ライフサイエンスコーパス: oxo

1 yclic ring or ring E, is equipped with 13(1)-oxo and 13(2)-carbomethoxy substituents.

2 derivative gave either the corresponding (1-oxo-1H-isochromen-3-yl)quinolinium derivative or the mes

3 a,omega-dicarboxylic, omega-hydroxy, omega-1-oxo, omega-1-methyl, 2-methyl, 2-methyl-2-enolic and 2,3

4 l shRNAs or blocker 1-[4-[(2,3,3-trichloro-1-oxo-2-propen-1-yl)amino]phenyl]-5-(trifluoromethyl)-1H-p

5 equences related to the observed elevated 12-oxo phytodienoic acid (OPDA), which was 9-fold higher in

6 howed higher levels of JA, JA-isoleucine, 12-oxo-phytodienoic acid, and arabidopsides in dgd1 mutants

7 the llb mutant including the JA precursor 12-oxo-phytodienoic acid.

8 Both 22-OH-MaR1 and 14-oxo-MaR1 incubated with human primary macrophages gave d

9 ture elucidation gave 22-hydroxy-MaR1 and 14-oxo-MaR1.

10 MaR1 further metabolites, 15-oxo-LXA4 and 14-oxo-MaR1.

11 rahydro-16-hydroxy-15-(methyl pentanoate)-14-oxo-2H-pyran-13-yl)-9-methyl-but-11-enyl benzoate (1), i

12 22-OH-MaR1 and approximately 25% at 1 pM 14-oxo-MaR1, whereas 14-oxo-MaR1 was less active than MaR1

13 , whereas the main macrophage product was 14-oxo-MaR1.

14 ximately 25% at 1 pM 14-oxo-MaR1, whereas 14-oxo-MaR1 was less active than MaR1 at higher concentrati

15 15-epi-LXA4 and MaR1 further metabolites, 15-oxo-LXA4 and 14-oxo-MaR1.

16 natural product class and include (1R,10S)-2-oxo-3,4-dehydroneomajucin, (2S)-hydroxy-3,4-dehydroneoma

17 fenolide, (-)-jiadifenin, and (-)-(1R,10S)-2-oxo-3,4-dehydroxyneomajucin (ODNM) along the synthetic p

18 mutations as well as treatment with 2,2'-(2-oxo-1,3-propanediyl)bis[(phenylmethoxy)carbonyl]-l-leucy

19 Interaction of 4,5-dimethyl-2-(2-oxo-1,2-diphenyl)ethoxy-1,3,2-dioxaphospholane, bearing

20 yclic systems is developed by employing 3-(2-oxo-2-arylethylidene)oxindoles and 1,4-benzoxazinone as

21 tion, and mechanism of action of phenyl 4-(2-oxo-3-alkylimidazolidin-1-yl)benzenesulfonates (PAIB-SOs

22 are N-dealkylated into cytotoxic phenyl 4-(2-oxo-3-imidazolidin-1-yl)benzenesulfonates (PIB-SOs) in C

23 e addition of enolate derived from acyclic 2-oxo-butanoate 10 to 2H-azirine phosphine oxide 1 led to

24 A general six-step approach to alkyl 2-oxo-2,3,6,7-tetrahydro-1H-1,3-diazepine-5-carboxylates a

25 c ring-opening copolymerization of 2-alkyl-2-oxo-1,3,2-dioxaphospholanes provided functional polymers

26 that is activated by redox mechanisms and 2-oxo acids.

27 tionally regulated by redox mechanisms and 2-oxo acids.

28 ith AOX1A activity being more increased by 2-oxo acids than that of AOX1C and AOX1D.

29 idine TNA triphosphate analogue (1,3-diaza-2-oxo-phenothiazine, tCfTP) that maintains Watson-Crick ba

30 roach using alpha-(2,4-dimethylphenylethyl-2-oxo)-indole-3-acetic acid (auxinole), alpha-(phenylethyl

31 tion of cyclic enolates derived from ethyl 2-oxo-cyclopentanecarboxylate 2 to phosphorated 2H-azirine

32 Previously, we explored 1-hydroxy-2-oxo-1,2-dihydro-1,8-naphthyridine-3-carboxamides as an I

33 dues, CysI and CysII, are both involved in 2-oxo acid activation, with AOX1A activity being more incr

34 4'-bromophenyl)aminocyclohex-3-en-6-methyl-2-oxo-1-oate (E139), an anticonvulsant enaminone, has anti

35 9-bis(4-(1,1-dicyanomethylene)-3-methylene-2-oxo-cyclopenta[b]thiophen)-5,5,11 ,11-tetrakis(4-hexylph

36 2-Nitro-N-alkyl-N-(2-oxo-2-phenylethyl)benzenesulfonamide compounds are known

37 azomethine imines, 7-methyl- and 7-phenyl-2-oxo-Delta(7)-hexahydropyrazolo[1,5-a]pyridin-8-ium-1-ide

38 acetic acid (auxinole), alpha-(phenylethyl-2-oxo)-indole-3-acetic acid (PEO-IAA), and 5-fluoroindole-

39 -substituted phenylacetones to substituted 2-oxo-2H-pyran-3-carbonitriles at room temperature under a

40 known to be involved in regulation by the 2-oxo acids pyruvate and glyoxylate) and propose that this

41 ues (1 and 4) lacking both the N-1 and the 2-oxo substituents were also introduced in lieu of His24.

42 ro-2-fluorophenyl)-1'-(cyclopropylme thyl)-2-oxo-1,2,3',3'a,4',5',6',6'a-octahydro-1'H-spiro[indole-3

43 Iron(II)- and 2-(oxo)-glutarate-dependent oxygenases catalyze diverse oxi

44 yranosyl)oxy]-dammar-24-en-19-al; (3beta)-28-oxo-28-(phenylmethoxy)oleanan-3-yl 2-O-beta-d-galactopyr

45 er 60 min exposure, a fully assembled (mu(3)-oxo)Tris[(mu(2)-peroxo)(mu(2)-glutamato-kappaO:kappaO')]

46 en N-substituted 5-aminopyrazoles 1 and 3-(3-oxo-2-benzofuran-1(3H)-ylidene)pentane-2,4-dione (2).

47 iophe ne-2,7-diyl)bis(methanylylidene))bis(3-oxo-2,3-dihydro-1H-indene-2,1-diylidene))d imalononitril

48 es Hydra to specifically modify long-chain 3-oxo-homoserine lactones into their 3-hydroxy-HSL counter

49 trong electron-withdrawing 2-(5,6-difluoro-3-oxo-2,3-dihydro-1H-inden-1-ylidene)malononitrile to yiel

50 yl))bis(methanylylidene)) bis(5,6-difluoro-3-oxo-2,3-dihydro-1H-indene-2,1-diylidene))dimalononitrile

51 reated N-butyryl-homoserine lactone and n-(3-oxo-dodecanoyl)-homoserine lactone, exhibited marked att

52 ally acyl homo serine lactones, such as N-(3-oxo-dodecanoyl)-l-homoserine lactone (3O-C12-HSL), that

53 l has been developed for the generation of 3-oxo-3-(hetero)arylpropanenitriles via a carbonylative pa

54 The (3-oxo-2-butenyl)triphenylphosphonium zwitterion, a commonl

55 l for a P450, which typically uses an Fe(4+)-oxo intermediate known as compound I for the insertion o

56 e, 7-(2-hydroxypropan-2-yl)-4-[2-methyl-3-(4-oxo-3,4-dihydroquinazolin-3-yl)phenyl]- 9H-carbazole-1-c

57 enals (fumaraldehyde, 4-oxo-2-hexenal, and 4-oxo-2-nonenal) and phenolic compounds (resorcinol and 2-

58 ucts are the 4-oxo-beta-apo-carotenals and 4-oxo-beta-apo-carotenones.

59 ctanoic acid (4-OOA, 10.4 mug bee(-1)) and 4-oxo-decanoic acid (4-ODA, 13.3 mug bee(-1)) at a 0.78 ra

60 The reaction between 4-oxo-2-alkenals (fumaraldehyde, 4-oxo-2-hexenal, and 4-ox

61 avable cross-linker (containing a 1,3-bis-(4-oxo-butyl)-urea group, BuUrBu) generating characteristic

62 well as the kynurenic acid analog 7-chloro-4-oxo-1H-quinoline-2-carboxylic acid (7-chlorokynurenic ac

63 imidinone inhibitor 2-((1-(3-chlorophenyl)-4-oxo-4,5-dihydro-1H-pyrazolo [3,4-d]-pyrimidin-6-yl)thio)

64 MAO-B inhibitors were N-(3'-chlorophenyl)-4-oxo-4H-chromene-3-carboxamide (20) (IC50 = 403 pM) and N

65 no-3-(trifluoromethyl)phenyl)-5,5-dimethyl-4-oxo-2-thioxoimida zolidin-1-yl)-2-fluorophenyl)-N-hydrox

66 no-3-(trifluoromethyl)phenyl)-5,5-dimethyl-4-oxo-2-thioxoimidazolidin -1-yl)-2-fluoro-N-(7-(hydroxyam

67 C50 = 403 pM) and N-(3',4'-dimethylphenyl)-4-oxo-4H-chromene-3-carboxamide (27) (IC50 = 669 pM), acti

68 ies of 5,6-epoxy-beta-ionone and 5,6-epoxy-4-oxo-beta-apo-11-carotenal, no other epoxides were detect

69 The determination of ethyl [4-oxo-8-(3-chlorophenyl)-4,6,7,8-tetrahydroimidazo[2,1-c][

70 n between 4-oxo-2-alkenals (fumaraldehyde, 4-oxo-2-hexenal, and 4-oxo-2-nonenal) and phenolic compoun

71 initiated by 2-pentenal, 2,4-heptadienal, 4-oxo-2-pentenal, 4,5-epoxy-2-heptenal, or 4,5-epoxy-2-dec

72 he reference compound, 1-(4-methoxybenzyl)-4-oxo-1,4-dihydroquinoline-3-carboxylic acid (BQCA) (1), b

73 allosteric modulator, 1-(4-methoxybenzyl)-4-oxo-1,4-dihydroquinoline-3-carboxylic acid, enhanced ace

74 ligands xanomeline and 1-(4-methoxybenzyl)-4-oxo-1,4-dihydroquinoline-3-carboxylic acid.

75 A synthetic route to cis-2-methyl-4-oxo-6-alkylpiperidines has been developed using a 6-endo

76 ide the basis to understand the removal of 4-oxo-2-alkenals by phenolic compounds in foods.

77 with canthaxanthin these products are the 4-oxo-beta-apo-carotenals and 4-oxo-beta-apo-carotenones.

78 The active compounds were 4-oxo-octanoic acid (4-OOA, 10.4 mug bee(-1)) and 4-oxo-de

79 The lead prodrug, isopropyl 6-diazo-5-oxo-2-(((phenyl(pivaloyloxy)methoxy)carbonyl)amino)hexan

80 show that glutaminase inhibitors, 6-Diazo-5-oxo-L-norleucine (DON) or CB-839, hypersensitize cancer

81 The glutamine antagonist 6-diazo-5-oxo-l-norleucine (DON, 1) has shown robust anticancer ef

82 The glutamine antagonist 6-diazo-5-oxo-l-norleucine (DON, 14) attenuates glutamate synthesi

83 argeting glutamine metabolism with 6-diazo-5-oxo-l-norleucine uniformly sensitized MM cell lines and

84 umor effect of a glutamine analog (6-diazo-5-oxo-L-norleucine) as an adjuvant treatment to sensitize

85 w four other amine heterocycles: 6-methyl, 5-oxo-2,3,4,5-tetrahydropyridine (6M5OTP), 2-acetylpyrrole

86 N-[2-methylphenyl]-[4-furan-3-yl]-2-methyl-5-oxo-1,4,5,6,7,8-hexahydro-quinoline -3-carboxamide; MQC)

87 onkey leukocytes synthesize and respond to 5-oxo-ETE and that 230 is a potent antagonist of the OXE r

88 ognizes the C-2-amino group, but not the C-6-oxo group, N-1-hydrogen, or N-7-nitrogen, of GDP for the

89 11-dicarboxy-8-hydroxy-9-methoxy-2-hydroxy-6-oxo-6-phenyl-hexa-2,4-dienoate.

90 ich is transformed into the oncometabolite 6-oxo-cholestan-3beta,5alpha-diol (OCDO) by 11beta-hydroxy

91 '-(morpholinomethyl)-[1,1'-biphenyl]-3-yl)-6-oxo-4 -(trifluoromethyl)-1,6-dihydropyridine-3-carboxami

92 The intermediate 7-oxo-5-enones underwent a highly diastereoselective (dr >

93 8-oxo-7,8-dihydroguanine was incorporated into RNA to unde

94 8-oxo-7,8-dihydroxy-2'-deoxyguanosine (8-oxo-dG) has high

95 as 8-oxo-2'-deoxyguanosine (8-oxo-dG) and 8-oxo-2'-deoxyadenosine (8-oxo-dA) in diseased RPE could p

96 hyl)-2'-deoxyuridine, 2'-deoxyuridine, and 8-oxo-2'-deoxyguanosine.

97 ines are the main DNA oxidation sites, and 8-oxo-7,8-dihydro-2-deoxyguanosine (8-oxodG) is the initia

98 and AMD donors averaged 0.54 and 0.96, and 8-oxo-dA averaged 0.04 and 0.05 adducts per 10(6) bases, r

99 s and AMD donors averaged 170 and 188, and 8-oxo-dA averaged 11 and 17 adducts per 10(6) bases, respe

100 for simultaneous analysis of 8-oxo-dG and 8-oxo-dA in human retinal DNA.

101 nt a divergent synthesis of pyrimidine and 8-oxo-purine nucleotides starting from a common prebiotic

102 tional relationship between pyrimidine and 8-oxo-purine nucleotides suggests that 8-oxo-purine ribonu

103 asurement of oxidative DNA lesions such as 8-oxo-2'-deoxyguanosine (8-oxo-dG) and 8-oxo-2'-deoxyadeno

104 modified templates or DNA lesions, such as 8-oxo-2'-deoxyguanosine or cyclobutane pyrimidine dimer, e

105 ve damage to the genome can yield the base 8-oxo-7,8-dihydroguanine (OG).

106 ne of the two G residues is substituted by 8-oxo-7,8-dihydroguanine, a modification that is suggested

107 imately 2-fold higher than that induced by 8-oxo-dG adduct.

108 s and kinetic data that fully characterize 8-oxo-dG bypass by Pol lambda.

109 lar nucleotide pool of oxidatively damaged 8-oxo-dGTP, preventing mutagenesis by this nucleotide.

110 ne (8-oxo-dG) and 8-oxo-2'-deoxyadenosine (8-oxo-dA) in diseased RPE could provide important insights

111 A lesions such as 8-oxo-2'-deoxyguanosine (8-oxo-dG) and 8-oxo-2'-deoxyadenosine (8-oxo-dA) in diseas

112 8-oxo-7,8-dihydroxy-2'-deoxyguanosine (8-oxo-dG) has high mutagenic potential as it is prone to m

113 n species (ROS), primarily as 7, 8-dihydro-8-oxo-2'-deoxyguanosine (8-oxoG), which is repaired by 8-o

114 amaged DNA and the DNA lesions 7,8-dihydro-8-oxo-2'-deoxyguanosine and cyclobutane pyrimidine dimer b

115 with the template base (dG) or 7,8-dihydro-8-oxo-2'-deoxyguanosine with a significant propeller twist

116 e pro-mutagenic behavior of 8-oxo-guanine (8-oxo-G) by quantifying the ability of high-fidelity and s

117 ymerase to perform TLS with 8-oxo-guanine (8-oxo-G), a highly pro-mutagenic DNA lesion formed by reac

118 tT, a nucleotide sanitizer that hydrolyzes 8-oxo-dGTP to the monophosphate, or that lack MutM and Mut

119 erythroid-related factor 2, and increased 8-oxo-7,8-dihydro-2'-deoxyguanosine, a marker of DNA damag

120 osylases that process base pairs involving 8-oxo-dGTP.

121 with XBD173 (N-benzyl-N-ethyl-2-(7-methyl-8-oxo-2-phenylpurin-9-yl)acetamide) to determine nondispla

122 nerated here for replicating the miscoding 8-oxo-G are compared to those published for the replicatio

123 avenging the mutagenic oxidised nucleotide 8-oxo-dGTP.

124 The oxidized nucleotide, 8-oxo-7,8-dihydro-2-deoxyguanosine (8-oxoG), is one of the

125 th the efficiency and error-free bypass of 8-oxo-7,8-dihydrodeoxyguanosine (8-oxoG) lesions by PrimPo

126 n addition to increased urine secretion of 8-oxo-deoxyguanosine.

127 ometry method for simultaneous analysis of 8-oxo-dG and 8-oxo-dA in human retinal DNA.

128 In nuclear DNA, the levels of 8-oxo-dG in controls and AMD donors averaged 0.54 and 0.96

129 In mtDNA, the levels of 8-oxo-dG in controls and AMD donors averaged 170 and 188,

130 rt evaluates the pro-mutagenic behavior of 8-oxo-guanine (8-oxo-G) by quantifying the ability of high

131 nic exposure to ATZ increases the level of 8-oxo-guanine in the nucleus of meiotic cells, reflecting

132 LRMT also shows a high C-->A error rate on 8-oxo-dG templates ( approximately 10(-4)).

133 pproach was used to detect a uracil (U) or 8-oxo-7,8-dihydroguanine (OG) in codon 12 of the KRAS gene

134 ions by excising adenine from promutagenic 8-oxo-7,8-dihydroguanine (OG):A mismatches.

135 esis catalyzed by pol eta when replicating 8-oxo-G.

136 ficiency and low fidelity when replicating 8-oxo-G.

137 intain genomic integrity, post-replicative 8-oxo-dG:dA mispairs are removed through DNA polymerase la

138 and 8-oxo-purine nucleotides suggests that 8-oxo-purine ribonucleotides may have played a key role in

139 al incompatibility by recognizing that the 8-oxo-purines share an underlying generational parity with

140 In human DKD, increased urine 8-oxo-deoxyguanosine was associated with rapid DKD progres

141 made between the incoming nucleotide with 8-oxo-G.

142 idelity DNA polymerase to perform TLS with 8-oxo-guanine (8-oxo-G), a highly pro-mutagenic DNA lesion

143 hat ROS-mediated oxidation of DNA to yield 8-oxo-7,8-dihydroguanine (OG) in gene promoters is a signa

144 NA by reactive oxygen species (ROS) yields 8-oxo-7,8-dihydroguanosine (8-oxodG) as primary oxidation

145 s, the carbonyl double bonds break affording oxo-radicals that can be stabilized within the conjugate

146 tially reversed within tens of seconds after oxo-M washout, and not relieved by a strong depolarizati

147 or transformation product (TP) of MIA, alpha-oxo MIA was likely formed via a two-step oxidation react

148 s fragmentation into a highly reactive alpha-oxo gold carbene intermediate.

149 finylnitrene RS(O)N, a highly reactive alpha-oxo nitrene, has been rarely investigated.

150 an acid-catalyzed Nef-type reaction to alpha-oxo-carbonyls.

151 in m-xylene solvent to form transient alpha-oxo-ortho-quinodimethanes or "ortho-quinoid ketene methi

152 ich the protein might dictate an alternative oxo position.

153 high-energy intermediates and catalyzing an oxo transfer, including an enantioselective 1,2-hydride

154 from the modified alumina while donating an oxo ligand to the support.

155 Whether the green intermediate is an oxo atom donor adduct, Fe-O horizontal lineI-Ph, or an F

156 Here we report an oxo-vanadium(V) aminotriphenolate complex that was found

157 the course of the reaction indicated that an oxo-Mn(V) species is responsible for hydrogen-atom abstr

158 tent with dicarbonyls, hydroxycarbonyls, and oxo-carboxylic acids.

159 ing a series of NU-1000-supported bimetallic-oxo,hydroxo,aqua clusters.

160 deprotonation/electrophilic trapping of both oxo functions, and the catalytic behavior was studied in

161 pling between an oxyl radical and a bridging oxo.

162 e voltage dependency under the inhibition by oxo-M.

163 C-H bond activation mediated by oxo-iron (IV) species represents the key step of many he

164 350 nm irradiation of a cyclopropenone caged oxo-dibenzocyclooctyne (photo-ODIBO) biotin yields forma

165 The rhenium(V) oxo complex oxo(triphenylphosphine) (bis(3,5-di-tert-butyl-2-phenoxo

166 Cu oxo clusters stabilized in NU-1000 provide an active, fi

167 lysts, including intermediacy of terminal Cu oxo/oxyl species, are prevalent in the literature; howev

168 The Cu-loaded MOF contained Cu-oxo clusters of a few Cu atoms.

169 II) acetate, and pyridine to form the cubane oxo cluster MnCo3O4(OAc)5py3 (OAc = acetate, py = pyridi

170 state as an exchangeable fully deprotonated oxo bridge [Perez Navarro, M.; et al.

171 ith IL-4 followed by ADMA showed exaggerated oxo-nitrative stress and potent induction of the cellula

172 ts the formation of terminal and reactive Fe-oxo intermediates.

173 ggest that similar protonated high-valent Fe-oxo species may occur in the active sites of proteins.

174 e substrates in front of the reactive ferryl-oxo species.

175 h IL-4 followed by ADMA and investigated for oxo-nitrative stress and resultant mitochondrial toxicit

176 Furthermore, oxo-M-induced suppression of Na(+) currents remains unch

177 cyclic skeleton from an omega-isocyano-gamma-oxo-aldehyde via a sequence of an unprecedented C-C bond

178 Thus heterobimetallic oxo intermediates provide a promising route for enhancin

179 be linearly related to covalency, and M(III) oxo inductive effects on Co(IV) oxo bonding can tune the

180 ent reaction pathways (Fe(IV)-oxo vs Fe(III)-oxo) might be responsible for the observed arene hydroxy

181 pon reduction of the oxyanions, an iron(III)-oxo is formed, which in the presence of protons and elec

182 is a potential proton donor to reactive iron-oxo species during substrate decarboxylation.

183 nctionalized by placing it close to the iron-oxo haem complex.

184 provide direct evidence of high-valent iron-oxo intermediates as the product of the first hole-trans

185 suggest the formation of a high valent iron-oxo species as the catalytic intermediate.

186 Along with an isoelectronic oxo, we quantify the electronic structure of this 5f(1)

187 d Co diketiminate complexes affords isolable oxo species with M2 O2 "diamond" cores, including the fi

188 confirming the nucleophilic character of its oxo ligand.

189 , and M(III) oxo inductive effects on Co(IV) oxo bonding can tune the covalency of high-valent sites

190 Here we describe a series of Mn(IV) -oxo complexes with N5 pentadentate ligands that modulate

191 For these Mn(IV) -oxo complexes, the rate enhancements are correlated with

192 While many synthetic Mn(IV) -oxo species are mild oxidants, other members of this cla

193 rPP-n (n = 1, 2), featuring infinite Zr(IV) -oxo chains linked via polyphenolate groups on four perip

194 ot formed and NaClO oxidizes 3 to an iron(IV)oxo derivative.

195 e resulted in the formation of a rare Ce(IV)-oxo complex, that was stabilized by a supramolecular, te

196 Terminal cobalt(IV)-oxo (Co(IV)-O) species have been implicated as key inter

197 dies indicate that protonation of the Fe(IV)-oxo complex most likely occurs on the tripodal ligand, w

198 h-spin Fe(III)-superoxo and high-spin Fe(IV)-oxo complexes, respectively, in agreement with published

199 cyclopenin formation uses a high-spin Fe(IV)-oxo intermediate to carry out epoxidation.

200 The same protonated Fe(IV)-oxo species can be prepared via oxidation, suggesting th

201 reversible protonation of a synthetic Fe(IV)-oxo species containing a tris-urea tripodal ligand.

202 ing oxo<-->hydroxo tautomerism of the Fe(IV)-oxo species in the non-heme iron enzyme catalysis.

203 1 and 5, different reaction pathways (Fe(IV)-oxo vs Fe(III)-oxo) might be responsible for the observe

204 experiments identified a transient iron(IV)-oxo (ferryl) complex.

205 traction of hydrogen from carbon by iron(IV)-oxo (ferryl) complexes.

206 = CF3 (4)) promoted by the nonheme iron(IV)-oxo complex [(N4Py)Fe(IV) horizontal lineO](2+) occurs b

207 retching frequencies of high-valent iron(IV)-oxo complexes [(L)Fe(O)(X)](2+/+) (L = TMC, N4Py, PyTACN

208 yl sulfides promoted by the nonheme iron(IV)-oxo complexes [(N4Py)Fe(IV) horizontal lineO](2+) and [(

209 and compare reactivities of various iron(IV)-oxo complexes generated as dications or monocations (bea

210 Manganese(IV)-oxo complexes are often invoked as intermediates in Mn-c

211 activity in the presence of (*)OH and Mn(IV)-oxo species by channeling these oxidants toward the synt

212 ccinate ferryl precursor, and a vanadium(IV)-oxo mimic of the ferryl intermediate in the l-arginine 3

213 gations of an exclusive tetracarbene ligated oxo-iron(IV) complex, [L(NHC)Fe(IV)(O)(MeCN)](2+) (1).

214 U(III) salts forms highly symmetric, linear, oxo-bridged trinuclear U(V) /Ln(III) /U(V) , Ln(III) /U(

215 om transfer from the Ru(IV) horizontal lineO oxo species to the uncoordinated nitrogen of the NPM lig

216 e for the spin state reactivity in manganese-oxo corrolazine complexes.

217 nese cubane cluster with a pendant manganese-oxo moiety.

218 es use of light to generate a putative metal oxo intermediate.

219 wever, intermediacy of late transition metal oxo species would be remarkable given the high d-electro

220 itu transformation of the complex to a metal-oxo species, which is capable of catalyzing subsequent o

221 gen, metal-superoxo, metal-peroxo, and metal-oxo adducts.

222 Inorganic aqueous metal-oxo clusters are both functional "molecular metal oxides

223 faces with a wide variety of catalytic metal-oxo species.

224 zyme by directed evolution to catalyze metal-oxo-mediated anti-Markovnikov oxidation of styrenes with

225 c-direction to yield heterobimetallic metal-oxo nanowires.

226 Activation of O-H bonds by inorganic metal-oxo complexes has been documented, but no cognate enzyma

227 cited higher spin state in a number of metal-oxo species can provide a lower energy barrier for oxida

228 esolve the precise atomic structure of metal-oxo species deposited in the MOF NU-1000 through ALD.

229 rized metal-superoxo, metal-peroxo, or metal-oxo species, but the instances of biomimetic Fe/Mn compl

230 control the reactivity of high-valent metal-oxo species is critical to both an understanding of meta

231 eroxo, -(hydro)peroxo, and high-valent metal-oxo species, are the basis for the various biological fu

232 ific bi-manganese cluster, likely a di-micro-oxo bridged pair of Mn(III) ions, as an assembly interme

233 The results show that isolated Mo-oxo species present after calcination are converted by C

234 The well-defined silica-supported molybdenum oxo alkyl species ( identical withSiO-)MoO(CH2(t)Bu)3 wa

235 d hexameric topologies dominate monoalkyltin-oxo cluster chemistry.

236 trans-peroxo, (T)P) and dicopper(III)-bis(mu-oxo) (L2Cu(III)2(O(2-))2: O) can be controlled through l

237 ically characterized heterobimetallic bis(mu-oxo) complex of two transition metals.

238 1e(-)) attack of a partially quenched bis(mu-oxo)-diiron(IV) intermediate (Q' pathway).

239 (a) electrophilic (2e(-)) attack by a bis(mu-oxo)-diiron(IV) species (Q pathway); (b) electrophilic (

240 most preferred, but the corresponding bis(mu-oxo)-diiron(IV) species is significantly destabilized an

241 gest that the intermediate possesses a di-mu-oxo diamond core structure with a terminal hydroxide lig

242 ) dimer selectively generates either a di-mu-oxo or mu-oxo-mu-hydroxo Mn(IV) complex.

243 s of structurally characterized dinuclear mu-oxo Ir(IV,IV) compounds without metal-carbon bonds.

244 mplex, the rare bimetallic complex Ir(IV)(mu-oxo)2Ir(IV), can react with chlorine to release O2 by th

245 n of our recently reported Ir(IV,IV) mono-mu-oxo dimers results in the formation of fully characteriz

246 The similarity of these mono-mu-oxo dimers to our Ir "blue solution" water-oxidation cat

247 o a series of well-defined Ir(IV,IV) mono-mu-oxo dimers, containing the closely related L2Ir-O-IrL2 u

248 due to the their heavy masses, while one mu-oxo bridge (O5) moves significantly.

249 lectively generates either a di-mu-oxo or mu-oxo-mu-hydroxo Mn(IV) complex.

250 The (mu-oxo)(mu-1,1-peroxo)diferric core structure associated wi

251 cy of iron(IV) species, which are usually mu-oxo-bridged dimers (Fe(IV)Fe(IV)), and this allows for t

252 active material, proposed to be a Ir(V,V) mu-oxo species, is formed on electrochemical oxidation of t

253 rol the synthesis of non-heme high-valent mu-oxo and mu-hydroxo Mn species from Mn(II) precursors and

254 multinucleated complex of manganese with mu-oxo units, which was able to mimic the properties of enz

255 rtionate with a Mn(II) precursor to yield mu-oxo and/or mu-hydroxo Mn(III) dimers.

256 ions in a cubic fashion linked by eight mu2-oxo and four mu2-OH groups.

257 Multimeric oxo-hydroxo Al clusters function as models for common mi

258 more rapid collapse is observed for the non-oxo counterpart (n = 4) hampering the observation of qua

259 ndem mass spectrometer by decarboxylation of oxo[4-(trimethylammonio)phenyl]acetic acid upon collisio

260 tral features are sensitive to the degree of oxo-mediated metal-metal coupling across Co4O4.

261 The LC1MS2 in (1)D allowed identification of oxo-TAG species by HRAM MS and quantification of 806.3 +

262 gle catalytic sites derived from metallic or oxo-metallic nodes.

263 xiamycin A, including enolate SET oxidation, oxo-vanadium oxidation, and atom-transfer cyclization.

264 regulated in a slow manner by oxotremorine (oxo-M) and angiotensin II in rat sympathetic neurons.

265 When compared with a topologically related oxo-iron(IV) complex bearing an equatorial N-donor ligan

266 fatty acid derivatives (including saturated oxo-fatty acids (C5-C18) and saturated hydroxy-fatty aci

267 re explored with many anions including small oxo anions like ReO4(-) and Cr2O7(2-) as well as anionic

268 in the context of using a series of strongly oxo-coupled homo- and heterometallic poly(f-block) chain

269 ydrocyclopeptin, reveals evidence supporting oxo<-->hydroxo tautomerism of the Fe(IV)-oxo species in

270 is a nucleophilic water attack on a terminal oxo (or oxyl) group.

271 developed a route to a new class of terminal oxo complexes of Re(III) supported by olefin moieties of

272 e2)N(Me)N2(amide)(Pr,Pr)](-) (8), shows that oxo atom donor reactivity correlates with the metal ion'

273 ent and isotope-effect studies, suggest that oxo-amino-acids of the protein serum albumin play a majo

274 ontrast, (t) BuNC was found to attack at the oxo moiety to produce isocyanate by oxygen atom transfer

275 idation involves initial coordination of the oxo atom donor to the metal ion.

276 yl Fe-O bonds, primarily by direction of the oxo group into one of two cis-related coordination sites

277 The reactivity of the oxo Re(V) beta-diketiminate, OReCl2 (BDI), with various

278 by the addition of capping moieties onto the oxo-frameworks, is critical for the development of the d

279 observations, which generally preserves the oxo palladium catalytic cycle widely accepted in the lit

280 nism have been studied and compared with the oxo-oxyl mechanism.

281 ation, leading to mitochondrial loss through oxo-nitrative stress and hypoxic response.

282 Not only Ti-MOFs but also Ti-oxo-clusters will be discussed and particular interest w

283 en a recent surge in discovery of aqueous Ti-oxo clusters but without extensive solution characteriza

284 e broadly the landscape of heterometallic Ti-oxo clusters.

285 rometals such as bismuth stabilize labile Ti-oxo sulfate clusters in aqueous solution.[Ti22 Bi7 O41 (

286 ontrast to the dominant corner-linking of Ti-oxo clusters.

287 center in AnCp3 (An=U, Np, Pu; Cp=C5 H5 ) to oxo-bind and reduce the uranyl(VI) dication in the compl

288 -ligated 8 is shown to be unreactive towards oxo atom donors, in contrast to imine-ligated 2.

289 the parent imido derivative and its tungsten oxo analogue.

290 migratory insertion to generate the tungsten-oxo alkylidene 2.

291 t selective functionalizations of the uranyl oxo by another actinide cation.

292 The rhenium(V) oxo complex oxo(triphenylphosphine) (bis(3,5-di-tert-but

293 At pH 13 iron(V)oxo is not formed and NaClO oxidizes 3 to an iron(IV)oxo

294 e structurally authenticated reactive iron(V)oxo units (Fe(V)O), wherein the iron atom is two oxidati

295 oxidation, suggesting that a putative Fe(V)-oxo species was initially generated.

296 In biology, high valent oxo-iron(IV) species have been shown to be pivotal inter

297 solvent interactions with the polar vanadium-oxo moiety.

298 so recorded the (77)Se NMR shift for a U(VI) oxo-selenido complex, [U(O)(Se)(NR2)3](-) (delta((77)Se)

299 (S2(Me2)N3(Pr,Pr))](+) (2), that reacts with oxo atom donors (PhIO, IBX-ester, and H2O2) to afford a

300 ing the synthesis of a range of ligated zinc oxo clusters, containing 4, 6 and 11 zinc atoms.