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1 nti-glomerular basement membrane disease, 2; oxalosis, 2; and miscellaneous, 2.
2                      Among the patients with oxalosis, 56 patients had a liver transplant followed by
3 kidney and many other tissues, with systemic oxalosis and ESRD being a common outcome.
4 nal failure early in life, advanced systemic oxalosis, and a formidable mortality rate.
5 mias, familial hypercholesterolemia, primary oxalosis, and factor IX deficiency, among others, might
6 nsplant recipients from 1988 to 1998 who had oxalosis as their primary diagnosis for their ESRD.
7                     Indications for LTx were oxalosis (four), congenital hepatic fibrosis (two), cyst
8                     Indications for KTx were oxalosis (four), drug-induced (four), polycystic kidney
9  although concern exists about recurrence of oxalosis in the transplanted kidney.
10 survival in renal transplant recipients with oxalosis is similar to other transplant recipients with
11                                              Oxalosis, or calcium oxalate deposition in the tissues,
12                                              Oxalosis patients receiving a KTA had a significantly wo
13  death-censored graft survival compared with oxalosis patients who receive a cadaveric or living-dono
14 indicating poor renal allograft survival for oxalosis patients who receive a renal transplant alone.
15 -censored graft survival (76%) compared with oxalosis patients who received a KTA (47.9%, P<0.001) an
16                                 In addition, oxalosis patients who received a living-donor KTA had si
17  death-censored graft survival compared with oxalosis patients who received a LKTx (22% vs. 64%, P<0.
18                                 In contrast, oxalosis patients who received a LKTx had a significantl
19 Unadjusted death-censored graft survival for oxalosis patients with a cadaveric or living-donor KTA o
20                                          For oxalosis patients with minor enzyme deficiencies, renal
21               In this study, a case of renal oxalosis probably secondary to excessive parenteral vita
22                         Patient survival for oxalosis recipients with a KTA or a LKTx was not signifi
23                                    Secondary oxalosis represents a possible cause of delayed recovery
24 raft survival for transplant recipients with oxalosis to a reference group with ESRD secondary to glo
25                                Patients with oxalosis who receive a LKTx have superior death-censored
26 th-censored graft survival for patients with oxalosis who received a LKTx or a KTA.
27 leading to renal failure, followed by tissue oxalosis with life-threatening complications.

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