ライフサイエンスコーパス: pancreatic endocrine tumor

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1 traductal papillary mucinous neoplasms and 3 pancreatic endocrine tumors).

2 y be caused by a gastric carcinoid and not a pancreatic endocrine tumor.

3 uctal adenocarcimomas but are never found in pancreatic endocrine tumors.

4 tumors and 7 of 8 (88%) cases of multifocal pancreatic endocrine tumors.

5 n A was identified as a promising marker for pancreatic endocrine tumors.

6 ttle is known of its expression in malignant pancreatic endocrine tumors.

7 hepatic metastases in patients with sporadic pancreatic endocrine tumors.

8 thogenic role in the development of sporadic pancreatic endocrine tumors.

9 ctor expression in both human and mouse MEN1 pancreatic endocrine tumors.

10 cystadenomas, 13 other pancreatic cysts, 27 pancreatic endocrine tumors, 16 chronic pancreatitis, 9

11 a radiologic response rate of 25% (45% among pancreatic endocrine tumors, 33% among pheochromocytomas

12 welve mutations were detected in 31 sporadic pancreatic endocrine tumors (34%).

13 oRNA expression patterns in normal pancreas, pancreatic endocrine tumors and acinar carcinomas to eva

14 H) at 11q13 in approximately 40% of sporadic pancreatic endocrine tumors and hypothesize that MENIN i

15 se characterized by parathyroid hyperplasia, pancreatic endocrine tumors, and pituitary adenomas.

16 Pancreatic endocrine tumors are rare but have long held

17 l intestinal carcinoid tumors and multifocal pancreatic endocrine tumors arise independently or wheth

18 sion in isolated adult pancreatic islets and pancreatic endocrine tumor cell lines was determined to

19 t deletion of chromosome 1 may contribute to pancreatic endocrine tumor formation.

20 The molecular pathogenesis of pancreatic endocrine tumors is largely unknown.

21 , 107 received sunitinib (carcinoid, n = 41; pancreatic endocrine tumor, n = 66).

22 Pancreatic endocrine tumors occur both sporadically and

23 Forty-two patients with advanced pancreatic endocrine tumors or carcinoid tumors were tre

24 Overall objective response rate (ORR) in pancreatic endocrine tumor patients was 16.7% (11 of 66

25 Carcinoid tumors and pancreatic endocrine tumors (PETs) are uncommon neuroend

26 Pancreatic endocrine tumors (PETs) have long fascinated

27 with gastrinomas as well as other functional pancreatic endocrine tumors (PETs) in whom the excess-ho

28 icated in the pathogenesis of sporadic human pancreatic endocrine tumors (PETs).

29 ns were present in both benign and malignant pancreatic endocrine tumors, suggesting that a MENIN gen

30 locus at chromosome 3p25 may harbor a novel pancreatic endocrine tumor suppressor gene, and allelic

31 study, this regimen appeared more active in pancreatic endocrine tumors than in carcinoid tumors.

32 Thirty-one sporadic pancreatic endocrine tumors were analyzed for mutation o

33 Pancreatic endocrine tumors were found in all patients a

34 Twenty-nine sporadic and MEN1 pancreatic endocrine tumors were studied for loss of het

35 stinal carcinoid tumors and eight multifocal pancreatic endocrine tumors, were studied.

36 Neuroendocrine tumors include carcinoids and pancreatic endocrine tumors, which share a number of com

37 Patients with pancreatic endocrine tumors with liver metastases should

38 there is continued inclusion and analysis of pancreatic endocrine tumors with small intestinal neuroe

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