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1 o significantly induce levels of p130 and/or p16 protein.
2 unohistochemical analysis, all NFs expressed p16 protein.
3 ease the IC0.5 approximately fivefold in the p16 protein.
4 nohistochemistry (IHC) for expression of the p16 protein.
5 mas and two low-grade gliomas that expressed p16 protein.
8 umor specimens that stained positive for the p16 protein, an indicator that human papillomavirus had
9 in which is equivalent to 0.49 ng mL(-1) for p16 protein and 28 cells for HeLa cervical cancer cells.
11 mors, 16 (34.7%) of the 46 showed detectable p16 protein and mRNA; of these, 12 had no DNA abnormalit
16 ressor protein (residues 84-103 of the human p16 protein) can bind to cdk4 and cdk6 and inhibit cdk4-
17 as and that the restoration of the wild-type p16 protein could have clinical and therapeutic utility.
18 ecular mechanisms underlying this absence of p16 protein expression are not completely understood.
21 of RB function has been associated with high p16 protein expression in several other tumor types.
22 There is considerable evidence that lack of p16 protein expression is a frequent event in human glio
32 ptosis; and 6) HaCaT cells have undetectable p16 protein (hypermethylation of the promoter), dysfunct
34 antibody has confirmed an over-expression of p16 protein in cervical cancer cells and its association
39 ines directed the biosynthesis of functional p16 protein in the majority of the exposed cells, signif
41 oviral-mediated overexpression of functional p16 protein, indicating the existence of a defect(s) dow
46 ontaining the p16 gene (AdRSVp16) produced a p16 protein that suppressed cellular proliferation and i
51 le arrest was associated with binding of the p16 protein to cyclin-dependent kinase 4 and dephosphory
53 e HMEC express readily detectable amounts of p16 protein, whereas normal or E6-expressing HMEC that e
54 and detection limit of 1.3 ng mL(-1) for GST-p16 protein which is equivalent to 0.49 ng mL(-1) for p1
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