ライフサイエンスコーパス: oxygen

1 doperoxides 1-O2 upon oxidation with singlet oxygen.

2 vironment, including the rise of atmospheric oxygen.

3 e highly active oxides can come from lattice oxygen.

4 ified, with subjects breathing either air or oxygen.

5 eveloped that enables tumor-specific singlet oxygen ((1) O2 ) generation for cancer therapy, based on

6 lipid bilayer via the production of singlet oxygen ((1)O2).

7 The oxygen-18 isotopic ((18)O) composition in CO2 provides a

8 elative to Co(3+) and by the shift of active oxygen 2p states.

9 h the Co inserts into the C-H bond while the oxygen abstracts the leaving H-atom in a concerted, four

10 An oxygen adatom and a vicinal Co-atom form a metal-oxygen

11 evere COPD, and (2) to test whether low flow oxygen administration improves endothelial function and

12 the oxygen evolution reaction (OER), surface oxygen adsorption plays an indispensable role in the ele

13 s COPD admissions, previous use of long-term oxygen, age, and BMI.

14 ies in the presence and absence of molecular oxygen allow us to conclude that the imidazolium salts i

15 Sixty-four patients (28 in home oxygen alone and 36 in home oxygen plus home NIV) comple

16 proton relay mechanism between a ketone-like oxygen and a pyridine-like nitrogen across the vacancy s

17 strong association between reduced pressure/oxygen and fatigue on high-altitude flights.

18 controlling the insertion and extraction of oxygen and hydrogen ions independently of each other can

19 talase, suggesting that HDM-induced reactive oxygen and nitrogen species can be neutralized by antiox

20 apoptosis, heme turnover, inflammation, and oxygen and nutrient sensing have been discovered for org

21 acclimatisation to this trio of temperature, oxygen and pH changes through heterotrophic plasticity.

22 ve energy of formation) has been extended to oxygen and sulfur heterocycles.

23 ogen bonds between the oxazolidinedione ring oxygen and the CA II protein backbone.

24 cept for systems containing a high amount of oxygen and thus decreased conductivity.

25 by low temperature and that the exclusion of oxygen and water from the seed by the suberin and tannin

26 iratory support (RS) (receiving supplemental oxygen and/or positive-pressure RS); among those, oxygen

27 nt activity, produces a leak of electrons to oxygen, and completely blocks the binding of UQ and the

28 The rates at which wine consumes oxygen are important technological parameters for whose

29 metabolize formate and facilitate the use of oxygen as a terminal electron acceptor, respectively.

30 eduction in oxidase activity using molecular oxygen as the electron acceptor and a small increase in

31 the increase in water uptake is a result of oxygen atoms occupying the vacancies in the hydrophilic

32 lectrodes obtained by replacing stepwise the oxygen atoms with sulfur atoms in the carboxylate groups

33 Reduced oxygen availability (hypoxia) leads to increased product

34 y involve activation of an integrin/reactive oxygen axis.

35 evails in water-contaminated aprotic lithium-oxygen batteries is revealed.

36 synergistically to decrease both HbA and HbS oxygen binding affinity.

37 ediator- and membrane-free enzymatic glucose/oxygen biofuel cell based on transparent and nanostructu

38 , the latter kink can be ascribed only to an oxygen-breathing phonon.

39 non-haem metal not only donates electrons to oxygen but also activates it for efficient O-O bond clea

40 cient, having evolved well after the rise of oxygen (ca. 867 million years ago), and thus cannot be p

41 cates that proton has a good mobility in MC, oxygen can rotate around carbon to facilitate the proton

42 Nevertheless, the field of oxygen chemoreception still presents the general observe

43 ssion of many phosphorescent compounds, thus oxygen concentration could in many cases be derived dire

44 Above the 3-8% oxygen concentration typical of most tissues, we find th

45 ssue-specific variations in redox stress and oxygen concentration.

46 tions are that ellagitannins are the fastest oxygen consumers of the different oenological tannins, f

47 efficiency defined as stroke work/myocardial oxygen consumption (r=0.63-0.65; all P<0.01).

48 e micelles, have been determined in terms of oxygen consumption by a Clark electrode in an oxygen-tig

49 As the temperature dependence of oxygen consumption depends on activity levels, these fin

50 Oxygen consumption in freshly isolated mitochondria from

51 ndent and -independent energy biogenesis and oxygen consumption in mice without a concomitant increas

52 uffer from a severe deficit in mitochondrial oxygen consumption in response to the respiratory comple

53 Net flux (oxygen consumption rate) is determined by demand for ATP

54 As a consequence, oxygen consumption rates and intracellular ATP concentra

55 errucosus continuously showed 15-36% reduced oxygen consumption rates indicating metabolic depression

56 Average oxygen consumption was 1.1 +/- 0.2 mL/kg per minute.

57 edance (ie, global afterload) and myocardial oxygen consumption were reduced by -11% and -12% (P=0.03

58 limitation on exercise testing (reduced peak oxygen consumption, 24+/-1.3 versus 31+/-1.3 mL/kg/min,

59 FpEF displayed worse exercise capacity (peak oxygen consumption, 7.7+/-2.3 versus 10.0+/-3.4 and12.9+

60 lower valvuloarterial impedance, myocardial oxygen consumption, and improved myocardial efficiency d

61 , whereas loss of NLRX1 results in increased oxygen consumption, oxidative stress, and subsequently a

62 surements of extracellular acidification and oxygen consumption.

63 hdrawing groups, halogens, and nitrogen- and oxygen-containing heterocycles, as well as aromatic-cont

64 basic compounds, naphthenic acids, and other oxygen-containing species, carbazoles, sulfones, and thi

65 of ex situ methods for determining the true oxygen content.

66 ing treatments are used to produce different oxygen contents in the films, which has resulted in sign

67 ] sub-units controlled by the adaptive Ln/Fe oxygen coordination and the Fe(2/3+) redox.

68 eristics and reactivity trends of the copper-oxygen cores discussed.

69 Compared to its oxygen counterpart, the sulfur atom displays a remarkabl

70 Thus, AVD ensures increased oxygen delivery to active muscle fibres by reducing upst

71 d whether transfusion could augment cerebral oxygen delivery, particularly in vulnerable brain region

72 irculation that combine to reduce myocardial oxygen demand and to increase supply, thereby attenuatin

73 e duration of oxygen therapy and the rate of oxygen dependence at 36 weeks rose, and airflows at 8 ye

74 Here, oxygen-dependent changes in C. jejuni physiology were st

75 room-temperature-phosphorescence (RTP)-based oxygen detection platform by constructing core-shell nan

76 insight into the influence of cholesterol on oxygen diffusion across and within the membrane.

77 ealing ability of the ceramic's oxides, slow oxygen diffusion and a dense and gradient distribution o

78 hat oxygen vacancy rows are not only natural oxygen diffusion channels, but also preferred sites for

79 On the basis of dissolved oxygen (DO), ammonium, nitrite, and nitrate profiles wit

80 to reveal highly variable and heterogeneous oxygen dynamics at a high, quasi-continuous resolution a

81 system enables highly detailed insights into oxygen dynamics in various aquatic and terrestrial envir

82 Oxygen electrocatalysis, including the oxygen-reduction

83 here and a novel principle for the design of oxygen electrocatalysts is proposed.

84 ever, the origin of the advanced activity of oxygen electrocatalysts is still somewhat controversial.

85 Oxygen encapsulated nanosize carboxymethyl cellulosic na

86 The central importance of oxygen, energetics, and redox homeostasis in immune cell

87 In ambient oxygen environments, SRB-13 signaling impacts gene expre

88 The ultrahigh oxygen evolution activity and strong durability, with su

89 e and cost-effective electrocatalysts toward oxygen evolution and hydrogen evolution reactions in wat

90 rded in parallel, is due to both overlapping oxygen evolution and the oxidation of Ni(OH)2 to NiOOH.

91 00 nm were achieved with incorporation of Co oxygen evolution catalysts.

92 ately 95% were measured in the gas phase for oxygen evolution in alkaline media at an Inconel 625 all

93 ysts, which is reflected by 1.2 times higher oxygen evolution reaction (OER) activity than that of Ru

94 d on 3D transition-metal-based materials for oxygen evolution reaction (OER) and hydrogen evolution r

95 d-activity Ni/Fe (oxy)hydroxide (Ni(Fe)OxHy) oxygen evolution reaction (OER) catalysts, yet its preci

96 for the oxygen reduction reaction (ORR) and oxygen evolution reaction (OER) in fuel cells or metal-a

97 In the oxygen evolution reaction (OER), surface oxygen adsorpti

98 academic and practical interest, namely, the oxygen evolution reaction at Ni and Ni/Fe electrodes.

99 ection of oxide materials for catalyzing the oxygen evolution reaction in acid-based electrolyzers mu

100 ding the oxygen-reduction reaction (ORR) and oxygen-evolution reaction (OER), is a critical process f

101 ultrafast in the oxygen-rich chloroplasts of oxygen-evolving photosynthetic organisms.

102 Following oxygen exposure, preterm infants frequently develop chro

103 e developed a composite rat model of UPI and oxygen-fluctuations and removed premature birth as a con

104 reaction of iodoalkyl radical with molecular oxygen: for instance, CH2I + O2 --> CH2OO + I.

105 nt in tumor tissue prevents the formation of oxygen free radicals.

106 their oxidation states, which are related to oxygen fugacity (fO2).

107 achieved by establishing ion channels and an oxygen-functional-group gradient in graphene oxide nanor

108 ss/charge transport capability, easy release oxygen gas bubbles, and strong structural stability, whi

109 sive microbubbles (MagMBs), consisting of an oxygen gas core and a phospholipid coating functionalise

110 The oxygen generating capability of the polymer coated NPs w

111 gnificant potential as an in situ method for oxygen generation to enhance the efficacy of treatments

112 platform can accurately generate and control oxygen gradients, eliminates complex microfluidic handli

113 participation of light, photosensitizer, and oxygen greatly hinders the broad application of PDT as a

114 High-flow nasal oxygen has recently shown survival benefits in unselecte

115 water characteristics (e.g., high dissolved oxygen, high pH, and enrichment of (13)C in CO2) indicat

116 ny fish lineages, allowing animals to obtain oxygen in hypoxic aquatic environments.

117 ical (depth) EEG (dEEG), partial pressure of oxygen in interstitial brain tissue (Pbto2), and regiona

118 ew article, we consider the use of molecular oxygen in reactions mediated by polyoxometalates.

119 lso efficiently function to generate singlet oxygen in situ (PhiDelta approximately 20 %) and has app

120 machinery, and consequently suppressing the oxygen-independent degradation of HIF-1alpha.

121 abrogate angiogenesis in the mouse models of oxygen-induced retinopathy and laser-induced choroid neo

122 etinal and choroidal angiogenesis, including oxygen-induced retinopathy, laser-induced choroidal neov

123 t role of FEN1 phosphorylation to counteract oxygen-induced stress in the heart during the fetal-to-n

124 In response to gradual changes in oxygen influx, this model abruptly transitions between a

125 cterization and reactivity of important iron-oxygen intermediates.

126 ace vanadium oxides and diffusion of surface oxygen into the metal bulk are both important mechanisms

127 ings are consistent with the hypothesis that oxygen is a limiting factor shaping the temperature-size

128 Oxygen is an efficient quencher of emission of many phos

129 Evolved oxygen is detected by reduction at a Pt UME, allowing fo

130 otosynthetic organisms, in which exposure to oxygen is low, chlorophyll-to-carotenoid triplet-triplet

131 re we analyze a new high-resolution deep-sea oxygen isotope (delta(18)O) record from the South Atlant

132 Blood oxygen level dependent (BOLD) signal differences in resp

133 ward accumulated within a context, and blood oxygen level dependent (BOLD) signals in the ventromedia

134 Blood oxygen level-dependent imaging demonstrated increased fr

135 photosensitive epilepsy, alpha-related blood oxygen level-dependent signal changes demonstrated lower

136 asures of human brain activity are the blood-oxygen-level dependent (BOLD) signal and surface field p

137 gger sounds elicit greatly exaggerated blood-oxygen-level-dependent (BOLD) responses in the anterior

138 Reduced oxygen levels and ATP consumption rates were simulated t

139 ntial signalling pathways by which a fall in oxygen levels might initiate Type I cell activation.

140 biospheric productivity and ocean-atmosphere oxygen levels over time.

141 t has also been suggested that environmental oxygen levels then crashed to very low levels during the

142 s accompanied by a decrease in intramuscular oxygen levels, resulting in induction of HIF-1alpha.

143 e SRMs with uniform size distribution, large oxygen loading capacity, high LLL12 encapsulation effici

144 ions, total antioxidant capacity or reactive oxygen metabolites.

145 s of the oceans and particularly abundant in oxygen minimum zones (OMZs).

146 Without oxygen, most vertebrates die within minutes as they cann

147 Furthermore, polar (oxygen-, nitrogen-, and sulfur-containing) analytes foun

148 Regions of hypoxia (low oxygen) occur in most solid tumours and cells in these a

149 roup theory identifies how configurations of oxygen octahedral rotation patterns, ordered cation arra

150 en bonding interactions with the nonbridging oxygen of the scissile phosphate.

151 rtheless, the influence of triplet-quenching oxygen on PL and a photon correlation analysis of aggreg

152 arly stages of the first rise in atmospheric oxygen on the Earth (the Great Oxidation Event).

153 nety patients in each group (high-flow nasal oxygen or standard oxygen) were matched according to the

154 We measured arterial oxygen partial pressure continuously with a fast intra-v

155 esults show that He ion irradiation leads to oxygen penetration into the irradiated areas, and diffus

156 water-soluble polymethyloxazoline shells and oxygen-permeable polystyrene cores crosslinked with meta

157 investigate the magnitude of these proposed oxygen perturbations using selenium (Se) geochemistry, w

158 the local electronic structure of the copper-oxygen plane.

159 om the nanometre-spaced confinement of extra oxygen planes that enhances the unscreened Coulomb inter

160 ents (28 in home oxygen alone and 36 in home oxygen plus home NIV) completed the 12-month study perio

161 Changing the oxygen pressure to 10(-6) Torr enriched the Al site occu

162 emonstrated in aqueous solution with minimal oxygen produced at pH7.4, whereas it increased significa

163 Visible-light-driven oxygen production by an organic chromophore for up to 1

164 ond, and solid K3PO4 interacts with carbonyl oxygen, promoting intermolecular nucleophilic attack by

165 egetable intake or total antioxidant intake (oxygen radical absorbance capacity).

166 pport interactions can significantly improve oxygen reduction activity without inducing shape, alloyi

167 Catalysts at the air cathode for oxygen reduction and evolution reactions are central to

168 and protons are found to be involved in the oxygen reduction and evolution reactions, and lithium hy

169 ts high activity and superior durability for oxygen reduction and evolution reactions.

170 activity than that of Ru/C and a comparable oxygen reduction reaction (ORR) activity to that of Pt/C

171 ce noble metal Pt and RuO2 catalysts for the oxygen reduction reaction (ORR) and oxygen evolution rea

172 Herein we report the first study of the oxygen reduction reaction (ORR) catalyzed by a cofacial

173 dissolution of platinum, resulting from the oxygen reduction reaction (ORR) or hydrogen peroxide red

174 (carbonized at 800 degrees C) exhibited high oxygen reduction reaction activity with an onset potenti

175 imately 6 times higher mass activity for the oxygen reduction reaction than Pt/C, and twice the mass

176 A high-performance nonprecious-metal oxygen-reduction electrocatalyst is prepared via in situ

177 Oxygen electrocatalysis, including the oxygen-reduction reaction (ORR) and oxygen-evolution rea

178 chlorophyll switch; pufQ is found within the oxygen-regulated pufQBALMX operon encoding the reaction

179 Definitions using oxygen requirement alone as the criterion at various pos

180 e, during, and after the procedure to record oxygen requirement, oxygen saturation, respiratory rate,

181 r reasons independent of their physiological oxygen requirements.

182 k identifies bacterial formate oxidation and oxygen respiration as metabolic signatures for inflammat

183 e compared with those using the criterion of oxygen/respiratory support (RS) (receiving supplemental

184 econds range, whereas it is ultrafast in the oxygen-rich chloroplasts of oxygen-evolving photosynthet

185 n and/or positive-pressure RS); among those, oxygen/RS at 36 weeks had the highest AOR and area under

186 o "control" (FIO2 0.3, adjusted for arterial oxygen saturation >/= 90%) and "hyperoxia" (FIO2 1.0 for

187 ia and signs of severe respiratory distress, oxygen saturation <93% (when not at high altitude), mode

188 ysmography), heart rate (electrocardiogram), oxygen saturation (pulse oximetry), and brachial artery

189 HR and oxygen saturation changes were not significantly associa

190 al dysfunction, 24-h AHI, CAI, and time with oxygen saturation of <90% were independent predictors of

191 A drop in oxygen saturation that required supplemental oxygen was

192 the procedure to record oxygen requirement, oxygen saturation, respiratory rate, consciousness level

193 ssential for acute but not chronic pulmonary oxygen sensing by triggering mitochondrial hyperpolariza

194 together with plant Cys oxidases, act as an oxygen-sensing mechanism.

195 rofluidic device with integrated optical pH, oxygen sensors and algal fluorescence.

196 PHD1 to PHD3 are molecular oxygen sensors and increasingly considered as putative t

197 en adatom and a vicinal Co-atom form a metal-oxygen site-pair that cleaves the C-H bond via a sigma b

198 ncreases in mitochondrially-derived reactive oxygen species (ROS) activate an adaptive stress respons

199 esponses, such as the production of reactive oxygen species (ROS) and mitogen-activated protein (MAP)

200 Liver damage, reactive oxygen species (ROS) and paracrine tumor necrosis factor

201 gnificantly increased production of reactive oxygen species (ROS) and upregulated expression of proin

202 a) leads to increased production of reactive oxygen species (ROS) by the electron transport chain.

203 Reactive oxygen species (ROS) can damage DNA, proteins, and lipid

204 nduction by YC-1 involved excessive reactive oxygen species (ROS) generation.

205 Reactive oxygen species (ROS) have been suggested as such trigger

206 CF), despite abundant production of reactive oxygen species (ROS) in lung tissue.

207 that PA increased the production of reactive oxygen species (ROS) in podocytes and that NAC (N-acetyl

208 ar ATP concentrations decreased and reactive oxygen species (ROS) increased.

209 Reactive oxygen species (ROS) play a critical role in cell signal

210 l pathogens are exposed to damaging reactive oxygen species (ROS) produced from a variety of sources

211 oxidases contribute to LPS-induced reactive oxygen species (ROS) production and modulate TLR respons

212 Reactive oxygen species (ROS) production, degranulation, and phag

213 which is reported to cause reduced reactive oxygen species (ROS) production, predisposes to SLE (odd

214 osinophil apoptosis associated with reactive oxygen species (ROS) production.

215 now demonstrated that mitochondrial reactive oxygen species (ROS) signal to support adipocyte thermog

216 utrophils generate higher levels of reactive oxygen species (ROS) when presented with planktonic orga

217 n Caco-2 cells (MTT, cell cycle and reactive oxygen species (ROS)) were evaluated in Colombian coffee

218 essment of viability, mitochondrial reactive oxygen species (ROS), membrane damage, mitochondrial DNA

219 TRX1 inhibition elevates reactive oxygen species (ROS), p53 levels and cell death in andro

220 Oxidative DNA bases modified by reactive oxygen species (ROS), primarily as 7, 8-dihydro-8-oxo-2'

221 n mice, in the setting of hyperoxia/reactive oxygen species (ROS)-induced lung injury.

222 eration of intra- and extracellular reactive oxygen species (ROS).

223 n (p66Shc) is a master regulator of reactive oxygen species (ROS).

224 excess mitochondrial production of reactive oxygen species (ROS).

225 sis of hormones, and homeostasis of reactive oxygen species (ROS).

226 ggesting an increased production of reactive oxygen species (ROS).

227 und to: 1) induce the production of reactive oxygen species (ROS); 2) decrease succinate dehydrogenas

228 oxide to mitochondria could prevent reactive oxygen species accumulation, limiting downstream oxidati

229 verload and increased production of reactive oxygen species after reoxygenation.

230 and resulted in the accumulation of reactive oxygen species and cell death in the absence of p53.

231 ate the production of mitochondrial reactive oxygen species and contribute to alcohol-induced hepatoc

232 myocytes subjected to I/R increased reactive oxygen species and necrotic cell death, both of which we

233 ochondrial DNA along with increased reactive oxygen species and reduced superoxide dismutase 2 (SOD2)

234 ted to well-characterized examples of copper-oxygen species but seeks to provide a thorough picture o

235 do X-ray absorption spectroscopy showed that oxygen species can survive in the bulk of the catalyst d

236 h acidic environment which contains reactive oxygen species due to the mycobacterial genomes encoding

237 chondrial dysfunction, indicated by reactive oxygen species expression, reduced expression of the mit

238 ries protect human cells from toxic reactive oxygen species for up to a week.

239 through their role in respiration, reactive oxygen species generation, and energy production.

240 gh glucose-induced NOX4 expression, reactive oxygen species generation, and, matrix laminin expressio

241 destructive phase, where a burst of reactive oxygen species induces loss of E-cadherin-mediated cell

242 Such glycolytic flux and elevated reactive oxygen species is supported by increased antioxidant; gl

243 This oxygen species is the most probable candidate for the sl

244 that result from diverse chemicals, reactive oxygen species or UV light.

245 essed cytotoxicity, nitric oxide or reactive oxygen species production, and phagocytosis.

246 phosphate/NADPH levels, phagocytic reactive oxygen species production, neutrophil extracellular trap

247 prevented by co-treatment with the reactive oxygen species scavenger Tempol.

248 miR-193b also stimulated reactive oxygen species signaling by targeting the antioxidant me

249 e in endocytosis, scavenging of the reactive oxygen species, and in the response to endoplasmic retic

250 econd messengers-cytosolic calcium, reactive oxygen species, and nitric oxide.

251 sonophagocytosis, and production of reactive oxygen species, closely mimicking the defective immune f

252 Compound SA-2 scavenged reactive oxygen species, inhibited proliferation and migration of

253 troponin I and elevated amounts of reactive oxygen species, lower phosphorylated extracellular recep

254 aluate cell viability, formation of reactive oxygen species, mitochondrial health, as well as cell mo

255 groups and allows 5-HT to intercept reactive oxygen species, preventing membrane oxidation.

256 phosphatase activity by scavenging reactive oxygen species, thus preventing spleen tyrosine kinase d

257 tenuated cell invasion and elevated reactive oxygen species, whereas such phenotypes were reversed by

258 s are achieved as a balance between reactive oxygen species-regulated effects on polymerization and g

259 ffects of relatively high levels of reactive oxygen species.

260 tivity, and increased production of reactive oxygen species.

261 brane potential and a high level of reactive oxygen species.

262 sis factor, interleukin (IL)-6, and reactive oxygen species.

263 results in increased production of reactive oxygen species.

264 sequence contexts after exposure to reactive oxygen species.

265 ce oxygen vacancy concentration and superior oxygen storage capacity.

266 r obstruction during a VOC leads to impaired oxygen supply to the periphery and ischemia reperfusion

267 newal, and differentiation are influenced by oxygen supply, an environmental regulator of stem cell a

268 This process requires an adequate oxygen supply, typically delivered by packed red blood c

269 OH) derivatives of the steroids were used as oxygen surrogates.

270 and selectivity, fast response time and low oxygen-, temperature- and pH- dependencies.

271 nges in ankle brachial index, transcutaneous oxygen tension, rest pain, and walking capacity after ce

272 The first significant buildup in atmospheric oxygen, the Great Oxidation Event (GOE), began in the ea

273 asive ventilation over time, the duration of oxygen therapy and the rate of oxygen dependence at 36 w

274 time in the use of assisted ventilation and oxygen therapy during the newborn period and in lung fun

275 xygen consumption by a Clark electrode in an oxygen-tight cell.

276 of treatments that depend on the presence of oxygen to elicit a cytotoxic effect.

277 ed these measurements to quantify 6 steps of oxygen transport and utilization (the O2 pathway) in eac

278 oxygen within the membrane but enhances the oxygen transport parameter (solubility-diffusion product

279 5-furandicarboxylic acid (FDCA) using aerial oxygen under mild reaction conditions.

280 ontribution of both regulation mechanisms to oxygen up-regulation likely varies over depth.

281 in healthy men and women.The MFO and maximal oxygen uptake ([Formula: see text]O2 max) were determine

282 The primary end point was a change in peak oxygen uptake (Vo2) from baseline to 16 weeks.

283 e test of aerobic fitness to measure maximal oxygen uptake (VO2Max).

284 aired testing, the percent predicted maximum oxygen uptake decreased (69 +/- 14% vs. 61 +/- 16%; p <

285 The oxygen uptake promotes a reconstructive shearing of the

286 ratory fitness (commonly measured by maximal oxygen uptake, VO2 max ), a trait with wide-ranging impa

287 The accumulation of oxygen vacancies at the Cu2O/Cu interface drives the col

288 ng and phase-field simulations, we show that oxygen vacancies can move under a stress-gradient-induce

289 An electron gas stabilized by oxygen vacancies is confined within the domain wall, dis

290 Oxygen vacancies, especially their distribution, are dir

291 ls, but also preferred sites for the induced oxygen vacancies.

292 rface area to volume ratio, reactive surface oxygen vacancy concentration and superior oxygen storage

293 uced in VO2 by ionic liquid gating is due to oxygen vacancy formation rather than to electrostatic do

294 Our results show that oxygen vacancy rows are not only natural oxygen diffusio

295 VIVID(VVD) protein is a Light-Oxygen-Voltage(LOV) domain in circadian clock system.

296 oxygen saturation that required supplemental oxygen was the most common SAE.

297 ch group (high-flow nasal oxygen or standard oxygen) were matched according to the propensity score,

298 ristics and used for in vivo measurements of oxygen with high resolution in the brain extracellular s

299 rapy is limited to normobaric and hyperbaric oxygen, with no available antidotal therapy.

300 holesterol reduces the overall solubility of oxygen within the membrane but enhances the oxygen trans