ライフサイエンスコーパス: pallidum

1 sal ganglia (including the putamen and right pallidum).

2 s that receive cortical input and project to pallidum.

3 nucleus accumbens projections to the ventral pallidum.

4 on of motor variability first emerges in the pallidum.

5 cuits in nucleus accumbens (NAc) and ventral pallidum.

6 ctyostelium fasciculatum and Polysphondylium pallidum.

7 s associated with macrolide resistance in T. pallidum.

8 orsal striatum, prelimbic cortex, or ventral pallidum.

9 er, with both cell types innervating ventral pallidum.

10 a X that sits within the surrounding striato-pallidum.

11 s likely role in metal ion homeostasis in T. pallidum.

12 lost in D2, but not D1 inputs to the ventral pallidum.

13 y undescribed projection of the avian dorsal pallidum.

14 tor functioned when infused into the ventral pallidum.

15 involving the nucleus accumbens and ventral pallidum.

16 -insula and the ventral striatum and ventral pallidum.

17 statement of cocaine seeking via the ventral pallidum.

18 teral BF, in regions occupied by the ventral pallidum.

19 iMSNs than their projections to the ventral pallidum.

20 lasticity in the dorsal and ventral striatum/pallidum.

21 ity to culture and genetically manipulate T. pallidum.

22 mus (-0.82 [0.19] vs 0.41 [0.24]; P = .001), pallidum (-0.78 [0.18] vs 0.43 [0.48]; P = .001), and hi

23 , ventral striatum (VST) (+14%; p<0.01), and pallidum (+11%; p=0.02).

24 he globus pallidus (+9%; p=0.06) and ventral pallidum (+11%; p=0.1), whereas binding was slightly low

25 philis was defined as detection of Treponema pallidum 16S RNA in CSF or CSF white blood cells (WBCs)

26 D3-rich regions, variability was low in the pallidum (6%) but higher in substantia nigra (19%), thal

27 68 T. pallidum Ab-positive and 100 of 102 T. pallidum Ab-negative samples, and the HCV assay correctl

28 hilis assay correctly identifies 67 of 68 T. pallidum Ab-positive and 100 of 102 T. pallidum Ab-negat

29 Furthermore, left pallidum activity was significantly elevated after EX wh

30 negative, triggers a confirmatory treponemal pallidum agglutination test.

31 uster spanning the ventral striatum, ventral pallidum, amygdala, midbrain, and orbitofrontal cortex (

32 PFC functional connectivity with the ventral pallidum, an AVP receptor-rich region previously associa

33 Treponema pallidum, an obligate parasite of humans and the causati

34 ses in accumbens volume and local changes in pallidum and caudate volume occurred in patients defined

35 brain regions studied (hippocampus, ventral pallidum and cerebellum), or of the effects of chronic k

36 gton's disease was decreased in striatum and pallidum and increased in motor thalamic nuclei, compare

37 osynaptic inhibitory currents in the ventral pallidum and lateral habenula, though the net effects on

38 lume, but larger bilateral caudate, putamen, pallidum and lateral ventricle volumes.

39 s including the syphilis bacterium Treponema pallidum and Lyme disease pathogen Borrelia burgdorferi,

40 the interplay of the timing of STN input to pallidum and pallidal neuronal dynamics, resulting in se

41 ophy, particularly in the thalamus, caudate, pallidum and putamen, and this was most apparent in seco

42 onse in the DRD3-rich regions of the ventral pallidum and substantia nigra.

43 al size and shape; focusing on the striatum, pallidum and thalamus.

44 lized to the posterior third of the internal pallidum and theta power correlated with proximity to th

45 es send GABAergic projections to the ventral pallidum and were found to differentially promote cue-in

46 (Borrelia burgdorferi), syphilis (Treponema pallidum) and leptospirosis (Leptospira interrogans), an

47 tantial GABAergic innervation to the ventral pallidum, and chemogenetic inhibition of ventral pallida

48 fic neurons in nucleus accumbens and ventral pallidum, and how these changes are associated with posi

49 based shape metrics of the caudate, putamen, pallidum, and nucleus accumbens in 53 depressed patients

50 including the sensorimotor cortex, putamen, pallidum, and substantia nigra.

51 nction for D1-MSN innervation of the ventral pallidum, and suggest that losing LTDGABA in D2-MSN, but

52 We show that the striatum, pallidum, and thalamus each follow curvilinear trajector

53 subcortical structures such as the striatum, pallidum, and thalamus has remained poorly described--de

54 entral tegmentum, nucleus accumbens, ventral pallidum, and the orbitofrontal prefrontal cortex), the

55 ilis, the Elecsys Syphilis assay detected T. pallidum antibodies for 3 patients for whom antibodies w

56 ic reader for detection of HIV and Treponema pallidum antibodies in 450 previously characterized seru

57 and compared to reference HIV and Treponema pallidum antibody detection methods.

58 For visual T. pallidum antibody detection, the test sensitivity was 94

59 cts IgG and IgM antibodies against Treponema pallidum antigens in human serum and plasma.

60 gest that theta oscillations in the internal pallidum are robustly associated with dystonic symptoms

61 ous models, we propose that the striatum and pallidum are stretched along the rostrocaudal axis of th

62 rticularly the nucleus accumbens and ventral pallidum as well as actions within the ascending seroton

63 mygdala, hippocampus, thalamus, putamen, and pallidum), as well as insular cortex, is associated with

64 ging revealed that right ventral putamen and pallidum atrophy correlated with higher reward-seeking s

65 s-induced mu-opioid activation (left ventral pallidum, bilateral anterior cingulate cortices, right h

66 a suggest an important role for TP0126 in T. pallidum biology and syphilis pathogenesis.

67 o similar to poor ex vivo phagocytosis of T. pallidum by host macrophages reported previously.

68 d ulcers were tested for HSV-2 and Treponema pallidum by polymerase chain reaction (PCR).

69 n this study, we analyzed the proteome of T. pallidum by the isoelectric focusing (IEF) and nonequili

70 Unlike many pathogenic bacteria, Treponema pallidum cannot synthesize riboflavin; we recently descr

71 sterior cingulate cortex, thalamus, putamen, pallidum, caudate, hippocampus, and brain stem.

72 T. pallidum cells appeared to form flat waves, did not cont

73 es also provided the first observation of T. pallidum chemoreceptor arrays, as well as structural det

74 pmental shift in bilateral posterior putamen/pallidum clusters from preferential connectivity with th

75 alamus, medial prefrontal cortex, and globus pallidum compared to euglycemia for both PASL-MRI and PE

76 Blood T. pallidum concentrations were determined by real-time pol

77 highest opsonic activity had lower blood T. pallidum concentrations.

78 mple, our findings suggest that the external pallidum could be a new target for cell-based therapies

79 sus D2-MSN GABAergic synapses in the ventral pallidum could explain the differential regulation of VP

80 footprinting assay, recombinant TP0262, a T. pallidum CRP homologue, was shown to bind specifically t

81 syphilis was defined as undetectable CSF T. pallidum, CSF WBCs </=5/uL and nonreactive CSF-VDRL.

82 l pallidum (RVP), but not the caudal ventral pallidum (CVP), were robustly Fos activated during cue-i

83 cranial volumes (d=-0.12), as well as larger pallidum (d=0.21) and lateral ventricle volumes (d=0.37)

84 athogens to directly capture whole-genome T. pallidum data in the context of human infection.

85 the swimming speeds of B. burgdorferi and T. pallidum decrease with increases in viscosity of the ext

86 The product of the tp0971 gene of T. pallidum, designated Tp34, is a periplasmic lipoprotein

87 atrophy was not observed in the caudate and pallidum despite marked amyloid accumulation.

88 e able to cause or contribute to disease, T. pallidum differs in that it is able to rapidly dissemina

89 , suggesting a role for these proteins in T. pallidum dissemination and tissue invasion.

90 e dorsolateral subcompartment of the ventral pallidum (dlVP) and through the direct pathway to the me

91 Treponema pallidum DNA was isolated from 158 patients with syphili

92 Because T. pallidum does not possess other enzymes for ROS detoxifi

93 n immune evasion of the obligate pathogen T. pallidum during infection.

94 distinguish area X from the ventral striato-pallidum during singing.

95 ubregions of medial accumbens shell, ventral pallidum, elements of extended amygdala, and lateral sep

96 rs facility, finding 5.5% reactive Treponema pallidum enzyme immunoassay (EIA) tests.

97 was recovered from 28 (57%) specimens and T. pallidum from none; one woman showed serologic evidence

98 irus gene (HIV), and the syphilis (Treponema pallidum) gene.

99 inding sites and corresponding to as many T. pallidum genes, were identified.

100 The T. pallidum genome sequence has revealed a few open reading

101 e small number of proteins encoded by the T. pallidum genome with sequence similarity to well-charact

102 rtex, and in contralateral amygdala, ventral pallidum, globus pallidus, and hippocampus, as well as d

103 ysis confirmed prior results showing that T. pallidum glycolipids are not immunoreactive, and (iii) l

104 ith standard laboratory tests (the Treponema pallidum haemagglutination assay [TPHA] and the RPR test

105 ce of reactive combined serology (positive T pallidum haemagglutination test and rapid plasmin reagin

106 ance for syphilis research, suggests that T. pallidum has appropriated a paradigmatic global regulato

107 ry in both the nucleus accumbens and ventral pallidum has been reported to mediate taste-reactivity r

108 Thus, T. pallidum has evolved an extraordinarily robust, broad-sp

109 T. pallidum has long been regarded as a stealth pathogen be

110 opto-MOR in GABAergic neurons of the ventral pallidum hedonic cold spot led to real-time place aversi

111 nome sizes (Mycoplasma pneumoniae, Treponema pallidum, Helicobacter pylori, Campylobacter jejuni, Syn

112 (CSF-TPPA) is sensitive and a CSF Treponema pallidum hemagglutination assay (CSF-TPHA) titer of >/=1

113 The dorsal pallidum in birds is considered similar, if not homologo

114 e chain reaction (PCR) testing for Treponema pallidum in cerebrospinal fluid (CSF) samples.

115 ratory test (CSF-VDRL), (ii) detection of T. pallidum in CSF by reverse transcriptase PCR, or (iii) n

116 between opsonic activity and detection of T. pallidum in CSF or CSF-VDRL reactivity.

117 alamus, medial prefrontal cortex, and globus pallidum in response to hypoglycemia.

118 ays and connectivity patterns related to the pallidum in schizophrenia.

119 HD-related increases in DC in right striatum/pallidum, in contrast with ASD-related increases in bila

120 T. pallidum-infected rabbits mount a vigorous antibody resp

121 reactive CIA specimens may represent true T. pallidum infection and may be found after seroreversion

122 t-pathogen interactions that occur during T. pallidum infection.

123 an inflammatory eye disease due to Treponema pallidum infection.

124 Treponema pallidum infections can have severe complications if not

125 ategy has unveiled a scenario of discreet T. pallidum interstrain single-nucleotide-polymorphism-base

126 However, the ventral pallidum is a heterogeneous structure, and how this comp

127 The ventral pallidum is centrally positioned within mesocorticolimbi

128 Treponema pallidum is cleared from sites of infection by opsonizat

129 tional regulation in Treponema pallidum ssp. pallidum is poorly understood, primarily because this or

130 The spirochete Treponema pallidum subsp. pallidum is the causative agent of syphilis, a chronic,

131 Treponema pallidum subsp. pallidum is the causative agent of syphilis, a sexually

132 f the etiologic agent of syphilis, Treponema pallidum, is compact and devoid of many metabolic enzyme

133 an extended inflexible structure, and, in T. pallidum, is tightly bound to the protoplasmic cylinder.

134 is research, the Nichols strain of Treponema pallidum, isolated in 1912, has been the most widely stu

135 ession sites, among eight T. pallidum subsp. pallidum isolates (Nichols Gen, Nichols Sea, Chicago, Se

136 hina, Ireland, and Madagascar and from 15 T. pallidum isolates.

137 horesis revealed a modular Bam complex in T. pallidum larger than that of Escherichia coli.

138 and brainstem targets, including the ventral pallidum, lateral and magnocellular preoptic nuclei, lat

139 ions with the hippocampus, amygdala, ventral pallidum, lateral hypothalamus, and ventral tegmental ar

140 ate, left orbitofrontal cortex, left ventral pallidum, left amygdala, and left inferior temporal cort

141 protein shares similarity with the Treponema pallidum LRR (LRR(TP)) family of proteins and contains s

142 rge dorsomedial neurons of the caudal dorsal pallidum may be involved in sensory processing, in that

143 A in D2-MSN, but not D1-MSN input to ventral pallidum may promote cue-induced reinstatement of cocain

144 ficiencies explicit, and suggest that the T. pallidum network topology is inconsistent with evolution

145 Ventral pallidum neurons do not ordinarily fire vigorously to a

146 se in behavior and firing signals of ventral pallidum neurons, and likewise, they increased incentive

147 When the T. pallidum subsp. pallidum Nichols strain genome was initially annotated,

148 but not to volumes of the thalamus, putamen, pallidum, nucleus accumbens, or caudate nucleus.

149 of kappa-opioid receptors within the ventral pallidum or mu-opioid receptors with the specific mu-opi

150 Extrapolated to T. pallidum, our model enables us to explain how individual

151 ups of candidate rare OMPs, the predicted T. pallidum outer membrane proteome (OMPeome), which we pos

152 fference between children and adults for the pallidum (p=0.79) or thalamus (p=0.89).

153 here was no difference in volume size in the pallidum (p=0.95) and thalamus (p=0.39) between people w

154 nescence immunoassay (CLIA), and a Treponema pallidum particle agglutination (TP-PA) test.

155 d by rapid plasma reagin (RPR) and Treponema pallidum particle agglutination (TP.PA) testing (n = 231

156 ng with rapid plasma reagin (RPR), Treponema pallidum particle agglutination (TPPA), and fluorescent

157 ay [Zeus Scientific, Raritan, NJ], Treponema pallidum particle agglutination [TP-PA; Fujirebio Diagno

158 Sure EIA) and three manual assays (Treponema pallidum particle agglutination [TP-PA], fluorescent tre

159 ggest that the cerebrospinal fluid Treponema pallidum particle agglutination assay (CSF-TPPA) is sens

160 y underwent reflexive testing with Treponema pallidum particle agglutination assay (TP-PA) and were c

161 tive serology were tested with the Treponema pallidum particle agglutination assay (TP-PA) at Kaiser

162 ted using the rapid plasma reagin, Treponema pallidum particle agglutination, and chemiluminescence i

163 d plasma reagin (RPR) test and the Treponema pallidum passive particle agglutination (TP-PA) assay.

164 sly believed to be exclusively located in T. pallidum periplasm.

165 81%) participants-either H ducreyi (n=42), T pallidum pertenue (yaws; n=19), or coinfection with both

166 We investigated T pallidum pertenue and another bacterium known to cause s

167 n lesional swabs to detect the presence of T pallidum pertenue and H ducreyi.

168 . pallidum protein spots, representing 88 T. pallidum polypeptides; 63 of these polypeptides had not

169 Differences in the putamen and pallidum (positive association) were significant after a

170 The dorsal pallidum projects to both thalamic and midbrain targets

171 ode the major genetic mechanisms by which T. pallidum promotes immune evasion and survival, and demon

172 We determined the identity of 148 T. pallidum protein spots, representing 88 T. pallidum poly

173 Previously, the T. pallidum proteins, Tp0750 and Tp0751 (also called pallil

174 s accumbens, amygdala, caudate, hippocampus, pallidum, putamen and thalamus.

175 , particularly between the left IFG and left pallidum, putamen, and insular cortex, is associated wit

176 s accumbens, amygdala, caudate, hippocampus, pallidum, putamen, thalamus, and lateral ventricle).

177 rtex, caudate, frontal cortex, hypothalamus, pallidum, putamen, thalamus, and ventral striatum) showe

178 Regionally, we found higher CBF in the right pallidum/putamen of the cannabis users compared with non

179 s associated with a significant reduction in pallidum/putamen responses to pictures of high-calorie f

180 (r=0.34, p=0.03), VST (r=0.36, p=0.02), and pallidum (r=0.30, p=0.05) across all subjects.

181 Treponema pallidum rapidly disseminates from a genital site of ino

182 Identification of Treponema pallidum rare outer membrane proteins (OMPs) has been a

183 Definitive identification of Treponema pallidum rare outer membrane proteins (OMPs) has long el

184 In addition to being bona fide T. pallidum rare outer membrane proteins, TprC/D and TprI r

185 Treponema pallidum reacts poorly with the antibodies present in ra

186 l changes in the dorsal and ventral striatum/pallidum relate to or predict therapeutic response to EC

187 l as the parental ortholog for the Treponema pallidum repeat (Tpr) family in the syphilis spirochete.

188 TP0117/TP0131 (TprC/D), a member of the T. pallidum repeat (Tpr) family, was a highly ranked candid

189 tprE, tprG and tprJ, three members of the T. pallidum repeat (tpr) gene family (subfamily II).

190 nt length polymorphism (RFLP) analysis of T. pallidum repeat (tpr) subfamily II genes, (3) RFLP analy

191 We previously identified Treponema pallidum repeat proteins TprC/D, TprF, and TprI as candi

192 , suggesting a role for TP0092 in helping T. pallidum respond to harmful stimuli in the host environm

193 ainst specific TprK epitopes expressed on T. pallidum, resulting in immune selection of new TprK vari

194 , precunei, cunei, bilateral putamena, right pallidum, right thalamus, cerebellum, middle frontal, mi

195 projections to VTA from the rostral ventral pallidum (RVP), but not the caudal ventral pallidum (CVP

196 ward within the nucleus accumbens to ventral pallidum segment of mesocorticolimbic circuitry.

197 This rapid assay detected HIV and Treponema pallidum serum antibodies with sensitivities of 100% (95

198 direct local field potentials from the human pallidum simultaneously with whole head magnetoencephalo

199 Impaired T. pallidum-specific immune responses could contribute to d

200 Serum T. pallidum-specific opsonic activity is significantly lowe

201 individuals with syphilis were tested for T. pallidum-specific opsonic activity.

202 Transcriptional regulation in Treponema pallidum ssp. pallidum is poorly understood, primarily b

203 We describe an enhanced T. pallidum strain typing system that shows biological and

204 Recently, important differences among T. pallidum strains emerged; therefore, we sequenced and an

205 None of the T. pallidum strains examined had both point mutations.

206 e A2058G or the A2059G mutation among the T. pallidum strains were 35.6, 51.2, and 13.2%, respectivel

207 oth copies of the 23S rRNA gene in Treponema pallidum strains.

208 This double dissociation in ventral pallidum subregional roles in drug seeking is likely to

209 or presence of ulcers negative for Treponema pallidum subsp pertenue on PCR, and active yaws was defi

210 olates (Gauthier, CDC2, and Samoa D), one T. pallidum subsp. endemicum isolate (Iraq B), the unclassi

211 allidum, T. pallidum subsp. pertenue, and T. pallidum subsp. endemicum), Treponema paraluiscuniculi,

212 rial infection, which is caused by Treponema pallidum subsp. pallidum (TPA), has been re-emerging glo

213 The spirochete Treponema pallidum subsp. pallidum is the causative agent of syphi

214 Treponema pallidum subsp. pallidum is the causative agent of syphi

215 as the tprK expression sites, among eight T. pallidum subsp. pallidum isolates (Nichols Gen, Nichols

216 When the T. pallidum subsp. pallidum Nichols strain genome was initi

217 the three Treponema pallidum subspecies (T. pallidum subsp. pallidum, T. pallidum subsp. pertenue, a

218 Treponema pallidum subsp. pallidum, the agent of syphilis, is chal

219 In Treponema pallidum subsp. pallidum, the agent of syphilis, the TP0

220 n surface-exposed proteins, and in Treponema pallidum subsp. pallidum, the syphilis agent, it was rep

221 Dal-1, Street14, UW104, and UW126), three T. pallidum subsp. pertenue isolates (Gauthier, CDC2, and S

222 subspecies (T. pallidum subsp. pallidum, T. pallidum subsp. pertenue, and T. pallidum subsp. endemic

223 use of DNA sequencing to identify Treponema pallidum subsp. pertenue-specific sequences in a patient

224 Although the three Treponema pallidum subspecies (T. pallidum subsp. pallidum, T. pal

225 ence that TP0126 is fully conserved among T. pallidum subspecies and strains, these data suggest an i

226 Haemophilus ducreyi (HD) and Treponema pallidum subspecies pertenue (TP) are major causative ag

227 Yaws, caused by Treponema pallidum subspecies pertenue and diagnosed by the presen

228 f resistance to macrolides against Treponema pallidum subspecies pertenue was seen.

229 for Disease Control and Prevention (CDC) T. pallidum subtyping method.

230 iting in another slime mold, Polysphondylium pallidum, suggests organism-specific idiosyncrasies in t

231 orrelia burgdorferi) and syphilis (Treponema pallidum) swim through viscous fluids, such as blood and

232 ly for multiple antigens of HIV-1, Treponema pallidum (syphilis), and hepatitis C virus (HCV) in a co

233 onorrhoeae, Chlamydia trachomatis, Treponema pallidum (syphilis), herpes simplex virus 2, and HIV.

234 In Treponema pallidum (T. pallidum), the causative agent of syphilis,

235 nema pallidum subspecies (T. pallidum subsp. pallidum, T. pallidum subsp. pertenue, and T. pallidum s

236 splenium of the corpus callosum, left globus pallidum, thalamus and hippocampus (P < 0.01) were signi

237 or ADHD, including the hippocampus, putamen, pallidum, thalamus, midbrain with pons (comprising a reg

238 increased 18F-AV-1451 uptake in the putamen, pallidum, thalamus, midbrain, and in the dentate nucleus

239 onic bacterial infection caused by Treponema pallidum that is endemic in low-income countries and and

240 A total of 129 specimens PCR positive for T. pallidum that were obtained from an azithromycin resista

241 y for the detection of antibodies against T. pallidum The performance of this assay was investigated

242 In Treponema pallidum (T. pallidum), the causative agent of syphilis, the TprK Ag

243 Treponema pallidum subsp. pallidum, the agent of syphilis, is challenging to study

244 In Treponema pallidum subsp. pallidum, the agent of syphilis, the TP0092 gene is pred

245 Treponema pallidum, the bacterial agent of syphilis, is predicted

246 cryo-ET) was utilized to visualize Treponema pallidum, the causative agent of syphilis, at the molecu

247 nto the genomic adaptive traits of Treponema pallidum, the causative bacterium of syphilis, have long

248 is topographically distinct from the dorsal pallidum, the homolog of the globus pallidus interna.

249 d proteins, and in Treponema pallidum subsp. pallidum, the syphilis agent, it was reported to affect

250 The ability of Treponema pallidum, the syphilis spirochete to colonize various ti

251 he native cellular organization of Treponema pallidum, the syphilis spirochete.

252 linking this suppression to the striatum and pallidum, these results provide compelling functional ev

253 n host-associated Treponema that includes T. pallidum, this pathway is found in neither bacteria nor

254 In Treponema pallidum, this protein (TatT) is a water-soluble trimer

255 into abnormal information processing in the pallidum through alterations in oscillatory activity.

256 nown about the mechanisms by which Treponema pallidum (Tp), the causative agent of syphilis, copes wi

257 tion of the outer membrane (OM) of Treponema pallidum (Tp), the noncultivable agent of venereal syphi

258 oding the putative TRAP-T components from T. pallidum, tp0957 (the SBP), and tp0958 (the symporter),

259 which is caused by Treponema pallidum subsp. pallidum (TPA), has been re-emerging globally in the las

260 We found that in T. pallidum, TprC is heat modifiable, trimeric, expressed i

261 cated neuronal subpopulations in the ventral pallidum tracked signal enhancements for hedonic impact

262 This is the first characterization of a T. pallidum transcriptional modulator that influences tpr p

263 ce similarity at its C terminus to Treponema pallidum TroR.

264 vector (pHSVsiLA1) infused into the ventral pallidum, unrelated to TLR4.

265 ut supports non-vocal behaviors: the striato-pallidum ventral to area X (VSP), our focus here.

266 onic seizures correlated with a reduction of pallidum volume (r = -0.71; P = .03).

267 Putamen and pallidum volume augmentations were positively associated

268 escents following PCE (P = .03), whereas the pallidum volume was smaller in adolescents following pre

269 chizophrenia-specific leftward asymmetry for pallidum volume.

270 pinal tract, and reduced thalamic and globus pallidum volumes relate to deficits in cognitive functio

271 Here, we identify the ventral pallidum (VP) as a site of convergence of medium spiny n

272 Neural activity in the ventral pallidum (VP) has been shown to encode changes in the va

273 The ventral pallidum (VP) is a target of dense nucleus accumbens pro

274 Ventral pallidum (VP) is a well-established locus for the reinfo

275 The ventral pallidum (VP) is necessary for drug-seeking behavior.

276 The ventral pallidum (VP) is posited to contribute to reward seeking

277 present evidence suggesting that the ventral pallidum (VP) may participate in this process.

278 albumin-positive (PV) neurons in the ventral pallidum (VP) projecting to either the lateral habenula

279 GABAergic neurons in the ventral pallidum (VP) provide a major input to VTA neurons.

280 The ventral pallidum (VP) receives orexin projections from lateral h

281 that cue-evoked neural firing in the ventral pallidum (VP) reflected the strength of incentive motiva

282 Ventral pallidum (VP) serves important roles in reward and motiv

283 w that optogenetic inhibition of the ventral pallidum (VP), a region known for processing reward, imp

284 s accumbens (NAc), lateral habenula, ventral pallidum (VP), and amygdala.

285 he NAc-S projects prominently to the ventral pallidum (VP), and in the current experiments, we assess

286 The dorsal-lateral BF, including the ventral pallidum (VP), contains reward-sensitive neurons, some o

287 lves a projection from area X to the ventral pallidum (VP), which in turn projects to dopaminergic re

288 t (GPe), internal segment (GPi), and ventral pallidum (VP)-in 8 HD cases compared with 7 matched cont

289 nucleus of the amygdala (BLA) or the ventral pallidum (VP).

290 n of a downstream target of BLA, the ventral pallidum (VP).

291 ucture of the mesolimbic system, the ventral pallidum (VP).

292 ugh its output to the rostral medial ventral pallidum (VP-m).

293 In participants in whom blood T. pallidum was detectable, those with the highest opsonic

294 ion amplification of the tp0574 gene, and T. pallidum was detected in cerebrospinal fluid (CSF) by re

295 HSV-2 and T. pallidum were detected by serum antibody testing.

296 halamus, medial prefrontal cortex and globus pallidum) were obtained using PASL-MRI and [(15)O] water

297 ctures such as nucleus accumbens and ventral pallidum (where opioid/endocannabinoid/orexin signals ca

298 tive TprC and TprI are surface-exposed in T. pallidum, whereas their MOSP(N)-like domains are tethere

299 rom Sodalis glossinidius and Polysphondylium pallidum, which are phylogenetically related to the Salm

300 TP0326, the sole protein in T. pallidum with sequence homology to a Gram-negative OMP,