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1 inase cascade which include RAF-1, MEK-1 and p42 MAP kinase.
2  like Rsk1, forms a heteromeric complex with p42 MAP kinase.
3 llin, and extracellular-related kinase 2 (or p42 MAP kinase) accounted for the major changes occurrin
4 that of the alpha(2A)AR homodimer, while p44/p42 MAP kinase activation was unaffected.
5 ced, whereas ISO became competent to inhibit p42 MAP kinase activation.
6 stimulation and inhibition, respectively, of p42 MAP kinase activity.
7 ied by the nearly simultaneous activation of p42 MAP kinase and Cdc2/cyclin B.
8 nal cell line induces phosphorylation of p44/p42 MAP kinase and GSK3beta.
9 pproximately 95% with specific inhibitors of p42 MAP kinase and p38 SAP kinase function, respectively
10 the Rsk2 isozyme in the M phase functions of p42 MAP kinase and provide tools for further examining R
11 to be essential for Ag-induced activation of p42 MAP kinase and release of arachidonic acid were unaf
12 ndicate that the sustained activation of p44/p42 MAP kinase and subsequent arachidonate release by cy
13 AP kinase negatively regulated activation of p42 MAP kinase and the responses mediated by this kinase
14 ereas other pathways including Stat5 and p44/p42 MAP kinase are activated normally.
15  with PD 098059 and SB 203580 indicated that p42 MAP kinase, but not p38 MAP kinase, contributed to t
16                               Stimulation of p42 MAP kinase by Ag resulted in relatively sustained ac
17                                Activation of p42 MAP kinase by growth factors was blunted by pretreat
18 ransduction molecules, including Akt and p44/p42 MAP kinases, by both EGF and IGF, whereas each indiv
19                 Finally, we demonstrate that p42 MAP kinase can activate recombinant Rsk2 in vitro to
20              The phosphorylation of cPLA2 by p42 MAP kinase correlated with an approximately 1.5-fold
21 action is mediated through activation of the p42 MAP kinase (erk2).
22   We also observed a transient activation of p42 MAP kinase following activation of Mer by Gas6.
23 ve examined whether Cdc2 activation requires p42 MAP kinase function.
24 e phosphorylation or enzymatic activation of p42 MAP kinase in eosinophils after IL-5 treatment.
25  the rapid but transient activation of ERK2 (p42 MAP kinase) in CD34(+) cells, and we used the MAP ki
26                 These findings indicate that p42 MAP kinase is an essential component of the M phase
27  Mos-induced Cdc2 activation requires active p42 MAP kinase, is inhibited by a MAP kinase phosphatase
28 opus oocyte extract system demonstrated that p42 MAP kinase (MAPK) exhibits a sharp, sigmoidal stimul
29                     Here we demonstrate that p42 MAP kinase (MAPK), the Xenopus ortholog of ERK2, is
30 he inhibition or the depletion of endogenous p42 MAP kinase resulted in defective spindle structures
31 ucleotide phosphate (NADPH)-oxidase, via p44/p42 MAP kinase signaling, and upregulated pro-fibrotic g
32                                        Thus, p42 MAP kinase was activated by increasing intracellular
33 In PHX cells, the ability of PHE to activate p42 MAP kinase was dramatically reduced, whereas ISO bec
34  phosphorylation of Stat1 and Stat3, whereas p42 MAP kinase was phosphorylated regardless of the pres

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