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1 athways interact in the establishment of the pair bond.
2 onditioned partner preference, observed as a pair bond.
3 ng that is necessary to maintain the initial pair bond.
4 n, expression, and maintenance of monogamous pair bonds.
5 n species differences in the ability to form pair bonds.
6 productive activation or to the formation of pair bonds.
7 anges that result in the formation of stable pair bonds.
8 attributed to paternal provisioning based on pair bonds.
9 er pairs typically show strong and selective pair bonds.
10 ion (STI) in populations with such ephemeral pair bonds.
11 ial sex differences in the CRF modulation of pair bonding.
12 nsible for the enduring nature of monogamous pair bonding.
13 thin these subregions differentially mediate pair bonding.
14 ned the effects of repeated AMPH exposure on pair bonding.
15 direct evidence for epigenetic regulation of pair-bonding.
16 tion of selective aggression associated with pair-bonding.
19 ocially monogamous rodent species that forms pair bonds after mating, a behavior in which central dop
20 ially monogamous rodent that forms long-term pair bonds after mating--we demonstrate that amphetamine
23 aracterized their aggression associated with pair bonding and examined the related neuronal activatio
24 ributors to the effects of VP-OT peptides on pair bonding and gregariousness; reveal previously unkno
25 mine receptor subtype-specific influences on pair bonding and how dopamine receptor activation may in
29 overlap between the processes that underlie pair bonding and those that mediate responses to abused
30 reeding grounds is important for maintaining pair bonds and is achieved by pairs that remain together
31 y predicts that in species lacking long-term pair bonds and menopause, males should not exhibit a pre
33 appear to be important for the initiation of pair bonds and parental behaviors as well as the infant'
35 Unbalanced ASRs are predicted to influence pair-bond and mating behavior, since the rarer sex in th
37 ociation between variation in OXTR and human pair-bonding and other social behaviors, possibly indica
38 cin receptor gene (OXTR) are associated with pair-bonding and related social behaviors in humans.
39 uptake were found between males in long-term pair-bonds and lone males in areas including the nucleus
40 on, (c) motivation for parental behavior and pair bonding, and (d) the neural consequences of social
41 female-specific deficits in gregariousness, pair bonding, and nest cup ownership; reduces side-by-si
44 ch individuals form pairs just to mate (i.e. pair bonds are ephemeral) and later move on to sexually
49 cus of such research, especially its role in pair bonding between mates in species that display monog
52 of human origins is the emergence of strong pair-bonding between males and females accompanied by a
53 h levels of affiliative behaviors, including pair-bonding, biparental care, and cooperative breeding.
54 ions and beyond the mother-offspring, kin or pair bond broadens the generality of the social bufferin
55 te change by pair members and/or breaking of pair bonds by unmated individuals is more frequent when
58 chrogaster) are monogamous rodents that form pair bonds characterized by a preference for a familiar
59 ominent scenarios of the human transition to pair-bonding: communal care, mate guarding, food for mat
60 (i.e., alpha-helix N conformation, DNA base pair bonding, conformation of protein residues in the vi
62 effects are observed for social preferences, pair bonding, courtship, maintenance behaviors, or anxie
63 gitudinal relationship between precursors of pair-bonding during childhood and subsequent behavior in
66 are mediated by neural mechanisms regulating pair bond formation and not alcohol's effects on mating,
67 cies differences in the behaviors, including pair bond formation and paternal care, found selectively
68 potential role for CART in the regulation of pair bond formation between monogamous mates and suggest
69 se data demonstrate opposing regulation over pair bond formation by cAMP signaling within the NAcc sh
70 ing that AVP in the lateral septum regulates pair bond formation in male prairie voles and that this
72 es of action for CRF-induced facilitation of pair bond formation in prairie voles, as well as potenti
73 interaction of these circuits in a model of pair bond formation in rodents with a discussion of the
74 the regulation of alloparental behavior and pair bond formation in the socially monogamous prairie v
76 ntagonist in the lateral septum also blocked pair bond formation induced by either mating or AVP admi
77 tested the hypothesis that DA regulation of pair bond formation is mediated via the cAMP signaling c
78 how that dopamine transmission that promotes pair bond formation occurs within the rostral shell of t
79 the dorsomedial NAc shell appear to mediate pair bond formation through the positive hedonics associ
80 ve affiliation between adult mates, known as pair bond formation, as assessed by partner preference i
82 posite effects: D1-like activation prevented pair bond formation, whereas D2-like activation facilita
89 gh dopamine is necessary for mammalian adult pair-bond formation and maternal behavior, its function
93 that V1aR activation in this region promotes pair-bond formation via a mechanism similar to condition
95 ehaviors associated with monogamy, including pair-bond formation, are facilitated by the neuropeptide
96 icetines that differ in the extent of social pair-bond formation, Siberian (Phodopus sungorus) and Dj
97 that the mate-guarding hypothesis for human pair bonds gains strength from explicit links with our g
98 a monogamous rodent that forms long-lasting pair bonds, has proven useful for the neurobiological st
103 T) receptors (OTRs) impairs the formation of pair bonds in prairie voles (Microtus ochrogaster) and z
105 n OXTR was associated with traits reflecting pair-bonding in women in the TOSS and TCHAD samples.
107 rence (PP) formation (a laboratory proxy for pair bonding) in socially monogamous prairie voles (Micr
108 results based on assuming long-term, stable pair bonds, in which case one is capable of preventing o
109 Microtus ochrogaster) display mating-induced pair bonding indicated by social affiliation with their
113 and the subsequent development of monogamous pair-bonds is substantially predicted by influential imp
114 to forgoing access to resources, maintaining pair bonds led individuals to develop a flexible "scroun
116 le, male voles showed behavior indicative of pair bond maintenance-namely, selective aggression towar
119 attractive because humans maintain long-term pair bonds, making reproductive value (i.e. future repro
121 re we show that this selective aggression in pair-bonded male prairie voles is associated with increa
131 P in the regulation of aggression induced by pair-bonding or drug experience in socially monogamous m
135 dimorphic involvement of stress hormones in pair bonding provides a proximate mechanism for regulati
137 ed potential epigenetic mechanisms mediating pair-bond regulation and found that the histone deacetyl
138 ASR is strongly correlated with long-term pair bonds, since the divorce rate is higher in species
139 We investigated the sensitivity of jackdaws, pair-bonded social corvids that exhibit an analogous eye
140 s complex mammalian social behaviors such as pair bonding, social recognition and aggression causally
142 this structure is crucial for mating-induced pair bonding, suggesting an important role for the VNO i
143 ghly social, monogamous species and displays pair bonding that can be assessed by the presence of sel
144 crotus ochrogaster), mating induces enduring pair-bonds that are initiated by partner preference form
145 suggest that repeated AMPH exposure impairs pair bonding through an OT-mediated mechanism, and that
146 rgument that connects the evolution of human pair bonds to the male-biased mating sex ratios that acc
148 a partner preference, a choice test in which pair-bonded voles regularly prefer their partner to a co
149 ccumbens in the formation and maintenance of pair bonds was assessed in a series of experiments using
150 e bonding was attributed to the reduced lone pair bond weakening effect, LPBWE, upon substitutions wi
151 sioned females failed to show mating-induced pair bonding whereas intact and sham-lesioned females di
152 cies of vole, one of which is monogamous and pair bonds whereas the other species is promiscuous and
153 in the lateral septum blocked mating-induced pair bonding, whereas administration of AVP induced this
154 Prairie vole breeder pairs form monogamous pair bonds, which are maintained through the expression
155 prairie voles (Microtus ochrogaster) form a pair bond with a female partner after mating, and this b
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