcium oxalate crystal
239 hase mechanistic relationships leading up to oxalate production.
240 5 and 14.13 percent in phytin phosphorus and oxalate respectively.
241         Net intestinal absorption of dietary oxalate results from passive paracellular oxalate absorp
242 y of Al-O-Si linkages due to the presence of oxalate results in the promotion of both Al and Si relea
243                                  When fed an oxalate-rich diet, wild-type mice developed progressive
244 loropurine provided crystalline 2AMTA as the oxalate salt.
245 to test the hypothesis that SLC26A6-mediated oxalate secretion is defective in cystic fibrosis.
246        We conclude that defective intestinal oxalate secretion mediated by SLC26A6 may contribute to
247 e exhibited significantly less transcellular oxalate secretion than intestinal tissue of wild-type mi
248 ation with the profound defect in intestinal oxalate secretion, Cftr(-/-) mice had serum and urine ox
249  with colonic epithelium and induces colonic oxalate secretion, thereby reducing urinary oxalate excr
250 cretion and stimulated (>42%) distal colonic oxalate secretion.
251 dentify the derived factors inducing colonic oxalate secretion.
252             For the first time, we show that oxalate selectively attacks edge surface sites and enhan
253 e net extent of feldspar's dissolution, with oxalate showing a more prominent effect than acetate.
254 on oxalato complexes is likely a significant oxalate sink in the study region due to the ubiquity of
255 tions in order to investigate the effects on oxalate solubility.
256 nsistent 100% recovery, at pH 4, using 0.1 M oxalate solution as an effective stripping agent.
257    The adsorbent is fully regenerable, using oxalate solution.
258 contribute to the increased risk for calcium oxalate stone formation observed in patients with cystic
259 aluria and hypocitraturia can trigger Ca(2+)-oxalate stone formation, even in the absence of hypercal
260 te and citrate levels and may control Ca(2+)-oxalate stone formation.
261                                     However, oxalate strongly enhanced biotite dissolution and induce
262    The combination of oxalate adaptation and oxalate supplementation in the challenge medium resulted
263 colate oxidase, to deplete the substrate for oxalate synthesis.
264                        The phenols, phytate, oxalate, tannin and alkaloid profiles of the flours were
265 grandiflora had a very high content of total oxalates, tannins and dietary fibers, which reduced calc
266 ormation of a quinoxaline-di-N-oxide/Mn(III)/oxalate ternary complex in which Mn(III) functioned as t
267 ding affinity are due to the formation of Ni-oxalate ternary surface complexes.
268 first used to predict that NPs conjugated to oxalate Tf will exhibit a higher degree of cellular asso
269 efficacy of NPs conjugated to our engineered oxalate Tf.
270            Upon reaction with DFOB or DFOB + oxalate, the remaining solids became enriched in Cr rela
271 FDA-approved drugs tramadol and escitalopram oxalate, they release or uptake serotonin in a dose- and
272                                              Oxalate thus largely enhances Ni mobility, thereby incre
273 f ligands facilitated electron transfer from oxalate to carbadox.
274                 Seeds of mutants accumulated oxalate to levels threefold higher than the wild type, r
275 so significantly restricted the induction of oxalate transport by CM.
276 rmigenes-derived bioactive factors stimulate oxalate transport in intestinal cells through mechanisms
277 fonic acid completely blocked the CM-induced oxalate transport.
278                             Knockdown of the oxalate transporter SLC26A6 also significantly restricte
279 re, we examined the relationship between the oxalate transporter SLC26A6 and the citrate transporter
280 alate back secretion mediated by the SLC26A6 oxalate transporter.
281                   The formation of manganese oxalate trihydrate was detected after growth of S. himan
282  a copper complex converts carbon dioxide to oxalate under mild conditions.
283 m, O. formigenes CM significantly stimulated oxalate uptake (>2.4-fold), whereas CM from Lactobacillu
284 ture conditioned medium (CM) on apical (14)C-oxalate uptake by human intestinal Caco-2-BBE cells.
285 rocess for conversion of carbon dioxide into oxalate using a binuclear copper complex and a mild redu
286                                              Oxalate-vanadium complexes markedly reduced the vanadini
287              In the complex, the C-C bond in oxalate was cleaved to create CO(2)(-*) radicals, follow
288                         In addition, calcium oxalate was formed from the calcium present in the miner
289                                         Lead oxalate was precipitated by Aspergillus niger during bio
290                           In spinach, [(14)C]oxalate was the major product of [(14)C]ascorbate degrad
291                                  Acetate and oxalate were detected as transient intermediates during
292           pH 7 samples behaved like ammonium oxalate, which has a vapor pressure of approximately 10(
293 lated to give the tert-alkyl N-phthalimidoyl oxalate, which in the presence of visible-light, catalyt
294  scaled with the absorption spectrum of iron oxalate, which suggests that the reaction is at least in
295 h is known to contain significant amounts of oxalates, which are toxic and, if consumed regularly, ca
296 dine (a highly disordered feldspar) owing to oxalate, while the corresponding increase for albite (a
297 dical coupling of tert-alkyl N-phthalimidoyl oxalates with electron-deficient alkenes is terminated b
298 dditional sink for tartronate, pyruvate, and oxalate, with a complex photolysis contribution to overa
299 th carbon dioxide from air and fixes it into oxalate, with the oxalate ion bridging between two coppe
300 CO2(*-) produced can either dimerize to form oxalate within the nanogap between SECM tip and substrat

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