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1  the compounds' ability to rescue cells from oxidative stress induced cell death.
2 nds reported to protect human RPE cells from oxidative stress-induced cell death.
3 ant SOD1 (G93A) both conferred resistance to oxidative stress-induced cell death.
4 nce of CypD protects neurons from Abeta- and oxidative stress-induced cell death.
5 e to see whether they would inhibit light or oxidative stress-induced cell death.
6 ed by hydrogen peroxide, implicating CypD in oxidative stress-induced cell death.
7 ne peroxidase 4 (Gpx4) is a key regulator of oxidative stress-induced cell death.
8 are largely protected from Ca2+-overload and oxidative stress-induced cell death.
9 ncovers a novel signaling pathway regulating oxidative stress-induced cell death.
10  PARP-1 null fibroblasts were protected from oxidative stress-induced cell death.
11 dh2 increases NADPH levels and protects from oxidative stress-induced cell death.
12 silencing in lens cells results in increased oxidative-stress-induced cell death.
13 selective cation channel that is involved in oxidative stress-induced cell death and inflammation pro
14     These results indicate that NAC restores oxidative stress-induced cell death and severe functiona
15 indicate that p53 plays a functional role in oxidative stress-induced cell death and supports the pos
16 hat the protective effects of apomorphine on oxidative stress-induced cell death are, at least in par
17     Here, we show that DJ-1 protects against oxidative stress-induced cell death, but that its relati
18 expression could protect cardiomyocytes from oxidative stress-induced cell death by reducing reactive
19  insights into a functional role of MKP-1 in oxidative stress-induced cell death by regulating ERK1/2
20 lls, was tested using three model systems of oxidative stress-induced cell death: glutathione (GSH) d
21 man neuronal cells conferred protection from oxidative stress-induced cell death in a sirtuin-depende
22  that heat shock protein 27 (Hsp27) prevents oxidative stress-induced cell death in cerebellar granul
23 over, overexpression of ubiquilin suppressed oxidative stress-induced cell death in HeLa cell lines s
24 strate that the protein kinase MST1 mediates oxidative-stress-induced cell death in primary mammalian
25 he neuroprotective mechanism of EGCG against oxidative stress-induced cell death includes stimulation
26 terns of these genes, coupled with increased oxidative-stress-induced cell death on their deletion pr
27  We propose a model in which protection from oxidative stress-induced cell death requires the tyrosin
28 le TG isoforms, not only TG2, participate in oxidative stress-induced cell death signaling; and that
29 express HO-1 or NQO-1 were more resistant to oxidative stress-induced cell death than control cells.
30  (GSTp)-mediated cellular protection against oxidative stress-induced cell death, the effect of GSTp
31 o antagonizes YAP-FoxO1, leading to enhanced oxidative stress-induced cell death through downregulati
32 ival phosphatase preventing both thermal and oxidative stress-induced cell death, whereas studies in

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