ライフサイエンスコーパス: oxygen saturation

1 lator, in response to reduced blood arterial oxygen saturation.

2 airway hyperreactivity, and diminished blood oxygen saturation.

3 death at 36 weeks in the group with a lower oxygen saturation.

4 esults may help determine the optimal target oxygen saturation.

5 ogy but maintained normal levels of arterial oxygen saturation.

6 in tumor oxygen concentration and hemoglobin oxygen saturation.

7 ow supplemental oxygen to normalize arterial oxygen saturation.

8 not be used as surrogate for central venous oxygen saturation.

9 verity of disease, higher dyspnea, and lower oxygen saturation.

10 ion and to a lower or higher target range of oxygen saturation.

11 l lactate concentrations, and central venous oxygen saturation.

12 versely related to average asleep and waking oxygen saturation.

13 aging, limited blood flow and lowered tissue oxygen saturation.

14 ed apoptosis in the lungs, and a decrease in oxygen saturation.

15 and IgM but not IgG isotype predicted better oxygen saturation.

16 predicting low cardiac index or mixed venous oxygen saturation.

17 hemoglobin concentration, blood volume, and oxygen saturation.

18 with greater alterations accompanying lower oxygen saturation.

19 percentage of sleep time with less than 90% oxygen saturation.

20 tSo2 level were poor predictors of cerebral oxygen saturation.

21 ial pressure cardiac index, and mixed venous oxygen saturations.

22 al pressure, cardiac index, and mixed venous oxygen saturations.

23 icant differences were found in jugular vein oxygen saturation (83.2% [79.2-87.6%] vs. 86.7% [83.2-88

24 tion and to lower or higher target ranges of oxygen saturation (85 to 89% or 91 to 95%).

25 Health Stroke Scale score, 5; mean baseline oxygen saturation, 96.6%) were enrolled.

26 y 6-hr resuscitation period, masseter tissue oxygen saturation accurately identified patients with se

27 It remains uncertain what values of arterial oxygen saturations achieve this balance in preterm infan

28 alone, resulted in improvement in nocturnal oxygen saturation and apnea/hypopnea index.

29 n patients treated with CPAP, mean nocturnal oxygen saturation and baseline IL-1beta were independent

30 on between obtained values of femoral venous oxygen saturation and central venous oxygen saturation (

31 is lack of agreement between femoral venous oxygen saturation and central venous oxygen saturation i

32 We concurrently determined femoral venous oxygen saturation and central venous oxygen saturation i

33 we determined simultaneously femoral venous oxygen saturation and central venous oxygen saturation i

34 ients, the difference between femoral venous oxygen saturation and central venous oxygen saturation i

35 Results for changes of femoral venous oxygen saturation and central venous oxygen saturation w

36 Correlation and agreement of femoral venous oxygen saturation and central venous oxygen saturation w

37 luding the difference between femoral venous oxygen saturation and central venous oxygen saturation.D

38 Both masseter tissue oxygen saturation and deltoid tissue oxygen saturation b

39 our study was to determine if central venous oxygen saturation and femoral venous oxygen saturation c

40 n (n = 136); or level 4 (L4), which included oxygen saturation and heart rate (n = 135).

41 showed a significant decrease in local brain oxygen saturation and in brain tissue PO(2) alongside br

42 Lower oxygen saturation and intravenous iron may be modifiable

43 of BV5/TBV were directly related to resting oxygen saturation and inversely associated with both the

44 We compared the ability of central venous oxygen saturation and markers of anaerobic metabolism to

45 In the presence of pain, tissue oxygen saturation and perfusion index are further reduce

46 in must be considered when evaluating tissue oxygen saturation and perfusion index as markers of hypo

47 Tissue oxygen saturation and peripheral perfusion index are pro

48 sculature and providing images of hemoglobin oxygen saturation and pulsation.

49 nic therapy can improve microcirculation and oxygen saturation and reduce vessel calibers in patients

50 Healthcare providers should monitor oxygen saturation and requirements during and after IS c

51 Primary outcomes were nocturnal oxygen saturation and the apnea/hypopnea index; secondar

52 The reduction in mixed venous blood oxygen saturation and the increase in blood lactate seen

53 psis, the correlation between central venous oxygen saturation and tissue oxygen saturation at differ

54 ous pressure, respiratory rate, and arterial oxygen saturation and treatment with vasopressors target

55 Daily home monitoring of oxygen saturation and weight has been reported to improv

56 iratory rate, blood pressure, and peripheral oxygen saturation) and standard care; INDEX background w

57 roup analysis of children by malaria status, oxygen saturation, and age.

58 with oxygenation, ventilation, mixed venous oxygen saturation, and cardiac output.

59 Dyspnea intensity, inspiratory capacity, oxygen saturation, and cardiac, metabolic, and respirato

60 rfusion index, capillary refill time, tissue oxygen saturation, and forearm-to-fingertip skin tempera

61 FDNIRS) to measure hemoglobin concentration, oxygen saturation, and indices of cerebral blood flow an

62 2), improved hemodynamics and central venous oxygen saturation, and less organ dysfunction.

63 Concentrations of algal pigments, dissolved oxygen saturation, and pH rapidly declined following ces

64 ated through histological studies, degree of oxygen saturation, and responsiveness to carbachol.

65 attering exponent, hemoglobin concentration, oxygen saturation, and sampling depth are presented alon

66 well as global cardiac output, mixed venous oxygen saturation, and systemic and cerebral oxygen deli

67 Perfusion, Intravascular Venous Oxygen saturation, and T2* (PIVOT), a recently developed

68 nction derived from baseline CMR and resting oxygen saturation are associated with mortality in adult

69 bability that cardiac index and mixed venous oxygen saturation are normal and physical examination fi

70 al venous oxygen saturation or thenar tissue oxygen saturation are strong predictors of 28-day mortal

71 provide evidence that low asleep and waking oxygen saturations are associated with LV abnormalities

72 the curve 0.66; 0.46-0.86) or central venous oxygen saturation (area under the curve 0.56; 0.38-0.80)

73 curve 0.88; 0.77-0.98) but not thenar tissue oxygen saturation (area under the curve 0.66; 0.46-0.86)

74 c curve analysis showed that masseter tissue oxygen saturation (area under the curve 0.87; 0.75-0.98)

75 he curve 0.87; 0.75-0.98) and deltoid tissue oxygen saturation (area under the curve 0.88; 0.77-0.98)

76 us oxygen saturation >70% than thenar tissue oxygen saturation (area under the curve, 0.80; 95% confi

77 tory rate >/= 25/min, PaO2 </= 50 mm Hg, and oxygen saturation (arterial [SaO2] or measured by pulse

78 Changes in tumor hemoglobin oxygen saturation as a function of time after treatment

79 ypopnea index, <30 events per hour) + (nadir oxygen saturation as measured by pulse oximetry >82.5%)

80 onservative oxygenation strategy with target oxygen saturation as measured by pulse oximetry (SpO2) o

81 dicted; FEV(1)/FVC = 31.6 +/- 7.1%; exercise oxygen saturation as measured by pulse oximetry [Spo(2)]

82 ean pulmonary artery pressure, SpO(2) is the oxygen saturation as measured by pulse oximetry, and DLC

83 Nadir oxygen saturation as measured by pulse oximetry, apnea-h

84 ut also essentially stabilized gas exchange (oxygen saturation) as an overall measure of lung functio

85 eatment was associated with higher nocturnal oxygen saturation at 1630 m and 2590 m than placebo and

86 central venous oxygen saturation and tissue oxygen saturation at different levels.

87 Mean oxygen saturation at rest was 85.1 +/- 7.8%, and median

88 xygen therapy was 11.6 hours, and the median oxygen saturation at the end of the treatment period was

89 and tissue Doppler echocardiography, waking oxygen saturation averaged over 5 minutes, and overnight

90 Targeting an oxygen saturation below 90% with the use of current oxim

91 The primary outcome was lowest arterial oxygen saturation between induction and 2 minutes after

92 -saline group had significantly lower tissue oxygen saturation, brain tissue PO2, and venous oxygen s

93 was not predicted by baseline central venous oxygen saturation but by high baseline lactate and (P(v

94 tissue oxygen saturation and deltoid tissue oxygen saturation but not central venous oxygen saturati

95 ical coherence tomography and measurement of oxygen saturation by spectrophotometry.

96 venous oxygen saturation and femoral venous oxygen saturation can be used interchangeably during sur

97 have developed a system that quantifies the oxygen saturation, cell volume, and Hb concentration for

98 HR and oxygen saturation changes were not significantly associa

99 was associated with higher regional cerebral oxygen saturation compared with manual chest compression

100 comes of time to intubation, lowest arterial oxygen saturation, complications, and in-hospital mortal

101 We recorded central venous oxygen saturation continuously for 0 to 6 hrs of resusci

102 peritoneal BLM model as assessed by arterial oxygen saturation (control, 84.4 +/- 1.3%; C-188-9, 94.4

103 low 24-hr fluid output; and low mixed venous oxygen saturation correlated with knee mottling and high

104 including height, weight, respiratory rate, oxygen saturation, cough, or respiratory symptom scores.

105 cyanosis confirmed by medical staff when his oxygen saturation decreased to the 60% level, and he had

106 venous oxygen saturation and central venous oxygen saturation.Despite significant correlation betwee

107 kers of anaerobic metabolism, central venous oxygen saturation did not allow the prediction of whethe

108 e)V ratios were inversely related to resting oxygen saturation, diffusing capacity of carbon monoxide

109 e primary outcome was longitudinal change in oxygen saturation divided by the FIO2 (S/F) through day

110 We measured intubating conditions, oxygen saturation during and 5 mins following intubation

111 on does not seem to increase lowest arterial oxygen saturation during endotracheal intubation of crit

112 ring the temporal dynamics of blood flow and oxygen saturation during reactive hyperemia than by conv

113 -DLPFC GABA levels, but not Glx, and minimal oxygen saturation during sleep (r = 0.62, P = 0.0005).

114 ation with the apnea-hypopnea index, average oxygen saturation during sleep, and average respiratory

115 easures included noninvasive cerebral tissue oxygen saturation during the first transfusion, clinical

116 Clinical assessment, cerebral oxygen saturation, electrolyte abnormalities, adverse ev

117 id not change intraocular pressure, arterial oxygen saturation, end-tidal CO2, and respiration rate (

118 alveolar accumulation and improves arterial oxygen saturation even when administered 90 minutes afte

119 Tissue oxygen saturation (except cerebral) and perfusion index

120 p severe pneumonitis associated with reduced oxygen saturation, fibrosis, peripheral inflammation, hy

121 ould be considered instead of central venous oxygen saturation for starting hemodynamic resuscitation

122 % (15.8 vs. 25.0%; P = 0.22), or decrease in oxygen saturation greater than 3% (53.9 vs. 55.6%; P = 0

123 of 612), versus 27.5% of those in the higher-oxygen-saturation group (171 of 622) (relative risk, 1.1

124 8), and in 30.2% of the infants in the lower-oxygen-saturation group (185 of 612), versus 27.5% of th

125 al ventilation titrated to maintain arterial oxygen saturation > 90%), "hyperoxia" (standard resuscit

126 o "control" (FIO2 0.3, adjusted for arterial oxygen saturation >/= 90%) and "hyperoxia" (FIO2 1.0 for

127 presenting with isolated fast breathing and oxygen saturation >/=90% were randomly assigned to recei

128 ation was better predictor of central venous oxygen saturation >70% than thenar tissue oxygen saturat

129 tients with severe sepsis and central venous oxygen saturation >70%.

130 The presence of normal oxygenation (oxygen saturation >96%) decreased the likelihood of pneu

131 infants, those in the lower-target group for oxygen saturation had a reduced rate of retinopathy of p

132 R or noninvasive measurement besides resting oxygen saturation (hazard ratio, 0.90 [0.83-0.97]/%; P=0

133 Mean oxygen saturation, heart rate and hematocrit were not si

134 Oxygen saturation, heart rate and rhythm, and blood pres

135 Safety indicators included spirometry, oxygen saturation, heart rate, and symptoms.

136 ses in plasma NO levels, muscle interstitial oxygen saturation, hind leg glucose extraction, and musc

137 re no significant differences in heart rate, oxygen saturation, hospitalization rate, or other outcom

138 Secondary outcomes included vital signs, oxygen saturation, hospitalization, physician clinical i

139 Tissue hemoglobin oxygen saturation (i.e., oxygenation) is a functional im

140 venous oxygen saturation and central venous oxygen saturation in 30 surgical patients and in 30 crit

141 venous oxygen saturation and central venous oxygen saturation in a group of 100 stable cardiac patie

142 Pain (cold pressor test) reduces tissue oxygen saturation in all measurement sites (except cereb

143 , MSOT provided images reflecting hemoglobin oxygen saturation in blood vessels, clearly identifying

144 venous oxygen saturation and central venous oxygen saturation in both stable and unstable medical co

145 bserved higher cerebral blood flow and lower oxygen saturation in females compared to males.

146 viously that pulmonary function and arterial oxygen saturation in NY1DD mice with sickle cell disease

147 timisation of anaesthesia depth and cerebral oxygen saturation in older adults undergoing surgery.

148 The safest ranges of oxygen saturation in preterm infants have been the subje

149 The clinically appropriate range for oxygen saturation in preterm infants is unknown.

150 by making direct time-course measurement of oxygen saturation in the femoral/popliteal arteries and

151 venous oxygen saturation and central venous oxygen saturation including its range of variation dimin

152 The systolic blood pressure and mixed venous oxygen saturation increased from 75 (IQR:15) mm Hg to 10

153 , serum pH, and Paco(2) were associated with oxygen saturation index (p < .05); and 1/PaO2/Fio2, mean

154 validation data sets, given by the equation oxygen saturation index = 2.76 1 0.547*oxygenation index

155 Oxygen saturation index had a strong linear association

156 comparability of SpO2/Fio2 to PaO2/Fio2 and oxygen saturation index to oxygenation index in children

157 on, cerebral abscess was associated with low oxygen saturation (indicating greater right-to-left shun

158 egivers to record measurements of weight and oxygen saturation into a binder and requires families to

159 iratory rate, blood pressure, and peripheral oxygen saturation) into AN instability index value (INDE

160 le tissue from which the intravascular blood oxygen saturation is computed as %HbO(2).

161 sed on continuing resuscitation until venous oxygen saturation is normalized.

162 on less than 90% (44.7 vs. 47.2%; P = 0.87), oxygen saturation less than 80% (15.8 vs. 25.0%; P = 0.2

163 c oxygenation and usual care in incidence of oxygen saturation less than 90% (44.7 vs. 47.2%; P = 0.8

164 opnea index (AHI) and percent nighttime with oxygen saturation less than 90% (TSat(90)) were used as

165 Median night-time spent with oxygen saturation less than 90% at 2590 m was 13% (IQR,

166 rapid eye movement sleep and time spent with oxygen saturation less than 90% were associated with inc

167 ), and the percentage of sleep time with the oxygen saturation less than 90%.

168 significantly higher mean regional cerebral oxygen saturation levels during cardiopulmonary resuscit

169 f deterioration) during hospitalization when oxygen saturation levels were 90% or higher.

170 R: 13.2), hemoglobin < or =90 g/l (OR: 6.7), oxygen saturation < or =94% (OR: 3.0), and Q-wave on the

171 The presence of moderate hypoxemia (oxygen saturation </=96%; LR, 2.8 [95% CI, 2.1-3.6]; sen

172 gen saturation <70% predicted a mixed venous oxygen saturation <60% with a sensitivity 84%,specificit

173 Central venous oxygen saturation <70% predicted a mixed venous oxygen s

174 the area under the curve for central venous oxygen saturation <70%.

175 Episodes of hypoxemia (pulse oximeter oxygen saturation <80%) or bradycardia (pulse rate <80/m

176 the burden of hypoxemia (the time spent with oxygen saturation <90%) significantly predicted BNP conc

177 at chest radiography, very severe pneumonia, oxygen saturation <92%, C-reactive protein >/=40 mg/L, a

178 ia and signs of severe respiratory distress, oxygen saturation <93% (when not at high altitude), mode

179 ar systolic dysfunction had a central venous oxygen saturation<70%.

180 Oxygen saturation mapping in the intact lung yields uniq

181 h multispectral oximetry for two-dimensional oxygen saturation mapping.

182 howed some correlation between perfusion and oxygen saturation maps and the ability to sensitively mo

183 o capillaries and venules in two-dimensional oxygen saturation maps.

184 e the apparent diffusion coefficient, tissue oxygen saturation, mean transit time, and blood volume f

185 ficient (n = 11 rats per group); local brain oxygen saturation, mean transit time, and blood volume f

186 he effects of hypovolemia and pain on tissue oxygen saturation (measurement sites: cerebral, deltoid,

187 casian individuals) underwent retinal vessel oxygen saturation measurements using dual-wavelength oxi

188 y has a significant impact on retinal vessel oxygen saturation measurements using dual-wavelength ret

189 High flash intensities lead to supranormal oxygen saturation measurements with a magnified effect i

190 ificant difference in mean regional cerebral oxygen saturation (median % +/- interquartile range) in

191 medications (p = 0.02) and had lower resting oxygen saturation (median 89% vs. 95%, p < 0.001).

192 s in other clinical outcomes for either home oxygen saturation monitoring or home weight monitoring.

193 ntra-operative BiSpectral index and cerebral oxygen saturation monitoring to enable optimisation of a

194 ed on a physiological basis (with the use of oxygen-saturation monitoring in selected infants), at 36

195 ysplasia, confirmed by means of standardized oxygen-saturation monitoring, at a postmenstrual age of

196 nds, body position, electrocardiography, and oxygen saturation (n = 136); or level 4 (L4), which incl

197 re for various home monitoring strategies of oxygen saturation (n=494) or weight (n=472), adjusting f

198 he apnea-hypopnea index (AHI), and nocturnal oxygen saturation (O2sat) parameters, and relevant comor

199 Oxygen saturation of </=90%, tachypnea, and tachycardia

200 blood pressure of >110 mm Hg, and peripheral oxygen saturation of <85%) defined INSTABILITYmin.

201 al dysfunction, 24-h AHI, CAI, and time with oxygen saturation of <90% were independent predictors of

202 ls, we evaluated the effects of targeting an oxygen saturation of 85 to 89%, as compared with a range

203 mmends a permissive hypoxaemic target for an oxygen saturation of 90% for children with bronchiolitis

204 with suspected myocardial infarction and an oxygen saturation of 90% or higher were randomly assigne

205 breaths per min for children) and a room air oxygen saturation of 93% or more.

206 on during pain crisis could affect the local oxygen saturation of hemoglobin when oxygen delivery is

207 f mannitol on brain tissue PO2 and on venous oxygen saturation of the superior sagittal sinus (n = 5

208 gen saturation, brain tissue PO2, and venous oxygen saturation of the superior sagittal sinus values

209 In extremely preterm infants, targeting oxygen saturations of 85% to 89% compared with 91% to 95

210 with mild to moderate bronchiolitis and true oxygen saturations of 88% or higher.

211 f a lag phase, which could be overcome under oxygen saturation or by reoccupying the buried site with

212 arterial pressure, and either central venous oxygen saturation or lactate clearance.

213 ry hypertension, was not predicted by either oxygen saturation or sleep variables with multivariable

214 sue oxygen saturation but not central venous oxygen saturation or thenar tissue oxygen saturation are

215 We did not find any associations of home oxygen saturation or weight monitoring with mortality or

216 [CI, 1.31 to 1.77] per 20 mm Hg) and initial oxygen saturation (OR, 1.16 [CI, 1.01 to 1.33] per 5%).

217 No pulmonary emboli, alterations in oxygen saturation, or hemodynamics occurred with either

218 St George's Respiratory Questionnaire score, oxygen saturation, or lung function tests.

219 ygen saturation (p = .04) and deltoid tissue oxygen saturation (p = .002), and masseter tissue oxygen

220 was consistently higher than masseter tissue oxygen saturation (p = .04) and deltoid tissue oxygen sa

221 was consistently higher than deltoid tissue oxygen saturation (p = .04).

222 ed by respiratory inductive plethysmography, oxygen saturation, perfusion index, regional cerebral an

223 erial blood pressure, electrocardiogram, and oxygen saturation plethysmography activity were recorded

224 ysmography), heart rate (electrocardiogram), oxygen saturation (pulse oximetry), and brachial artery

225 a lower (85 to 89%) or a higher (91 to 95%) oxygen-saturation range.

226 ance of > or = 500 meters with a decrease in oxygen saturation received bosentan or placebo.

227 wine "forced ageing" process under different oxygen saturation regimes at 60 degrees C.

228 PVRI, whereas cardiac index and mixed venous oxygen saturation remained unchanged.

229 the procedure to record oxygen requirement, oxygen saturation, respiratory rate, consciousness level

230 venous oxygen saturation and central venous oxygen saturation (rs = 0.55; p < .001), the limits of a

231 infrared spectroscopy monitoring of cerebral oxygen saturation (rSo(2)) has become routine in many ce

232 kept between 50 and 60 mmHg, and peripheral oxygen saturation (Sao(2)) was kept above 80%.

233 The arterial oxygen saturation (SaO(2)), bicarbonate concentration, b

234 as a respiratory rate (RR) < 55 and room air oxygen saturation (SaO2) >/= 97%, and severe illness (n

235 Overnight nocturnal oxygen saturation (SaO2) is a clinically relevant and ea

236 t of postoperative IS on hypoxemia, arterial oxygen saturation (Sao2) level, and pulmonary complicati

237 ts and seconds with less than 90% hemoglobin oxygen saturation (Sao2) per hour of recording.

238 Distal occlusive pressure and toe oxygen saturation (Sao2) were measured for 5 minutes und

239 including end-tidal carbon dioxide (ETCO2), oxygen saturation (SaO2), intra-arterial blood pressure,

240 nd between: left thalamus mI/Cr and baseline oxygen saturation (SaO2); right putamen tCho/Cr and apne

241 Central venous oxygen saturation (ScvO2) in the superior vena cava is p

242 actate >4 mM), and continuous central venous oxygen saturation (Scvo2) monitoring for quantitative re

243 This suggests that oxygen saturation should not be the only factor in the d

244 Thus, femoral venous oxygen saturation should not be used as surrogate for ce

245 ur subjects and was also associated with low oxygen saturation (sleep or waking).

246 s of total hemoglobin concentration (Hb(T)), oxygen saturation (So(2)), and tissue reduced scattering

247 pectroscopy (FDNIRS-DCS) to measure cerebral oxygen saturation (SO2) and an index of cerebral blood f

248 y of PAI for analysis of placental and fetal oxygen saturation (sO2) in mice.

249 e used to compare change in tumor hemoglobin oxygen saturation (sO2) levels and BOLD signal in respon

250 oxyhemoglobin (HbO2), deoxyhemoglobin (HbR), oxygen saturation (sO2), blood flow (BF) and rate of oxy

251 rameters, the blood flow rate and hemoglobin oxygen saturation (sO2), must be measured together.

252 ding total hemoglobin concentration (C(Hb)), oxygen saturation (sO2), sO2 gradient (VsO2), flow speed

253 The optimal oxygen saturation (SpO2) target for extremely preterm in

254 ailure (partial oxygen pressure <60 mm Hg or oxygen saturation [SpO2] </=90% when breathing room air

255 eters (patients treated with oxygen if pulse oxygen saturation [SpO2] <94%) or modified oximeters (di

256 line flights (decreased peripheral capillary oxygen saturation [SpO2] and increased radiation exposur

257 b), total Hb (ctTHb = ctO(2)Hb + ctHHb), and oxygen saturation (stO(2) = ctO(2)Hb/ctTHb) in tumor and

258 aneous reflectance spectrophotometry (venous oxygen saturation StO2 and relative tissue hemoglobin co

259 h native endobronchial tissues, donor tissue oxygen saturations (Sto2) were reduced in the upper lobe

260 he cohort of 17 patients with baseline tumor oxygen saturation (%StO2) greater than the 77% populatio

261 Perfusion, venous oxygen saturation SvO2, and T2* were each quantified in

262 nagement of infants with bronchiolitis to an oxygen saturation target of 90% or higher is as safe and

263 Mortality was increased with the lower-oxygen-saturation target (22.1%, vs. 18.2% with the high

264 Use of an oxygen-saturation target range of 85 to 89% versus 91 to

265 ion target (22.1%, vs. 18.2% with the higher-oxygen-saturation target; relative risk, 1.25; 95% CI, 1

266 d assess the benefits and risks of different oxygen saturation targets in acute respiratory infection

267 A drop in oxygen saturation that required supplemental oxygen was

268 es of diffusing capacity of carbon monoxide, oxygen saturation, the 6-minute-walk distance, St George

269 For every 1% drop in the average asleep oxygen saturation, there was a 2.1 g/m(2.7) increase in

270 Infants who did not achieve predetermined oxygen saturation thresholds underwent echocardiography.

271 al pressure of oxygen or arterial hemoglobin oxygen saturation to individualized target values, with

272 ume (respiratory inductive plethysmography), oxygen saturation, transcutaneous carbon dioxide, and in

273 oembolism; oxygen therapy for those with low oxygen saturation; treatment of left ventricular failure

274 turation in cerebral tissue (cerebral tissue oxygen saturation [tSo2]) before, during, and after bloo

275 PAT was demonstrated by mapping the cerebral oxygen saturation via multi-wavelength irradiation in be

276 s 0.94 (0.89-1.05) (p = 0.027), jugular vein oxygen saturation was 79.2 (71.1-81.8) versus 83.3% (76.

277 Median lowest arterial oxygen saturation was 92% with apneic oxygenation versus

278 given via nasal tubes at 3 L/min if baseline oxygen saturation was 93% or less and at 2 L/min if oxyg

279 A lower (vs. higher) target range of oxygen saturation was associated with a lower rate of se

280 curves analyses showed that masseter tissue oxygen saturation was better predictor of central venous

281 Retinal arteriolar and venular oxygen saturation was comparable at flash settings of 27

282 n saturation (p = .002), and masseter tissue oxygen saturation was consistently higher than deltoid t

283 the 6-hr resuscitation period, thenar tissue oxygen saturation was consistently higher than masseter

284 Regional blood oxygen saturation was determined by near-infrared spectr

285 ctal pulse oximetry, while regional cerebral oxygen saturation was estimated using near-infrared spec

286 saturation was 93% or less and at 2 L/min if oxygen saturation was greater than 93%.

287 t was highly constrained, and red blood cell oxygen saturation was low and inappropriately variable.

288 eter in the Australian trial and those whose oxygen saturation was measured with a revised oximeter i

289 is outcome was evaluated among infants whose oxygen saturation was measured with any study oximeter i

290 ts at H0 and H6, whereas mean central venous oxygen saturation was preserved but significantly lower

291 Surviving Sepsis Campaign guidelines, tissue oxygen saturation was recorded every other hour at the l

292 hypoxia (as quantified by low central venous oxygen saturation) was not associated with major coagula

293 venous oxygen saturation and central venous oxygen saturation were assessed, including the differenc

294 rebral artery flow velocity and jugular vein oxygen saturation were measured at the end of each step.

295 ow velocities using Doppler and jugular vein oxygen saturation were measured in both strategies 18 ho

296 Heart rate (HR) and oxygen saturation were monitored.

297 venous oxygen saturation and central venous oxygen saturation were similar.

298 nopathy of prematurity when lower targets of oxygen saturation were used.

299 pressure, heart rate, respiratory rate, and oxygen saturation) were collected at the same time point

300 n the redox chemistry of Cu species, than at oxygen saturation, where the value was 0.6.

301 ral microvascular blood flow and dynamics of oxygen saturation with Perfusion, intravascular SvO2, an