ライフサイエンスコーパス: oxygen desaturation

1 uration of respiratory events, and degree of oxygen desaturation.

2 were accompanied by moaning, tachypnea, and oxygen desaturation.

3 tively correlated with severity of nocturnal oxygen desaturation.

4 s and none of the 10 controls had sleep-time oxygen desaturation.

5 in during intermittent hypoxia to accentuate oxygen desaturation.

6 Oxygen desaturation (353 patients [5.6%]) and vomiting (

7 rface displacements (32% vs. 56%; P = 0.01), oxygen desaturations (40% vs. 75%; P < 0.001), required

8 per airway collapse during sleep, leading to oxygen desaturation and disrupted sleep.

9 after completion of the vascular anatomoses oxygen desaturation and increased airway pressure was no

10 hypopnea cycles during sleep associated with oxygen desaturation and sleep disruption.

11 Subjects demonstrated a modest oxygen desaturation and tachycardia during the procedure

12 obesity, the frequency of obstructive apnea, oxygen desaturation, and arousal contributes to abnormal

13 n of the amygdala co-occurred with apnea and oxygen desaturation, and electrical stimulation of the a

14 s performed in response to an adverse event, oxygen desaturation, and vomiting.

15 or the presence of respiratory disturbances, oxygen desaturations, and cortical arousals within a 90-

16 y insufficient unless clinical findings like oxygen desaturation, apnea, and bradycardia are part of

17 Oxygen desaturations are accompanied by increases in ETC

18 Oxygen desaturation at rest was present in 4 patients.

19 Six children (0.9%) had brief periods of oxygen desaturation below 95%, none of which required ai

20 In these 19 seizures, all oxygen desaturations below 85% were accompanied by an in

21 Pulse oximetry showed oxygen desaturations below 90% in 101 (33.2%) of all sei

22 h SAHS, particularly in those with nocturnal oxygen desaturation, but the decrease is small.

23 , nocturnal REM sleep latency, and extent of oxygen desaturation-could reflect neurophysiological mec

24 ose tolerance was related to the severity of oxygen desaturation (DeltaSa(O(2))) associated with slee

25 The aim of this study was to assess oxygen desaturation during sleep in hepatopulmonary synd

26 m notifications were for clinically relevant oxygen desaturation, facilitating timely nursing respons

27 Oxygen desaturation greater than 5% from rest to peak ex

28 idence of postreinfusion nausea, emesis, and oxygen desaturation in comparison to unselected PBSC rei

29 Nineteen had SDB (oxygen desaturation index > 5 events/h).

30 Elevated oxygen desaturation index (>/=15 events/hour) and high p

31 indicating moderate-to-severe apnea) and the oxygen desaturation index (ODI; the number of times per

32 thickness was only associated with the mean oxygen desaturation index among the polysomnography para

33 sleep apnoea (Epworth score > 9, and a > 4% oxygen desaturation index of > 10 per h) who were assign

34 lycated hemoglobin level of 6.5-8.5%, and an oxygen desaturation index of 15 or more events per hour

35 ETCO2, supporting the assumption that ictal oxygen desaturation is a consequence of hypoventilation.

36 00 mm Hg, systolic blood pressure <90 mm Hg, oxygen desaturation <80%, and extubation.

37 These complications included oxygen desaturation <90% (n = 1), vital sign alterations

38 These complications included: oxygen desaturations <90%, vital sign alterations requir

39 fied with the apnea hypopnea index (AHI) and oxygen desaturation measures.

40 The most common complication was oxygen desaturation (n = 8).

41 ical rating scale), interface displacements, oxygen desaturations, need for ventilator support, and r

42 Conclusion: Oxygen desaturation occurred during sleep in 70% of hepa

43 Chills, fever, and arterial oxygen desaturation of >/= 3% occurred in 7% of the tran

44 Adverse respiratory event was defined as oxygen desaturation of at least 5%, pulmonary aspiration

45 number of obstructive events and less severe oxygen desaturations on the operative night.

46 itical vulnerability, resulting in saw-tooth oxygen desaturation possibly representing the earliest m

47 n adenosine that occur during jugular venous oxygen desaturations suggest that adenosine may play an

48 uat exposure with ventilatory equivalent and oxygen desaturation suggests that paraquat may be associ

49 f bronchial hemorrhage of > 30 mL; transient oxygen desaturation to < 90% in seven (8.4%) patients; h

50 with adverse respiratory events: 58 cases of oxygen desaturation, two pulmonary aspirations, 10 cases

51 We examined whether seizure-associated oxygen desaturation was a consequence of hypoventilation

52 Oxygen desaturation was a late phenomenon.

53 crog/mL), both Cheyne-Stokes respiration and oxygen desaturation were markedly attenuated.

54 Obesity and the magnitude of nocturnal oxygen desaturation, which is an important pathophysiolo

55 There were fewer episodes of oxygen desaturation with pentobarbital (0.2%) than with

56 ere coded as clinically relevant (i.e., true oxygen desaturation with SpO2<89 for >15s) or irrelevant