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1 s, at least for a time, for the treatment of palmar and axillary hyperhidrosis.
2                                          The palmar and dorsal ulnotriquetral and ulnolunate ligament
3           Immunostaining of Cx26 in lesional palmar and knuckle skin was weak or absent, although its
4 c signaling pathways in fibroblasts from the palmar and nonpalmar dermis of Dupuytren's patients and
5 ling arthritic changes, marked osteoporosis, palmar and plantar subcutaneous nodules and distinctive
6 rome include multiple basal cell carcinomas, palmar and/or plantar pits, odontogenic keratocysts, ske
7            Incompleteness of the superficial palmar arch (SPA) was present in 46%, the deep palmar ar
8                                  The rate of palmar arch incompleteness and the clinical consequences
9 lmar arch (SPA) was present in 46%, the deep palmar arch was complete in all patients.
10 upply is always preserved by a complete deep palmar arch.
11                                          The palmar arches serve as the most important conduits for d
12 romoted differentiation into specifically of palmar dermal fibroblasts from Dupuytren's patients in t
13 ith the peak systolic velocity of the second palmar digital artery (Pearson coefficient: 0.621; p < 0
14 CR) analysis and immunohistochemistry of the palmar epidermis demonstrated significantly increased ex
15 ratum granulosum of both normal and affected palmar epidermis, indicating that the altered AQP5 prote
16                                        Human palmar fascia fibroblasts and human fetal lung fibroblas
17 disabling fibroproliferative disorder of the palmar fascia, which leads to flexion contractures of th
18 nonpalmar dermis of Dupuytren's patients and palmar fibroblasts from non-Dupuytren's patients.
19                                              Palmar finger skin reacts to extension under mechanical
20  altered thumb print due to other causes and palmar hyperhidrosis were excluded.
21 bioenergetics in 13 patients with idiopathic palmar hyperhidrosis.
22 cise performance in patients with idiopathic palmar hyperhidrosis.
23 mediate phenotype most readily identified by palmar hyperlinearity and in some cases fine-scale and/o
24 mon skin disorder characterised by dry skin, palmar hyperlinearity and keratosis pilaris.
25 s of IV are fine-scale on the arms and legs, palmar hyperlinearity, and keratosis pilaris.
26 ong adduction of the thenar, hypothenar, and palmar interosseous muscles offer powerful rigidity to t
27 il thickening in PC-K6a and PC-K17; (3) more palmar keratoderma in PC-K16; (4) cysts primarily in PC-
28                       Our findings show that palmar keratoderma is a clinical indicator of recessive
29 pe of EPP which is characterized by seasonal palmar keratoderma, relatively low erythrocyte protoporp
30 ited Kingdom identified six individuals with palmar keratoderma.
31                                The digit and palmar pads are also represented by barrelettes located
32        Dose-limiting toxicities were grade 3 palmar plantar erythrodysesthesia (PPE), mucositis, and
33 nt but required early dose reductions due to palmar plantar erythrodysesthesia, and liver decompensat
34 ns affecting nails, glands, oral mucosa, and palmar-plantar epidermis.
35 , fatigue (24 [8%]), dyspnoea (21 [7%]), and palmar-plantar erythrodysaesthesia (18 [6%]) in the sora
36 n with sorafenib than with axitinib included palmar-plantar erythrodysaesthesia (PPE; 37 [39%] of 96
37 ] vs 7 [2%]), fatigue (36 [11%] vs 24 [7%]), palmar-plantar erythrodysaesthesia syndrome (27 [8%] vs
38 hoea (103 [21%] of 488 patients) followed by palmar-plantar erythrodysaesthesia syndrome (87 [18%]),
39  fatigue in the axitinib arm, and diarrhoea, palmar-plantar erythrodysaesthesia, and alopecia in the
40        PLD-treated patients experienced more palmar-plantar erythrodysesthesia (37%; 18% grade 3, 1 p
41 atigue (6% v 15%), hypertension (28% v 22%), palmar-plantar erythrodysesthesia (8% v 4%), and hematol
42                                              Palmar-plantar erythrodysesthesia (PPE) became evident a
43 cause of adverse events related to the drug (palmar-plantar erythrodysesthesia [PPE], n = 3; asthenia
44 ful neuropathy occurred in 7.5%; and grade 3 palmar-plantar erythrodysesthesia occurred in 2.5%.
45  fatigue, hypertension, febrile neutropenia, palmar-plantar erythrodysesthesia syndrome, and stomatit
46 ncidences of diarrhea, nausea, vomiting, and palmar-plantar erythrodysesthesia were higher with lapat
47  events were fatigue, weight loss, diarrhea, palmar-plantar erythrodysesthesia, and hypertension.
48 mg twice per day; n = 1); grade 3 mucositis, palmar-plantar erythrodysesthesia, and hypokalemia (400
49                                   Skin rash, palmar-plantar erythrodysesthesia, and thrombocytopenia
50  of whom had three dose-limiting toxicities: palmar-plantar erythrodysesthesia, cerebral ischaemia, a
51 associated adverse events included diarrhea, palmar-plantar erythrodysesthesia, decreased weight and
52 5% of patients) were diarrhea, nausea, rash, palmar-plantar erythrodysesthesia, mucositis, vomiting,
53 as 1,657 mg/m2/d with limiting toxicities of palmar-plantar erythrodysesthesia, nausea, vomiting, ver
54 ction, elevated thyroid stimulating hormone, palmar-plantar erythrodysesthesia, weight loss, and head
55                                      Painful palmar-plantar keratoderma (PPK) severely impairs mobili
56 on in a family with diffuse nonepidermolytic palmar-plantar keratoderma was shown to be the loss in o
57 cognized, but important, normal functions in palmar-plantar tissues.
58          The remarkable occurrence of severe palmar--plantar hyperkeratosis in both patients suggests
59 d protein electrophoretic features closer to palmar/plantar or mucosal-like epithelia.
60 erse metacarpal ligament (DTML) (n = 5), and palmar plate (n = 10) were analyzed.
61 ittal MR images were best for evaluating the palmar plate and the capsule.
62 n of lesions of the extensor hood, DTML, and palmar plate structures.
63 , radioscapholunate, dorsal radiotriquetral, palmar scaphotriquetral, and dorsal scaphotriquetral lig
64   By simulating skin deformations across the palmar surface of the hand and tiling it with receptors
65   Injection of capsaicin into the plantar or palmar surface of the paws produced a depression of brad
66                              We examined the palmar surfaces of the dominant and nondominant hands of
67                   Despite limited sensation, palmar tactile stimulation delivered 4 months post-trans
68                    The capitate head is more palmar than in all other known hominoids, permitting ext
69 rgone AHSCT, namely finger pad inflammation, palmar violaceous papules, and digital ulcerations.
70 rgone AHSCT, namely finger pad inflammation, palmar violaceous papules, and digital ulcerations.

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