ライフサイエンスコーパス: oxygen-free

1 and a sulfonated phenanthroline ligand in an oxygen-free 7:3:1 ethanol/water/toluene mixture at 70 de

2 ut oxidative hydrolysis of six samples in an oxygen-free atmosphere at 150 degrees C for 90 min.

3 We find no oxygen-free composition that fits the seismological data

4 itated enzyme stabilization and operation in oxygen free conditions.

5 rformance and possibilities for operation in oxygen-free conditions of an oxidase enzyme biosensor us

6 O is unable to react with nucleophiles under oxygen-free conditions, suggesting that its higher oxide

7 lic substrates, without the need for dry and oxygen-free conditions.

8 Supplemental oxygen-free days as a clinical endpoint may allow smalle

9 e LRTI had a median of 16 fewer supplemental oxygen-free days than those presenting with URTI (P < .0

10 Supplemental oxygen-free days were defined as any day while alive aft

11 ent conditions a significant fraction of the oxygen-free edge sites are neither H-terminated nor unad

12 conversion under ambient conditions as in an oxygen-free environment in several unsaturated polyphosp

13 However, the requirement for an oxygen-free environment when classic radical polymerizat

14 ng of various ligands to OAM crystals (in an oxygen-free environment) leads to transimination in the

15 ure to low-intensity laser irradiation in an oxygen-free environment.

16 paration and manipulation of reactants in an oxygen-free environment; trace quantities of O2 lead to

17 e functionality of oxidoreductase enzymes in oxygen-free environments.

18 ormation of methane by microbes that live in oxygen-free environments.

19 Here we present oxygen-free flow lithography via inert fluid-lubrication

20 The oxygen-free gas was routed through stainless steel tubin

21 g the fraction of substrate that reacts with oxygen free in solution.

22 -gas shift activity of platinum supported on oxygen-free multiwalled carbon nanotubes.

23 evolved hydrogen facilitates preparation of oxygen-free powders.

24 lity to sequester iron from participation in oxygen free radical formation is consistent with a cytop

25 se results directly demonstrate an increased oxygen free radical generation during hypoxia and sugges

26 The direct demonstration of oxygen free radical generation during hypoxia is the cri

27 The role of oxygen free radical generation during reversible focal c

28 the hypothesis that maternal hypoxia induces oxygen free radical generation in the fetal guinea pig b

29 ly decreased the hypoxia-induced increase in oxygen free radical generation in the term fetal guinea

30 reserved liver cells is not mediated by: (1) oxygen free radical generation or improved by antioxidan

31 Allantoin, a marker of oxygen free radical generation, decreased by 20% after a

32 f neutrophil phagocytosis and a reduction in oxygen free radical generation, which may contribute to

33 are deficient in myeloid cell activation and oxygen free radical generation.

34 TP metabolism are the primary candidates for oxygen free radical generation: (a) MPTP oxidation to MP

35 physiologic outcome following treatment with oxygen free radical inhibitors and scavengers.

36 ated metabolic changes leading to changes in oxygen free radical levels, which in turn lead to the in

37 asic pattern consisting of both acute phase (oxygen free radical mediated) and subacute phase (neutro

38 Oxygen free radical production also increased MCP-1 mRNA

39 ) isolated from piglet cortex to measure CEC oxygen free radical production and determine its role in

40 n is increased xanthine oxidase (XO)-derived oxygen free radical production and endothelial dysfuncti

41 tion in nNOS-/- mice may relate to decreased oxygen free radical production and related NO reaction p

42 plug capillary-sized pores and show enhanced oxygen free radical production may account for the exces

43 -/-) embryonic fibroblasts demonstrated high oxygen free radical production when exposed to hemin, hy

44 Blockade of oxygen free radical production with superoxide dismutase

45 inergic PC12 cell cultures, does not involve oxygen free radical production, but rather may be caused

46 n were increased in response to cytokines or oxygen free radical production, but the magnitude and du

47 A 55% increase in oxygen free radical production, determined by fluorescen

48 f energy metabolism, oxygen consumption, and oxygen free radical production, it becomes imperative to

49 (NO radical) exhibit changes in the rate of oxygen free radical production.

50 (+)/H(+) exchange blocker (amiloride), or an oxygen free radical scavenger (vitamin E).

51 se results support the concept of developing oxygen free radical scavengers for both AD and PD and fu

52 ectron oxidation of hydrogen peroxide to the oxygen free radical, superoxide.

53 ly of UV radiation-induced, but also of some oxygen free radical-induced, DNA lesions.

54 d to damage dopamine-secreting neurons by an oxygen free radical-mediated mechanism.

55 Thus, reductions in oxygen free radical-mediated vascular injury may contrib

56 In order to promote oxygen-free radical generation, hypoxanthine (n=9) or xa

57 in piglets have shown that the generation of oxygen free radicals (O(-)(2)) following traumatic brain

58 The generation of oxygen free radicals (OFR) and tissue infiltration by ac

59 t hypoxia induces an increased production of oxygen free radicals (OFR) in the brain of the guinea pi

60 Oxygen free radicals (OFRs) have been implicated in the

61 rophils by contact, leading to production of oxygen free radicals accompanied by release of granule p

62 (2) Oxygen free radicals also appear to mediate the hypoxic-

63 The UVA-dependent generation of oxygen free radicals also oxidized ASA at a 10(3) faster

64 y to alloxan, a generator of highly reactive oxygen free radicals and a potent beta-cell toxin.

65 ve stress; increased protein modification by oxygen free radicals and an elevated concentration of th

66 Because NFkappaB is a target of oxygen free radicals and Bcl-2 is an antioxidant gene, w

67 This injury is attributed in part to oxygen free radicals and has been partially ameliorated

68 ic newborn piglets through the generation of oxygen free radicals and induction of lipid peroxidation

69 Exercise increases the generation of oxygen free radicals and lipid peroxidation.

70 as previously believed to be accomplished by oxygen free radicals and other reactive oxygen species g

71 n-superoxide dismutase (SOD1) would diminish oxygen free radicals and reduce alcohol-induced liver in

72 elated cytokines; b) increased production of oxygen free radicals associated with ischemia/reperfusio

73 Generation of oxygen free radicals by treatment of telencephalic mitoc

74 The production of oxygen free radicals catalysed by non-haem iron was inve

75 ate that CECs produce significant amounts of oxygen free radicals following ischemia, primarily from

76 3'-blocking groups formed from the action of oxygen free radicals generated during normal cellular me

77 These results suggest that a burst of oxygen free radicals generated during the initial period

78 The involvement of oxygen free radicals has been suggested in c-fos activat

79 Oxygen free radicals have been implicated in beta-cell d

80 Elevated levels of oxygen free radicals have been implicated in the pathway

81 Oxygen free radicals have been invoked as mediators of t

82 Oxygen free radicals have been proposed to mediate amylo

83 These results not only support a role for oxygen free radicals in beta-AP toxicity but also highli

84 data, for the first time, suggest a role of oxygen free radicals in causing abnormality of female re

85 These data support a role of oxygen free radicals in promoting post-anoxic mitochondr

86 e, from the production and detoxification of oxygen free radicals in the mitochondrion to the efficac

87 f the Bad pathway after tFCI and the role of oxygen free radicals in the regulation of apoptosis rema

88 We tested the hypothesis that oxygen free radicals induce an increase in ODC activity

89 ment of S phase cells with agents that cause oxygen free radicals induces the dephosphorylation of DN

90 hat in tumor cells, endogenous production of oxygen free radicals may be a major factor in promoting

91 nhanced muscle fatigue, whereas formation of oxygen free radicals may be attenuated by endogenous pro

92 Oxygen free radicals may play a pivotal role in the mito

93 Oxygen free radicals oxidize arachidonic acid to a compl

94 However, the role of oxygen free radicals produced after ischemia/reperfusion

95 Short-lived oxygen free radicals react with the spin trap and produc

96 d coronary endothelial dysfunction caused by oxygen free radicals released during reperfusion.

97 ine and xanthine, xanthine oxidase generates oxygen free radicals that cause postischemic injury.

98 es are associated with the overproduction of oxygen free radicals that inflict cell damage.

99 Activation and oxygen free radicals were assessed using gp91(phox-/-) m

100 Oxygen free radicals were measured by ESR spectroscopy i

101 ble of generating significantly greater ROS (oxygen free radicals) than nondiabetic blood (P < 0.05).

102 such mucosal injury is initially mediated by oxygen free radicals, and because mitogen-activated prot

103 se is especially vulnerable to inhibition by oxygen free radicals, and the upstream metabolites, pyru

104 MPP(+), unlike rotenone, did not produce oxygen free radicals, but rather blocked ATP production

105 Oxygen free radicals, generated by cerebral ischemia, ha

106 Anticancer modalities based on oxygen free radicals, including photodynamic therapy and

107 (1) Oxygen free radicals, particularly hydroxyl radical (OH.

108 Variable factors, including accumulation of oxygen free radicals, protein conformational changes, de

109 t the idea that MPTP toxicity is mediated by oxygen free radicals, we assessed lipid peroxidation and

110 pathophysiologic production of cytokines and oxygen free radicals, which potentiate organ injury in s

111 nt in tumor tissue prevents the formation of oxygen free radicals.

112 n thiols and inhibits cellular damage due to oxygen free radicals.

113 are related to its role in the generation of oxygen free radicals.

114 s in PD involves the accumulation of harmful oxygen free radicals.

115 stnatal exposure to environmental sources of oxygen free radicals.

116 tients may be related to oxidative damage by oxygen free radicals.

117 smutases (SODs) protect cells from damage by oxygen free radicals.

118 of MCP-1 may be stimulated by cytokines and oxygen free radicals.

119 disorders linked to excessive generation of oxygen free radicals.

120 philic response, which causes the release of oxygen free radicals.

121 are mediated via the formation of NE-derived oxygen free radicals.

122 cytochrome oxidase, and (5) the formation of oxygen free radicals.

123 Both STGP and LTGP gels produced oxygen free radicals.

124 , at least in part through the generation of oxygen free radicals.

125 nhance phagocytic activity and generation of oxygen free radicals.

126 ave been proposed as a significant source of oxygen free radicals.

127 he affected tissues produce large amounts of oxygen-free radicals and NO.

128 hypoxic-ischemic brain injury by generating oxygen-free radicals during reperfusion.

129 Oxygen-free radicals formed during normal aerobic cellul

130 Superoxide anion and other oxygen-free radicals have been implicated in the pathoge

131 ): Each does indeed bind copper and scavenge oxygen-free radicals in vivo.

132 e hypoxia results in increased generation of oxygen-free radicals including nitric oxide (NO), expres

133 We examined the role of XO in generating oxygen-free radicals that cause brain injury, hypothesiz

134 These results suggest that oxygen-free radicals, especially superoxide anions, are

135 and which are formed in DNA by the action of oxygen-free radicals.

136 In this study, the oxygen-free reaction of U(VI) with varying quantities of

137 in inorganic oxides and hydroxides using an oxygen-free solution containing a biradical polarization

138 escent (Phi = 0.2-0.3) in a room-temperature oxygen-free solution.

139 enzymatic systems were designed to maintain oxygen-free solutions in open, small volume electrochemi

140 esponse over prolonged (6-7 h) operations in oxygen-free solutions, indicating no depletion of the in

141 provide convenient biosensing of glucose in oxygen-free solutions.

142 h Eubacteria and Archeabacteria in the early oxygen-free stages of the earth's evolution.

143 uboxic (0.2% oxygen), and anoxic conditions (oxygen-free with nitrate).