ライフサイエンスコーパス: oxygen demand

1 ar tone to match local tissue perfusion with oxygen demand.

2 ally at the expense of increasing myocardial oxygen demand.

3 rcise-induced increases in muscle energy and oxygen demand.

4 ion in which O2 supply is inadequate to meet oxygen demand.

5 ssure product, dP/dt, -dP/dt, and myocardial oxygen demand.

6 ncreased right ventricular (RV) workload and oxygen demand.

7 ell with high removal efficiency of chemical oxygen demand.

8 lature to match oxygen delivery to increased oxygen demand.

9 F response is mediated by factors other than oxygen demand.

10 n of large predatory fish, animals with high oxygen demand.

11 ft ventricular (LV) afterload and myocardial oxygen demand.

12 myocardial perfusion and reducing myocardial oxygen demand.

13 t worsening major determinants of myocardial oxygen demand.

14 ontractility to balance oxygen delivery with oxygen demand.

15 rictor nerve activity and reductions in limb oxygen demand.

16 is the major contributor to the increase in oxygen demand.

17 increases red blood cell mass to meet tissue oxygen demands.

18 like flying insects that have high metabolic oxygen demands.

19 with removals of 97% of the soluble chemical oxygen demand, 97% NH3-N, and 91% of total bacteria (bas

20 IPB system removed more than 92% of chemical oxygen demand, 98% of ammonium nitrogen, and 82% of phos

21 However, chemical oxygen demand, a common measure of gross residual organi

22 more likely to result from preventing excess oxygen demand after surgery rather than from deciding wh

23 h the alternation of the influent biological oxygen demand and ammonium nitrogen load to the aerated

24 when added to leachate; five-day biochemical oxygen demand and biochemical methane potential results

25 approaches that address the balance between oxygen demand and delivery; the manipulation of cardiopu

26 Chemical oxygen demand and dissolved organic carbon concentration

27 changes decrease LV afterload and myocardial oxygen demand and reduce the number of angina episodes,

28 abolites signalling a mismatch between blood/oxygen demand and supply in exercising muscles.

29 s occur in the setting of a mismatch between oxygen demand and supply, as with severe hypotension.

30 ignificant correlation of HAAs with chemical oxygen demand and THMs with FC was observed.

31 irculation that combine to reduce myocardial oxygen demand and to increase supply, thereby attenuatin

32 wastewater temperature, influent biological oxygen demand, and ammonium nitrogen load, was investiga

33 (NTG) improves myocardial perfusion, reduces oxygen demand, and may enhance low-dose dobutamine to im

34 well as a detector for measuring biochemical oxygen demand (BOD) using Rhodotorula mucilaginosa UICC

35 aracterize phosphorus, nitrogen, biochemical oxygen demand (BOD), and fecal coliform pollution from h

36 mical oxygen demand (COD) 90-95%, biological oxygen demand (BOD5) 94-98%, total nitrogen (TN) 70-80%,

37 ) and a 4-fold increase in 5-day biochemical oxygen demand (BOD5).

38 ng with a local hypoxia induced by increased oxygen demands by proliferating cells which supports chr

39 ly been associated with increased myocardial oxygen demand, cardiac arrhythmias, and mortality in a v

40 DNA degradation rate declined as biochemical oxygen demand, chlorophyll, and total eDNA (i.e., from a

41 g in large values of chemical and biological oxygen demand (COD and BOD) in the aquatic systems into

42 high biological elimination rates (chemical oxygen demand (COD) 90-95%, biological oxygen demand (BO

43 Treatment efficiency (chemical oxygen demand (COD) and ammonia removal), Ag dissolution

44 Greater than 95% reductions in chemical oxygen demand (COD) and ammonium ion were achieved withi

45 d the 24-h and 7-day Pb toxicity to chemical oxygen demand (COD) and NH3-N removal, bacterial viabili

46 ystem oxidized only 10% of influent chemical oxygen demand (COD) and recovered up to 55% of incoming

47 Both MFCs removed 65-70% chemical oxygen demand (COD) at a hydraulic retention time (HRT)

48 method for the determination of the chemical oxygen demand (COD) in heterogeneous solid or semisolid

49 used and mainly contribute to high chemical oxygen demand (COD) in textile effluents.

50 rs applied to the rapid analysis of chemical oxygen demand (COD) in urban waste waters.

51 te, the SCMFC acted as a sensor for chemical oxygen demand (COD) in water.

52 Condensate pH was 6.2 and its chemical oxygen demand (COD) level was 36,000 ppm.

53 synthetic acetate solution having a chemical oxygen demand (COD) of 320 mg/L.

54 ed as total organic carbon (TOC) or chemical oxygen demand (COD), though these parameters do not prov

55 tified accounted for only 2.1 mg of chemical oxygen demand (COD)/L (16% of total SMP as COD) because

56 the highest solubilization (0.16 mg chemical oxygen demand (COD)/mg volatile solids (VS), at 2.13 mg

57 ter column features arising from respiratory oxygen demand during organic matter degradation in strat

58 t adequately restore tissue oxygenation when oxygen demand exceeds supply.

59 idation result in reduced economy (increased oxygen demand for a given speed) at velocities that tran

60 tive to riverine nutrient loads and sediment oxygen demand from settled organic carbon.

61 explained by its ability to reduce cerebral oxygen demand has been replaced by an increasingly docum

62 western basin bloom growth and central basin oxygen demand in distinct ways that merit further invest

63 the diaphragm to augment blood flow to match oxygen demand in response to contractile activity and co

64 The importance of increase in myocardial oxygen demand in the genesis of ischemia in both men and

65 The comorbidities that increase oxygen demands in these infants, like bronchopulmonary d

66 ater consumption, discharge of COD (chemical oxygen demand) in effluent water, cumulative COD and dil

67 Significant increases in myocardial oxygen demand, including systolic blood pressure, occur

68 oals of treatment are to decrease myocardial oxygen demand, increase coronary blood flow and oxygen s

69 h the influent total phosphorus and chemical oxygen demand instead of geographical factors (e.g. lati

70 forms, E. coli, enterococci, and biochemical oxygen demand (Kendall's tau = 0.348 to 0.605, p < 0.05)

71 ischemia induced by a sustained increase in oxygen demand may not progress to necrosis but may inste

72 logy similar to that of a modern sponge, its oxygen demands may have been met well before the enhance

73 ncreases in these determinants of myocardial oxygen demand, myocardial perfusion decreased by 30% (10

74 To meet the extreme oxygen demand of insect flight muscle, tracheal (respira

75 carbon-paste matrix is shown to satisfy the oxygen demand of the enzymatic reaction and to provide c

76 ter so that significant reduction of initial oxygen demand of wastewater by 61% was observed.

77 ue and specialized trait associated with the oxygen demands of flying, their endothermic metabolism a

78 However, the effects of remote increases in oxygen demand on a circulation with limited ability to r

79 are associated with an increased myocardial oxygen demand on reperfusion.

80 e observed the effect of remote increases in oxygen demand on splanchnic and renal blood flow in hemo

81 ion was related to a reduction in myocardial oxygen demand or preservation of myocardial oxygen suppl

82 ponses were expressed relative to myocardial oxygen demand (rate-pressure product).

83 DO level was attributable to biofilm-induced oxygen demand rather than changes in oxygen diffusivity.

84 ogens, which led to the decrease in chemical oxygen demand removal.

85 We hypothesized that increased myocardial oxygen demand resulting from hypotension and reflex tach

86 Increased myocardial oxygen demand resulting from hypotension and reflex tach

87 tized, hemorrhaged dog, increased peripheral oxygen demand results in further redistribution of blood

88 f MFCs were proposed earlier (as biochemical oxygen demand sensing) only lately a myriad of new uses

89 Besides, the use of MFCs as biochemical oxygen demand sensors (perhaps the main analytical appli

90 on rate, renal oxygen consumption, and renal oxygen demand/supply relationship, i.e., renal oxygen ex

91 domestic wastewater having a total chemical oxygen demand (tCOD) of 210 +/- 11 mg/L.

92 Secondary-treated PPM effluent has lower oxygen demand than primary-treated effluent, but ASB acc

93 ations and functions for estimating sediment oxygen demand that are linked to settled organic carbon

94 whole-limb blood flow is related to a lower oxygen demand that is independent of tissue mass.

95 However, the heart has intrinsic oxygen demands that must be met to maintain effective co

96 A2 class of adenosine receptors and reducing oxygen demand through A1 adenosine receptors (A1AR).

97 investigation illustrated that the chemical oxygen demand, total nitrogen, and total phosphorus remo

98 ciencies of total suspended solids, chemical oxygen demand, total phosphorus, and total nitrogen were

99 d minor effect on determinants of myocardial oxygen demand, vasodilator stress myocardial perfusion i

100 Statistical analysis indicated that cerebral oxygen demand was maintained to an Hct of 0.14, 0.11, an

101 iency of total suspended solids and chemical oxygen demand was observed for recovered aluminum (85-60

102 ples where water temperature and biochemical oxygen demand were measured.

103 These increases in oxygen demand were met by increases in both oxygen deliv

104 ad (aortic pressure, P=0.030) and myocardial oxygen demand were seen (tension-time index, P=0.024; ra

105 r side, high heart rates increase myocardial oxygen demand, which can be a problem in patients with f

106 ulation was used to increase lower extremity oxygen demand while lower extremity, splanchnic, renal b

107 Balancing oxygen demand with availability is crucial to cardiac fu

108 ormal pigs, there is no change in myocardial oxygen demand with CPAP, whatever the change in cardiac

109 calized vascular damage and increased tissue oxygen demand, wound healing occurs in a relatively hypo