ライフサイエンスコーパス: p44 mitogen-activated protein kinase

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1 hosphatidylcholine selectively activated p42/p44 mitogen-activated protein kinase.

2 of gap junctions by bFGF is mediated by p42/p44 mitogen-activated protein kinases.

3 ether with failure in phosphorylation of p42/p44 mitogen-activated protein kinases.

4 utyrate, palytoxin does not activate the p42/p44 mitogen-activated protein kinases.

5 ng pathways, including activation of the p42/p44 mitogen-activated protein kinases.

6 Inhibition of p42/p44 mitogen-activated protein kinase activation by U0126

7 receptors do not affect IGF-I-stimulated p42/p44 mitogen-activated protein kinase activation or phosp

8 nduced phosphorylation and activation of p42/p44 mitogen-activated protein kinase, although this acti

9 l and a 4-fold decrease in the activation of p44 mitogen-activated protein kinase and Akt.

10 ese SCFAs function as both activators of p42/p44 mitogen-activated protein kinase and as inhibitors o

11 ctively blocked prolactin stimulation of p42/p44 mitogen-activated protein kinases and transcription

12 E-associated proteins, BubR1 and phospho-p42/p44 mitogen-activated protein kinase, and mitotic centro

13 e C (at residues Ser(14) and Thr(63)) and by p44 mitogen-activated protein kinase (at residues Ser(27

14 stimulated phosphorylation/activation of p42/p44 mitogen-activated protein kinases by an initially (1

15 ation, Whn levels were suppressed by the p42/p44 mitogen-activated protein kinase cascade, and this s

16 otein kinases and to a lesser extent p42 and p44 mitogen-activated protein kinase isoforms.

17 NE caused rapid and transient p42-p44 mitogen-activated protein kinase (MAPK) activation t

18 exhibit increased expression of phospho-p42/p44 mitogen-activated protein kinase (MAPK) after ischem

19 The p42/p44 mitogen-activated protein kinase (MAPK) cascade incl

20 ell proliferation via specific activation of p44 mitogen-activated protein kinase (MAPK) in the p21(r

21 We examined the role of p38, p42, and p44 mitogen-activated protein kinase (MAPK) isoforms and

22 Although p42/p44 mitogen-activated protein kinase (MAPK) negatively m

23 In VSMC, p42/p44 mitogen-activated protein kinase (MAPK) pathway acti

24 was to determine the involvement of the p42/p44 mitogen-activated protein kinase (MAPK) pathway and

25 nduced activation of signaling pathways (p42/p44 mitogen-activated protein kinase (MAPK), c-Jun NH2-t

26 h signals by potentiating EGF-stimulated p42/p44 mitogen-activated protein kinase (MAPK), p38 MAP kin

27 ceptors, and a pertussis toxin-sensitive p42/p44 mitogen-activated protein kinase (MAPK)-mediated pat

28 necrosis factor (TNF) activates both p42 and p44 mitogen-activated protein kinases (MAPK) in human FS

29 d activators of transcription 5 (STAT5), p42/p44 mitogen-activated protein kinase [MAPK; extracellula

30 ctor (TNF)-induced activation of the p42 and p44 mitogen-activated protein kinases (MAPKs) (known as

31 The p42 and p44 mitogen-activated protein kinases (MAPKs) are phosph

32 c and Ras caused upregulation of the p42 and p44 mitogen-activated protein kinases (MAPKs), only Ras

33 PMT stimulation of p42/p44 mitogen-activated protein kinase occurs upstream via

34 ression were reduced, without changes in p42/p44 mitogen-activated protein kinase or IRS-1.

35 rations of the activation of the p38 and p42/p44 mitogen-activated protein kinases or the phosphoryla

36 Also, p42/p44 mitogen-activated protein kinase (p42/p44mapk), acti

37 cells (1) and now report a key role for p42/p44 mitogen-activated protein kinases (p42/p44MAPK) in t

38 residues for activating S1P(1)-dependent p42/p44 mitogen-activated protein kinase phosphorylation, wh

39 y MDC was dependent on the activation of p42/p44 mitogen-activated protein kinases, Src-like protein

40 independent of activation of the p38 and p42/p44 mitogen-activated protein kinases, the phosphorylati

41 ATP or insulin, on the activity state of p42/p44 mitogen-activated protein kinases, while in combinat

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