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1 t cell types following infection with Brugia pahangi.
2 sponses after infection with B. malayi or B. pahangi.
3 was cloned from the filarial nematode Brugia pahangi.
4  from the filarial parasitic nematode Brugia pahangi.
5 ions in gerbils chronically infected with B. pahangi.
6 he killing of Wolbachia is detrimental to B. pahangi.
7 pared with those in animals infected with B. pahangi alone.
8 in many filarial nematodes, including Brugia pahangi and Brugia malayi.
9           Gerbils were then infected with B. pahangi, and the effect of the polarized Th1 responses o
10     Apoptosis of human monocytes with Brugia pahangi antigen (BpA) was demonstrated by scoring severa
11 ne construct the expression of hsp83 from B. pahangi (Bp-hsp83) was studied by transfection of COS-7
12 llels the down regulation of experimental B. pahangi granulomas.
13  against Acanthocheilonema viteae and Brugia pahangi in jirds.
14 ntraperitoneal (i.p.) infections with Brugia pahangi in Mongolian gerbils, or jirds (Meriones unguicu
15 iminates or reduces Wolbachia bacteria in B. pahangi in vivo.
16 s and spleens compared with the levels in B. pahangi-infected gerbils not treated with tetracycline.
17   However, similar findings were noted in B. pahangi-infected gerbils treated with ivermectin, sugges
18 icient and immunocompetent mice following B. pahangi infection suggest that B cells are required for
19 itment to the peritoneal cavity following B. pahangi infection.
20  into the course of Brugia malayi and Brugia pahangi infections in immunodeficient mouse models, we f
21 ion following primary intraperitoneal Brugia pahangi infections in mice.
22 cytes in antifilarial immunity, using Brugia pahangi infections in the murine peritoneal cavity as a
23 ment did not inhibit the establishment of B. pahangi intraperitoneally.
24  a challenge infection with infective Brugia pahangi L3 in an accelerated manner, whereas cohorts imm
25 d to define early tissue migration of Brugia pahangi L3 in the gerbil (Meriones unguiculatus) and mea
26 ld-type mice that had been immunized with B. pahangi L3 protected athymic recipients from challenge i
27 ally inoculated in the hind limb with 100 B. pahangi L3, and necropsies were performed at various tim
28 c target of granuloma-mediated killing of B. pahangi L3.
29 expressed in the three main stages of the B. pahangi life cycle: microfilariae, infective larvae and
30 la and vertebrates are not present in the B. pahangi sequence.
31 Animals infected with both B. abortus and B. pahangi showed increased IFN-gamma and interleukin-10 (I
32  of primary and challenge infections with B. pahangi third-stage larvae (L3).

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