ライフサイエンスコーパス: pair-fed

1 es from control counterpart Na-exposed rats (pair-fed 216 ppm sodium acetate).

2 in fortified ethanol-containing liquid diet, pair fed a calorically equivalent sucrose-containing die

3 nd the control animals in each instance were pair fed a diet containing the basal requirement of thes

4 ment, transgenic and nontransgenic mice were pair fed a nutritionally complete liquid diet for 16 wee

5 hanol-containing Lieber-DeCarli diet or were pair-fed a control diet.

6 Rats were pair-fed a modified Lieber-DeCarli liquid diet containin

7 administered by osmotic minipumps for 8 d to pair-fed adult rats.

8 (<1 mg Zn/kg) or zinc-adequate (35 mg Zn/kg, pair-fed) adult male rats, and zinc levels were manipula

9 ipose triglycerides with deuterium, and then pair-fed alcohol or control diet for 2 weeks.

10 i liquid diets, with matched control animals pair fed an isocaloric alcohol-free diet to ensure equal

11 om gestational day (G) 6 through G21 or were pair fed an isocaloric nonalcoholic liquid diet.

12 Mice were pair-fed an alcohol or control liquid diet for 8 weeks t

13 Male 129S mice were pair-fed an alcohol or isocaloric maltose dextrin liquid

14 Mice were pair-fed an alcohol-containing liquid diet for 4 weeks.

15 nein-knockout and wild-type 129/Sv mice were pair-fed an ethanol-containing liquid diet for 12 weeks,

16 dam's diet, whereas control rats were either pair-fed an iso-caloric diet or given food ad libitum.

17 ion, respectively), as compared with animals pair-fed an isocaloric control diet containing the same

18 th an ethanol-containing liquid diet (Et) or pair-fed an isocaloric non-alcoholic diet (Ct).

19 Compared with those for isocaloric pair-fed and ad lib chow control animals, the results fo

20 Compared to isocaloric pair-fed and ad libitum control groups, the alcohol-expo

21 )Cr release assay, as compared with those in pair-fed and ad libitum-fed animals.

22 Controls were pair-fed and received dextrose isocaloric to ethanol.

23 ivity, in the splenocytes of ad libitum-fed, pair-fed, and ethanol-fed Sprague Dawley male rats.

24 Pair-fed animals lost one-fourth of the weight lost by t

25 eight changes were also monitored and yoked, pair-fed animals were used to control for possible chang

26 ntake and weight gain in hyperleptinemic and pair-fed animals, identifiable fat tissue was completely

27 measuring percent HbA1c in BDNF-treated and pair-fed animals, we show that the effects of BDNF on no

28 libitum-fed exercise (AL+Exe), exercise but pair-fed at the amount as controls (PF+Exe), 20% DCR, an

29 -gamma treatment inhibited liver fibrosis in pair-fed but not in ethanol-fed mice.

30 ntrations in ethanol-fed cells compared with pair-fed cells, without significant differences in total

31 Western blots of renal membrane protein from pair-fed CON and OVX revealed bands at 129-135 kD, but t

32 back in a nonrestricted manner (N) or under pair-fed conditions (A+) to yield weight-matched rats.

33 Wistar male rats were pair-fed control (C) or 5% ethanol (E) Lieber-DeCarli li

34 e also obtained in two hypothalamic areas of pair-fed control animals.

35 ere fed an ethanol-containing liquid diet or pair-fed control diet for 4 (11% total kcal;early respon

36 were fed an ethanol-containing or isocaloric pair-fed control diet for 8 weeks, followed by DC isolat

37 2 days with 32.4% of calories as ethanol or pair-fed control diet.

38 Female C57BL6/J mice were fed ethanol or pair-fed control diets and treated or not with HA35.

39 ss to 2% (vol/vol) ethanol (11% calories) or pair-fed control diets for 2 days, 2 weeks or 5 weeks an

40 d free access to ethanol-containing diets or pair-fed control diets for 4 or 25 days.

41 /- mice were fed ethanol-containing diets or pair-fed control diets for 6 weeks.

42 arli ethanol-containing diets for 6 weeks or pair-fed control diets.

43 tal iron/kg body weight as iron dextran) and pair-fed control groups.

44 Mice were fed an ethanol or isocaloric pair-fed control liquid diet followed by immunizations w

45 Mice were fed an ethanol or isocaloric, pair-fed control liquid diet for 8 weeks, followed by im

46 The bone marrow of pair-fed control mice receiving intraperitoneal saline s

47 cellular domain compared with ECs of AVFs in pair-fed control mice.

48 mary cultures of Kupffer cells compared with pair-fed control mice.

49 s increased susceptibility was reproduced in pair-fed control mitochondria pretreated with diethylmal

50 Body weight in RYGB pigs and sham-operated, pair-fed control pigs developed similarly.

51 red saline) for 12-14 days (VEH; n = 10); 3) pair-fed control rats given a daily food ration matching

52 The pair-fed control rats that ate the same amount of food a

53 as evaluated in muscles of acidotic, CKD and pair-fed control rats under physiologic conditions and i

54 Pair-fed control rats were generated for both the pre- a

55 streptozotocin-treated and vehicle-injected, pair-fed control rats were studied.

56 content was 0, compared with 14 ng/islet in pair-fed control rats, we coperfused a 2:1 oleate:palmit

57 -fold in livers from uremic rats compared to pair-fed control rats.

58 eptin binding in anorectic tumor-bearing and pair-fed control rats.

59 mg/d s.c. for 3-4 d) and were compared with pair-fed control rats.

60 ia induced by subtotal nephrectomy, and from pair-fed control rats.

61 nucleus by 30% compared with saline-treated, pair-fed controls (P < 0.05).

62 ate gyrus compared to both zinc-adequate and pair-fed controls (p<0.05).

63 inhibitor diethyldithiocarbamate to that of pair-fed controls abolished APAP toxicity in the 10-day

64 levation of ubiquitin above that in cells of pair-fed controls and this difference exceeded the relat

65 et-induced obese C57BL/6J mice compared with pair-fed controls and was associated with suppressed exp

66 s of adipose tissue inflammation relative to pair-fed controls independent of increased body weight o

67 Pair-fed controls lost 24-50% less body weight than C75-

68 r S-nitrosylated mitochondrial proteins from pair-fed controls or alcohol-fed rat livers were subsequ

69 rats were fed a K-deficient diet for 8 d and pair-fed controls received a K-replete diet.

70 ts after hyperleptinemia disappears, whereas pair-fed controls regain their weight in 2 weeks.

71 atocytes from long-term ethanol-fed rats and pair-fed controls were stimulated with EGF (0.5-20 nmol/

72 egradation in ethanol-fed rats compared with pair-fed controls with no effect of ethanol on triglycer

73 Compared with the pair-fed controls, COX activity was decreased with expos

74 Compared with pair-fed controls, RYGB rats had significant reduction i

75 ignificantly lower in hyperleptinemic versus pair-fed controls, while fatty acid and glucose levels w

76 seline and in response to LPS, compared with pair-fed controls.

77 seline and in response to LPS, compared with pair-fed controls.

78 ion, SDH) from these rats were compared with pair-fed controls.

79 P-1 mRNA levels, compared with sham-operated pair-fed controls.

80 d mdr2 mRNA levels to 57% +/- 2 (P <.001) of pair-fed controls.

81 ls, while no such increases were observed in pair-fed controls.

82 renal failure (CRF) and sham operated (SO), pair-fed controls.

83 ver cytosol from ethanol-fed rats but not in pair-fed controls.

84 g ethanol using the Lieber-DeCarli diet with pair-fed controls.

85 roduction in isolated KCs when compared with pair-fed controls.

86 male Wistar rats was decreased compared with pair-fed controls.

87 inhibition of respiration relative to their pair-fed controls.

88 cause of fatty acid oxidation, compared with paired-fed controls.

89 terol, and phospholipid than obese and lean (pair-fed) controls that were fed high-fat diets.

90 In contrast, the estrous cycles of their pair-fed counterparts remained disrupted.

91 cocaine HCl daily on gestational Days 8-20, pair-fed dams injected with saline, or nontreated contro

92 ith restoration toward normal in livers from pair-fed db/db mice.

93 T and liver-to-body weight ratio compared to pair-fed DEN-injected mice.

94 Animals were pair-fed either a 6 or 24% casein-based diet for 7-10 da

95 A computerized pair-fed experiment showed that adjuvant-induced hypopha

96 Rats were pair-fed for 7 months with a liquid diet containing eith

97 nd OVX rats fed ad libitum for 6 and 9 wk or pair-fed for 9 wk were processed for transmission electr

98 y weight and food intake were examined and a pair-fed group was included to determine if fluoxetine-i

99 In the AGRP pair-fed group, a significant increase in the epididymal

100 +/- 8% of saline control; P < 0.01) and AGRP pair-fed groups (24 +/- 7% of saline control; P < 0.01).

101 ols in both the AGRP ad libitum fed and AGRP pair-fed groups (3.5 +/- 0.3 [saline] vs. 2.7 +/- 0.4 [A

102 Male rats were divided into 3 pair-fed groups (n = 13/group) as follows: Control rats

103 cids were elevated in both tumor-bearing and pair-fed groups, while circulating levels of triglycerid

104 re detected between the nicotine-treated and pair-fed groups.

105 n plasma insulin noted in vehicle-treated or pair-fed groups.

106 also treated prediabetic, ad libitum-fed and pair-fed Lean-huIAPP transgenic males.

107 ts, 12 months of age, were fed an ethanol or pair-fed liquid diet, or rat chow for a period of 10, 20

108 Rats were pair-fed liquid diets containing either ethanol (36% of

109 Rats were pair-fed liquid diets containing either ethanol or isoca

110 Rats were pair-fed liquid diets containing either ethanol or isoca

111 cient ones in these experiments, even though pair fed, makes it difficult to isolate effects of zinc

112 nd were either allowed to feed ad libitum or pair-fed matched (PF SAL) to COC subjects to control for

113 Pair-fed Mc4r-null females maintained body weights inter

114 levels were elevated in both male and female pair-fed Mc4r-null mice compared with WT mice.

115 nges nor the MS were present in age-matched, pair-fed MG(-) mice.

116 g; P < 0.001) and lean mass (P < 0.001) than pair-fed mice at 22 degrees C.

117 Despite a similar calorie intake, pair-fed mice at 27 degrees C (PF27) were heavier (28.3

118 Pair-fed mice experienced reductions of glucose and insu

119 xposed to CIH for 12 weeks and compared with pair-fed mice exposed to intermittent air (IA, n = 15).

120 in CNTF(Ax15)-treated UCP1-DTA compared with pair-fed mice of similar body weight.

121 Pair-fed mice were provided either a CR diet or a high-A

122 x15) treatment of CNTF(Ax15)-treated but not pair-fed mice, followed by a gradual regain in body weig

123 attenuated in ethanol-fed mice compared with pair-fed mice, which was due to reduced natural killer g

124 is of HSCs in ethanol-fed mice compared with pair-fed mice.

125 ell cycle arrest and apoptosis compared with pair-fed mice.

126 of MTII is increased to levels comparable to pair-fed mice.

127 Three groups of male Wistar rats were pair fed NIH-31 diets for 14 days to which were added 30

128 9-kD liver UT-A protein by 36% compared with pair-fed nonacidotic rats.

129 Compared to controls pair-fed normal chow, carbonyl iron-fed rats had elevate

130 o 20% higher in ad libitum-fed obese than in pair-fed obese group.

131 ted HSC-specific NOX4 KO mice and these were pair-fed on alcohol diet.

132 icarboxylic acid (TCA) cycle flux, rats were pair-fed on diets consisting of 1) 59% safflower oil, 2)

133 -resistant diabetes and obesity, were either pair fed or treated with the Sglt inhibitor phloridzin,

134 s were sham-operated animals who were either pair-fed or ad libitum-fed.

135 ECs from ethanol-fed rats when compared with pair-fed or chow-fed rats.

136 d RYGB and VSG compared with those that were pair-fed or fed ad lib.

137 eated with TNF-alpha or leptin or in animals pair-fed over a 7-day time course using 11,000-gene micr

138 ad libitum fed] vs. 2.1 +/- 0.2 ng/ml [AGRP pair-fed]; P < 0.01).

139 ncreased the available amount of ACh in both pair-fed (PF) and PTD rats, it did so to a greater exten

140 pocampus and the amygdala of PTD-treated and pair-fed (PF) control rats while they were tested on a s

141 Zucker rats were studied: RYGB, sham surgery pair-fed (PF), and sham surgery ad libitum (AL) fed rats

142 tion of region (FC, RSC) and treatment (PTD, pair-fed [PF]).

143 Pair-fed pregnant animals exposed to ambient conditions

144 Serum IGF-I levels in both hypoxic and pair-fed pups were significantly lower than in control a

145 f the study, body weights of the hypoxic and pair-fed pups were significantly lower than the weights

146 In related studies, in pair fed rats fed a low calorie diet for 4 days, the con

147 r) in vitro compared with Kupffer cells from pair-fed rats (< 150 pg/10(6) Kupffer cells/24 hr).

148 f TUNEL-labeled cells in the SGZ compared to pair-fed rats (p<0.05).

149 cultures of Kupffer cells from ethanol- and pair-fed rats after treatment with HA35.

150 Pair-fed rats were treated with either FK506 (1 mg/kg/da

151 ease in oxygen consumption compared with the pair-fed rats, but there were no changes in oxygen consu

152 In pair-fed rats, effects similar to fluoxetine treatment w

153 effects were greater than those observed in pair-fed rats, suggesting that although Rb1's antihyperg

154 y 2-fold over baseline in Kupffer cells from pair-fed rats.

155 in the sera of alcoholic rats but not in the pair-fed rats.

156 chlearis muscles from CRF and sham-operated, pair-fed rats.

157 bese controls, RYGB, and sham-operated obese pair-fed rats.

158 rate (HR) were evaluated in radiotelemetered pair-fed sham-operated (SO), ovariectomized (OVX), and O

159 (jejunectomy), ileal resection (ileectomy), pair-fed sham-operated, and nonoperated controls.

160 ssessed 30 days postsurgery in GIBP and sham pair-fed (sham.PF) groups.

161 ct ligation (BDL) for 4 weeks (n = 5) and in pair-fed, sham-operated control rats (n = 4).

162 kDa protein increased 1.9-fold compared with pair-fed, sham-operated rats, whereas the 51- and 39-kDa

163 ortacaval anastomosis rat and sham-operated, pair-fed Sprague-Dawley rats treated with ammonia-loweri

164 Pair-fed Sprague-Dawley rats were fed a low-sodium (0.03

165 e housed at 27 degrees C or 22 degrees C and pair fed the same diet for 21 weeks (95% of ad libitum i

166 All control animals were pair fed the same diets as ethanol-fed rats except that

167 red these with saline-infused rats that were pair-fed the amount of food consumed by the leptin-treat

168 Additionally, a group of inactive rats was pair-fed the amount of food consumed on the previous day

169 containing 5 ppm total zinc; and group 3 was pair-fed the control diet but restricted in amount to th

170 lated mitochondria from mice brain that were pair-fed the ethanol (4% v/v) and control liquid diets f

171 RAGE and p66(shc) levels compared with mice pair-fed the regular (Reg(AGE)) diet.

172 Rats from each group were pair-fed the same diet for 20 to 22 d.

173 ietary AGEs promote AD, we evaluated WT mice pair-fed three diets throughout life: low-AGE (MG(-)), M

174 , another group of STZ-injected animals were pair fed to the intake of those receiving leptin.

175 fed rats and rats administered AGRP and then pair-fed to a saline control group.

176 compared to sham-operated rats fed ad lib or pair-fed to animals that received RYGB.

177 Control rats were pair-fed to BDL rats, and all rats were fed an artificia

178 er, when corticosterone-treated animals were pair-fed to control intake, aiming to prevent the cortic

179 Control, Whey, Lactalbumin, Lactoferrin, or pair-fed to lactoferrin.

180 Control rats were pair-fed to leptin-treated animals.

181 termates fed ad libitum (AL); and db/db mice pair-fed to match the intake of db/m mice.

182 ther the subjects were allowed to overeat or pair-fed to the BCD (P>0.05).

183 libitum, Fat/Sucrose ad libitum, Fat/Sucrose pair-fed to the caloric intake of CHO, or Fat/Sucrose at

184 d either BCD ad libitum, HPD ad libitum, HPD pair-fed to the caloric intake of the BCD, or the HPD at

185 n hyperleptinemic rats, whereas control rats pair-fed to the hyperleptinemic rats retained approximat

186 ntrols were either fed ad libitum (n = 8) or pair-fed to the intake of the leptin-treated group (n =

187 ibitum, whereas the control (Ctrl) mice were pair-fed to the KE group.

188 .5 microl/hr for 12 days), and a subset were pair-fed to vehicle-infused controls.

189 umption by Mc4r-null mice was restricted to (pair-fed to) that consumed by wild-type (WT) mice.

190 RYGB or VSG compared with rats fed ad lib or pair-fed, whereas glucose clearance was similar in all g

191 r extent after ethanol feeding compared with pair-fed wild-type mice.

192 Obese rats were also pair fed with lean controls to prevent hyperphagia.

193 e and littermate loxP control (WT) mice were pair-fed with either an ethanol-containing diet or an et

194 Results were compared to control groups pair-fed with ethanol-free liquid diet and trained to se

195 t was detected in the control rats that were pair-fed with isocaloric amounts of dextrose.

196 C57BL/6J mice (n = 12/group) were isocaloric pair-fed with Lieber-DeCarli liquid diet containing eith

197 disease-free) and the MTX-treated group were pair-fed with positive controls (i.e., untreated AIA rat

198 ther caused by hyperphagia because they were pair-fed with the control group nor caused by increased

199 r half were maintained unchallenged and were pair-fed with the infected fish.

200 sham-operated rats, fed either ad libitum or pair-fed with the VSG group, were used as controls.

201 ormalized linear growth rate to that seen in pair-fed WT littermate controls.

202 lamic NPY levels by nearly 50% compared with pair-fed young rats, whereas there were no changes in th

203 y groups: zinc-deficient, zinc-adequate, and pair-fed zinc-adequate.