ライフサイエンスコーパス: ozone

1 f difenoconazole and linuron from carrots by ozone.

2 n the two-pollutant multivariable model with ozone.

3 er disinfection by chlorine, chloramines, or ozone.

4 dants besides OH and NO3 radicals as well as ozone.

5 e preparation of ozonides without the use of ozone.

6 anced UV-B due to depletion of stratospheric ozone.

7 s in the atmosphere, are readily oxidized by ozone.

8 d three-center electron delocalization as in ozone.

9 gen species (ROS) from oxygen, peroxides, or ozone.

10 ith 60 s of activation), and a high level of ozone (13 ppm with 60 s of activation).

11 the IL-33 receptor, ST2, and then exposed to ozone (2 ppm for 3 hr).

12 ion from exposure to common indoor levels of ozone (23 +/- 2 ppb).

13 Ozone, a strong oxidant and disinfectant, seems ideal to

14 via an abiotic process, and implies that the ozone activated halogenation of a variety of natural and

15 on of these materials can involve the direct ozone activated seawater halogenation of N-methylbipyrro

16 Here we use ozone-activated silver-gold alloys in the form of nanopo

17 d 10 parts per billion (10-9) in warm-season ozone (adjusted by PM2.5) were statistically significant

18 particulate matter (PM2.5) and tropospheric ozone air pollution, affecting human health, crop yields

19 Thus, NOx emission controls to improve ozone air quality have a significant cobenefit in reduci

20 y of spas treated with chlorine, bromine, or ozone, along with pools treated with these same disinfec

21 Obesity and ozone also interacted to promote type 2 cytokine product

22 NO is a precursor to tropospheric ozone, an air pollutant and greenhouse gas.

23 alyzer (CAPS) for NO2 measurements, and a UV ozone analyzer.

24 ation are modest, mostly less than 1 ppb for ozone and 0.5 mug m(-3) for fine particulate matter (PM2

25 mong which 853 were precursors attenuated by ozone and 165 were TPs.

26 g-linear concentration-response function for ozone and an integrated exposure-response model for PM2.

27 We explored the associations between ozone and daily cause-specific mortality in China.

28 Associations between ozone and daily mortality due to respiratory and chronic

29 y amine pharmaceuticals that they react with ozone and form the corresponding N-oxides.

30 layer undergoes frequent rapid depletions in ozone and gaseous elemental mercury due to reactions wit

31 Ozone and H2O2 will not be important photochemical OH so

32 controlled by the reactivity of TrOCs toward ozone and HO(*) radicals.

33 lating the effects of input uncertainties on ozone and NO2 output, to allow robust global sensitivity

34 nificant differences in associations between ozone and nonaccidental mortality according to region, s

35 ined with anthropogenic emissions to produce ozone and particulate matter has long been recognized.

36 dependent of other common pollutants such as ozone and particulate matter.

37 ental Protection Agency (EPA), not including ozone and particulate matter.

38 an vegetation and corresponding ground-level ozone and particulate matter.

39 The largest reductions for ozone and PM occur in urban areas due to lower mobile so

40 these surfaces, can have complex impacts on ozone and PM2.5 concentrations.

41 possible association between both long-term ozone and PM2.5 exposure and depression onset.

42 ding sodium hypochlorite, hydrogen peroxide, ozone and sodium periodate, are described in this review

43 aerosols and of atmospheric oxidants such as ozone and the hydroxyl radical, which controls the self-

44 The distribution of gases such as ozone and water vapour in the stratosphere - which affec

45 increase of 10 mug/m3 in PM2.5 (adjusted by ozone) and 10 parts per billion (10-9) in warm-season oz

46 ambient fine particulate matter (PM2.5) and ozone, and at levels below the current daily NAAQS, and

47 mass spectrometry (CIMS) during the Bromine, Ozone, and Mercury Experiment (BROMEX) near Barrow, Alas

48 he prevalence of volatile organic compounds, ozone, and mercury in the Arctic lower troposphere.

49 luding exposures to fine particulate matter, ozone, and nitrogen dioxide.

50 rate of the reaction of the fatty acid with ozone, and that lyotropic-phase formation also occurs in

51 urally informative product ions arising from ozone- and collision-induced dissociation.

52 We observed ozone-associated increases in all respiratory, asthma, a

53 namic diameter less than 2.5 mum (PM2.5) and ozone at an approximate 11 km x 11 km resolution with sa

54 rse effects related to exposure to PM2.5 and ozone at concentrations below current national standards

55 Ozone attack on resins, asphaltenes, and soil organic ma

56 was proposed for gallic acid degradation by ozone based on Criegee mechanism.

57 h NO and isoprene emissions, NO2 photolysis, ozone BCs, and deposition velocity being among the most

58 ich is important for the global tropospheric ozone budget.

59 The uptake of ozone by forest is estimated through several meteorologi

60 ay a common spatial pattern in the uptake of ozone by forests with a marked change in the magnitude,

61 rC and BC slow the net rate of production of ozone by up to 18% and lead to reduced concentrations of

62 Exposure to ozone caused an additional 254 000 (95% UI 97 000-422 00

63 RESULTS AND DISCUSSION: Ozone caused greater increases in BAL IL-33, neutrophils

64 by other environmentally relevant oxidants (ozone, chlorine dioxide, and phosphate and carbonate rad

65 owever, our analysis shows that tropospheric ozone concentration and subtropical intrusions account

66 ge of pesticides increases with increases in ozone concentration and the time of treatment.

67 d find that an additional day with a maximum ozone concentration greater than 120 ppb is associated w

68 12% +/- 0.83% relative to a day with maximum ozone concentration less than 60 ppb.

69 The highest ozone concentration observed during that period was appr

70 level, a 10-mug/m(3) increase in 8-h maximum ozone concentration was associated with 0.24% [95% poste

71 An increase of 10 mug/m(3) in weekly ozone concentration was associated with a decrease in FV

72 that included the negative control exposure (ozone concentrations 1 day after the emergency departmen

73 oximately 45% of the variability in JJA MDA8 ozone concentrations and approximately 30% variability i

74 in the relationship between rice yields and ozone concentrations and find that an additional day wit

75 e correlation between power measurements and ozone concentrations as well as ozone quenching effects

76 Changes in 8-h ozone concentrations depend on the potential change in U

77 ime-differentiated pricing aimed at reducing ozone concentrations had particulate matter reduction co

78 ugust (JJA) daily maximum 8-h average (MDA8) ozone concentrations in the eastern United States based

79 Mean 4-hour afternoon tropospheric ozone concentrations in Western Kenya increased by up to

80 Multiphase reaction kinetics at gaseous ozone concentrations of 131, 480, and 965 parts-per-bill

81 amage from ozone, we find that the increased ozone concentrations result in an increase in AOT40 expo

82 other seasons potentially because of higher ozone concentrations that may interfere with GOM samplin

83 ivity approach (i.e., modeling the change of ozone concentrations that result from modifying precurso

84 ratures in the East, which increases surface ozone concentrations there.

85 We model ozone concentrations under baseline and policy condition

86 Four-month averaged CO and ozone concentrations were negatively associated with FEV

87 Measured PM2.5, nitrogen dioxide, and ozone concentrations were spatially interpolated using e

88 We find that increases in mean ozone concentrations, SUM60, and AOT40 during panicle fo

89 ts of increased NO emissions on tropospheric ozone concentrations.

90 ng the potential health benefits of reducing ozone concentrations.

91 s precursor at low, atmospherically relevant ozone concentrations.

92 The first was mainly ozone controlled, while the second phase was more relate

93 evising the conventionally accepted value of ozone cross section at 253.65 nm.

94 ospheric concentration of dichloromethane-an ozone-depleting gas not controlled by the Montreal Proto

95 ons in the atmospheric concentration of many ozone-depleting gases, such as chlorofluorocarbons.

96 limate change is masking ozone recovery from ozone-depleting substances in some regions and will incr

97 ts, the atmospheric loading of anthropogenic ozone-depleting substances is decreasing.

98 ts significant contribution to stratospheric ozone depletion and its potent greenhouse effect, nitrou

99 the Montreal Protocol due to concerns about ozone depletion and provide an illustration of how emiss

100 bromine chemistry has been shown to initiate ozone depletion events, and it has long been hypothesize

101 o those associated with modern stratospheric ozone depletion over Antarctica-plausibly link the Mount

102 ide (N2 O) is a powerful greenhouse gas with ozone depletion potential.

103 I2 levels are able to significantly increase ozone depletion rates, while also producing iodine monox

104 cate that the eruptions led to stratospheric ozone depletion.

105 However, 8-h policy-relevant ozone design values increase in all cases.

106 r by plants, and its emissions contribute to ozone destruction in the stratosphere.

107 interannual variability of the tropospheric ozone distribution over the central-eastern Pacific Ocea

108 study characterized the effect of different ozone dosages (0-1.0 mg O3/mg dissolved organic carbon)

109 e almost linearly correlated to the specific ozone dose for higher ozone doses.

110 ozonation formed little bromate at specific ozone doses of </=0.4 mg O3/mg DOC, while the bromate yi

111 er, appreciable abatements (>50% at specific ozone doses of 0.5 gO3/gDOC to approximately 100% at >/=

112 elated to the specific ozone dose for higher ozone doses.

113 e with input values for these variables plus ozone dry deposition velocity chosen according to a 576

114 uggest that the offspring of dams exposed to ozone during implantation had reduced growth compared wi

115 ed the approach using a time-series study of ozone effects on asthma emergency department visits.

116 f that isoprene and the likelihood of a high-ozone episode the following day.

117 imately 30% variability in the number of JJA ozone episodes (>70 ppbv) when averaged over the eastern

118 Exposure to pollutants, such as ozone, exacerbates airway inflammation and hyperresponsi

119 Ozone-exposed dams also had lower serum glucose and high

120 increase in mean of same and previous 3 days ozone exposure (lag03) = 2.7%, 95% CI: 1.5, 3.9; ARI ERl

121 Ozone exposure also increased circulating mitochondrial

122 No inverse association was found between ozone exposure and PEF.

123 idemiological studies suggest that increased ozone exposure during gestation may compromise fetal gro

124 ereas lung function decreases with increased ozone exposure in schoolchildren.

125 Ozone exposure increased the risk of PROM on the day of

126 Long-term ozone exposure is associated with development of ARDS in

127 In spite of recent developments in ozone exposure metrics, from a concentration-based to a

128 We aimed to investigate effects of personal ozone exposure on various respiratory outcomes in school

129 URTI were also significantly associated with ozone exposure over the whole year, but typically more c

130 To assess whether peri-implantation ozone exposure resulted in sustained pulmonary or system

131 The association of ozone exposure with respiratory outcomes has been invest

132 sity augments the pulmonary effects of acute ozone exposure.

133 ammation and proinflammatory cytokines in an ozone-exposure model.

134 May-September ozone exposures were predicted by interpolating concentr

135 This increase in tropospheric ozone flux over the Pacific Ocean may affect the radiati

136 re, the interspecific variation in slopes of ozone flux-response relationships was considerably lower

137 results indicate that improved estimates of ozone fluxes require a better representation of plant ph

138 d to filtered air, 0.4 ppm ozone, or 0.8 ppm ozone for 4 h/d during implantation, on gestation days (

139 amplification in the catalytic loss rates of ozone for the dominant halogen, hydrogen, and nitrogen c

140 he contribution of biogenic VOC emissions to ozone formation is lower in June (9-11%) and August (6-9

141 Therefore, the secondary organic aerosol and ozone formation potential of the exhaust does not depend

142 e relationship between isoprene emission and ozone formation, there is a positive feedback loop betwe

143 llustrated by the discovery of a convenient, ozone-free synthesis of bridged secondary ozonides from

144 linear regression model to predict JJA MDA8 ozone from 1980 to 2013, using the identified SST and SL

145 e results illuminate the mechanisms by which ozone gas interacted with the weathered petroleum residu

146 nfluence of the operating frequency over the ozone generated by a DBD-plasma reactor is studied by va

147 report the experimental characterization of ozone generation in dielectric barrier discharges as a f

148 male fetuses from litters exposed to 0.8 ppm ozone had lower lean mass and fat mass than pooled contr

149 tly modest, the impact of dichloromethane on ozone has increased markedly in recent years and if thes

150 n summer, when intercontinental influence on ozone has typically been found to be weakest, nearly 3 p

151 evated (per 10-parts-per-billion increase in ozone, hazard ratio (HR) = 1.06; 95% confidence interval

152 depressant use was used to define cases (for ozone, HR = 1.08; 95% CI: 1.02, 1.14; for PM2.5, HR = 1.

153 ecific variation in LMA and that large-scale ozone impact assessment could be greatly improved by con

154 ociation with short-term exposure to ambient ozone in China.

155 am and up to 15 ppb for seasonal maximum 1 h ozone in Indonesia.

156 IL-33 contributes to augmented responses to ozone in obese mice.

157 IL-33 drives augmented responses to ozone in obese mice.

158 We find that anomalously high JJA ozone in the East is correlated with these springtime pa

159 ated with more summertime heatwaves and high ozone in the East.

160 ating the interannual variability of surface ozone in this region.

161 TSG-6 deficiency protects against AHR after ozone (in vivo) or sHA (in vitro and in vivo) exposure.

162 obesity-related increases in the response to ozone, including increases in type 2 cytokines.

163 In obese mice, ozone increased lung IL-13+ innate lymphoid cells type 2

164 Ozone increased ST2+ gammadelta T cells, indicating that

165 Ozone increases IL-33 in the lungs, and obesity augments

166 icy relevant, to be weighed against possible ozone increases.

167 is essential for the development of HA- and ozone-induced AHR.

168 Anti-ST2 reduced ozone-induced airway hyperresponsiveness and inflammatio

169 e of the NE genotype was more susceptible to ozone-induced changes compared with the IE plants.

170 Serum amyloid A: an ozone-induced circulating factor with potentially import

171 address this challenge, we have implemented ozone-induced dissociation (OzID) in-line with LC, IMS,

172 Ozone-induced dissociation (OzID) is a novel ion activat

173 fined by oxygenated addends, we propose that ozone-induced functionalization decreases the size of gr

174 This model explains ozone-induced increase in emission energies yielding flu

175 Obesity also augmented ozone-induced increases in BAL CXCL1 and IL-6, and in BA

176 with controls, possibly as a consequence of ozone-induced vascular dysfunction.

177 ere to investigate the effects of short-term ozone inhalation during implantation on fetal growth out

178 hase reaction between mass-selected ions and ozone inside a mass spectrometer to assign sites of unsa

179 was undertaken to allow the introduction of ozone into the high-pressure trapping ion funnel region

180 In the aqueous bulk, oxidation of bromide by ozone involves a [Br*OOO(-)] complex as intermediate.

181 Ozone is a pollutant that has a relatively smooth spatia

182 Tropospheric ozone is considered the most detrimental air pollutant f

183 Ozone is produced from ambient oxygen in situ and is res

184 atospheric chlorine levels are declining and ozone is projected to return to levels observed pre-1980

185 ate constants for the reactions of CBDs with ozone (kO3) (<0.1-7.9 x 10(3) M(-1) s(-1)) or with hydro

186 the second order reaction rate constant with ozone (kO3).

187 models to estimate the cumulative effects of ozone (lagged over 0-3 d) on mortality in each city, and

188 chlorine-containing chemicals contribute to ozone layer depletion.

189 Accordingly, the stratospheric ozone layer is expected to recover.

190 hydrogen radicals, destroy the stratospheric ozone layer, such that Earth's surface receives high dos

191 otocol, further delaying recovery of Earth's ozone layer.

192 ining an aerobic atmosphere and creating the ozone layer.

193 Elevated tropospheric ozone leads to no reduction of forest productivity and c

194 21353817 of 22433862), and 91.1% of days had ozone levels below 60 parts per billion, during which 93

195 Warm-season ozone was defined as ozone levels for the months April to September of each y

196 Daily PM2.5 and ozone levels in a 1-km x 1-km grid were estimated using

197 such products in the presence of significant ozone levels might materially influence personal exposur

198 We conclude that heightened surface ozone levels will potentially lead to reductions in rice

199 Modeled ozone levels yield a median of 11 deaths per 100000 peop

200 ut it would also entail new risks, including ozone loss and heating of the lower tropical stratospher

201 water vapor concentration causing additional ozone loss and surface warming.

202 engages the element of risk associated with ozone loss in the stratosphere over the central United S

203 er, thereby extending the time for catalytic ozone loss over the Great Plains.

204 to nitrogen dioxide (NO2) in the presence of ozone, making it impossible to directly measure (15)N in

205 This study suggests that tropospheric ozone may not diminish forest carbon sequestration capac

206 on between short-term exposures to PM2.5 and ozone (mean of daily exposure on the same day of death a

207 2.2) x 10(-4) and (4.0 +/- 2.2) x 10(-4) for ozone mixing ratios (MRO3) of 50 and 25 ppb, respectivel

208 Atmospheric Infrared Sounder (AIRS) CO, Aura Ozone Monitoring Instrument (OMI) aerosol index, and Aur

209 incident satellite measurements, made by the Ozone Monitoring Instrument (OMI) on NASA's Aura satelli

210 99-2004), nitrogen dioxide (NO2) (2006), and ozone (O3) (2002-2004) concentrations were linked to the

211 abundance of tropospheric oxidants, such as ozone (O3) and hydroxyl (OH) and peroxy radicals (HO2 +

212 ed NO through NO conversion to NO2 in excess ozone (O3) and subsequent NO2 collection in a 20% trieth

213 spectrometry is used to investigate how the ozone (O3) concentration, relative humidity (RH), and pa

214 ric chemistry and causing sharp tropospheric ozone (O3) depletion in polar regions and significant O3

215 In this context, the role of iodide-mediated ozone (O3) deposition over seawater and marine halogen c

216 Relative risk estimates for long-term ozone (O3) exposure and respiratory mortality from the A

217 Level of surface ozone (O3) has been increasing continuously in China in

218 The present study aimed to optimize ozone (O3) treatments, as gas and dissolved in water, to

219 nitrogen oxides (NOx) produces tropospheric ozone (O3), and NOx is traditionally considered to be di

220 itrogen dioxide (NO2), sulfur dioxide (SO2), ozone (O3), and particulate matter with aerodynamic diam

221 of extended summer (April-September) surface ozone (O3), fine particulate matter (PM2.5), and maximum

222 trous acid (HONO), hydrogen peroxide (H2O2), ozone (O3), formaldehyde (HCHO), and acetaldehyde (CH3CH

223 Ground-level ozone (O3), harmful to most living things, is produced f

224 exoplanets including molecular oxygen (O2), ozone (O3), water vapor (H2O), carbon dioxide (CO2), nit

225 tions of fine particulate matter (PM2.5) and ozone (O3).

226 Concentrations of ground-level ozone ([O3 ]) over much of the Earth's land surface have

227 he role of IL-33 in the augmented effects of ozone observed in obese mice.

228 M2.5 of less than 12 mug per cubic meter and ozone of less than 50 ppb, the same increases in PM2.5 a

229 This study assesses the impact of ozone on forest composition and ecosystem dynamics with

230 e impact of increased air pollution (surface ozone) on rice yields in Southeast China.

231 mpounds that either deplete the stratosphere ozone or have significant global warming potential.

232 n, TSG-6 is necessary for AHR in response to ozone or sHA, in part because it facilitates rapid forma

233 G-6(-/-) and TSG-6(+/+) mice were exposed to ozone or short-fragment HA (sHA), and AHR was assayed vi

234 A chemically driven increase in polar ozone (or "healing") is expected in response to this his

235 s rats were exposed to filtered air, 0.4 ppm ozone, or 0.8 ppm ozone for 4 h/d during implantation, o

236 reaction network that reduces stratospheric ozone over the Arctic.

237 n reactions of unsaturated hydrocarbons with ozone (ozonolysis).

238 hree factors affect the relative benefits of ozone policies with household income: (1) unequal ozone

239 portionately affected by ozone pollution and ozone policy.

240 eholds may be disproportionately affected by ozone pollution and ozone policy.

241 ir Quality (CMAQ) simulations of a July 2006 ozone pollution episode in the UK were made with input v

242 the United States the probability of severe ozone pollution when there are heat waves could be up to

243 ress the acute health effects of atmospheric ozone pollution.

244 munities and ozone, which further aggravates ozone pollution.

245 53.7 nm, 310-410 nm), and oxidized (UV-H2O2, ozone) poultry litter extracts.

246 us studies, extend the influence of regional ozone precursors emissions and, thus, greatly enhance O3

247 trogen oxides (NOx), which are key PM2.5 and ozone precursors.

248 well as the tagging approach (i.e., tracking ozone produced from specific O3 precursors emitted from

249 entify the effect of the dielectric barrier, ozone production curves corresponding to ten dielectric

250 eekend-weekday effect, which locally changes ozone production rates by 40%.

251 cross-section measurements directly on pure ozone provide strong evidence for revising the conventio

252 urements and ozone concentrations as well as ozone quenching effects at extreme power conditions are

253 s of recovery, and climate change is masking ozone recovery from ozone-depleting substances in some r

254 Here we discuss the nature and timescales of ozone recovery, and explore the extent to which it can b

255 Ozone reductions are highest among low-income households

256 irst application to evaluate the benefits of ozone reductions for low-income households.

257 Ozone reductions were relatively twice as beneficial for

258 its more than a policy delay or differential ozone reductions with income.

259 Our analyses suggest that current ozone reductions, although significant, cannot substanti

260 policies with household income: (1) unequal ozone reductions; (2) policy delay; and (3) economic val

261 nited States to estimate the distribution of ozone-related health impacts across nine income groups.

262 are associated with about 38,000 PM2.5- and ozone-related premature deaths globally in 2015, includi

263 ding approximately 174,000 global PM2.5- and ozone-related premature deaths in 2040.

264 in 2015, including about 10 per cent of all ozone-related premature deaths in the 28 European Union

265 nto its positive phase even if stratospheric ozone returns to normal levels, so that climate conditio

266 the extra-tropical tropopause and transport ozone rich dry stratospheric air into the tropics.

267 evant stomatal flux-based index, large-scale ozone risk assessment is still complicated by a large an

268 ated by a large and unexplained variation in ozone sensitivity among tree species.

269 e results show that much of the variation in ozone sensitivity among woody plants can be explained by

270 Here, we explored whether the variation in ozone sensitivity among woody species can be linked to i

271 by isoprene-emitting species (which tolerate ozone stress better than non-emitters).

272 ding to a difference of 25% in the amount of ozone that enters the leaves.

273 This study evaluates the effects of ozone therapy (OT) on the early healing period of deepit

274 nue into the future, the return of Antarctic ozone to pre-1980 levels could be substantially delayed.

275 5) formed quickly, following the addition of ozone to the smoke aerosol, and ClNO2(g) production prom

276 We found that ozone tolerance at the leaf level was closely linked to

277 henic acid (NA) species from unprocessed and ozone-treated oil sands process-affected water (OSPW) wa

278 impaired, but not in fish that had received ozone-treated OSPW.

279 Ozone treatment in aqueous GO suspensions yields the add

280 Furthermore, ozone treatment provides a versatile approach to control

281 In this work, we utilize ozone treatment to controllably tune the band gap of GO,

282 size decrease of ordered sp(2) domains with ozone treatment, whereas semi-empirical PM3 calculations

283 ere analyzed using a resistance model of net ozone uptake and found the second-order rate coefficient

284 for PM2.5 and 10 parts per billion (ppb) for ozone using a two-pollutant Cox proportional-hazards mod

285 1,3-butadienes (CBDs) in aqueous solution by ozone, UV-C(254 nm) photolysis, and the corresponding ad

286 ransistors (TFTs) with a simple, low-cost UV-ozone (UVO)-treated polymeric gate dielectric is reporte

287 this configuration, a high number density of ozone was achieved and thus abundant and diagnostic OzID

288 Warm-season ozone was defined as ozone levels for the months April t

289 oduction from indoor chemical reactions with ozone was found.

290 ut the 2013-2014 academic year, during which ozone was measured using personal samplers.

291 T40, a metric for assessing crop damage from ozone, we find that the increased ozone concentrations r

292 Effects of ozone were also assessed in obese and lean mice deficien

293 ug per cubic meter in PM2.5 and of 10 ppb in ozone were associated with increases in all-cause mortal

294 than 50 ppb, the same increases in PM2.5 and ozone were associated with increases in the risk of deat

295 c diameter of less than 2.5 mum [PM2.5]) and ozone were estimated according to the ZIP Code of reside

296 hort-term exposures to PM2.5 and warm-season ozone were significantly associated with increased risk

297 feedback loop between forest communities and ozone, which further aggravates ozone pollution.

298 nthropogenic coupling expands the daily high-ozone window and likely has an opposite O3-NOx response

299 ys with low morning isoprene then have lower ozone with a more typical afternoon peak.

300 exposure for users owing to the reaction of ozone with terpene ingredients.