ライフサイエンスコーパス: pain perception

1 t afferent nociceptive signals into a stable pain perception.

2 (SNP) rs563649 and individual variations in pain perception.

3 n, context, injury) can separately influence pain perception.

4 triction, reduction of edema, and diminished pain perception.

5 rve as a neural center for the modulation of pain perception.

6 nsity on afferent nociceptive processing and pain perception.

7 te to subsequently emerging abnormalities in pain perception.

8 ical responses, including blood clotting and pain perception.

9 upporting the behavioural results of reduced pain perception.

10 be important targets for agents that modify pain perception.

11 n endogenous cannabinoid tone that modulates pain perception.

12 laces VRs in a much broader perspective than pain perception.

13 early places VR1 in a much broader role than pain perception.

14 ies in locomotor activity, visual tasks, and pain perception.

15 neuropathic pain, as well as in fundamental pain perception.

16 ermal pain thresholds are related to anginal pain perception.

17 ate the spinal cord dorsal horn and modulate pain perception.

18 sic and counteranalgesic pathways modulating pain perception.

19 e control of memory, cognition, movement and pain perception.

20 n the brain's own mechanisms for controlling pain perception.

21 g of the functional postnatal development of pain perception.

22 channel subtype that has been implicated in pain perception.

23 al nociceptive circuit and are essential for pain perception.

24 al nociceptive network and are essential for pain perception.

25 close relationship between inflammation and pain perception.

26 ffective and cognitive factors can influence pain perception.

27 , weight of surgical specimen, and patients' pain perception.

28 nomas and is implicated in heart failure and pain perception.

29 ems become sensitized, leading to heightened pain perception.

30 domains including working memory, mood, and pain perception.

31 phin that is implicated in the modulation of pain perception.

32 le contribution to individual differences in pain perception.

33 ntense than the aversiveness associated with pain perception.

34 alysis on neuroimaging studies of empathetic pain perception.

35 we investigated which computations underlie pain perception.

36 ntitatively the temporal dynamics of thermal pain perception.

37 en GBOs and the cortical activity subserving pain perception.

38 nwanted abnormalities in mechanosensation or pain perception.

39 entral role in cognition, affective mood and pain perception.

40 of nociceptive inputs by SpVc, and regulate pain perception.

41 nd's adjuvant (CFA), without affecting basal pain perception.

42 ent chest pain caused by heightened coronary pain perception.

43 tigated whether vision of the body modulates pain perception.

44 E(2), and capsaicin as well as reduced cold pain perception.

45 endogenous nNOS-2 activity acted to minimize pain perception.

46 ansient receptor potential (TRP) channels in pain perception?

47 bo hyperalgesia is an increase in subjective pain perception after a patient or subject underwent an

48 There are limited data on pain perception after periodontal or implant surgery or

49 regulate diverse brain functions, including pain perception, alcoholism, and substance addiction.

50 at while inequality per se did not influence pain perception, altruistic behavior had an intrinsic an

51 dulation (CPM), may play a role in enhancing pain perception among some RA patients.

52 e, painful stimulation can lead to increased pain perception and activation in pain-processing brain

53 opioid receptor (OPRM1) plays a key role in pain perception and addiction.

54 in motor control, in motor behavior, and in pain perception and also predict involvement of Go in Ca

55 ting many physiological functions, including pain perception and analgesia, responses to stress, aggr

56 These results further the understanding of pain perception and are potentially relevant for the dec

57 rticularly in regions of the cord related to pain perception and autonomic tone.

58 gated the effects of attention on esophageal pain perception and brain activity.

59 of controllability and uncontrollability on pain perception and cerebral pain processing.

60 genetic polymorphisms are mediators of human pain perception and clinical pain phenotypes.

61 Our study indicates for the first time that pain perception and expectation elicit different hemodyn

62 While the relation between pain perception and homeostatic regulation of bodily sys

63 is fundamental to our understanding of both pain perception and how opioids modulate pain.

64 le of voltage-gated sodium channel Nav1.7 in pain perception and how we can advance our understanding

65 tablished the participation of the cortex in pain perception and identified a long list of brain stru

66 common features that reliably contribute to pain perception and its modulation.

67 eptor (KOR) may represent a means to control pain perception and modulate reward thresholds.

68 to, as well as coordinate, effects of SP on pain perception and mood.

69 Pain perception and QOL were evaluated by using specific

70 rtex (rACC), a region putatively involved in pain perception and regulation.

71 Pain perception and related cortical activation patterns

72 whereas their acute activation reduces basal pain perception and relieves inflammatory and neuropathi

73 tion between the brain mechanisms underlying pain perception and representation of the body.

74 other populations, our results suggest that pain perception and severity are important when evaluati

75 The relation between pain perception and spatial representation of the body i

76 s finding provides fresh insights into human pain perception and suggests a new avenue for the develo

77 te diverse physiological responses including pain perception and the control of vascular tone.

78 vations illustrate the complexity of nNOS in pain perception and the existence of opposing nNOS syste

79 Continued research into the neurobiology of pain perception and the placebo effect has also played a

80 MC1R variants may influence orofacial pain perception and, in turn, predispose individuals to

81 ls are believed to play an important role in pain perception, and anesthetic steroids such as alphaxa

82 and (b) evaluate the quality of life (QOL), pain perception, and efficacy in terms of time to local

83 ntromedial medulla bidirectionally influence pain perception, and locus coeruleus activity mirrors th

84 ponse, muscle atrophy, exercise intolerance, pain perception, and mitochondrial energy metabolism.

85 reatment sessions and surgical intervention, pain perception, and procedure time.

86 enabled light-inducible inhibition of acute pain perception, and reversed mechanical allodynia and t

87 ver activation in rACC leads to control over pain perception, and that these effects were powerful en

88 play important roles in cognitive function, pain perception, and the reinforcing properties of nicot

89 lved in modulation of dopaminergic circuits, pain perception, and thermoregulation.

90 ments strongly suggest that these changes in pain perception are predominantly based on altered perce

91 ensory neurons transducing thermal, itch and pain perception are specified in early development is un

92 Thus, our finding suggests pain perception as an aspect of the nervous system that

93 eta-band power are covarying with subsequent pain perception, as well as lowered frontolateral theta-

94 enhanced sensitivity to morphine in tests of pain perception attributable to impaired desensitization

95 but not nitroglycerin reduced aggregate and pain perception averaged over four distention levels.

96 athways specific to chronic and acute sickle pain, perception-based targets of "top-down" mechanisms

97 ect cortical processing involved directly in pain perception, because their magnitude correlates with

98 servationally learned information can affect pain perception, both consciously and non-consciously.

99 -gated sodium channel is implicated in human pain perception by genetics.

100 nnabinoids may affect memory, cognition, and pain perception by means of this cellular mechanism.

101 pose that the DLPFC exerts active control on pain perception by modulating corticosubcortical and cor

102 It is known that pain perception can be altered by mood, attention and co

103 creased mortality, but it is unknown whether pain perception can directly affect aging.

104 thyltransferase (COMT) is a key regulator of pain perception, cognitive function, and affective mood.

105 these effects of prestimulus connectivity on pain perception covary with pain-relevant personality tr

106 ide range of behaviors, including cognition, pain perception, drug addiction, and memory consolidatio

107 reas related to the consequence of increased pain perception during CS, we found that only cortical a

108 e 1:100 000 to a physiologic level decreases pain perception during periocular, subcutaneous anesthes

109 It is well known that pain perception for patients and normal subjects can be

110 While the static magnitude of thermal pain perception has been shown to follow a power-law fun

111 Pain perception has evolved as a warning mechanism to al

112 citance on atrial defibrillation success and pain perception has not been studied in patients.

113 showed greatly reduced thermal inflammatory pain perception in AQP1(-/-) mice evoked by bradykinin,

114 oved efficacy of almost 10-fold in relieving pain perception in diabetic neuropathic rats as compared

115 Hypervigilance is considered important in pain perception in functional gastrointestinal disorders

116 ur understanding of the neural correlates of pain perception in humans has increased significantly si

117 connectivity patterns related to subsequent pain perception in humans, we contrasted painful with no

118 e that variation in the TRPA1 gene can alter pain perception in humans.

119 oint to a mechanism by which the body blocks pain perception in moderate states of tissue damage, all

120 ithin the central nervous system, increasing pain perception in patients.

121 ecally administered 8d significantly reduced pain perception in the formalin model of rat sensory ner

122 ed significant alterations in locomotion and pain perception in the knock-out mouse.

123 In mice, it attenuates visceral pain perception, indicating an antinociceptive effect, p

124 We argue that pain perception involves some of the representational pr

125 tion of the stress hormone axis by conscious pain perception is a likely explanation, but the magnitu

126 after periodontal or implant surgery or how pain perception is affected by presurgical anxiety.

127 r periodontal surgery and implant treatments pain perception is affected by the level of presurgical

128 Pain perception is one of the most complicated measurabl

129 Pain perception is strongly influenced by descending pat

130 Sex dependency in pain perception is well documented and is thought to be

131 ce has severe consequences on TRPV1-mediated pain perception leading to altered capsaicin consumption

132 Visceral hyperalgesia or heightened central pain perception may contribute to pain in chronic pancre

133 rectal barostat studies to evaluate visceral pain perception measured with a visual analog scale.

134 ll as potentially contributing to changes in pain perception, memory and synaptic plasticity.

135 sex differences in somatic but not visceral pain perception, motility, and central processing of vis

136 Ratings of muscle pain perception (MPP) and perceived exertion (RPE), musc

137 ng machine-learning algorithms to infer that pain perception occurred.

138 ce and hyperalgesia, without affecting basal pain perception or locomotor functions.

139 urgical specimen (P = 0.54) or the patients' pain perception (P = 0.28).

140 differences between repeated VAS scores for pain perception (P = 0.91) or anxiety (P = 0.75) from tw

141 There were significant improvements in pain perception (P:=0.026) and role limitation resulting

142 ility in the GABAB pathway of inhibition, in pain perception pathways via opioid receptors, and is al

143 on in acute pain decreases the activation in pain perception regions while activating the pain modula

144 malities encompass emotional, autonomic, and pain perception regions, implying that they likely play

145 We propose that human pain perception relies on an ancient chemical sensor con

146 portance of genes versus experience in human pain perception remains unclear, rodent populations disp

147 oid hormone signaling to insulin release and pain perception, respectively.

148 enetic variability clearly appears to affect pain perception, response to analgesics and predispositi

149 id receptor agonists alter food consumption, pain perception, responses to stress, and drug reward.

150 cotransporter NKCC1 in hearing, salivation, pain perception, spermatogenesis, and the control of ext

151 the CNS and can serve as a means to modulate pain perception, stress responses, and affective reward

152 result in deficits in explicit and implicit pain perception, supporting a critical role of anterior

153 subdivisions for monetary reward and thermal pain perception tasks: pshell signaling impending pain a

154 Pain perception temporarily exaggerates abrupt thermal s

155 d with angina; symptoms have been related to pain perception thresholds.

156 e hypothesis that catastrophizing influences pain perception through altering attention and anticipat

157 atastrophizing has been suggested to augment pain perception through enhanced attention to painful st

158 o be equally diverse and range from roles in pain perception to male aggression.

159 The role of H(3) receptors in regulating pain perception was further demonstrated using other str

160 Pain perception was measured at the same time intervals

161 Heat pain perception was never affected.

162 Pain perception was separately scored to a pain-inducing

163 rgeting the human Nav1.7 channel involved in pain perception, we present a protein-engineering strate

164 y hypnotizable subjects (HHs) who eliminated pain perception were included in the present study.

165 In contrast, no changes in coordination or pain perception were observed using the rotarod or hot-p

166 Presurgical anxiety and surgical pain perceptions were measured by visual analog scale (V

167 cephalographic (EEG) response correlate with pain perception when stimuli are presented in isolation,

168 brain NMDA receptors can therefore influence pain perception, which suggests that forebrain-selective

169 strongly supports the phenomenon of reduced pain perception whilst attention is distracted away from

170 ing studies to date have confounded visceral pain perception with anticipation.