ライフサイエンスコーパス: oxygen delivery

1 an reduction in circulating blood volume and oxygen delivery.

2 rtially related to hypoxemia and compromised oxygen delivery.

3 on is dependent on continuous and controlled oxygen delivery.

4 oxygen saturation, and systemic and cerebral oxygen delivery.

5 situations in which disease reduces cardiac oxygen delivery.

6 sition can alter cell circulation and impede oxygen delivery.

7 , and optimizing cardiovascular function and oxygen delivery.

8 on blood vessel diameter, tissue volume, and oxygen delivery.

9 systemic blood pressure, cardiac output, and oxygen delivery.

10 essure, while maintaining cardiac output and oxygen delivery.

11 ls has been demonstrated to improve cerebral oxygen delivery.

12 ic hypertension, reduced cardiac output, and oxygen delivery.

13 lone, resulting in improved hemodynamics and oxygen delivery.

14 esult in molecules better suited for in vivo oxygen delivery.

15 uring detachment in addition to insufficient oxygen delivery.

16 nesis, osteogenesis, coronary perfusion, and oxygen delivery.

17 n, it is unwise to concentrate on maximizing oxygen delivery.

18 ng interest in perioperative optimization of oxygen delivery.

19 the gut oxygen consumption beyond splanchnic oxygen delivery.

20 ne and a reduction in both cardiac index and oxygen delivery.

21 a measure of the efficiency of microvascular oxygen delivery.

22 nticoagulation, and optimization of systemic oxygen delivery.

23 of circulating blood and thus improve tissue oxygen delivery.

24 d enhance exercise performance by increasing oxygen delivery.

25 ues as goals or when therapy did not improve oxygen delivery.

26 1) was administered to enhance the patient's oxygen delivery.

27 local HCT(m) has also been shown to increase oxygen delivery.

28 poxia to examine the relationship of CBF and oxygen delivery.

29 cannot differentiate cerebral from systemic oxygen delivery.

30 tained without the need for increased CBF or oxygen delivery.

31 on, with no effective improvement in maximal oxygen delivery.

32 er than the optimal concentration needed for oxygen delivery.

33 in oxygen consumption, despite an increased oxygen delivery.

34 ne did not significantly reduce RBF or renal oxygen delivery.

35 s of perfusion nor diminishes with increased oxygen delivery.

36 ble to the profound impact of AngII on renal oxygen delivery.

37 ing to provide enough material for efficient oxygen delivery.

38 on, thereby increasing tissue blood flow and oxygen delivery.

39 y after major surgery is associated with low oxygen delivery.

40 uires rapid removal of CO and restoration of oxygen delivery.

41 asis on improvements in biocompatibility and oxygen delivery.

42 required for blood circulation and systemic oxygen delivery.

43 ation regarding the quality of perfusion and oxygen delivery.

44 r cellular protection in the face of reduced oxygen delivery.

45 uction from parasite sequestration decreases oxygen delivery.

46 only occur if oxygen consumption depends on oxygen delivery.

47 utomatically increases to preserve sustained oxygen delivery.

48 including influence on systemic and cerebral oxygen deliveries.

49 7 mL/min/kg [95% CI, 39-57], p = 0.002), and oxygen delivery (7.6 mL O2/min/kg [95% CI, 6.4-9.0] vs 5

50 erichia coli suggest that instead of aerobic oxygen delivery, a dioxygenase converts NO to NO3(-) and

51 or muscle blood flow and systemic and muscle oxygen delivery accompanies marked dehydration and hyper

52 resulted in no change in cardiac output and oxygen delivery after bypass.

53 fabrication of a polymer-based intravascular oxygen delivery agent.

54 e no differences in myocardial blood flow or oxygen delivery among groups; however, at 45 min of isch

55 eltaMAP 51 +/- 5 mmHg), and leg and systemic oxygen delivery and (.)VO2 .

56 a significant decrease in cardiac output and oxygen delivery and a significant increase in pulmonary

57 mpaired peripheral O2 extraction, constrains oxygen delivery and aerobic capacity in ePVH.

58 Maintenance of systemic oxygen delivery and alleviation of pulmonary hypertensio

59 Global hypoxia-ischemia interrupts oxygen delivery and blood flow to the entire brain.

60 and directly measured parameters of systemic oxygen delivery and blood flow, NIRS can certainly assis

61 Oxygen delivery and blood gases were measured during bot

62 hyxia result in brain injury from inadequate oxygen delivery and constitute a major and growing world

63 Better determinations of oxygen delivery and consumption are needed to guide clin

64 ent directed at achieving survivor values of oxygen delivery and consumption in critically ill patien

65 hree dimensions is crucial for understanding oxygen delivery and consumption in normal and diseased b

66 due to inadequate tools to quantify cerebral oxygen delivery and consumption non-invasively and in re

67 Myocardial oxygen delivery and consumption were greater among survi

68 lid tumors arising from an imbalance between oxygen delivery and consumption.

69 al muscle vascular tone during mismatches in oxygen delivery and demand (e.g. exercise) via binding t

70 rum malaria result from an imbalance between oxygen delivery and demand.

71 There was no relationship between oxygen delivery and disease severity (P = .64) or outcom

72 l hypoperfusion (following INDO) on cerebral oxygen delivery and extraction.

73 y, blood transfusion does not always improve oxygen delivery and is associated with ischemic events.

74 nd inotropic agents in an effort to increase oxygen delivery and lactate clearance.

75 cellular response to hypoxia which promotes oxygen delivery and metabolic adaptation to oxygen depri

76 impaired by hypoxia despite global cerebral oxygen delivery and metabolism being maintained.

77 impaired by hypoxia despite global cerebral oxygen delivery and metabolism being maintained.

78 adjusting for various variables of cerebral oxygen delivery and metabolism, the only statistically s

79 rebral oxygen diffusion or reflects cerebral oxygen delivery and metabolism.

80 blood flow, which maintains global cerebral oxygen delivery and metabolism.

81 blood flow, which maintains global cerebral oxygen delivery and metabolism.

82 omyocyte function and metabolism rather than oxygen delivery and microvascular function.

83 Hypovolemia with reduced oxygen delivery and microvascular obstruction have diffe

84 We investigated whether oxygen delivery and muscle metabolism of the lower extre

85 hospital cardiac arrest may lead to improved oxygen delivery and organ perfusion.

86 The identification of an increase in both oxygen delivery and oxygen consumption (oxygen supply de

87 sirable in clinical conditions of low tissue oxygen delivery and perfusion.

88 t will reflect the unique pathophysiology of oxygen delivery and peripheral oxygen offloading are nee

89 cells invoke adaptive mechanisms to enhance oxygen delivery and promote energy conservation.

90 g hypertrophy necessitate increased fuel and oxygen delivery and stimulate angiogenesis in the left v

91 ntage saturation of haemoglobin and hindlimb oxygen delivery and the increase in P(a,CO2) were sustai

92 o review recent publications in the field of oxygen delivery and tissue oxygenation.

93 development has challenged understanding of oxygen delivery and use.

94 examined the efficiency of coupling between oxygen delivery and utilization using the sd of the oxyg

95 etabolites, neural signaling, alterations in oxygen delivery and utilization, and by modifications in

96 erse processes that in turn orchestrate both oxygen delivery and utilization.

97 ation of an arteriovenous shunt may increase oxygen delivery and, hence, improve patients' functional

98 s little reserve to tolerate interruption of oxygen delivery and, thus, is at risk for hypoxemia duri

99 their relationship with cerebral blood flow, oxygen delivery, and carbon dioxide reactivity remain un

100 flow, regional cerebral blood flow, cerebral oxygen delivery, and cerebral metabolic rate of oxygen i

101 post-ischemic defects in neovascularization, oxygen delivery, and chemokine expression, and normalize

102 lation of tissue blood flow distribution and oxygen delivery, and could further reduce skeletal muscl

103 The effects of gestational age, oxygen delivery, and cysteine infusion or glutathione in

104 O(2) saturation of haemoglobin, and hindlimb oxygen delivery, and increases in P(a,CO2), haemoglobin

105 gen binding affinity of Hb, increases tissue oxygen delivery, and increases maximal exercise capacity

106 ial for many biological processes, including oxygen delivery, and its supply is tightly regulated.

107 ntly decreased stroke volume, cardiac index, oxygen delivery, and left-ventricular (LV) function plot

108 transcutaneous carbon dioxide tensions, low oxygen delivery, and low oxygen consumption developed in

109 olume, total peripheral resistance, systemic oxygen delivery, and organ blood flow were determined by

110 mum mixed venous oxygen saturation, systemic oxygen delivery, and systemic oxygen consumption were 33

111 res were similar but cardiac index, systemic oxygen delivery, and systemic oxygen consumption were in

112 CHD was linearly related to reduced cerebral oxygen delivery, and that cardiac lesions associated wit

113 The data suggest that blood flow, oxygen delivery, and tissue oxygenation of the nonsurviv

114 s influenced by many interactions, including oxygen delivery (angiogenesis, permeability, and HgB) an

115 thout an increase in hematocrit (eliminating oxygen delivery as an etiologic factor in myocyte surviv

116 y be associated with relatively lower tissue oxygen delivery as reflected in higher erythropoietin co

117 We defined "useful" oxygen delivery as the amount of oxygen above a notional

118 ver, higher SpvO2 and SaO2 enhanced systemic oxygen delivery, as demonstrated by improvement in oxyge

119 Perioperative periods of diminished cerebral oxygen delivery, as indicated by rSo(2), are associated

120 nsfusion resulted in a greater (16%) rise in oxygen delivery associated with reduction in oxygen extr

121 olids, arterial oxygen content, and systemic oxygen delivery below baseline (p <.05).

122 Hypoxia is a reduction in oxygen delivery below tissue demand, whereas ischemia is

123 e was a significant difference (p < 0.05) in oxygen delivery between baboons (+164 +/- 47 from 705 +/

124 20% and when therapy produced differences in oxygen delivery between the control and protocol groups.

125 ncreases in (.)Q , LBF, and systemic and leg oxygen delivery, but central venous pressure and muscle

126 hought to function primarily in nutrient and oxygen delivery, but recent evidence suggests that it ma

127 Additionally, given VP can only approximate oxygen delivery by capillaries, we show that their gener

128 Oxygen delivery by Hb is essential for vertebrate life.

129 me and cardiac output, and ensuring adequate oxygen delivery by maintaining arterial oxygen partial p

130 set physiological hypoxia and achieve normal oxygen delivery by means of higher blood flow enabled by

131 Oxygen delivery by the oxygenator was significantly incr

132 We report measurements of oxygen delivery by the retinal circulation (DO2_IR) and

133 We have explored the coupling of CBF and oxygen delivery by using two complementary methods.

134 HIF-1 controls oxygen delivery, by regulating angiogenesis and vascular

135 were placed for hemodynamic measurement and oxygen delivery calculations.

136 The oxygen delivery capacity of the ceria nanoparticles embe

137 severe reductions in cerebral blood flow and oxygen delivery capacity.

138 d flow (gCBF) increases to preserve cerebral oxygen delivery (CDO2) in excess of that required by an

139 erebral metabolic rate (CMRO2), and cerebral oxygen delivery (CDO2) was determined over a range of Hc

140 ow) and cerebral oxygen metabolism (cerebral oxygen delivery, cerebral metabolic rate of oxygen, and

141 Hemoglobin, systemic oxygen delivery, circulating blood volume, and plasma le

142 h may provide inferior systemic and cerebral oxygen deliveries compared with either of the 2 surgical

143 ased mean arterial pressure, hemoglobin, and oxygen delivery compared with animals resuscitated with

144 stention and lung volume, as well as improve oxygen delivery compared with half functional residual c

145 Oxygen delivery, consumption, and extraction ratio were

146 d gases; electrolytes; lactate; base excess; oxygen delivery, consumption, and extraction ratio; hema

147 hat the coupling between neural activity and oxygen delivery could be imaged at the single-RBC level

148 Oxygen delivery decreased by 50% after infusion of AngII

149 significantly change cardiac index, but the oxygen delivery decreased due to a hemodilution-induced

150 n extraction, whereas both cardiac index and oxygen delivery decreased for patients in the 546C88 coh

151 the while increasing in mass to provide the oxygen delivery demands of embryonic growth.

152 with CC+RB, the left ventricular myocardial oxygen delivery did not differ.

153 tent (CaO(2)) and systemic blood flow: total oxygen delivery (DO(2))=CaO(2)xQs.

154 ic capacity could be impaired by the fall in oxygen delivery (DO2) during endotoxic shock.

155 The effect of this therapy on oxygen delivery (DO2) has not been studied.

156 ssue hypoperfusion and hypoxemia, changes in oxygen delivery (DO2), oxygen consumption VO2), and oxyg

157 pressin levels were determined, and systemic oxygen delivery (Do2I) and extraction ratio were calcula

158 Impaired oxygen delivery due to reduced cerebral blood flow is th

159 rebral and other organ perfusion, as well as oxygen delivery during cardiopulmonary resuscitation.

160 chanism underlying impaired vasodilation and oxygen delivery during hypoxemia with advancing age.

161 The effect of myoglobin on oxygen delivery during vasomotion was also examined.

162 In general, there was no association of oxygen delivery (except for ventriculomegaly in the BDG

163 eased diffusion barriers may reduce cellular oxygen delivery following head injury and attenuate the

164 rest and during exercise, despite attenuated oxygen delivery following NO-PG blockade, due to an incr

165 .4 to 10.8 +/- 1.4 g/dL (12%; p < 0.001) and oxygen delivery from 5.0 (interquartile range, 4.4-6.6)

166 mechanism of improvement in VO2 is increased oxygen delivery from increased hemoglobin concentration.

167 coustic flowoxigraphy (FOG), which can image oxygen delivery from single flowing RBCs in vivo with mi

168 scussed with respect to its proposed in vivo oxygen delivery function.

169 rates that RBC transfusion improves cerebral oxygen delivery globally and particularly to vulnerable

170 ia, in which cerebral perfusion pressure and oxygen delivery have gained new importance.

171 ted at reducing mortality through increasing oxygen delivery have not been successful.

172 esulted in a significant decrease of hepatic oxygen delivery (hDO2, 63% and 12% of baseline, respecti

173 Systemic oxygen delivery improved dose-dependently with Hb-200 bu

174 col-driven therapy targeting optimization of oxygen delivery improves outcomes in the management of m

175 urons are threatened by markedly constrained oxygen delivery, improving the latter by increasing arte

176 es of SNO-Hb could be manipulated to enhance oxygen delivery in a hypoxic tumor.

177 tabolic demand and the relatively inadequate oxygen delivery in affected synovium, can both be object

178 Haemodynamic therapy aimed at increasing oxygen delivery in an effort to reduce oxygen debt, tiss

179 Mechanisms involving enhanced tissue oxygen delivery in comparison to Lowlander populations h

180 We hypothesised that impaired cerebral oxygen delivery in infants with CHD is a cause of impair

181 unction (defined as the capacity to increase oxygen delivery in response to ischemia) and oxygen cons

182 oxide (NO)-derived vasoactivity to optimize oxygen delivery in the arterial periphery.

183 sion and vasodilation (required for adequate oxygen delivery in the face of chronic anemia) are media

184 bin functions to deliver NO (thus maximizing oxygen delivery in the respiratory cycle).

185 the spatial distribution and extent of tumor oxygen delivery in vivo.

186 Cardiac output and oxygen delivery increased significantly from 4.1 L/min a

187 Renal oxygen delivery increased while renal oxygen consumption

188 rterial pressure, cardiac index and systemic oxygen delivery, increases in heart rate and systemic va

189 rterial oxygen content, it further threatens oxygen delivery increasing the risk of cerebral infarcti

190 tension to fractional inspired oxygen >200, oxygen delivery index >600 mL/min/m2, and oxygen consump

191 emic vascular resistance index), metabolism (oxygen delivery index and consumption index, oxygen extr

192 rdiac output/index, stroke volume index, and oxygen delivery index and increases in systemic vascular

193 (66%) of 187 patients achieved preoperative oxygen delivery (irrespective of intervention).

194 Breakdown or absence of vascular oxygen delivery is a hallmark of many common human disea

195 l cerebral blood flow is abnormal, postnatal oxygen delivery is decreased, and intraoperative support

196 Oxygen delivery is far from synonymous with tissue oxyge

197 ications for disease states in which cardiac oxygen delivery is impaired.

198 e local oxygen saturation of hemoglobin when oxygen delivery is limiting.

199 It is well known that oxygen delivery is not the only function of systemic cir

200 ed blood cell (RBC) unit storage duration on oxygen delivery is uncertain.

201 is, although Qp:Qs falls in both conditions, oxygen delivery is unchanged during hypoxia and increase

202 unds p:s assessment and compromises SaO2 and oxygen delivery, judicious use of inspired oxygen and PE

203 regions were defined as those with baseline oxygen delivery less than 4.5 mL/100 g/min.

204 to the same functional outcome of successful oxygen delivery, long-term persistence and high function

205 suggest that strategies to improve cerebral oxygen delivery may help reduce brain dysmaturation in n

206 itical illness, and the resulting deficit in oxygen delivery may play an important role in the pathog

207 During shock and reinfusion, oxygen delivery, mixed venous PO2, mixed venous oxygen s

208 ylephrine, probably because it reduced renal oxygen delivery more than did phenylephrine.

209 hagic shock, Hb-200 infusion may not improve oxygen delivery more than hetastarch, likely due to hemo

210 nary artery occlusion pressure of <18 mm Hg, oxygen delivery of >600 mL x min(-1) x m(-2), and oxygen

211 f < or =4.0 g/dL (< or =40.0 g/ L); systemic oxygen delivery of < or =320 mL/min/m2; redo operation;

212 shock, serum albumin of <4.0 g/dL, systemic oxygen delivery of <320 mL/ min/m2 before surgery, blood

213 model to investigate possible limitations of oxygen delivery on size.

214 O2 rather than a direct measurement of total oxygen delivery or cerebral oxygen metabolism.

215 nonresponders were unable to increase either oxygen delivery or oxygen consumption to the dobutamine.

216 associated with changes in renal blood flow, oxygen delivery, or histological appearance.

217 lar resistance index), metabolic parameters (oxygen delivery, oxygen consumption, arterial lactate),

218 Heart rate, blood pressure, cardiac output, oxygen delivery, oxygen consumption, SMA blood flow, ile

219 saturation, as well as significantly higher oxygen delivery, oxygen consumption, transcutaneous oxyg

220 turation and reduced pulmonary hypertension, oxygen delivery, oxygen extraction, oxygen consumption,

221 cardiac output, stroke volume, and systemic oxygen delivery (p < .05 vs. controls).

222 est, the responders had significantly higher oxygen delivery (p<.01) and oxygen consumption (p<.05) t

223 d whether transfusion could augment cerebral oxygen delivery, particularly in vulnerable brain region

224 reestablish baseline MAP, blood flow rates, oxygen delivery, PrCO2, and pHi.

225 numerous physiological traits comprising the oxygen delivery process.

226 Any reduction in myocardial oxygen delivery relative to its demands can impair cardi

227 emodynamic stability and monitoring cerebral oxygen delivery remain important goals of perioperative

228 n (6 times basal), only minor differences in oxygen delivery resulted between the sprouting and split

229 predict whether a fluid-induced increase in oxygen delivery results in an increase in oxygen consump

230 of insufficient numbers of erythrocytes for oxygen delivery, SCD patients constantly face hypoxia.

231 of results from clinical trials, unnecessary oxygen delivery should be avoided in critically ill vent

232 of results from clinical trials, unnecessary oxygen delivery should be avoided in ventilated stroke p

233 Oxygen delivery significantly increased in these 25 pati

234 ., the late stage), cardiac output, systemic oxygen delivery, stroke volume, total peripheral resista

235 Improved gas exchange and higher systemic oxygen delivery suggest that calpain inhibition may be a

236 Hemoglobin A (HbA), the oxygen delivery system in humans, comprises two alpha an

237 We hypothesised that individualised oxygen delivery targeted haemodynamic therapy (goal-dire

238 , but this was not affected by the use of an oxygen delivery targeted strategy.

239 a significant increase in cardiac output and oxygen delivery , the creation of an arteriovenous shunt

240 se of its essential role in gas exchange and oxygen delivery, the lung has evolved a variety of strat

241 rocytes has been investigated as a potential oxygen delivery therapeutic.

242 NOS controls blood pressure, blood flow and oxygen delivery through its effect on vascular smooth mu

243 an elaborate branched network essential for oxygen delivery throughout the larva.

244 Thus, AVD ensures increased oxygen delivery to active muscle fibres by reducing upst

245 or ascending vasodilatation ensure increased oxygen delivery to active skeletal muscle.

246 st be tightly balanced to guarantee adequate oxygen delivery to all tissues in the body.

247 n and prevents neuron apoptosis by promoting oxygen delivery to brain or by direct interaction with n

248 regulation of skeletal muscle blood flow and oxygen delivery to contracting skeletal muscle is comple

249 2+/-3 mL; p<or=.05), with worse matching of oxygen delivery to demand (p<.001).

250 n-utero hemodynamics and increasing cerebral oxygen delivery to enhance brain development.

251 ces a systemic response designed to increase oxygen delivery to hypoxic tissues.

252 red ability of the microvasculature to match oxygen delivery to increased oxygen demand.

253 is critical to ensure proper blood flow and oxygen delivery to metabolically active skeletal muscle.

254 tory and ventilatory responses that increase oxygen delivery to muscle during exercise.

255 During hypothermic blood cardioplegia, oxygen delivery to myocytes is minimal with ineffective

256 partial, allowing for a residual flow, hence oxygen delivery to partially occluded vessels could redu

257 is not known whether VB can provide adequate oxygen delivery to restore or maintain renal function.

258 that facilitate increases in blood flow and oxygen delivery to the active tissue and the sympathetic

259 t of this hypothesis, mathematical models of oxygen delivery to the brain have been described in whic

260 ermine the quality of cerebral perfusion and oxygen delivery to the brain.

261 o the placental vasculature that compromises oxygen delivery to the fetus.

262 Here we have shown that endotoxemia reduces oxygen delivery to the kidney, without changing tissue o

263 Increased oxygen delivery to the microcirculation might have contr

264 ndurance in smokers may result from impaired oxygen delivery to the mitochondria and ability of the m

265 r cessation of coronary blood flow such that oxygen delivery to the myocardium is insufficient to mee

266 , perfusion pressure was 5.8+/-3.3 mmHg with oxygen delivery to the organs in excess of 3.5 times the

267 t homeostatic regulation to maintain optimal oxygen delivery to the tissues.

268 Limited oxygen delivery to tissues (hypoxia) is common in a vari

269 viscosity, which results in both compromised oxygen delivery to tissues and cerebrovascular complicat

270 rculating blood and their promise to improve oxygen delivery to tissues supports the potential for th

271 a fundamental physiologic response ensuring oxygen delivery to tissues under metabolic stress.

272 ust be tightly controlled to ensure adequate oxygen delivery to tissues without causing thrombosis or

273 sidents, resulting in greater than sea level oxygen delivery to tissues.

274 ces a systemic response designed to increase oxygen delivery to tissues.

275 ysical mechanism, losartan improves drug and oxygen delivery to tumours, thereby potentiating chemoth

276 ely seem best to distribute flow to maximize oxygen delivery (total, upper body, or lower body), we f

277 the impact of hypoxia versus hypercarbia on oxygen delivery, under conditions of fixed minute ventil

278 ted to achieve their individual preoperative oxygen delivery value (goal-directed therapy) or standar

279 Achievement of preoperative oxygen delivery values in the postoperative phase was as

280 amine the effect of HF on the time course of oxygen delivery versus uptake (protocol 1) and on vasoco

281 The decline in oxygen delivery was associated with an increase in extra

282 Cerebral oxygen delivery was calculated using phase contrast angi

283 Systemic oxygen delivery was higher after calpain inhibition comp

284 Oxygen delivery was maintained at 76 +/- 13 mL/min at ba

285 ean arterial pressure suggests that cerebral oxygen delivery was maintained during ETS.

286 Total systemic oxygen delivery was markedly reduced in the hybrid palli

287 We also demonstrated that single-RBC oxygen delivery was modulated by changing either the inh

288 d hybrid 475 mL . min(-1) . m(-2)). Cerebral oxygen delivery was similarly lower in the hybrid pallia

289 Volume status and oxygen delivery were assessed using transpulmonary therm

290 lesions associated with the lowest cerebral oxygen delivery were associated with the greatest impair

291 epsis maintained cardiac output and systemic oxygen delivery, whereas it increased oxygen consumption

292 serve as initial therapy to maintain tissue oxygen delivery while awaiting the maximal effect of rec

293 omes in critically ill patients by enhancing oxygen delivery while minimizing the risks of toxic effe

294 These data demonstrate that restoration of oxygen delivery with a small volume of MP4 yields signif

295 The amount of oxygen delivery with AV-ECMO depends on arterial desatur

296 oad, afterload, and contractility to balance oxygen delivery with oxygen demand.

297 By providing oxygen delivery with slow, limited infusion, new hemoglo

298 However, CO2 reactivity and oxygen delivery (with 1 exception) were not associated w

299 esions most associated with reduced cerebral oxygen delivery would demonstrate the greatest impairmen

300 ction of whether a fluid-induced increase in oxygen delivery would result in an increase in oxygen co