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1 y disease leading to pain, maldigestion, and pancreatic insufficiency.
2 ly characterized by neutropenia and exocrine pancreatic insufficiency.
3 the parenchyma, fibrosis, pancreatitis, and pancreatic insufficiency.
4 ects were children aged 6-8 y who had CF and pancreatic insufficiency.
5 rotein and mRNA levels, thus suggesting mild pancreatic insufficiency.
6 ion of patients with deltaF508 genotypes and pancreatic insufficiency.
7 these mice died within 3 weeks of birth from pancreatic insufficiency.
8 o progressive diabetes mellitus and exocrine pancreatic insufficiency.
9 r preschoolers aged 2 to 6 years with CF and pancreatic insufficiency.
10 the genders in these parameters, as well as pancreatic insufficiency, age at diagnosis, mode of pres
11 direct and indirect costs, quality of life, pancreatic insufficiency, alternative pain scales, lengt
13 late had a significantly lower prevalence of pancreatic insufficiency and CF-related diabetes, and be
14 recessive disorder characterized by exocrine pancreatic insufficiency and hematopoietic dysfunction,
15 5-10 y with CF, mild pulmonary disease, and pancreatic insufficiency and of 26 healthy control child
17 ctal metaplasia occur, culminating in marked pancreatic insufficiency and the development of pancreat
18 llow-up period, especially the children with pancreatic insufficiency and those who were homozygous f
19 infection, in combination with correction of pancreatic insufficiency and undernutrition by multidisc
20 rioperative morbidity, a substantial risk of pancreatic insufficiency, and may overtreat noninvasive
22 enital onset with sequelae of early exocrine pancreatic insufficiency as in the diseases of cystic fi
23 ciency and subjects with cystic fibrosis and pancreatic insufficiency, as compared with values in hea
24 ltisystem disorder characterized by exocrine pancreatic insufficiency, bone marrow dysfunction, and m
25 me assembly and are associated with exocrine pancreatic insufficiency, chronic neutropenia and skelet
26 c colitis, steatorrhea secondary to exocrine pancreatic insufficiency, dietary lactose or fructose ma
27 tants reported to be associated with CF with pancreatic insufficiency do not support HCO3- transport,
28 zing inflammatory disease that can result in pancreatic insufficiency due to acinar atrophy and fibro
29 in the use of diagnostic tests for exocrine pancreatic insufficiency (EPI) confound interpretation o
31 BMI-z(5to10) was negatively correlated with pancreatic insufficiency, history of meconium ileus, and
33 and acinar cell loss may be responsible for pancreatic insufficiency in Jagged1-deficient mice and,
34 to 6 years (mean age, 3.8 years) with CF and pancreatic insufficiency (intervention, n = 36 and contr
38 ltisystem disorder characterized by exocrine pancreatic insufficiency, multilineage hematopoietic dys
39 >/=1 mo, and 38% of infants were ExFM22+) or pancreatic insufficiency (n = 70; 25% of infants were Ex
40 ticocutaneous fistula, exocrine or endocrine pancreatic insufficiency, need for additional radiologic
44 orption of fat and nitrogen in patients with pancreatic insufficiency related to cystic fibrosis.
45 is may be underappreciated, and both gut and pancreatic insufficiency represent modifiable targets in
46 sulin-treated neonatal diabetes and exocrine pancreatic insufficiency requiring enzyme replacement th
48 ice are viable but have defects that include pancreatic insufficiency, similarly to UBR1-/- human pat
49 K status in subjects aged 8-25 y with CF and pancreatic insufficiency taking various vitamin K supple
50 nd adolescents with cystic fibrosis (CF) and pancreatic insufficiency, the efficacy of routine vitami
51 ille syndrome patients also display exocrine pancreatic insufficiency, the pathobiology of which is u
52 yvitamin D in 10 adults with CF and exocrine pancreatic insufficiency was compared with that of 10 he
57 or (CFTR) gene typically cause pulmonary and pancreatic insufficiency while rarely causing pancreatit
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