ライフサイエンスコーパス: oxalate

1 -tetrahydronaphth-1-ylamino]-(2S)-2-propanol oxalate).

2 ilizing vanadinite and precipitating lead as oxalate.

3 on and renal excretion of both phosphate and oxalate.

4 um speciation, and the significant effect of oxalate.

5 least in part via the formation of ammonium oxalate.

6 by making cations unavailable to precipitate oxalate.

7 solution with the concomitant degradation of oxalate.

8 emperature and ambient pressure catalyzed by oxalate.

9 increasing amounts of Mn(III), carbadox and oxalate.

10 d with a solution of 0.05 mol L(-1) ammonium oxalate.

11 d measured transcellular secretion of [(14)C]oxalate.

12 tite and goethite at pH 7 in the presence of oxalate.

13 lating absorption or endogenous synthesis of oxalate.

14 mations of the corresponding N-phthalimidoyl oxalates.

15 weeks of treatment with either escitalopram oxalate (10-20 mg/d) or 16 sessions of manual-based cogn

16 Oxalate (11.09 mg/100 g) content was the major anti-nutr

17 (6.2-69.7% by BSW and 10.6-57.3% by BNW) and oxalate (14.7-88.9% by BSW and 14.5-87.3% by BNW) but sa

18 Phenolic ( approximately 0.2%), oxalate (2.2-3.4%) and saponin (2.6-3.0%) contents were

19 The fraction of [Fe(oxalate)2](-) and [Fe(pyruvate)](2+) is significantly hi

20 is simple but elegant mechanism explains how oxalate, a molecule that humans and most animals cannot

21 ry oxalate results from passive paracellular oxalate absorption as modified by oxalate back secretion

22 , urine and biopsy studies is complicated by oxalate accumulation in chronic renal failure, and heigh

23 autosomal recessive disease characterized by oxalate accumulation in the kidneys and other organs.

24 n a cellular model system that recapitulated oxalate accumulation, exposure to DECA reduced oxalate a

25 alate accumulation, exposure to DECA reduced oxalate accumulation, similar to pyridoxine treatment th

26 In the presence of both DFOB and oxalate, oxalate acted synergistically with DFOB to increase the

27 The combination of oxalate adaptation and oxalate supplementation in the ch

28 The formation of insoluble oxalate after incubation at 25 degrees C for 30min is a

29 The presence of oxalate alone caused the release of Cr, but not of Fe, f

30 plexed) Ni(2+) in solution suggests that the oxalate also alters Ni adsorption affinity.

31 ility field, with cationic lead(II) and lead oxalate also occurring.

32 pletely suppressed in the presence of 0.01 M oxalate, an organic ligand that can exist in GCS sites.

33 oxalate, and (4) a mixture of 0.2 M ammonium oxalate and 0.1 M ascorbic acid at 96 degrees C) are app

34 ure and enthalpy of vaporization of ammonium oxalate and adipate.

35 rapped: the covalent adducts between TPP and oxalate and between TPP and CO2.

36 effect of potassium citrate on urine calcium oxalate and calcium phosphate supersaturation and stone

37 Furthermore, calcium oxalate and calcium phosphate supersaturation were highe

38 d phosphate levels lead to increased calcium oxalate and calcium phosphate supersaturation.

39 trations to wild-type (WT) plants, but lower oxalate and CaOx(c) concentrations.

40 ar pathway that senses and tightly regulates oxalate and citrate levels and may control Ca(2+)-oxalat

41 he molecular mechanisms that control urinary oxalate and citrate levels are not understood completely

42 adiolabeling was performed by pumping (89)Zr-oxalate and DFO-Bz-trastuzumab into the microfluidic rea

43 ptation of E. coli K-12 at pH 5.5 with 50 mM oxalate and inclusion of 25 mM oxalate in pH 3.0 minimal

44 speed suggesting that an important route for oxalate and malonate entry in cloudwater is via some com

45 ium(IV) complex (NBu4)2[ReBr4(ox)] (1) (ox = oxalate and NBu4(+) = tetra-n-butylammonium cation) has

46 .24mgkg(-1)FW, respectively), in addition to oxalate and phytate (14+/-9and0.17+/-0.02mg/100gFW, resp

47 evaluated and the values of phenols, tannin, oxalate and phytate contents were 0.02-0.32, 0.04-0.53,

48 ess copper, excreted approximately 1.9x more oxalate and produced approximately 1.75x less water-solu

49 o negligible values, also to reduce phytate, oxalate and saponin contents, simultaneously enhanced th

50 se cases was an inverse relationship between oxalate and several metals (Fe, Mn, K, Na, Mg, Ca), espe

51 calcium, and potassium and with lower urine oxalate and supersaturation for calcium oxalate and uric

52 monstrate that competition between dissolved oxalate and the mineral surface for Ni overwhelms the en

53 ion causes the accumulation of intracellular oxalate and the primary hyperoxaluria type 1 (PH1).

54 in tomato, a model plant for fleshy fruits; oxalate and threonate are accumulated in leaves, as is o

55 n 50% of the total Fe(III) is coordinated by oxalate and up to 20% of total Fe(III) is bound in the n

56 rine oxalate and supersaturation for calcium oxalate and uric acid.

57 Three case flights show that oxalate (and no other organic acid) concentrations drop

58 ium dihydrogen phosphate, (3) 0.2 M ammonium oxalate, and (4) a mixture of 0.2 M ammonium oxalate and

59 trations of sea salt crustal tracer species, oxalate, and malonate were positively correlated with lo

60 Urine citrate, phosphate, oxalate, and pH levels were higher and urine calcium lev

61 levels; however, the increases in urine pH, oxalate, and phosphate levels lead to increased calcium

62 g of lithogenic substrates, such as calcium, oxalate, and phosphate, and of inhibitors of crystalliza

63 tion did not increase in proportion to total oxalate, and the phytate concentration in all foods was

64 enol, salicylic acid, catechol, maleic acid, oxalate, and urea), the DeltaEE obtained during As(III)

65 rphous calcium carbonate cystoliths, calcium oxalates, and silica phytoliths.

66 ructed from Jahn-Teller distorted Cu(2+) and oxalate anions, showing a strong antiferromagnetic inter

67 Alkyl oxalates are new bench-stable alcohol-activating groups

68 nt ECL enhancement in the presence of sodium oxalate as co-reactant in PBS at pH 7.2).

69 ECL was also observed using oxalate as the co-reactant, which was dissolved in the a

70 We theorize that this oxalate ATR could enhance the pathogenesis of virulent E

71 Unlike other SCOAs, this oxalate ATR is not a part of the RpoS regulon but appear

72 racellular oxalate absorption as modified by oxalate back secretion mediated by the SLC26A6 oxalate t

73 Because aluminum citrate blocks calcium oxalate binding and toxicity in human kidney cells, it m

74 Thus, in order to reduce soluble oxalate, bran samples (wheat, oat and barley) and bean s

75 lysis of the Cu(2+) interactions through the oxalate-bridges suggests a stripe mode pattern of coupli

76 t strategies target the formation of calcium oxalate but not its interaction with kidney tissue.

77 transported to the cytosol and converted to oxalate by lactate dehydrogenase, leading to kidney fail

78 plexes with cations, which increases soluble oxalate by making cations unavailable to precipitate oxa

79 etermined by atomic absorption spectroscopy, oxalate by titrimetry, phytate and tannin by colorimetri

80 uction of carbon dioxide to products such as oxalate (C2O4(2-)) is an active area of research, as the

81 suggest that photodissociation of iron(III) oxalate can lead to the formation of volatile oxidation

82 The dimerized species of CO2(*-), oxalate, can also be determined quantitatively.

83 FR2 in human and murine kidneys with calcium oxalate (CaOx) nephrocalcinosis-related CKD compared wit

84 Nephrocalcinosis, acute calcium oxalate (CaOx) nephropathy, and renal stone disease can

85 be stabilized within the conjugated uranium oxalate-carboxylate sheet.

86 However, the presence of ligands (i.e., oxalate, citrate, pyrophosphate) greatly retards the oxi

87 d mineral speciation as a function of pH and oxalate closely correlated with experimental conditions

88 ed significant reduction (P<0.05) in soluble oxalate compared to bean samples.

89 s smoothly with CO2 to generate the bridging oxalate complex [(TiX3 )2 (mu2 -C2 O4 -kappaO:kappaO'')]

90 s leads to the stabilization of a bismuth(I) oxalate complex and results in a core ion switch.

91 Importantly, iron(III) oxalate complexes absorb near-UV radiation (lambda > 350

92 Photolysis of Fe-oxalate complexes further enhances Fe dissolution with t

93 on photoreactivity in terms of iron-aqua and oxalate complexes, are not in accordance with our result

94 y the photodissociation of aqueous iron(III) oxalate complexes.

95 ely to involve -OH(-) exchange from zirconyl oxalate complexes.

96 ggests that this is dominantly the result of oxalate complexing and solubilizing Ni.

97 The oat bran sample had the highest soluble oxalate concentration at 79+/-1.3 mg/100 g, while total

98 ient to contribute to an increase in soluble oxalate concentration by binding calcium.

99 Soluble oxalate concentration did not increase in proportion to

100 Notably, ALN-GO1 reduced urinary oxalate concentration up to 50% after a single dose in t

101 hytase in all samples, but effect on soluble oxalate concentration varied.

102 mers due to its tendency to increase urinary oxalate concentration.

103 of calcium ions and this influences soluble oxalate concentration.

104 ed together with their soluble and insoluble oxalate concentrations in order to investigate the effec

105 t 79+/-1.3 mg/100 g, while total and soluble oxalate concentrations in the food samples studied range

106 thogenesis of virulent E. coli consumed with oxalate-containing foods like spinach.

107 modeling was associated with the decrease in oxalate content in transgenic fruit.

108 2-OXDC fruit showed up to a 90% reduction in oxalate content, which correlated with concomitant incre

109 verage) without affecting vegetables growth, oxalate contents and marketable quality.

110 ntion of starch, PEF treatment reduced tuber oxalate contents by almost 50% in some tissues and could

111 With time, a shift to oxalate coordination of bioaccumulated copper occurred i

112 high oxalate production resulting in calcium oxalate crystal formation and deposition in the kidney a

113 h this, deficiency of RIPK3 or MLKL prevents oxalate crystal-induced acute kidney injury.

114 y intravital microscopy in models of IRI and oxalate crystal-induced acute kidney injury.

115 Also in human oxalate crystal-related acute kidney injury, dying tubul

116 Oxalate-producing plants accumulate calcium oxalate crystals (CaOx(c)) in the range of 3-80% w/w of

117 Calcium oxalate crystals are widespread among animals and plants

118 In combination, calcium oxalate crystals in leaves can act as a biochemical rese

119 ellular matrix (ECM) from the fungus, and Ca oxalate crystals.

120 the wild type, resulting in the formation of oxalate crystals.

121 Oxalate decarboxylase (OxDC) catalyzes the Mn-dependent

122 Oxalate decarboxylase (OxDC) catalyzes the Mn-dependent

123 alyzed, in a substrate-specific reaction, by oxalate decarboxylase (OXDC), forming formic acid and ca

124 anced expression level of Cmoxdc1 coding for oxalate decarboxylase.

125 The calcium oxalate deficient 5 (cod5) mutant of Medicago truncatula

126 cidity-mimicking phenotypes such as improved oxalate degradation and increased antifungal activity at

127 reas plays a repressor role in regulation of oxalate degradation and possibly antifungal activity of

128 Improved efficacy in oxalate degradation by DeltaCmpacC-29 was consistent wit

129 On the basis of the stoichiometry of oxalate degradation in the PMS/CuFe(2)O(4), the radical

130 All species examined have abundant calcium oxalate deposits around the veins.

131 te-minus mutants, whereas supplementing with oxalate did not.

132 Zinc oxalate dihydrate was formed through oxalate excretion b

133 ch subsequently was transformed to manganese oxalate dihydrate.

134 As a result, the hydrogenation of dimethyl oxalate (DMO) to ethylene glycol, an important reaction

135 le of OXDC and the metabolic consequences of oxalate down-regulation in a heterotrophic, oxalic acid-

136 f box model studies we show that the loss of oxalate due to the photolysis of iron oxalato complexes

137 Aspergillus niger also precipitated lead oxalate during growth in the presence of lead carbonate,

138 on of CFTR stimulated SLC26A6-mediated Cl(-)-oxalate exchange in Xenopus oocytes.

139 es CM significantly reduced (>32.5%) urinary oxalate excretion and stimulated (>42%) distal colonic o

140 24 of 26 patients had steatorrhea and urine oxalate excretion averaged 69 mg/day, with a positive co

141 Zinc oxalate dihydrate was formed through oxalate excretion by the test fungi and the mineral surf

142 of the study was to determine whether urine oxalate excretion correlates with elements of fat balanc

143 The reduction in urinary oxalate excretion in hyperoxaluric mice treated with O.

144 Average urine oxalate excretion was 61 mg/day; there was no correlatio

145 Fat balance and urine oxalate excretion were measured simultaneously in 26 sev

146 oxalate secretion, thereby reducing urinary oxalate excretion, via an unknown secretagogue.

147 ysis and lead oxalate formation depending on oxalate excretion.

148 s no correlation between fecal fat and urine oxalate excretion.

149 tive correlation between fecal fat and urine oxalate excretions (r = 0.71, P < .001).

150 dings confirmed the role of mineral-specific oxalate exudation in ectomycorrhizal weathering to disso

151 mide, duloxetine hydrochloride, escitalopram oxalate, fluoxetine hydrochloride, mirtazapine, nortript

152 ting the electrogenic exchange of sulfate or oxalate for chloride or bicarbonate and electroneutral c

153 te acetylhydrolase gene in H915-1 eliminated oxalate formation but neither influence on pH decrease n

154 formation reaction commonly anticipated for oxalate formation by reductive coupling of CO2 on low-va

155 ing on organic phosphate hydrolysis and lead oxalate formation depending on oxalate excretion.

156 Oxalate forms mononuclear bidentate ligand with surface

157 ent oxalate oxidoreductase (OOR) metabolizes oxalate, generating two molecules of CO2 and two low-pot

158 were defined as fecal fat >7 g/day and urine oxalate >40 mg/day.

159 e coupled in a similar fashion through their oxalate half esters.

160 he citrate transporter NaDC-1 in citrate and oxalate homeostasis.

161 ophiles such as ethyl glyoxalate and diethyl oxalate in aqueous medium leads to the formation of benz

162 These results suggest that the dominance of oxalate in diacid concentrations measured in ambient aer

163 Soluble oxalate in foods is major concern for kidney stone forme

164 addition to the well-characterized C2O4(2-) oxalate in Li2C2O4 viable covalent CO2-based nets emerge

165 .5 with 50 mM oxalate and inclusion of 25 mM oxalate in pH 3.0 minimal challenge medium separately co

166 The presence of iron oxalate in seed aerosol is found to inhibit aerosol grow

167 ted in increased survival over adaptation or oxalate in the challenge medium alone.

168 ading to the deposition of insoluble calcium oxalate in the kidney.

169 This research highlights the importance of oxalate in vanadinite bioweathering and suggests a gener

170 Thus, oxalate increases the relative reactivity ratio of bioti

171 umulation of weathered calcium with secreted oxalate, increasing significantly in sequence: quartz, g

172 a different therapeutic approach to calcium oxalate-induced injury.

173 te defects in growth, stress resistance, and oxalate/insecticidal compound production, only a small d

174 Before and after RYGB, high oxalate intake contributed to the severity of hyperoxalu

175 redox coupling of tert-alkyl N-phthalimidoyl oxalate intermediates with electron-deficient alkenes is

176 ed activity of oxalate oxidase that converts oxalate into CO2 Similar results were also observed unde

177 from air and fixes it into oxalate, with the oxalate ion bridging between two copper atoms.

178 The bound oxalate ion is released as oxalic acid on treatment with

179 ysis demonstrated that Zr(IV) interacts with oxalate ions, and the fluoride adsorption mechanism is l

180 e of the (13)C(2) pair in [1-(18)O,(13)C(2)]-oxalate is 2-3 times longer than the spin-lattice relaxa

181 bon radicals from tert-alkyl N-phthalimidoyl oxalates is proposed that is based on earlier pioneering

182 ng of which occurs in the hereditary calcium oxalate kidney stone disease primary hyperoxaluria type

183 or inhibiting the crystallisation of calcium oxalate kidney stones in susceptible individuals.

184 1) in two unrelated individuals with calcium oxalate kidney stones.

185 (AGT), which allows glyoxylate oxidation to oxalate leading to the deposition of insoluble calcium o

186 ecretion, Cftr(-/-) mice had serum and urine oxalate levels 2.5-fold greater than those of wild-type

187 DECA and pyridoxine was additive in reducing oxalate levels.

188 bers and if PEF treatment could reduce tuber oxalate levels.

189 ed by Mn(II) and Cr(III) ions linked through oxalate ligands and a layer of [Fe(sal2-trien)](+) compl

190 ilibrated with aqueous solutions of acetate, oxalate, malonate, or citrate at 50 degrees C and 90 bar

191 The oxalate-minus mutants accumulated fumaric acid, produced

192 Here, we generated oxalate-minus mutants of S. sclerotiorum using two indep

193 The oxalate-minus mutants retained pathogenicity on plants,

194 ng the host tissue enhanced virulence of the oxalate-minus mutants, whereas supplementing with oxalat

195 to (13)C(2) singlet states in a symmetrical oxalate molecule by substituting one or more (16)O nucle

196 catalyzes the Mn-dependent conversion of the oxalate monoanion into CO2 and formate.

197 catalyzes the Mn-dependent conversion of the oxalate monoanion into CO2 and formate.

198 Here we examine this phenomenon in calcium oxalate monohydrate (COM) crystallization, a model syste

199 e proteins in the crystallization of calcium oxalate monohydrate (COM), the most prominent constituen

200 Some calcium oxalate monohydrate (whewellite) was formed with natural

201 Here we report that crystals of calcium oxalate, monosodium urate, calcium pyrophosphate dihydra

202 This investigation shows the oxalate motif is maintained when the concentration of li

203 eased incidence of hyperoxaluria and calcium oxalate nephrolithiasis.

204 We report two cases of renal allograft oxalate nephropathy in patients with remote histories of

205 ases in the transplant population, allograft oxalate nephropathy is likely to be an increasing cause

206 cation of bariatric surgery, with consequent oxalate nephropathy leading to chronic kidney disease an

207 lay progressive forms of nephrocalcinosis in oxalate nephropathy, such as primary hyperoxaluria.

208 in renal pathology are nephrocalcinosis and oxalate nephropathy.

209 re required to recognize and treat allograft oxalate nephropathy.

210 urgery, renal allografts are also at risk of oxalate nephropathy.

211 les as a fundamental initiating mechanism of oxalate nephropathy.

212 [Fe(sal2-trien)](+) complex cation into a 2D oxalate network in the presence of different solvents re

213 HBr3, or CH2Br2) intercalated between the 2D oxalate network.

214 could potentially aid the development of low oxalate oca-based foods.

215 Here we investigate effects of acetate and oxalate on alkali feldspar-brine interactions in a simul

216 gree of Al-Si order may affect the effect of oxalate on feldspar dissolution: a promotion of ~500% in

217 the effects of organic ligands (acetate and oxalate) on biotite dissolution and surface morphologica

218 eatment randomization to either escitalopram oxalate or cognitive behavior therapy for 12 weeks.

219 In the presence of both DFOB and oxalate, oxalate acted synergistically with DFOB to incr

220 Several forms of InsP(n) in the oxalate-oxalic acid extracted sediment were identified.

221 )) in sediment samples was tested, utilizing oxalate-oxalic acid extraction followed by determination

222 Tomato waste was treated with ammonium oxalate/oxalic acid by conventional extraction (CE), und

223 l decomposition and by increased activity of oxalate oxidase that converts oxalate into CO2 Similar r

224 the acidic conditions that are required for oxalate oxidase to function.

225 Combining an enzyme, oxalate oxidase, and an organic oxidation catalyst, 4-am

226 Thiamine pyrophosphate (TPP)-dependent oxalate oxidoreductase (OOR) metabolizes oxalate, genera

227 Cl), only produced by P. javanicus, and lead oxalate (PbC2O4), produced by A. niger and P. javanicus.

228 cium than other GLVs, because of low fibres, oxalate, phytate and tannin content.

229 toxic substances (nitrate, nitrite, cyanide, oxalate, phytate, and trypsin inhibitor) in tubers of Je

230 ntent of calcium absorption inhibitors, like oxalate, phytate, tannin and dietary fibres, and evaluat

231 s using co-precipitation of metal salts into oxalate precursors and subsequent thermal decomposition.

232 k in the study region due to the ubiquity of oxalate precursors, clouds, and metal emissions from shi

233 Alkyl oxalates, prepared from their corresponding alcohols, ar

234 ether calcium would combine with the soluble oxalate present in the spinach.

235 Oxalate-producing plants accumulate calcium oxalate crys

236 n UV-induced mutants that concomitantly lost oxalate production and pathogenicity.

237 demonstrate the ability of ALN-GO1 to reduce oxalate production in preclinical models of PH1 across m

238 ency in this enzyme leads to abnormally high oxalate production resulting in calcium oxalate crystal

239 hase mechanistic relationships leading up to oxalate production.

240 5 and 14.13 percent in phytin phosphorus and oxalate respectively.

241 Net intestinal absorption of dietary oxalate results from passive paracellular oxalate absorp

242 y of Al-O-Si linkages due to the presence of oxalate results in the promotion of both Al and Si relea

243 When fed an oxalate-rich diet, wild-type mice developed progressive

244 loropurine provided crystalline 2AMTA as the oxalate salt.

245 to test the hypothesis that SLC26A6-mediated oxalate secretion is defective in cystic fibrosis.

246 We conclude that defective intestinal oxalate secretion mediated by SLC26A6 may contribute to

247 e exhibited significantly less transcellular oxalate secretion than intestinal tissue of wild-type mi

248 ation with the profound defect in intestinal oxalate secretion, Cftr(-/-) mice had serum and urine ox

249 with colonic epithelium and induces colonic oxalate secretion, thereby reducing urinary oxalate excr

250 cretion and stimulated (>42%) distal colonic oxalate secretion.

251 dentify the derived factors inducing colonic oxalate secretion.

252 For the first time, we show that oxalate selectively attacks edge surface sites and enhan

253 e net extent of feldspar's dissolution, with oxalate showing a more prominent effect than acetate.

254 on oxalato complexes is likely a significant oxalate sink in the study region due to the ubiquity of

255 tions in order to investigate the effects on oxalate solubility.

256 nsistent 100% recovery, at pH 4, using 0.1 M oxalate solution as an effective stripping agent.

257 The adsorbent is fully regenerable, using oxalate solution.

258 contribute to the increased risk for calcium oxalate stone formation observed in patients with cystic

259 aluria and hypocitraturia can trigger Ca(2+)-oxalate stone formation, even in the absence of hypercal

260 te and citrate levels and may control Ca(2+)-oxalate stone formation.

261 However, oxalate strongly enhanced biotite dissolution and induce

262 The combination of oxalate adaptation and oxalate supplementation in the challenge medium resulted

263 colate oxidase, to deplete the substrate for oxalate synthesis.

264 The phenols, phytate, oxalate, tannin and alkaloid profiles of the flours were

265 grandiflora had a very high content of total oxalates, tannins and dietary fibers, which reduced calc

266 ormation of a quinoxaline-di-N-oxide/Mn(III)/oxalate ternary complex in which Mn(III) functioned as t

267 ding affinity are due to the formation of Ni-oxalate ternary surface complexes.

268 first used to predict that NPs conjugated to oxalate Tf will exhibit a higher degree of cellular asso

269 efficacy of NPs conjugated to our engineered oxalate Tf.

270 Upon reaction with DFOB or DFOB + oxalate, the remaining solids became enriched in Cr rela

271 FDA-approved drugs tramadol and escitalopram oxalate, they release or uptake serotonin in a dose- and

272 Oxalate thus largely enhances Ni mobility, thereby incre

273 f ligands facilitated electron transfer from oxalate to carbadox.

274 Seeds of mutants accumulated oxalate to levels threefold higher than the wild type, r

275 so significantly restricted the induction of oxalate transport by CM.

276 rmigenes-derived bioactive factors stimulate oxalate transport in intestinal cells through mechanisms

277 fonic acid completely blocked the CM-induced oxalate transport.

278 Knockdown of the oxalate transporter SLC26A6 also significantly restricte

279 re, we examined the relationship between the oxalate transporter SLC26A6 and the citrate transporter

280 alate back secretion mediated by the SLC26A6 oxalate transporter.

281 The formation of manganese oxalate trihydrate was detected after growth of S. himan

282 a copper complex converts carbon dioxide to oxalate under mild conditions.

283 m, O. formigenes CM significantly stimulated oxalate uptake (>2.4-fold), whereas CM from Lactobacillu

284 ture conditioned medium (CM) on apical (14)C-oxalate uptake by human intestinal Caco-2-BBE cells.

285 rocess for conversion of carbon dioxide into oxalate using a binuclear copper complex and a mild redu

286 Oxalate-vanadium complexes markedly reduced the vanadini

287 In the complex, the C-C bond in oxalate was cleaved to create CO(2)(-*) radicals, follow

288 In addition, calcium oxalate was formed from the calcium present in the miner

289 Lead oxalate was precipitated by Aspergillus niger during bio

290 In spinach, [(14)C]oxalate was the major product of [(14)C]ascorbate degrad

291 Acetate and oxalate were detected as transient intermediates during

292 pH 7 samples behaved like ammonium oxalate, which has a vapor pressure of approximately 10(

293 lated to give the tert-alkyl N-phthalimidoyl oxalate, which in the presence of visible-light, catalyt

294 scaled with the absorption spectrum of iron oxalate, which suggests that the reaction is at least in

295 h is known to contain significant amounts of oxalates, which are toxic and, if consumed regularly, ca

296 dine (a highly disordered feldspar) owing to oxalate, while the corresponding increase for albite (a

297 dical coupling of tert-alkyl N-phthalimidoyl oxalates with electron-deficient alkenes is terminated b

298 dditional sink for tartronate, pyruvate, and oxalate, with a complex photolysis contribution to overa

299 th carbon dioxide from air and fixes it into oxalate, with the oxalate ion bridging between two coppe

300 CO2(*-) produced can either dimerize to form oxalate within the nanogap between SECM tip and substrat