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1 ry rehabilitation programs, and supplemental oxygen therapy.
2 ing illness who are ineligible for long-term oxygen therapy.
3 correlated with the duration of supplemental-oxygen therapy.
4 were discharged on tube feedings and 22% on oxygen therapy.
5 anent mechanical ventilation or supplemental oxygen therapy.
6 ion to the hospital, and 24 had been on home oxygen therapy.
7 hanical ventilatory support and supplemental oxygen therapy.
8 py, and with oxygen therapy compared with no oxygen therapy.
9 development and healing of lungs injured by oxygen therapy.
10 esumption of normal feeding--and duration of oxygen therapy.
11 ith rectal bleeding, benefit from hyperbaric oxygen therapy.
12 roves oxygenation compared with conventional oxygen therapy.
13 eived high-flow therapy and 263 conventional oxygen therapy.
14 d hypoxaemia compared with standard low-flow oxygen therapy.
15 ow oxygen therapy, and 79 (35%) to high-flow oxygen therapy.
16 herapy, and ten (13%) had received high-flow oxygen therapy.
17 ildhood pneumonia and hypoxaemia is low-flow oxygen therapy.
18 s of conventional compared with conservative oxygen therapy.
19 ompared with standard low-flow and high-flow oxygen therapies.
20 d in 87 of 414 patients with high-flow nasal oxygen therapy (21.0%) and 91 of 416 patients with BiPAP
21 ventilation-free days compared with standard oxygen therapy (25.4 vs 23.2 days; absolute difference,
22 female sex, 56.9%) were assigned to standard oxygen therapy (367 patients), CPAP (346 patients), or N
23 ith BiPAP [5.5%] and 28 with high-flow nasal oxygen therapy [6.8%]; P = .66) (absolute difference, 1.
24 re respiratory support (55 [40] vs 54 [42]), oxygen therapy (88 [41] vs 91 [59]), and hospitalization
25 ortality between patients receiving standard oxygen therapy (9.8%) and those undergoing noninvasive v
26 reduced the postmenstrual age at last use of oxygen therapy (adjusted mean difference, -0.8 weeks; 95
28 est that conditioned high-flow nasal cannula oxygen therapy after extubation improves oxygenation com
30 early noninvasive ventilation compared with oxygen therapy alone did not reduce 28-day mortality.
31 s were evaluated: baseline with conventional oxygen therapy and high-flow nasal cannula at 20, 40, an
32 Infants with late PH had greater duration of oxygen therapy and increased mortality in the first year
33 ecessitating the institution of supplemental oxygen therapy and positive pressure mechanical ventilat
34 asive ventilation over time, the duration of oxygen therapy and the rate of oxygen dependence at 36 w
36 entional (n = 218) or conservative (n = 216) oxygen therapy and were included in the modified intent-
37 therapy by bubble CPAP, 67 (30%) to low-flow oxygen therapy, and 79 (35%) to high-flow oxygen therapy
39 scalation, intravenous fluid administration, oxygen therapy, and diagnostic tests were all increased
41 sisted ventilation provided, the duration of oxygen therapy, and oxygen requirements at 36 weeks of a
42 bubble CPAP, 16 (24%) had received low-flow oxygen therapy, and ten (13%) had received high-flow oxy
43 er with the available literature, normobaric oxygen therapy appears a promising therapy for short-las
45 ronic lung disease diagnosed by the need for oxygen therapy at a postmenstrual age of 36 weeks, need
48 and metabolic disturbance than does standard oxygen therapy but has no effect on short-term mortality
49 domly allocated 79 (35%) children to receive oxygen therapy by bubble CPAP, 67 (30%) to low-flow oxyg
50 h severe pneumonia and hypoxaemia to receive oxygen therapy by either bubble CPAP (5 L/min starting a
51 ients (33.4%), mainly the need for prolonged oxygen therapy by nasal cannula (n = 235; 19.6%) and ate
52 piratory failure, the use of high-flow nasal oxygen therapy compared with intermittent BiPAP did not
54 ng-lasting sensorimotor deficits, normobaric oxygen therapy completely prevented sensorimotor deficit
57 ed trial, patients were assigned to standard oxygen therapy, CPAP (5 to 15 cm of water), or NIPPV (in
58 ggestions of potential harm from unnecessary oxygen therapy, critically ill patients spend substantia
62 randomly assigned to receive high-flow nasal oxygen therapy delivered continuously through a nasal ca
63 r less to high-flow oxygen therapy, standard oxygen therapy delivered through a face mask, or noninva
65 rus coinfection (aOR, 1.4; 95% CI, 1.2-2.1), oxygen therapy during admission (aOR, 1.6; 95% CI, 1.1-2
66 f hospitalization, duration of symptoms, and oxygen therapy during admission, pneumococcal loads >/=1
67 time in the use of assisted ventilation and oxygen therapy during the newborn period and in lung fun
68 tegories: these included oxygen and reactive oxygen therapy; energy and radiation-based therapies; nu
69 increases in arterial O2 content, normobaric oxygen therapy experimentally induces significant increa
72 ational study of patients starting long-term oxygen therapy for COPD in Sweden between 1 October 2005
74 thy of prematurity is a major side effect of oxygen therapy for preterm infants, and is a leading cau
75 icoagulation for those with thromboembolism; oxygen therapy for those with low oxygen saturation; tre
77 apy group (11.6%) and 44 in the conventional oxygen therapy group (20.2%) died during their ICU stay
78 p had treatment failure than in the low-flow oxygen therapy group (relative risk [RR] 0.27, 99.7% CI
79 n the bubble CPAP and those in the high-flow oxygen therapy group (RR 0.50, 99.7% 0.11-2.29; p=0.175)
80 Occurrences were lower in the conservative oxygen therapy group for new shock episode (ARR, 0.068 [
81 group and 31 of 144 (21.5%) in the standard oxygen therapy group had died (absolute difference, -6.5
82 oup and in 66 of 145 (45.5%) in the standard oxygen therapy group within+ 7 days after randomization
83 oup, ten (15%) children died in the low-flow oxygen therapy group, and ten (13%) children died in the
85 ished to date, early-administered normobaric oxygen therapy had no significant effect on clinical out
87 luate the ability of low-pressure hyperbaric oxygen therapy (HBOT) to improve behavioral and neurobio
88 addition to the above therapies, hyperbaric oxygen therapy (HBOT) was implemented as adjuvant treatm
89 use of racemic epinephrine, steroids, helium-oxygen therapy (heliox), or noninvasive positive pressur
94 nce and guidelines for the use of hyperbaric oxygen therapy in carbon monoxide-poisoned infants and c
100 factors contributing to CO2 retention due to oxygen therapy in patients with acute exacerbations of C
102 that clinicians should prescribe continuous oxygen therapy in patients with COPD who have severe res
106 of carbon monoxide poisoning and hyperbaric oxygen therapy in the fetus, the newborn, the infant, an
107 he current evidence for the use of high-flow oxygen therapy, inhaled gases, and aerosols in the care
113 Objective: To test if high-flow conditioned oxygen therapy is noninferior to NIV for preventing post
120 m of this report to emphasize the point that oxygen therapy might have major adverse physiologic effe
122 frequent PPCs (eg, atelectasis and prolonged oxygen therapy need) deserve increased attention and int
125 Nevertheless, the effects of normobaric oxygen therapy on infarct volume in rodent models have b
127 ized to undergo either high-flow conditioned oxygen therapy or NIV for 24 hours after extubation.
128 cannulae with usual care (i.e., conventional oxygen therapy or noninvasive ventilation) in adults wit
130 There were no differences in duration of oxygen therapy (p = 0.74) or time to resolution of sympt
131 ere we tested the hypothesis that normobaric oxygen therapy prevents both selective neuronal loss and
132 adding home noninvasive ventilation to home oxygen therapy prolonged the time to readmission or deat
133 al cannula oxygen compared with conventional oxygen therapy reduced the risk of reintubation within 7
134 l surgery, use of NIV compared with standard oxygen therapy reduced the risk of tracheal reintubation
135 the children who received oxygen by low-flow oxygen therapy (RR 0.25, 95% CI 0.07-0.89; p=0.022).
136 red oxygen of 300 mm Hg or less to high-flow oxygen therapy, standard oxygen therapy delivered throug
139 clinical studies suggesting that keeping the oxygen therapy to an "acceptable" minimum in premature b
140 : Patients were randomly assigned to receive oxygen therapy to maintain Pao2 between 70 and 100 mm Hg
141 rations can have a valuable role in bringing oxygen therapy to patients and hospitals in countries wh
142 s were randomly assigned to receive standard oxygen therapy (up to 15 L/min to maintain SpO2 of 94% o
143 tibiotics, intravenous fluid administration, oxygen therapy, vasopressors, and diagnostic tests.
144 hours or longer, a conservative protocol for oxygen therapy vs conventional therapy resulted in lower
148 between noninvasive ventilation and standard oxygen therapy was death within 7 days after the initiat
149 assess whether routine prophylactic low-dose oxygen therapy was more effective than control oxygen ad
151 undergone extubation, high-flow conditioned oxygen therapy was not inferior to NIV for preventing re
153 s of active preoxygenation efforts with 100% oxygen therapy with a noncollapsing resuscitator bag and
154 , there is a wide variation in approaches to oxygen therapy within neonatal intensive care units.
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