ライフサイエンスコーパス: pacemaker cell

1 MP agonists were similar to the responses of pacemaker cells.

2 ells form chemical synapses on the relay and pacemaker cells.

3 anscriptional or translational events in SCN pacemaker cells.

4 ut not GABA(B), receptors are located on SCN pacemaker cells.

5 l types of ion channels expressed in cardiac pacemaker cells.

6 the pacemaker nucleus, i.e., relay cells and pacemaker cells.

7 cells, hematopoietic cells, and interstitial pacemaker cells.

8 a small structure containing <10,000 genuine pacemaker cells.

9 of rhythmic activity in cardiac and neuronal pacemaker cells.

10 and physiological automaticity of native SAN pacemaker cells.

11 n program may be especially important in fly pacemaker cells.

12 re initiated by a small number of sinus node pacemaker cells.

13 intercellular communications and the role of pacemaker cells.

14 l rhythmic, spontaneous action potentials by pacemaker cells.

15 ides a new perspective on the concept of SCN pacemaker cells.

16 r potassium current (I(K)) in guinea pig SAN pacemaker cells.

17 of the SR alter pacemaking in these primary pacemaker cells.

18 ends upon input, including PDF, from central pacemaker cells.

19 ate the firing rates of neuronal and cardiac pacemaker cells.

20 rrent fluctuations on the IBIs of sinoatrial pacemaker cells.

21 ed in other mammalian cells and in amphibian pacemaker cells.

22 ler (7-15 microm in diameter) than relay and pacemaker cells.

23 activation of a variety of ionic currents in pacemaker cells.

24 is abundant expression of endogenous Pak1 in pacemaker cells; (2) expression of constitutively active

25 cytes, as well as specialized conduction and pacemaker cells, agonist binding to muscarinic acetylcho

26 Primary pacemaker cells also have a SR-dependence of cardiac pac

27 In spontaneously beating pacemaker cells, an increase in subsarcolemmal intracell

28 s generated by endosomal Ca2+ release in the pacemaker cell and was propagated by gap junctional comm

29 emonstrate for chemosensing the existence of pacemaker cells and how the presence of gap junctions im

30 K channels control spike frequency in atrial pacemaker cells and inhibitory potentials in neurons.

31 Interstitial cells of Cajal (ICC) act as pacemaker cells and possess unique ionic conductances th

32 ontains two well-characterized neuron types: pacemaker cells and relay cells.

33 rapidly increasing mitochondrial calcium in pacemaker cells and that MCU-enhanced oxidative phoshory

34 are histochemically distinct from relay and pacemaker cells and that they receive electrotonic input

35 t a Pak1 signaling pathway exists in cardiac pacemaker cells and that this novel pathway plays a role

36 ndent differentiation of upper urinary tract pacemaker cells and the efficient flow of urine from the

37 myenteric ICC (ICC-MY) in human jejunum are pacemaker cells and whether these cells actively propaga

38 iption factors could be utilized to generate pacemaker cells, and suggest ISL1 mutations may underlie

39 to convert quiescent heart-muscle cells into pacemaker cells, and the successful generation of sponta

40 connexin expression phenotypes in sinus node pacemaker cells, and to define the spatial distribution

41 acent to the inner nerve ring where swimming pacemaker cells are located.

42 These findings suggest that cardiac pacemaker cells are physically segregated and molecularl

43 Pacemaker cells are specialized cell types that drive bi

44 erlying muscles fail to propagate beyond the pacemaker cell, are slow, initiate in abnormal locations

45 secondary heart fields, we found that chick pacemaker cells arise from a discrete region of mesoderm

46 e anatomical and neurochemical properties of pacemaker cells, as well as in the mechanisms of clock-g

47 at Isl1 is a specific marker for a subset of pacemaker cells at developmental stages examined, and su

48 Electrical coupling of pacemaker cells at gap junctions appears to play an impo

49 pled-clock system that drives normal cardiac pacemaker cell automaticity.

50 Cardiac pacemaker cells autonomously generate electrical impulse

51 medial septum/diagonal band of Broca (MSDB) pacemaker cells, because, in addition to previously desc

52 ten terminated at the periphery of relay and pacemaker cell bodies.

53 d in both sino-atrial node and latent atrial pacemaker cells but not in working atrial myocytes.

54 tomaticity in isolated sinoatrial node (SAN) pacemaker cells, but the potential role of NCX1 in deter

55 Sinoatrial node myocytes act as cardiac pacemaker cells by generating spontaneous action potenti

56 t interstitial cells of Cajal (ICC) serve as pacemaker cells, conduits for active transmission of ele

57 Cardiac pacemaker cells create rhythmic pulses that control hear

58 subunit also affects the reliability of the pacemaker cell; cycle timing is often irregular.

59 These results link defective pacemaker cell differentiation with hydronephrosis and p

60 e preparations revealed three populations of pacemaker cells distinguished on the basis of connexin i

61 These results indicate that subsets of pacemaker cells express distinct connexin phenotypes.

62 of the hypothalamus are described as master pacemaker cells for biological rhythms.

63 sarcoplasmic reticulum (SR) regulate cardiac pacemaker cell function by activation of electrogenic Na

64 lar Ca(2+) cycling dynamics regulate cardiac pacemaker cell function on a beat-to-beat basis remains

65 ar from these studies that ICC serve as: (i) pacemaker cells, generating the spontaneous electrical r

66 In cardiac pacemaker cells, hetero-tetramer GIRK1/4 channels and ho

67 Putative pacemaker cells, identified by immunoreactivity for hype

68 vri is expressed in circadian pacemaker cells in larval and adult brains.

69 ells of Cajal (ICCs) have been identified as pacemaker cells in the gastrointestinal (GI) tracts of v

70 acemaker potentials from isolated sinoatrial pacemaker cells in the presence of endogenous cAMP conce

71 terstitial cells of Cajal (ICC) are putative pacemaker cells in the rabbit urethra.

72 The spontaneous activity of pacemaker cells in the sinoatrial (SA) node controls hea

73 vity decreases heart rate (HR) by inhibiting pacemaker cells in the sinoatrial node (SAN).

74 Interstitial cells of Cajal (ICC) are pacemaker cells in the small bowel, and therefore this c

75 ration of oscillations in neural activity of pacemaker cells in the suprachiasmatic nucleus (SCN).

76 CRY overexpression in brain pacemaker cells increases behavioral photosensitivity, a

77 ditis elegans, a periodic calcium spike in a pacemaker cell initiates a calcium wave in the intestine

78 ctivity and neoplastic transformation of gut pacemaker cells (interstitial cells of Cajal).

79 by gene transfer or by delivering engineered pacemaker cells into normally quiescent myocardium.

80 ntracellular Ca2+ release to automaticity of pacemaker cells isolated from cat right atrium.

81 strointestinal (GI) muscles are generated by pacemaker cells, known as interstitial cells of Cajal (I

82 Pacemaker cells, known as interstitial cells of Cajal (I

83 us, the biological clock of sinoatrial nodal pacemaker cells, like that of many other rhythmic functi

84 in physiology and behavior are controlled by pacemaker cells located in the suprachiasmatic nucleus (

85 right atrium coexpressed the sinoatrial node pacemaker cell marker HCN4.

86 titial cells of Cajal, suggesting that these pacemaker cells may also be involved in neural reflexes.

87 n concert with the autonomic nervous system, pacemaker cells, myogenic mechanisms, and/or electrotoni

88 Putative pacemaker cells near the plexus showed immunoreactivity

89 In pacemaker cells of the brain, PER and TIM proteins rise

90 ulated rhythmically in nuclei of eyes and in pacemaker cells of the brain.

91 e 5-HT(2B) receptor is also expressed on the pacemaker cells of the gastrointestinal tract, the inter

92 The pacemaker cells of the heart initiate the heartbeat, sus

93 tivated potassium current (IKACh) channel in pacemaker cells of the heart.

94 e sequential activation of ionic currents in pacemaker cells of the sinoatrial node.

95 the idea that Ca(2+) regulates CL in cardiac pacemaker cells on a beat-to-beat basis, and suggest a m

96 eretofore focused on intrinsically bursting "pacemaker" cells operating in conjunction with synaptic

97 s and Ca transient decay to insure fail-safe pacemaker cell operation within a wide range of rates.

98 because they either modify the rhythm of the pacemaker cell or are essential for pattern generation i

99 Streptozotocin-treated mice had increased pacemaker cell ox-CaMKII and apoptosis, which were furth

100 lock spontaneous slow waves, suggesting that pacemaker cells populate all regions of the circular mus

101 A single isolated sinoatrial pacemaker cell presents intrinsic interbeat interval (IB

102 s driving heart rate acceleration in cardiac pacemaker cells remain incompletely understood.

103 icantly lower input resistances in relay and pacemaker cells, respectively, exhibiting drastically di

104 es instruct the motor steps and regulate the pacemaker cell's authority and reliability.

105 on the surface membrane of sinoatrial nodal pacemaker cells (SANCs) are the proximal cause of an act

106 polarization (DD) in rabbit sinoatrial nodal pacemaker cells (SANCs) generate an inward current (I(NC

107 Their isolated peripheral SAN pacemaker cells showed a reduced Na+ channel expression

108 A similar oscillator is present in pacemaker cells such as the interstitial cells of Cajal

109 electrical rhythmicity, which originates in pacemaker cells surrounding the myenteric plexus, called

110 ficantly less ox-CaMKII, exhibited increased pacemaker cell survival, maintained normal heart rates,

111 slow waves that are thought to originate in pacemaker cells termed interstitial cells of Cajal (ICC)

112 ity mediates axonal growth of the Drosophila pacemaker cells, termed "small ventrolateral neurons" (s

113 n the interstitial cells of Cajal (ICC), the pacemaker cells that control smooth muscle contraction.

114 Interstitial cells of Cajal (ICC) are pacemaker cells that generate electrical activity to dri

115 In the gastrointestinal (GI) tract, ICC are pacemaker cells that generate spontaneous electrical slo

116 Lipsius demonstrated that in cat subsidiary pacemaker cells the late phase of diastolic depolarizati

117 ER in two significant clusters of behavioral pacemaker cells: the large and the small ventral lateral

118 in survival, proliferation, and function of pacemaker cells throughout development.

119 ural and technological systems, from cardiac pacemaker cells to coupled lasers.

120 hrough a polysynaptic circuit extending from pacemaker cells to PI neurons.

121 flow of temporal information from circadian pacemaker cells to selective behaviors.

122 esponsiveness reflects the properties of the pacemaker cell type, not the receptor.

123 ," whose compositions differ between E and M pacemaker cell types.

124 l cells of Cajal (ICC), the gastrointestinal pacemaker cells, underlie a number of gastrointestinal m

125 In the present study, in mouse SA node pacemaker cells, we investigated Na+ currents under phys

126 The s-LNvs are the principal circadian pacemaker cells, whereas recent evidence indicates that

127 A variety of receptors are found on SCN pacemaker cells which permit the clock mechanism to resp

128 Pacemaker cells within the hamster suprachiasmatic nucle

129 or circadian rhythms of bioluminescence from pacemaker cells within the head for several days in indi

130 shift activity rhythms by acting directly on pacemaker cells within the SCN.